Leitlinienprogramm OnkologieProstate Cancer

7. Diagnosis and treatment of recurrent or metastatic prostate carcinoma – Prostate Cancer

Version 6.2 · October 2021 · AWMF 043/022OL
Leitlinienprogramm Onkologie · Deutsche Krebsgesellschaft · Deutsche Krebshilfe · AWMF

7.1 Definition and diagnosis of tumor recurrence

7.1 · Evidence-based Recommendation · checked 2018

In asymptomatic patients having undergone treatment with curative intent, the determination of serum PSA shall be used for follow-up.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (97%)
References
[190]
Evidence tables (1)7.1 EvT 7.1
7.2 · Evidence-based Statement · checked 2018

After radical prostatectomy, a PSA level confirmed in at least two measurements at > 0.2 ng/ml indicates biochemical recurrence.

Level of evidence
4
Consensus
Strong consensus (97%)
References
[190] [740] [741] [742]
Evidence tables (1)7.1 EvT 7.2
7.3 · Evidence-based Statement · checked 2018

After radiotherapy alone, a PSA rise of > 2 ng/ml, confirmed in at least two measurements, over the postinterventional PSA baseline denotes biochemical recurrence.

Level of evidence
4
Consensus
Strong consensus (97%)
References
[96] [743]
Evidence tables (1)7.1 EvT 7.3
7.4 · Evidence-based Statement · checked 2018

Biopsy-based confirmation of biochemical recurrence following RPE is not required.

Level of evidence
4
Consensus
Strong consensus (100%)
References
[101] [744] [745]
Evidence tables (1)7.1 EvT 7.4
7.5 · Evidence-based Recommendation · checked 2018

Biopsy-based confirmation of biochemical recurrence should be sought in patients having undergone radiotherapy with the option of local treatment for recurrence.

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
4
Consensus
Strong consensus (97%)
References
[101] [746] [747]
Evidence tables (1)7.1 EvT 7.5
7.6 · Evidence-based Recommendation · checked 2018

In patients with biochemical recurrence after undergoing primary treatment with curative intent and a treatment option for local recurrence, a differentiation shall be made between local and systemic recurrence. 

For this purpose:

  • the PSA doubling time;
  • the latency time to primary treatment with curative intent; and 
  • the Gleason score 

will be used. 

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (98%)
References
[661]
7.7 · Evidence-based Recommendation · checked 2018

In asymptomatic patients with biochemical recurrence, bone scintigraphy should not be performed if the PSA level is < 10 ng/ml.

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
4
Consensus
Strong consensus (97%)

Background information

Re: Recommendation 7.1

The diagnosis of a recurrence is primarily based on the detection of a PSA increase, i.e. biochemically. A PSA rise following radical prostatectomy or radiotherapy with curative intent may be due to either local recurrence or metastases. The localization of the recurrence is only of crucial importance if treatment for the local recurrence is an option for the patient. If this is ruled out because of a high risk of complications, serious concomitant diseases, or the patient's refusal to undergo this measure, no diagnosis is necessary because it is of no consequence. Over the further course of the disease, examinations should be symptom-oriented.

 

Re: Recommendation 7.2

Several studies have addressed the question of the PSA level at which recurrence is clearly present following radical prostatectomy. There have been investigations into the PSA values at which the following are probable: further increases in PSA over time; local recurrence; or metastasis. Since PSA values in the range of the detection limit do not necessarily have to increase further (e.g. in the case of residual benign glands) and measurement inaccuracies can occur in the low-PSA range, an increasing PSA value of > 0.2 ng/ml, measured at an interval of at least two weeks, confirmed in at least two measurements, was determined to be a suitable definition for significant PSA recurrence. This recommendation is identical to the current version of the European Society of Urology guidelines. However, two rising values < 0.2 ng/ml can also be taken to mean biochemical recurrence [740], [741], [742].

 

Re: Recommendation 7.3

The previous ASTRO definition of PSA recurrence after radiotherapy - three consecutive increases - was widely criticized: it was not of sufficient predictive value with regard to further clinical progression or overall survival, nor was it of reliable value in patients currently undergoing/having undergone hormone-ablation therapy. Moreover, there was a possibility of bias in disease-free survival rates calculated according to Kaplan-Meier by backdating the timing of PSA progression [743]. Among the alternative models investigated and retrospectively evaluated in large patient groups, the ASTRO definition is now: baseline plus 2 ng/ml ("Phoenix definition"). This was shown to be more sensitive and specific in detecting PSA recurrence after primary radiotherapy [748]. The dating of the recurrence is thereby determined "at call", i.e. at the documented time of exceeding the threshold (the second measurement is to be taken at an interval of approximately three months). This definition is applicable following teletherapy and brachytherapy, and has also proven to be valid in case of additional (neo)adjuvant short-term hormone-ablation therapy and/or shorter follow-up times [749], [750]. Compared to the previous definition, the "Phoenix definition" is significantly less sensitive with regard to false positive findings, which can be caused, for example, by the so-called "PSA bounce" (= reversible PSA increase after radiotherapy) [751].

 

Re: Statement 7.4

Several studies have shown that biopsy of the vesico-urethral anastomosis is an inaccurate method for detecting local recurrence [744], [745]. Since this is especially true for the low PSA range (therapy of PSA recurrence).

 

Re: Recommendation 7.5

Prior to local therapy, the diagnosis of a local recurrence should be confirmed by transrectal or transperineal punch biopsy. The histological specimen should be evaluated by a pathologist familiar with pathomorphological changes following radiotherapy in order to avoid false positive findings [747]. The biopsy should be performed once any PSA bounce phenomenon (= reversible PSA increase after radiotherapy) has been ruled out [752], [753], i.e. only in the case of a confirmed PSA recurrence (see chapter Definition and diagnosis of tumor recurrence).

 

Re: Recommendation 7.6

The most important factors for differentiating a local recurrence from a systemic recurrence are the PSA doubling time, the Gleason grade of the prostatectomy specimen and the time interval between surgery and recurrence detection. Systemic progression is indicated by a short PSA doubling time (e.g., B.)  7 makes systemic progression likely in recurrence. The shorter the time interval between PSA recurrence and detectable PSA level, the more likely systemic progression is present [661], [754].

 

Re: Recommendation 7.7

The probability of metastasis in relation to the PSA level in cases of recurrence following primary local treatment with curative intent is not known with any precision. In cases presenting following primary radiotherapy and/or brachytherapy with proven recurrence, metastasis should therefore be ruled out as being unlikely by means of the existing available options before any possible salvage surgery. In the absence of clear data on this issue, a PSA threshold of 10 ng/ml is considered to warrant bone scintigraphy. 

In cases having undergone radical prostatectomy where salvage radiotherapy is intended, this will be indicated anyway at low PSA values. The available data show that bony metastasis virtually does not occur at PSA < 7 ng/ml, is very rare at PSA < 10 ng/ml, and only at a PSA level of 20 ng/ml or higher do nuclear medical examinations show positive findings with sufficient probability. Therefore, the recommendation of a threshold of 10 ng/ml is a realistic compromise in order to avoid unnecessary examinations [755]

7.2 Therapy of PSA recurrence

The following recommendations and statements refer to recurrence assessed as localized. For any suspected distant metastasis, see chapter 7.3 onwards.

In addition, by way of introduction, please refer to chapter 5.3 Staging, in particular Recommendation 5.29 on diagnosis of recurrence regarding the use of PSMA-PET-CT in the diagnosis of PSA progression following radical prostatectomy.

Summary of the evidence base on radiotherapy for PSA recurrence following radical prostatectomy.

A recently published systematic review is available [756]. Randomized trials of radiotherapy for progression following radical prostatectomy (so-called salvage radiotherapy, SRT) which include comparison with a control group do not exist. For SRT, a controlled study is available [757], in which SRT was retrospectively compared with a "wait-and-see" strategy. It showed a carcinoma-specific survival advantage for SRT, but no survival advantage [757]. However, methodological flaws diminish the value of this study. In addition, two randomized trials are available in which SRT alone was compared with SRT+short-term hormone-deprivation therapy (GETUG-AFU 16) [758] and SRT compared with SRT+long-term therapy with bicalutamide 150 mg / d (RTOG 96-02) [759]. In these two randomized trials, the RTOG trial showed a significant survival benefit (primary endpoint) for combination therapy, and the GETUG trial showed an improvement in metastasis-free survival over 6 months with short-term ADT [759][758]. Thus, the value of SRT as a curative therapy can be considered to be well established.

7.8 · Evidence-based Statement · modified 2021

In patients with PSA recurrence and favorable prognostic criteria (PSA doubling time of > 12 months, Gleason score < 8 (ISUP <4)), wait-and-see is an option.

Level of evidence
2++
Consensus
Strong consensus (97%)
References
[760] [756]

The natural history of PSA progression following radical prostatectomy (RP) can be long-term even without any treatment intervention, whereby patients do not develop clinically significant symptoms. In a large retrospective series, approximately 2,000 patients were followed up for a median of 5.3 years (0.5-15 years) after RP [661]. 15 % of patients (n=315) developed PSA recurrence [661]. Of these, distant metastases occurred in 103 (34%) during the study period [661]. The median time between PSA recurrence and the diagnosis of distant metastases was 8 years. Among patients with distant metastases, these led to death after a median of 5 years [661]. Based on the data collected, the expected rate of patients with metastasis-free progression after 15 years was calculated to be 82% [661]. Overall, however, this is a favorable patient population with a relatively low proportion of patients at high risk of progression [661]. Only 6% had lymph node involvement, 5 % had seminal vesicle infiltration, and 93% of patients had a Gleason score of max. 7 [661]. It can therefore be assumed that significantly higher rates of PSA progression occur in collectives with more advanced tumors.

Salvage radiotherapy is a therapeutic option with curative potential. However, as an invasive procedure, it is also associated with potential adverse side effects. In this regard, it should be kept in mind that the rate of severe late effects > RTOG 2 (Radiation Morbidity Scoring Criteria) is comparatively low with modern RT technology (Intensity-Modulated Radiation Therapy – IMRT) [674][758]. In the series described [661], patients with favorable prognostic criteria (PSA doubling time > 10 months, occurrence of PSA recurrence later than 2 years after RP, primary Gleason score < 8) had significantly less PSA progression than those with unfavorable prognostic criteria.

In the systematic review on salvage radiotherapy cited [756], patients with a PSA doubling time of < 12 months and a Gleason score > 8 had significantly worse overall survival with biochemical progression without therapy than those with a doubling time of > 12 months and a Gleason score < 8 [756]. Accordingly, two risk groups (European Association of Urology biochemical recurrence risk groups) were defined. This classification was externally validated by a retrospective study of 1,125 patients from the Martini-Klinik in Hamburg, Germany, treated between 1992 and 2006 [760]. This showed that both cancer-specific survival and metastasis-free survival were independent predictors of subsequent outcome [760]. This systematic review and external validation form the basis of the EAU Prostate Cancer Guidelines Panel recommendations for salvage radiotherapy [761].

This work forms the background of the formulated statement. It is important to note here that many additional points must be included in this decision such as patient age, the level of PSA prior to possible salvage radiotherapy, and the patient's concomitant diseases and life expectancy. It must always be kept in mind that from a PSA level of > 0.5 ng/ml prior to initiation of SRT, the curative potential is significantly reduced or lost.

7.2.1 Treatment of PSA recurrence and PSA persistence following radical prostatectomy

7.9 · Evidence-based Recommendation · modified 2021

Percutaneous salvage radiotherapy (SRT) (min. 66 Gy) shall be offered as a treatment option following radical prostatectomy for a PSA rise from the zero range in the pN0/NX category.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
2++
Consensus
Consent (95%)
References
[762] [763] [759] [758] [764]

The recommendation for percutaneous salvage radiotherapy (SRT) is based on a large number of mostly retrospective studies, with some comparative studies with a wait-and-see approach, as well as two randomized phase-III studies, in which, however, SRT as standard was compared with an additional antihormonal therapy [758][759][762][763][764]. The RTOG 96-02 trial demonstrated improved survival with combined hormone and radiotherapy compared with salvage therapy alone [759]; to that extent, SRT alone is also thought to be superior to a "wait-and-see" strategy. Salvage radiotherapy either with or without antihormonal therapy is the only curative therapeutic option in this situation.

The optimal dosage of SRT has not been clarified. The minimum recommendation of 66 Gy reflects both the existing retrospective studies [762][763][764] and the dosing of the prospective randomized phase-III studies of the RTOG and GETUG groups [758][759]. It should be noted that at 64.8 Gy, the dose of the RTOG trial, was slightly lower but specified in a different manner [759]. For these reasons, the recommendation has included SRT, with a recommendation grade A, at a minimum total dose of 66 Gy. In a meta-analysis of all major case series on SRT (including patients with persistent PSA), it was shown that an increase in the total dose by 1 Gy can be expected to result in a calculation-based advantage in terms of biochemical progression-free survival of 2% [765]. Also, starting SRT earlier (for each 0.1 ng/ml of PSA level) suggested an improved biochemical progression-free rate of approximately 2% [765]. These results support the minimum dose requirement. This question is being tested in a current phase-III trial, for which recruitment has been completed, which is randomizing to 64 Gy versus 70 Gy [766][767]. Final results are pending.

7.10 · Evidence-based Recommendation · new 2021

Patients at high risk of progression (PSA before SRT > 0.7 ng/ml) shall be offered androgen deprivation therapy (ADT) or bicalutamide in addition to percutaneous salvage radiotherapy (SRT).

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1-
Consensus
Strong consensus (94%)
References
[759]

The recommendation for combination therapy of SRT with androgen deprivation is based on two previously described randomized trials [759][758]. In the RTOG 96-02 trial, patients having undergone RP with PSA persistence or PSA rise from the zero range were randomized to SRT alone (GD 64.8 Gy) and an experimental arm in which antihormonal therapy with bicalutamide 150 mg/d for 2 years was applied in addition to SRT at the same total dose [759]. A total of 771 patients with primary tumor stage pT3N0 or pT2R+ were involved. The median PSA level before SRT was 0.6 ng/ml, which was high. The median follow-up time in the 2017 publication was 13 years. A halving of cancer-specific mortality was found in the bicalutamide group at 12 years compared with the comparison group (5.4% vs. 13.4%; hazard ratio (HR) 0.49 (95% confidence interval (CI) 0.32-0.74); p<0.001). Overall survival (OS) at 12 years was higher in the bicalutamide group compared with the placebo group (OS: 76.3% vs. 71.3%; HR 0.77 (95% CI 0.59-0.99; P=0.04)) [759]. Prespecified subgroup analysis showed that patients with a pre-SRT PSA level > 0.7 ng/ml had a significant 12-year survival advantage over PSA levels < 0.7 ng/ml [759]. An unplanned re-analysis of this study re-examined overall patient survival [768]. Secondary endpoints of the study were cardiovascular and neurological late effects, RTOG grade 3-4. In the subgroup with a pre-SRT PSA level of <0.6 ng/ml (n=389), there was no survival benefit from two years of bicalutamide 150 mg/d. In addition, there was significant evidence of increased mortality in this group (HR: 1.94) due to the higher-grade neurologic and cardiovascular late effects. Even despite limited ability to evaluate this due to the design of this study, it still provides clear evidence on the use of additional antihormonal therapy but only if strictly indicated. These potential serious side effects of  antihormonal therapy are well documented in the treatment of PCa [674].

In the French randomized phase-III GETUG-AFU-16 trial, with a total of 742 patients, the risk profile was much more favorable compared to the RTOG trial: the median pre-SRT PSA was 0.3 ng/ml [758]. Only 27 % of patients had a PSA doubling time shorter than 6 months, only 11 % had a Gleason Score > 8. In this case, after a median follow-up of almost 10 years, there was no significant difference in overall survival, but a (small) significant difference in metastasis-free survival of 75 % in the SRT + 6 months LH-RH analog group vs. 69 % in the SRT alone group (p= 0.034, HR 0.73) [758]. In the overall assessment of the randomized trials available, the results of the RTOG 96-02 trial and to a lesser extent the GETUG-16 trial support the use of antihormonal therapy in patients with a high risk of progression after RP (to an evidence level of 1), whereby this was defined in the RTOG trial at a PSA level > 0.7 ng/ml [758].

7.11 · Evidence-based Recommendation · new 2021

Percutaneous salvage radiotherapy (SRT) (min. 66 Gy) shall be offered in combination with androgen deprivation therapy (ADT) as a treatment option after radical prostatectomy for a PSA rise from the zero range and unfavorable prognostic criteria (PSA doubling time < 12 months, Gleason 8-10 (ISUP 4-5)) in the pN0/NX category.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
2++
Consensus
Strong consensus (97%)
References
[756] [759] [758] [766]

This recommendation should be taken in addition to the preceding recommendation. In the randomized RTOG study 96-02, a pre-SRT PSA level of >0.7 ng/ml was associated with improved patient survival if treatment was with combined hormone and radiotherapy [759]. Thus, the prerequisite for optimal salvage therapy after RP is to identify the so-called high-risk patients whose PSA level rises from the zero range or persists above the PSA-zero range. In a systematic review [756] two risk groups were classified (European Association of Urology biochemical recurrence risk groups), whereby the second group had a significantly higher risk of dying earlier with a PSA doubling time of < 12 months and a Gleason score ≥ 8. These patients require intensified therapy. This is a recommendation for the patient group presented in the named systematic review that was externally validated [756]. Therefore, the cited recommendation is made with a recommendation grade of A. However, it has not yet been substantiated from randomized trials, so the level of evidence is 2++. Accordingly, this score has found its way into the EAU guideline [761]. Regardless of this recommendation, there is considerable debate as to which antihormonal therapy (LH-RH analog, LH-RH antagonist or an androgen receptor inhibitor) should be used, as well as regarding the duration of ADT [758][759][769].

7.12 · Evidence-based Recommendation · new 2021

If androgen deprivation therapy (ADT) is added to percutaneous radiotherapy (SRT), the duration of ADT can be based on the risk of progression (high risk of progression: 24 months; or increased risk of progression: 6 months).

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
2-
Consensus
Strong consensus (97%)
References
[770] [759] [769] [758]

Of the two randomized phase-III studies cited [759][758], the RTOG 96-02 study suggests the efficacy of 24 months of antihormonal therapy with the androgen receptor inhibitor bicalutamide in an unfavorable patient population (median pre-SRT PSA of 0.6 ng/ml). The second study (GETUG-16) had a patient cohort with a much more favorable risk profile (pre-SRT PSA median 0.3 ng/ml, only 11% of patients had a Gleason score ≥8 and also only 11% of patients had seminal vesicle infiltration) [758]. There was a group of patients in this case with an increased risk of progression (measured by the EAU risk score [756] but with a lower risk of progression than the patients in the RTOG-96-02 study) [759]. These patients were treated with 6 months of LH-RH analog therapy, i.e., short-term ADT [758]. The result was no improvement in survival, but a (moderate) improvement in metastasis-free survival [758]. To overcome the difficulty of the question of the optimal use of ADT, two systematic reviews [770][769] are available, in which attempts were made to develop evidence-based approaches by also analyzing non-randomized studies.

Overall, the review by Spratt et al. results in the proposal to recommend three therapeutic options depending on the resection margin (negative vs. positive), Gleason score/ISUP grade (ISUP 1 vs. ISUP 2.3 vs. ISUP 4.5), and pre-SRT PSA (0.1-0.5 vs. 0.6 -1.0 vs. > 1.0). These cover SRT alone without additional antihormonal therapy, SRT in combination with short-term ADT, and SRT in combination with long-term ADT. From the authors' point of view, SRT alone is possible in a PSA range of 0.1 ng/ml to 0.6 ng/ml, depending on the risk factors; for PSA values > 1.0 ng/ml, SRT is generally recommended in combination with long-term ADT. It should be noted that the long-term results of the GETUG 16 study could not yet be included in this systematic review [769]. By analogy to these data, the present evidence-based recommendation was formulated, (increased risk of progression: short-term ADT – 4 to 6 months and high risk of progression: 24 months). Based on the evidence presented, a "can" recommendation was made. Of particular importance is that individual patient characteristics such as age and general condition as well as progression risk are ideally determined as part of an interdisciplinary tumor board where a collaborative recommendation is made.

7.13 · Evidence-based Recommendation · checked 2021
a.       Salvage radiotherapy shall be started as early as possible (PSA before SRT
(A)

b.       With initial stage pN0 and early start to radiotherapy, lymphatic drainage pathways should not be irradiated.
(B)
Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
2++

to


3
Consensus
Strong consensus (97%)
References
[762] [763] [771] [765]

A large number of retrospective cohort studies are available on the question of the timing of the start of SRT [762][763]. In these, it is reported that early SRT (onset at a PSA level <0.5 ng/ml) without additional use of antihormonal therapy significantly improves biochemical freedom from progression after 5 and 10 years. This effect is most likely not due to earlier onset versus later onset ("lead-time bias"), but rather an effect of earlier tumor destruction [771]. This effect also has an impact (albeit retrospectively) on metastasis-free survival and overall survival [762][763]. In a large study of 2,460 patients [762] with a median follow-up of 60 months, this effect was also seen in patients irradiated with a pre-SRT PSA level < 0.2 ng/ml (n=441) versus a level of 0.2-0.5 ng/ml (n=822). The 5-year biochemical progression-free rate here was 75% versus 65% (p<0.05) [762]. Therefore, the trend continues toward starting SRT as early as possible, ideally when PSA is rising out of the zero range and confirmed by two seperate follow-up tests [674].

From the studies cited above [674][763][771], the recommendation is that SRT of the prostate region alone should be performed at initial pN0 stage, with no treatment of the pelvic lymphatic drainage, and that treatment initiation should be early for a pre-SRT PSA value <0.5 ng/m. From the published data, there is no evidence of a significant advantage for coirradiation of the pelvic lymphatic drainage areas. Randomized studies on this question have not been published.

7.14 · Evidence-based Recommendation · modified 2021

Treatment of persistent PSA levels (above the defined zero range) following radical prostatectomy should follow the above principles of treatment for PSA recurrence.

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
2++
Consensus
Consent (94%)
References
[772] [773] [774] [775] [768]

There are two clinical situations to distinguish between postoperatively following radical prostatectomy: A persistent PSA level after RP, meaning the PSA level does not reach the defined zero range after surgery (usually at a PSA level > 0.1 ng/ml) [674]; or the PSA level reaches the defined zero range and later rises again from the zero range as a consequent sign of progression of the underlying disease. A persistent PSA level is usually associated with a more advanced tumor stage, a higher Gleason grade, a higher preoperative PSA level and/or positive resection margins, associated with a less favorable prognosis [774]. Basically, a retrospective monocentric evaluation of the Hamburg cohort showed that patients with a persistent PSA value per se had a worse prognosis. This was true for metastasis-free survival, carcinoma-specific survival and overall survival [772]. This was a retrospective cohort analysis of a total of 11,604 men, of whom 1,025 had a persistent  PSA level after RP [772]. Of the patients with persistent PSA levels, 570 (55.6%) received salvage radiotherapy. This salvage radiotherapy proved to be a significant influencing factor with respect to improvements in overall survival [772]. Further retrospective cohort studies showed that analogous treatment of patients with persistent PSA and PSA rising from the zero range have comparable biochemical progression rates after SRT, especially in the low pre-SRT PSA range (<0.5 ng/ml) [775]. Based on the available data, the guideline group believes that salvage radiotherapy, both with and without additional ADT, is a valid treatment option in patients with persistent PSA levels after RP. However, the lack of randomized trials does not allow a recommendation beyond recommendation grade B to be made.

7.2.2 Treatment for PSA progression following radiotherapy

7.15 · Evidence-based Statement · checked 2018

Salvage prostatectomy is a treatment option for PSA recurrence after primary percutaneous radiotherapy or brachytherapy when PSA progression is highly unlikely to be due to metastasis. 

Functional outcomes in terms of potency and continence are significantly worse than with primary surgery.

Level of evidence
2++

to


3
Consensus
Strong consensus (100%)
References
[747] [776]
Evidence tables (1)7.2.2 EvT 7.12
7.16 · Evidence-based Recommendation · checked 2018

Biopsy-based confirmation should be sought before salvage prostatectomy.

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
3
Consensus
Strong consensus (97%)
References
[747]
Evidence tables (1)7.2.2 EvT 7.13
7.17 · Evidence-based Statement · checked 2018

Salvage prostatectomy should be performed only by experienced surgeons.

Level of evidence
4
Consensus
Strong consensus (97%)
7.18 · Evidence-based Recommendation · modified 2018

a) HIFU therapy can be used to treat histologically-confirmed isolated local recurrence following percutaneous radiotherapy. 

b) The patient shall be informed about the experimental character of this procedure as a salvage therapy, and about the treatment alternatives.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
3

refers to a)


4

refers to b)

Consensus
Consent (92%)
References
[777] [778] [779]
Evidence tables (1)7.2.2 EvT 7.15

Background information

Re: Statement 7.15

There is currently no standard strategy for the treatment of PSA recurrence after primary radiotherapy. Treatment planning is largely dependent on the assessment of whether the recurrence in question is localized.

The following factors are positively associated with treatment response to local interventions in the literature i.e. suggest the presence of local recurrence [747][776]:

·         Initial "low-risk" tumor: clinical T1c or T2a category, Gleason score

·         Interval to PSA recurrence > 3 yrs;

·         PSA doubling time after primary therapy > 12 mo;

·         PSA increase < 2.0 ng/ml/y;

·         Number of pos. biopsies < 50 %;

·         Type of previous radiotherapy (previous seeds implantation) [764].

If the recurrence is assessed as localized, salvage prostatectomy is the treatment option that has been practiced the longest internationally (first reports are available from the 1960s). In 1990-2009, 15 case series with a total of 563 patients were published on this procedure [747][780].

Incontinence rates of 41 % on average (17-67 %), rectal injuries between 0 and 10 % (4.7 % on average), and bladder neck strictures between 0 and 41 % (24 % on average) are reported for salvage prostatectomy. These complication rates are far higher than the adverse events that occur after radical prostatectomy as primary therapy. Thereby, a decrease predominantly in rectal complication rates over time can be observed from the studies [747]. Current series show an average OR time of about two hours and a blood loss of about 360-500 ml on average [776][780].

In addition to salvage prostatectomy, there have been case series on the use of cryotherapy since the late 1990s (nine studies with a total of 540 patients). The use of cryotherapy is generally not recommended in this guideline (see Chapter 6.3.5 and Chapter 6.4.5).

For salvage brachytherapy, the systematic review – since 2003 – reported small case series (five studies with 124 patients), as well as for salvage HIFU therapy (three studies with n=71). Another study on salvage HIFU with n=167 patients was published in 2008 [781].

The 2011 EAU guideline lists cryotherapy and brachytherapy as possible salvage therapy procedures when salvage prostatectomy cannot be used [419]. According to the EAU guideline, HIFU may also be considered as an option, although patients should be made aware of the experimental nature of this procedure.

 

Re: Recommendation 7.16

Prior to salvage prostatectomy as local therapy, the diagnosis of local recurrence should be confirmed by transrectal punch biopsy. The histological specimen should be evaluated by a pathologist familiar with pathomorphological changes following radiation therapy to avoid false positive findings [747]. The biopsy should only be performed once any PSA bounce phenomenon has been excluded (reversible PSA increase after radiotherapy) [752][753] i.e. only in the presence of a confirmed PSA recurrence (see Chapter 7.1). Biopsy should be performed as early as possible after diagnosis of PSA recurrence, as the chances of cure by salvage prostatectomy correlate closely with the PSA level at the time of surgery [746][782]. The complication rate of transrectal punch biopsy after radiation or brachytherapy is not higher than that of conventional biopsy [783].

An imaging technique (choline PET/CT) cannot yet be recommended as an alternative to biopsy due to the lack of data.

If the biopsy is negative, but the PSA trend and clinical characteristics indicate a local recurrence, the therapeutic approach (wait versus radical prostatectomy) should be discussed with the patient on an individual basis.

 

Re: Statement 7.17

Due to the more difficult surgical conditions in pre-irradiated tissue, salvage prostatectomy should only be performed by experienced surgeons. This corresponds to an expert consensus also reached in other countries: the Dutch guideline calls for centralization of this surgical procedure. The published series so far come exclusively from centers [747][776][780].

 

Re: Recommendation 7.18

For HIFU in the salvage setting, one systematic review [777] and two complementary primary studies [778][779] were identified. All cohorts included patients with recurrence following radiotherapy.

The reviews include predominantly small patient cohorts with short follow-up and no overall or cancer-specific survival rates. For both endpoints, the larger of the primary studies (n=418) reported values at 7 years of 72% and 82%, respectively. Further salvage treatment was required in 63% of all patients in this study, with graded levels between risk groups (low risk: 46%, high risk: 77%). Although the efficacy of HIFU treatment in the salvage setting is significantly inferior compared with the present data in primary therapy, the procedure can be performed after consideration of the available therapeutic alternatives. For the time being, it remains experimental in nature.

7.2.3 Hormone-ablation therapy for PSA recurrence or progression

7.19 · Evidence-based Statement · checked 2018

Hormone-ablation therapy is not a standard treatment for PSA recurrence or progression.

Level of evidence
4
Consensus
Strong consensus (97%)
References
[101]
Evidence tables (1)7.2.3 EvT 7.16

Background information

Three primary studies were identified on hormone-ablation therapy for PSA recurrence or progression. The only randomized-controlled trial on the topic (endocrine therapy for PSA recurrence after prostatectomy, [784]) investigated surrogate parameters and also has a low number of cases for the research question (intervention group n=47). Furthermore, two retrospective case series are available. Moul et al. 2004 [785] showed an effect in terms of metastasis-free survival for PSA recurrence after prostatectomy (T1-T2) only for patients with tumors with a high-risk profile (Gleason score > 7 or PSA doubling time  7 months) versus late hormone-ablation therapy for PSA progression following percutaneous radiotherapy. The early treatment group showed statistically significant improvement for both overall survival and disease-specific survival and hormonal control.

For the above recommendation, due to the lack of available studies, reference was made to the NICE guideline [101] which provides indirect evidence for this indication based on a systematic review by Wilt et al. 2001 [734] on early versus late hormonal therapy for locally-advanced prostate cancer.

Accordingly, antiandrogenic therapy should only be used in the case of:

  • a PSA doubling time < 3 months;
  • symptomatic local progression;
  • proven distant metastasis. 

7.3 Treatment of hormone-sensitive metastatic prostate carcinoma (mHSPC)

7.20 · Evidence-based Recommendation · new 2021

Patients with metastatic hormone-sensitive prostate cancer (mHSPC) shall be classified as high and low-volume as well as high and low-risk.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Strong consensus (98%)
References
[786] [787] [788]
Evidence tables (2)Sweeney_NEJM_2015; Fizazi_NEJM_2017_Fizazi_LancetOncol_2019
7.21 · Evidence-based Recommendation · checked 2021

Part of the patient education about androgen deprivation alone or combination therapy shall include the following points in particular:

•the palliative nature of the therapy;

•impact on quality of life;

•the adverse effects.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (98%)
References
[728] [729] [730] [733] [789]
Evidence tables (1)7.3 EvT 7.18
7.22 · Evidence-based Recommendation · modified 2021

a.     Patients in good general health (ECOG 0-1) with metastatic (M1), hormone-sensitive, prostate cancer (mHSPC) shall be offered hormone therapy with apalutamide in addition to androgen deprivation.

(A)


b.      Patients in good general health (ECOG 0-1) with metastatic (M1), hormone-sensitive, prostate carcinoma (mHSPC) shall be offered hormone therapy with enzalutamide in addition to androgen deprivation.

(A)


c.     Patients in good general condition (ECOG 0-1) with newly-diagnosed (de novo), metastatic (M1), hormone-sensitive, high-risk prostate cancer (mHSPC) shall be offered hormone therapy with abiraterone (plus prednisone/prednisolone) in addition to androgen deprivation.

(A)

 

d.      Patients in good general condition (ECOG 0-1) with metastatic (M1), hormone-sensitive, high-volume prostate carcinoma (mHSPC) shall be offered chemotherapy with docetaxel in addition to androgen deprivation, with education about the higher toxicity compared with new hormonal agents.

(A)


e.      Patients in good general health (ECOG 0-1) with metastatic (M1), hormone-sensitive, low-volume prostate cancer (mHSPC) can be offered chemotherapy with docetaxel in addition to androgen deprivation.

(0)

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Strong consensus (a. 98 %, b. 92 %, c. 86 %, d. 93 %, e. 95)
References
[790] [791] [792] [786] [787] [793] [788] [794]
Evidence tables (6)Sweeney_NEJM_2015; Agarwal_LancetOncol_2019_Chi_NEJM_2019; Armstrong_JClinOncol_2019; Gravis_LancetOncol_2013; James_Lancet_2016; Fizazi_NEJM_2017_Fizazi_LancetOncol_2019
7.23 · Consensus-based Statement · modified 2021
The therapeutic decision should be made depending on patient preferences, side effects and concomitant diseases.
Consensus
Strong consensus (100%)
7.24 · Evidence-based Recommendation · modified 2021
a.     If combined treatment of androgen deprivation and apalutamide is decided upon, apalutamide administration shall begin within 3 months of initiation of androgen deprivation. Therapy should be given at a dose of 240 mg/day.

b.      If combined treatment of androgen deprivation and enzalutamide is decided upon, enzalutamide administration shall begin within 3 months of the start of androgen deprivation. Therapy should be given at a dose of 160 mg/day.

c.     If combined treatment of androgen deprivation and abiraterone is chosen, abiraterone administration shall begin within 3 months of initiation of androgen deprivation. Therapy shall be given at a dose of 1000 mg/day in combination with prednisone or prednisolone (5 mg/day).

d.      If combined treatment of chemotherapy and androgen deprivation is decided, docetaxel administration shall begin within 4 months of the start of androgen deprivation. 6 cycles every three weeks at a dose of 75mg/m² shall be given.

e.      Reasons for discontinuation of therapy shall include: patient request, progression, or intolerable side effects.
 
Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Strong consensus (a. 97 %, b. 90 %, c. 98 %, d. 98 %, e 97)
References
[791] [792] [786] [787] [789] [36] [728] [729]
Evidence tables (4)7.3 EvT 7.21; Agarwal_LancetOncol_2019_Chi_NEJM_2019; Armstrong_JClinOncol_2019; Fizazi_NEJM_2017_Fizazi_LancetOncol_2019
7.25 · Evidence-based Recommendation · modified 2021
a.  Patients who are not eligible for combination treatment shall be recommended for androgen deprivation.
(A)

b.   Androgen deprivation can be performed by medication or surgery.
(0)

c.  Androgen deprivation should be performed continuously if the PSA level does not fall below 4 ng/mL after at least 7 months.
(B)

d.   If the PSA level drops below 4 ng/mL, intermittent hormone therapy can be offered as an alternative after detailed patient education is provided.
(0)
Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1++
Consensus
Strong consensus (97%)
References
[728] [729] [730] [733] [36] [795] [796] [797] [789]
Evidence tables (1)7.3 EvT 7.22

Treatment of metastatic (M1), hormone-sensitive prostate carcinoma has fundamentally changed in recent years. After the CHAARTED study showed an advantage for early combined chemohormonal therapy [788], this could later also be shown for combination therapies of androgen deprivation (AD) with the new generation of hormone drugs (abiraterone, enzalutamide, apalutamide) [790][786][798][792][791]. Corresponding to what is seen with simple androgen deprivation, individual studies that also included locally-advanced high-risk tumors (e.g., STAMPEDE) suggest that patients at earlier tumor stages may also see a similar benefit from the combination therapies.

 

Re: Recommendation 7.20

In the trials that demonstrated a benefit from combined hormone therapy with docetaxel or abiraterone (plus prednisone/prednisolone) in hormone-sensitive metastatic prostate cancer, the study populations were defined in different ways based on metastatic pattern or Gleason score. For combined chemohormonal therapy (AD+docetaxel), patients were divided into high and low volume (CHAARTED) [788]. High volume includes those patients in whom the following were present:

·         at least four bone metastases, including at least one outside the axial skeleton or pelvis

·         and/or visceral metastases.

Only patients who had de novo metastatic disease and who met the following high risk criteria were included in the pivotal study with abiraterone (LATITUDE) [786][798]:

At least two of the three parameters:

·         Gleason 8 – 10

·         at least three bone metastases

·         visceral metastases.

Studies in which patients were classified according to both high and low volume as well as high and low risk show a little more than 80% agreement between high volume and high risk. Nevertheless, differentiation or classification of patients with metastatic hormone-sensitive prostate cancer into both groups is reasonable, as clear advantages for chemohormonal therapy have especially been shown in high-volume patients, and the approval of abiraterone is limited to high-risk patients.

 

Re: Recommendation 7.21

The recommendation on patient education is set against the background of informed decision-making as it corresponds to good clinical practice as well as the requirements of the Patient Rights Act. Patients should make decisions together with the informing physician regarding the difficult question of weighing up the risks. The typical and frequent side effects of androgen deprivation and, if applicable, combined chemotherapy or new hormone therapy listed in Table 18 and Table 19 (see Chapter 7.7) are part of the informed consent discussion.

 

Re: Recommendation 7.22 a-e and 7.24 a-e

The following paragraph summarizes the state of the literature for the combinations of AD and docetaxel or the new generation hormone drugs (abiraterone, apalutamide and enzalutamide) in mHSPC. For the combination hormonal therapies, the recommendations are based on the respective pivotal trials and the resulting regulatory texts; these take into account the patient populations included, some of which differed considerably in the trials and are therefore presented in detail in each case below.

For example, although the data from the STAMPEDE trial suggest that abiraterone is effective beyond the patient population with high-risk criteria, the guideline experts did not see a need to make a recommendation beyond the approval in view of the numerous treatment alternatives. Accordingly, Recommendations 7.19 and 7.21 take into account the indication and range of treatment approaches of the pivotal studies, for which data from RCTs are accordingly available.

Recommendations 7.24a (apalutamide) and 7.24b (enzalutamide) each consider a period of 3 months between the onset of AD and the initiation of apalutamide or enzalutamide therapy. However, in the respective studies, a period of 6 months was allowed for androgen therapy, provided that patients were still receiving docetaxel chemotherapy (up to 6 cycles) before the initiation of the study treatment. The proportion of patients with prior docetaxel chemotherapy was 11 and 18%, respectively, such that the subgroup analyses do not allow us to determine whether additional chemotherapy before starting the new androgen receptor blocker is appropriate.

The duration of therapy is long-term for all hormone combination therapies, but according to good clinical practice, it should be discontinued or modified in case of disease progression or the occurrence of intolerable side effects.

 

Re: 7.22a and 7.24a (apalutamide)

The combination of apalutamide in addition to AD was studied in TITAN, a randomized, double-blind, placebo-controlled phase 3 trial with a total of 1,052 patients [791]. Patients with documented mHSPC with an ECOG performance status of 0 or 1 who had received prior AD of up to 3 months in duration or up to six cycles of docetaxel with AD of up to 6 months in duration were included [791]. Patients with metastatic disease after local therapy with curative intent (e.g., prostatectomy, local radiation therapy) were also included in the study, provided it had been completed at least one year before randomization [791]. Excluded patients included those with severe angina pectoris, myocardial infarction, heart failure, thromboembolic events, or ventricular arrhythmias [791].

Patients in the intervention group received apalutamide in addition to AD, and patients in the control group received placebo in addition to AD [791]. Primary endpoints were radiographic progression-free survival (rPFS) and overall survival (OS); secondary endpoints were time to chemotherapy, time to worsening pain, time to long-term opioid use, and time to skeletal-associated events [791].

Overall, there is a significant benefit for the combination of apalutamide + AD for the primary study endpoints of rPFS and OS. The total number of adverse events (AEs) and grade 3-5 AEs differed only slightly between intervention and control groups. Skin rashes were observed significantly more frequently with apalutamide. A summary of the results, including adverse events, can be found in Table 15 below [790][791].

Apalutamide for the indication of mHSPC was also subjected to a benefit assessment according to §35a SGB V by IQWIG. As part of this procedure, the G-BA defined the appropriate comparative treatment (aCT) as chemohormonal therapy with docetaxel or, for patients with high-risk criteria, abiraterone. In the absence of a direct comparative study between aCT and apalutamide, an adjusted indirect comparison (AD as "bridge comparator") was implemented to compare apalutamide with docetaxel data from the STAMPEDE trial. Overall, no statistically significant difference in overall survival was found, but a positive effect was shown for apalutamide in terms of adverse events. Overall, IQWIG therefore saw evidence of an additional benefit that could not be quantified [799][800]. The G-BA, on the other hand, considered an additional benefit as not proven, since an advantage for apalutamide was only seen for the endpoint of serious adverse events in the indirect comparison and this did not allow an additional benefit to be determined with sufficient certainty [801][802].

 

Table 16: Results from the RCT TITAN on combination therapy with apalutamide, median observation time 22.7 months [Chi et al. 2019; Agarwal et al. 2019]


Number of patients: n=1052

AD + apalutamide

AD + placebo

Hazard ratio (HR) (confidence interval), significance

Benefit:

rPFS at 2 years (95% CI)

68.2% (62.9 – 72.9)

47.5 % (42.1-52.8)

HR 0.48 (0.39-0.60), p<0.001

Overall survival at 2 years (95% CI)

82.4 % (78.4-85.8)

73.5 % (68.7-77.8)

HR 0.67 (0.51-0.89), p=0.005

Harm:

Side effects

 

42.2 %

27.1 %

22.7 %

19.7 %

17.7 %

17.4 %

17.4 %

40.8 %

8.5 %

16.3 %

16.7 %

15.6 %

19.4 %

14.8 %

Grade 3-4 events

Skin rash

Hot flashes

Fatigue

Hypertension

Back pain

Arthralgia

Deaths during the course of treatment

10 (1.9 %)

16 (3.0 %)

 

 

Re: 7.22b and 7.24b (enzalutamide)

The combination of enzalutamide in addition to AD versus AD alone was studied in ARCHES, a randomized, double-blind, placebo-controlled phase 3 trial involving a total of 1,150 patients with pathologically confirmed mHSPC [792]. Adult patients with an ECOG performance status of 0 or 1 who received prior AD of up to 3 months or up to six cycles of docetaxel with AD of up to 6 months pretreatment were included [792]. As in the TITAN trial (apalutamide), patients with metastatic disease after local therapy with curative intent (e.g., prostatectomy, local radiation treatment) were eligible for study participation. Patients with clinically significant cardiac disease (e.g., myocardial infarction within 6 months prior to inclusion) were excluded [792]. The primary endpoint assessed was radiographic progression-free survival (rPFS) [792]. Secondary endpoints were time to PSA progression, time to initiation of new antineoplastic therapy, PSA rate below detection limit, objective response rate, time to worsening of symptoms related to the urinary tract, overall survival (OS), time to skeletal-related symptoms, time to castration resistance, time to worsening of quality of life, and time to worsening of pain symptoms [792].

After a median observation time of 14.4 months, there was a significant benefit for the combination of enzalutamide + AD for the primary study endpoint of rPFS. An OS benefit was not demonstrated in this data set (October 14, 2018) (HR 0.81; 95% CI 0.53-1.25) [792]. The total number of adverse events (AEs) and grade 3-5 AEs differed little between intervention and control groups. A summary of the results, including additional adverse events, is provided in Table 16 below.

For time to worsening quality of life, one of the secondary endpoints, a median of 11.3 months is reported for the intervention group and a median of 11.1 months for the control group (HR 0.96 (0.81-1.14) p=0.6548) [792].

Table 17: Results from the ARCHES RCTs on combination therapy with enzalutamide (median observation time 14.4 months) [Armstrong et al. 2019]

Number of patients: n=1150

AD + enzalutamide

AD + placebo

HR (confidence interval), significance

Benefit:

Median rPFS (months) (95% CI).

NM

19.0 (16.6 - 22.2)

HR 0.39 (0.30-0.50), p<0.001

Median overall survival* (months) (95% CI).

NM

NM

HR 0.81 (0.53-1.25), p=0.3361

Harms:

Side effects

 

24.3 %

27.1 %

19.6 %

12.2 %

7.5 %

8.6 %

4.5 %

6.5 %

26.4 %

25.6 %

22.3 %

15.3 %

10.6 %

10.8%

6.3 %

2.1 %

4.2 %

27.7 %

Grade 3-4 events

Hot flashes

Fatigue

Arthralgia

Back pain

Hypertension

Cognitive disorders

Fractures

Diseases of the musculoskeletal system

Deaths in the course of treatment

14 (2.4 %)

10 (1.7 %)

 

* Secondary study endpoint; NM – not met.

 

Re: 7.22c and 7.24c (abiraterone)

Data on the combination therapy of abiraterone and AD are available from two randomized clinical trials, STAMPEDE and LATITUDE [798][803].

Only patients in good general health (ECOG 0-1) were included in the STAMPEDE trial (whereas LATITUDE also included ECOG 2). Similar to the docetaxel arm of STAMPEDE (see 7.19 d), there was no difference in metastatic burden in the metastatic group in the abiraterone arm. In terms of overall survival (OS), there was a statistically significant advantage for the combination therapy with a hazard ratio (HR) of 0.63 (95% CI 0.52-0.76) [803]. There was also a significant advantage in failure-free survival between the treatment and placebo groups (HR 0.29; p<0.001) [803] (see Table 17).

The pivotal LATITUDE study included patients with newly-diagnosed, pathologically confirmed mHSPC (high risk) and ECOG 0-2 who had at least two of three designated risk factors (Gleason score of 8-10, at least three bone lesions, measurable visceral metastases) [798][786].

Patients who received prior chemotherapy, radiotherapy, or surgery for the treatment of metastatic prostate cancer were excluded, with the exception of AD with luteinizing hormone releasing hormone (LH-RH) analogues for a duration of less than three months, orchidectomy, or palliative radiotherapy or palliative surgery for symptomatic treatment, respectively [786][798].

The primary endpoint considered was OS and radiographic progression-free survival (rPFS) [786][798]. The evaluation was performed as part of an initial interim analysis in October 2016, the results of which resulted in unblinding as well as a protocol adjustment to an open-label extension phase of the study with the possibility of a change in treatment group in February 2017 [786][798]. In the final evaluation, the change of patients from the control group to the intervention group after unblinding (crossover, n=72) has to be considered [786].

The final evaluation was performed as of August 15, 2018, with a median observation time of 51.8 months [786]. The benefit in terms of overall survival was confirmed (HR 0.62 and 0.66, respectively, see Table 17). Evaluation of rPFS was only performed for the first interim analysis showing a clear advantage of combination therapy at 33.0 vs. 14.8 months, HR 0.47 [786][798].

The side effects in Table 17 are taken by example from the STAMPEDE study.

In accordance with the treatment regimen of the studies [798][803], abiraterone is recommended at the dosage of 1000 mg/day plus prednisone or prednisolone 5 mg/day starting within 3 months after initiation of androgen deprivation.

Table 18: Results of RCT on combination therapy with abiraterone [Fizazi et al. 2017/2019; James et al. 2017]

 

 

AD + Abiraterone

AD (study)

HR (confidence interval); significance

Benefit:

STAMPEDE

Overall survival at 3 years*

LATITUDE

Overall survival at 3 years (interim analysis)

Median survival final, months (95% CI)

 

83%

 

 

66%

 

53.3 (48.8 – NE)

 

76%

 

 

49%       (LATITUDE**]

 

36.5 (33.5-40.0)

 

0.63; p<0.001

 

 

0.62; p<0.001

 

0.66 (0.56-0.78), p<0.0001

 

STAMPEDE

failure-free survival, months

LATITUDE

progression-free survival, months (interim analysis)

 

43.9

 

33.0

 

30.0      (STAMPEDE)

 

14.8      (LATITUDE**)

 

13.9 mo; HR 0.29; p<0.001

 

18.2 mo; HR 0.47; p<0.001

Harms:

Side effects

(Exemplary from STAMPEDE study because larger patient population and longer follow-up)

47%

14%

10%

7%

5%

7%

5%

5%

4%

33%

14%

4%

5%

4%

1%

3%

2%

2%

Grade 3-5 events

endocrine diseases

cardiovascular Diseases

musculoskeletal diseases

gastrointestinal diseases

hepatic diseases

general diseases

respiratory diseases

abnormal laboratory values

Deaths during the course of treatment

9

5

3            (STAMPEDE)

4            (LATITUDE**]

 

* each for the entire study population; LATITUDE and STAMPEDE differed in inclusion criteria.

** Interim analysis data

 

Re: 7.22d, e and 7.24d, e (docetaxel)

Three randomized clinical trials and a meta-analysis [804] were identified to assess the effectiveness of combination therapy of docetaxel and AD. Two of three trials evaluating combination therapy of docetaxel with concurrent AD showed significant prolongation of overall survival (OS) by 15 and 13.6 months (60 vs. 45 and 57.6 vs. 44 months; 2,962 and 790 patients, respectively) [788][794]: differences in outcomes in a third trial (OS 62.1 vs. 48.6 months; 385 patients) were not statistically significant [793]. Progression-free survival (PFS) or survival without treatment failure was significantly prolonged by combination therapy in all three trials (progression: by 10 and 8.5 months, respectively; treatment failure: by 17 months). Two of three studies (CHAARTED and GETUG) performed a subgroup analysis for patients with high tumor burden (defined in both studies as "visceral metastases or ≥4 bone lesions with ≥ 1 beyond vertebral bodies and pelvis", in GETUG only as post-hoc analysis) and find significantly better results for this subgroup. The study with the largest population (STAMPEDE) does not perform this subgroup evaluation a priori and comes to a significant result for the whole group. In a retrospective analysis of imaging at study inclusion with a subsequent classification according to high and low volume criteria (corresponding to CHAARTED), an evaluation with regard to metastatic burden was also performed for the STAMPEDE study. In contrast to the CHAARTED study, there was no heterogeneity between the treatment groups with different tumor volumes (interaction p-value: 0.827), i.e., according to the authors, the benefit of chemotherapy was independent of tumor burden.

None of the three studies performed subgroup analyses regarding symptomatic versus asymptomatic patients. Given the restrictive inclusion criteria of the trials and prognostically favorable factors such as a median age of 63.5-65 years and the increased grade 3-5 toxicities observed in all trials in the respective docetaxel arm, the recommendation is for patients in good general health with ECOG scores of 0 or 1 in whom the metastatic pattern meets high-volume criteria.

Conflicting data are available for low-volume metastasis. While in some studies or meta-analyses no influence is seen for the "metastatic volume" on the efficacy of early chemohormonal therapy, in other evaluations, no advantage for low-volume patients can be demonstrated [805][806]. In view of the numerous treatment alternatives now available and the toxicity profile of chemohormonal therapy compared with combined hormone therapy, the recommendation for low-volume metastasis has been weakened ("can").

The dosing recommendation of 75 mg/m² every three weeks for six cycles for docetaxel administration in chemohormonal therapy is consistent with the dosing and prioritized frequency used in RCTs addressing this question [788][794] and has thus found its way into the European Medicines Agency's SmPC. In contrast to metastatic castration-refractory prostate cancer (cf. Recommendation 7.39), docetaxel administration at 50 mg/m² every two weeks at the metastatic hormone-sensitive stage has not been investigated in prospective studies and is therefore not recommended. According to the longest time period available in the included studies, the start of chemotherapy is recommended no later than 4 months after the start of androgen deprivation.

Table 19: Results of the RCT on combined hormone chemotherapy

 

AD + docetaxel

AD (study)

Difference, Significance

Benefit:

Overall survival [months]

62.1

57.6

60

48.6      [GETUG)

44              (CHAARTED)

45          (STAMPEDE)

13.5 M., n.s.

13.6 M., p<0.001

15 M., p=0.005

Subgroup with high tumor burden

39.8

49.2

35.1      (GETUG)

32.2              (CHAARTED)

4.7 M., n.s.

17 M., p<0.001


progression-free survival/
failure-free survival

22.9

20.2

37

12.9      (GETUG)

11.7              (CHAARTED]

20          (STAMPED)

10 M., p=0.005

8.5 M., p<0.001

17 M., p<0.001

Harm:

Side effects

(exemplary from STAMPEDE study, because largest patient collective and good documentation)

52%

15%

12%

10%

8%

7%

6%

5%

4%

3%

3%

1%

32%

1%

0%

12%

3%

4%

6%

2%

6%

3%

2%

0%

Grade 3-5 events febrile neutropenia

neutropenia

endocrine disorders

gastrointestinal diseases

general diseases

musculoskeletal diseases

respiratory diseases

renal diseases

cardiac disorders

diseases of the CNS

nail changes

Treatment-related deaths

4

1

1

0            (GETUG)

0              (CHAARTED)

0            (STAMPED)

 

 

Re: Recommendation 7.23

There are now numerous combination therapies for mHSPC for which superiority over AD alone has been demonstrated. The various therapies differ, among other things, in terms of approval status or indication as well as spectrum of side effects. No statement can be made regarding the advantages of one or the other variant of combination therapy for specific patient groups, as there are no directly comparative studies. Only in the multi-arm STAMPEDE trial was there a docetaxel and an abiraterone arm, and a stronger effect in terms of time to treatment failure was reported with abiraterone than with docetaxel, but again these were not in a direct comparison. In contrast, there was no difference in overall survival or tumor-specific survival. There is also an indirect comparison for apalutamide to the docetaxel arm of the STAMPEDE study (see above); an IQWIG assessment of this comparison found evidence of a non-quantifiable added benefit for apalutamide due to advantages in the side effects category. Overall, the new generation of hormone drugs seems to have a more favorable side-effect profile; on the other hand, the duration of therapy is longer, and for high-risk patients taking abiraterone, the long-term administration of glucocorticoids should be considered as compared to docetaxel chemotherapy.

Overall, the choice of therapy should therefore be made for the appropriate indication, taking into account patient preferences, possible side effects, and the existing individual comorbidity profile after participatory decision making. It should be taken into account that relatively long survival times can also be achieved with a "simple" AD, such that, for example, a combination treatment does not necessarily have to be administered in cases of presumed limited life expectancy. In this sense, the restriction of the recommendations to patients with good performance status (ECOG 0-1) should also be understood, since it is important to doubt that patients who do not improve accordingly in performance status within 3 months on an initial hormone therapy will actually benefit from early combination treatment.

 

Re: Recommendation 7.25

a)     Immediate hormone-ablation therapy is associated with prolonged progression-free survival [734]. However, as stated in Chapter 6.7 "Primary Hormone Ablative Therapy and Watchful Waiting" in non-metastatic prostate cancer, the results are inconclusive in both non-metastatic and metastatic disease with respect to overall survival. However, due to the good response rates and prolongation of progression-free survival in the symptomatic metastatic stage, a strong recommendation is made for immediate hormone-ablation therapy. Cause-oriented treatment is clearly preferable to symptomatic treatment. In addition to prolonging progression-free survival, there is evidence that early initiaton of androgen deprivation reduces complications due to progression of the underlying disease (e.g., due to pathological fractures) [737].

With regard to both the indication and other aspects of androgen deprivation (AD), the situation of patients with localized prostate cancer cannot be reliably differentiated from that of patients with metastatic PCa on the basis of published analyses. Furthermore, there is no evidence that hormonal patients in localized tumor stages behave differently with respect to response to AD than those with metastatic PCa. Accordingly, both the meta-analysis by Wilt 2001 [734] and the ASCO guidelines of 2004 and 2007 [36][737], respectively, and this guideline, used study results from patients with localized and advanced stages for the recommendations [806].

 

b)     A similar recommendation can be found in the chapter Watchful Waiting and Hormone-Ablation Therapy Alone in Non-metastatic Prostate Cancer. The recommendation cites agents that have been effectively used for AD in randomized-controlled trials. The systematic review by Kunath et al. 2019 [789] includes studies of orchidectomy and LH-RH agonists. In addition, estrogens or DES have been used in the VACURG [739] studies. Iversen 2006 [728] uses bicalutamide; Studer 2006 [729] also uses LH-RH agonists or orchidectomy. The use of GnRH blockers has been derived from its effect on lowering testosterone levels, as seen in LH-RH agonists. Of the GnRH antagonists, Abarelix has been approved since 2005, and Degarelix since February 2007 for the indication of hormone-ablation therapy in advanced prostate cancer (Jakob et al. 2017/2021) [807]. Monotherapy with steroidal antiandrogens is associated with shorter progression-free survival compared with LH-RH-analog therapy and should not be recommended [36].

 

c)       Two meta-analyses [795][796] form the basis for this recommendation, each summarizing primary studies comparing continuous and intermittent AD. The majority of included studies, including the largest study with more than one thousand patients [797], had an inclusion criterion of a drop in PSA level below 4 ng/ml following an induction period (up to 7 months) to then be randomized to a continuous or intermittent treatment group. For patients with higher levels after the AD induction phase, the guideline group believes there are insufficient data on the efficacy and safety of intermittent AD, and therefore it is not recommended for this indication.

 

(d)     The available pooled studies, mostly of moderate quality, included patients of different stages and appropriate subgroup analyses were not performed. The meta-analyses, as well as the largest study, which included only metastatic stages, cannot demonstrate clear inferiority or superiority of any of the treatment options in terms of overall or cancer-specific survival, as well as time to progression. However, the Hussain trial showed a non-significant survival benefit of a median 5.8 vs. 5.1 years for continuous AD, with clear limitations. Data are also unclear or insufficient regarding the potential for harm from side effects as well as effects on quality of life; therefore, special consideration should be given to the individual patient's circumstances.

7.4 Treatment of androgen-independent or castration-resistant prostate cancer (CRPC)

7.26 · Evidence-based Recommendation · checked 2021

Patients with castration-resistant prostate cancer shall be provided with information about the following:

  • A cure cannot be achieved.
  • Various options are available for further treatment.
Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (100%)
7.27 · Evidence-based Recommendation · checked 2021

In patients with symptomatic progressive disease undergoing castration with medication, therapeutic options and the therapeutic approach should be discussed and determined on an interdisciplinary basis.

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
4
Consensus
Strong consensus (97%)
7.28 · Evidence-based Recommendation · checked 2021

The following factors shall be considered when making a treatment decision:
•   symptoms
•   side effects of treatment options
•   patient preference
•   comorbidities, life expectancy, and quality of life
•   progression over time
•   localization of metastases and general tumor burden.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (100%)
7.29 · Evidence-based Statement · checked 2021

Suitability for treatment with chemotherapy is not a clearly defined variable. Therefore, there is a lack of threshold values above which suitability for treatment can be determined or otherwise.

Level of evidence
4
Consensus
Strong consensus (97%)
7.30 · Evidence-based Statement · checked 2021

A geriatric assessment is useful for decision making before initiating tumor-specific therapy in multimorbid patients older than 70 years.

Level of evidence
4
Consensus
Consent (95%)
7.31 · Consensus-based Recommendation · checked 2021

Serum testosterone levels shall be monitored in patients with progressive disease undergoing surgical or drug castration therapy.

Consensus
Strong consensus (100%)

Re: Recommendations 7.26 - 7.28

Treatment for patients with advanced prostate cancer has changed fundamentally in recent years. In particular, the introduction of new drugs for systemic therapy has led to an increase in survival at different stages of the disease.

The treatment of castration-resistant prostate carcinoma is a palliative therapy. This fact must be taken into account when determining the indication for therapy. Patients should be informed accordingly.

The therapeutic options involve different disciplines. An interdisciplinary tumor conference for castration-resistant prostate cancer includes urology, internal oncology, pathology, radiotherapy and radiology, and depending on the symptoms and the therapeutic option to be discussed, also nuclear medicine, orthopedics, neurosurgery or other specialist disciplines.

The goals of therapy are to prolong survival, relieve symptoms, improve or maintain quality of life, and prevent complications. Therapy-associated side effects must be critically weighed up against the outcomes of therapy. Decision-making requires comprehensive patient education, which must include all aspects to be weighed up for a treatment decision.

Local treatment options for symptomatic or symptom-prone lesions relate to surgical and/or radiotherapeutic interventions. In addition to its use in the treatment of bone metastases, palliative radiotherapy is used for symptomatic locoregional pelvic tumors (primary tumor or lymphoma), especially for painful, obstructing, and/or bleeding tumors (can also be applied as re-radiation), in rare cases for pulmonary metastases (in the presence or threat of dyspnea), or as irradiation of cerebral metastases [808][809].

Systemic treatment options in patients with castration-resistant prostate cancer include antihormonal treatments, chemotherapy, and radionuclide therapy, as well as bone-modifying agents and supportive therapy measures.

The supporting literature is set out in the specific recommendations that follow. The order is based on clinical indications:

-       Non-metastatic castration-resistant prostate cancer (nmCRPC).

-       Metastatic castration-resistant prostate cancer (mCRPC).

o    Asymptomatic or mildly-symptomatic patients.

o    Symptomatic patients

o    Treatment sequence following initial therapy with at least one new hormonal agent (new hormonal agent)

o    Treatment sequence following docetaxel

o    Treatment sequence following androgen receptor-targeted treatment

 

Re: Statement 7.29

Chemotherapy is a therapy with relatively limited therapeutic breadth. The occurrence of adverse drug reactions (ADRs) is the rule, not the exception, even with standard-dose therapy. The toxicity of systemic therapy is classified using Common Terminology Criteria for Adverse Event (CTCAE)[http://evs.nci.nih.gov/ftp1/CTCAE/About.html]. In addition to this objective toxicity, the subjective burden on the patient plays a significant role. Both have to be recorded and weighted up in a structured way in the context of treatment decisions (continuation of therapy, dose modification and discontinuation of therapy). Therapeutic burdens are more likely to be accepted if the (supposed) advantages of therapy are great. Discontinuation of therapy, a high rate of toxicity, and dose modifications are therefore indications of low suitability for treatment.

 

Re: Statement 7.30

Given that aging is a highly heterogeneous process and conventional clinical history taking and physical examination often miss age-associated changes [810][811] and these changes are associated with outcome variables [812], in patients with cancer aged 70 years and older, performance of an assessment prior to initiation of chemotherapy is recommended by national and international professional societies [813][814][815][816]. Specific recommendations for patients with prostate cancer have been made by the International Society of Geriatric Oncology (SIOG) [418][817].

In the CRASH (Chemotherapy Risk Assessment for High Aged Patients) study, Extermann et al. analyzed variables associated with the occurrence of grade 3-4 nonhematological toxicities and grade 4 hematologic toxicities according to CTCAE criteria [818]. 518 patients aged 70 years and older treated with chemotherapy were included [818]. Using the following variables, 4 risk groups (low = 7 %; intermediate-low = 23 %; intermediate-high = 54 % and high = 100 %) could be created for the occurrence of grade 4 hematologic toxicities: Lymphocytes, ASAT, Instrumental Activities of Daily Living (IADL), LDH, diastolic blood pressure, and MAX-2 value of chemotherapy [818]. For the occurrence of grade 3-4 non-hematologic toxicities, 4 risk groups (low = 33 %; medium-low = 46 %; medium-high = 67 % and high = 93 %) could be identified using the variables: Hb, albumin, creatinine clearance, ECOG-PS, Mini-Mental Status Examination (MMSE), Mini-Nutritional Assessment (MNA), and MAX-2 value of chemotherapy form [818]. Docetaxel 75 mg/m² body-surface 3-weekly has a score of 2 on the CRASH score, while docetaxel 35 mg/m² body-surface weekly has a score of 0 on the CRASH score [818].

In a prospective cohort study that included 500 patients aged 65 years and older, median 73 years (range 65–91 years), Hurria et al. examined predictors of chemotherapy associated toxicity grade 3-5 [819]. Among others, geriatric assessment variables (mobility limitation, falls, need for assistance in instrumental activities of daily living (IADL), limitation of social activities) were predictors of low, intermediate, or high risk of grade 3-5 toxicities [819].

In a prospective cohort study that included patients aged 70 years and older who were scheduled to receive first-line chemotherapy, Soubeyran et al. analyzed geriatric assessment variables for their role in predicting death within the first six months [820]. Advanced disease, limitation in the Mini-Nutritional Assessment, and limitations in mobility were associated with 6-month mortality [820].

 

Re: Recommendation 7.31

The background to this recommendation is the fact that androgen-independent or castration-resistant disease can only be assumed once adequate castration-equivalent testosterone suppression has been implemented. Therefore, a serum testosterone level below 20-50 ng/dl is the target, and this is documented (definition of castration resistance based on the EAU guideline: serum testosterone <50 ng/dl with concomitant biochemical (three consecutive PSA increases one week apart yielding two increases of 50 % above baseline, and a PSA level >2 ng/mL)) or radiological progression [674][821]. In progressive disease, testosterone levels should be monitored. Progression of disease may be evident clinically, biochemically (see definition above), or on imaging.

7.4.1 Non-metastatic castration-resistant prostate cancer (nmCRPC)

7.32 · Evidence-based Recommendation · new 2021

Patients with non-metastatic castration-resistant prostate cancer (nmCRPC) and a high risk of metastasis (PSA doubling time of <10 months) shall be offered apalutamide, darolutamide, or enzalutamide (alphabetical order) in addition to androgen deprivation therapy (ADT).

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Strong consensus (96%)
References
[822] [823] [824] [825] [826] [827] [828] [829] [830]
Evidence tables (3)Saad_LancetOncol_2018_Small_AnnOncol_2019_Smith_NEJM_2018_Smith_EurUrol_2020; Fizazi_NEJM_2019_Fizazi_NEJM_2020; Hussain_NEJM_2018_Sternberg_NEJM_2020_Tombal_LancetOncol_2019
7.33 · Consensus-based Recommendation · new 2021

The indication for intensified treatment according to 7.32 should take into account presumed life expectancy (based on age, among other factors) as well as potential adverse effects and quality of life.

Consensus
Strong consensus (97%)

Re: Recommendation 7.32          

The definition of castration-resistant prostate cancer includes [821]:

-       therapy with a GnRH agonist, a GnRH antagonist, or status post bilateral orchidectomy; and

-       PSA rise in three consecutive analyses at least one week apart, and

-       PSA level >2 ng/ml or

-       radiological progression (as defined by the Prostate Cancer Working Group) and 

-       testosterone level <50 ng/dl.

Prostate cancer patients with an increase in PSA when taking sufficient androgen deprivation treatment represent a heterogeneous group. The clinical picture ranges from initial clinical symptoms (e.g., pain) with synchronous evidence of metastases on imaging, to delayed evidence of metastases on imaging and long, protracted courses with exclusively "PSA disease". Unfavorable prognostic factors include [831][661]:

-       short interval between radical prostatectomy and biochemical progression.

-       Gleason score >8

-       Baseline PSA level >2 ng/ml

-       PSA doubling time <10 months.

Three androgen receptor pathway inhibitors are approved for systemic therapy of non-metastatic castration-resistant prostate cancer in patients with a PSA doubling time of <10 months: Apalutamide, Darolutamide and Enzalutamide. This entails the creation of a new indication. The basis for the pivotal studies was conventional imaging. In this case, enlarged pelvic lymph nodes <2 cm were not an exclusion criterion. The results of the three substances are comparable (see below). Even with the new options, therapy is not curative. Results are discussed in alphabetical order of the approved drugs:

Apalutamide was approved for use in the European Union (EU) in January 2019. It was based on SPARTAN, an international, multicenter, randomized, placebo-controlled phase-III study of 1,207 patients [825][822][823][824][802]. Randomization was 2:1 in favor of the verum arm. The median age was 74 years [825]. After the primary study endpoint was met, the study was unblinded in July 2017, allowing crossover [825]. 76 patients (19%) crossed over from the control to the intervention arm [825].

Apalutamide + androgen deprivation (ADT) significantly prolonged overall survival (median 14 months; HR 0.78; p=0.016) [824]. Crossover of 19% of patients into the apalutamide arm may mean the impact of apalutamide on survival is underestimated. This assumption is supported by mathematical corrections using the Rank Preserving Structure Failure Time Model (RPSFTM) and Inverse Probability of Censoring Weighting (IPCW).

Radiologically-assessed metastasis-free survival was the primary endpoint of the pivotal study [825]. Imaging was performed every 16 weeks. Apalutamide significantly delayed the appearance of metastases detectable on imaging with a median metastasis-free survival of 40.5 vs. 16.2 months, hazard ratio 0.28 [825]. At 12 weeks, PSA had decreased by at least 50% in 89.7% of patients in the apalutamide arm, and had increased in 40.2% of patients in the control arm at this time [825]. The median time to PSA progression was significantly prolonged by apalatumide, hazard ratio 0.06 [825].

Time to symptomatic progression was significantly prolonged in the apalutamide arm with a hazard ratio of 0.45 [825]. Symptomatic progression was defined as time to the occurrence of a bone-associated event, pain progression, or worsening of disease-associated symptoms leading to initiation of a new systemic therapy, or time to the occurrence of significant clinical symptoms due to local or regional tumor progression leading to surgery or radiation treatment [825]. Data on quality of life and patient-reported outcome showed few overall differences between treatment arms; changes in favor of apalutamide were found on the pain scale.  

The rate of severe CTCAE grade 3/4 adverse events was 45.1% in the ADT/apalutamide arm and 34.2% in the ADT/placebo arm [825]. More common among apalutamide were hypertension (14.3%), rash (5.2%), weight loss (1.1%), tendency to fall (1.7%), and fractures (2.7%) [825]. Among the non-severe adverse events, confusion, hypothyroidism, and mental changes occurred in clusters [825]. Therapy-associated mortality was 1.2% with apalutamide and 0.3% in the control arm [825]. The discontinuation rate due to adverse events was 10.6% in the apalutamide arm versus 7.0% in the placebo arm [825].

Darolutamide was approved for the EU in March 2020. The basis of the approval is ARAMIS, an international, multicenter, randomized, placebo-controlled phase III study of 1,509 patients [826][827][832]. Randomization was 2:1 in favor of the verum arm [826]. The median age was 73.6 years [826]. After completion of the treatment phase, treatment was unblinded [826]. Patients in the placebo arm were allowed to switch to the verum arm (crossover, switching) [826].

Overall survival was significantly prolonged by darolutamide, HR 0.69 (p=0.003) [827]. Median survival was not reached at the last data cutoff point in either study arm [827]. Crossover of 170 patients (30.6 %) from the control to the intervention arm may mean the impact of darolutamide on survival is underestimated. This assumption is supported by mathematical corrections using the Rank Preserving Structure Failure Time Model (RPSFTM) and Iterative Parameter Estimation.

Radiologically-assessed metastasis-free survival was the primary endpoint of the pivotal study [826]. Imaging was performed every 16 weeks. Darolutamide significantly delayed the occurrence of metastases detectable on imaging, with a median metastasis-free survival of 40.4 vs. 18.4 months, hazard ratio 0.41 [827]. The median time to PSA progression was prolonged by darolutamide from 7.3 to 33.2 months, hazard ratio 0.13 [827]. 

Time to pain progression was significantly prolonged in the darolutamide arm with a hazard ratio of 0.65, as was time to cytotoxic therapy (HR 0.43) and time to symptomatic bone-associated event (HR 0.43) [826]. Quality of life and patient-reported outcome showed few differences between treatment arms. Changes in favor of darolutamide were found in terms of worsening quality of life, pain intensity, and micturition discomfort scales.

The rate of serious adverse events was comparable in the two study arms [826]. The rate of CTCAE grade 3/4 adverse events was 24.7% in the ADT/darolutamide arm and 19.5% in the ADT/placebo arm [826]. More frequently found in darolutamide patients were arterial hypertension, coronary artery disease, and arrhythmias. However, these figures do not take into account the more than two-fold increase in exposure time in the darolutamide arm [826]. The discontinuation rate due to adverse events was 8.9 % in the darolutamide arm vs.8.7% in the placebo arm [826].

Enzalutamide was approved for this indication in October 2018. It was based on PROSPER, an international, multicenter, randomized, placebo-controlled phase-III study [828][829][830][833]. Patients were randomized 2:1 in favor of the verum arm [829]. The median age was 74 years [829]. After the primary study endpoint was met, the study was unblinded to all patients and entered an open-label period [829]. Within this period, all eligible patients received enzalutamide at the investigator's discretion [829]. At the time of the third data cutoff, 87 patients had crossed over from the control to the enzalutamide arm [828].

Enzalutamide resulted in a 10.7-month increase in median overall survival (67.0 vs. 56.3 months; HR 0.73; p=0.001)) compared with placebo [828]. The switch of 87 patients (18.6%) from the control to the enzalutamide arm may lead to an underestimation of the impact of enzalutamide on overall survival. Analysis of the impact of crossover was performed using the Rank Preserving Structure Failure Time Model (RPSFTM), and confirms the significant impact of enzalutamide on overall survival.

Radiologically-assessed metastasis-free survival was the primary endpoint of the pivotal study [829]. Imaging was performed every 16 weeks. Enzalutamide significantly delayed the appearance of metastases detectable on imaging, with a median metastasis-free survival of 36.6 vs. 14.7 months, hazard ratio 0.29 [828]. The PSA response rate (decline >50 %) was 76.3 % in the enzalutamide arm vs. 2.4 % in the placebo arm, p<0.0001 [829]. At a higher threshold (decrease >90%), the PSA response rate was 55.9% in the enzalutamide arm versus 0.4% in the placebo arm, p<0.0001 [829].

Data on quality of life and patient-reported outcome were collected using the EORTC QLQ-PR25 and FACT-P disease-related questionnaires [829]. There were few differences between treatment arms. Changes in favor of enzalutamide were found with respect to time period to first worsening of micturition symptoms; and to the disadvantage of enzalutamide with respect to the side effects of antihormone therapy. A positive influence of enzalutamide on metastasis-associated symptoms such as pain, fatigue, etc. was not found. 

The rate of severe CTCAE grade 3/4 adverse events was 48% in the ADT/enzalutamide arm and 27% in the ADT/placebo arm [829]. When corrected for exposure time, the differences were not significant (17% versus 20%). More common among enzalutamide than in the control arm were (all severities): fatigue (46%), musculoskeletal events (34%), hypertension (18%), falls (18%), impaired cognition and memory (8%), cardiovascular events (6%), ischemic heart disease (6%), and second neoplasms (5%) [829]. The discontinuation rate due to adverse events was 17% in the enzalutamide arm versus 9% in the placebo arm [829].

In the absence of data from direct comparisons of the three approved androgen receptor pathway inhibitors, a recommendation for a specific drug is not made.

 

Re: Recommendation 7.33

Intensified treatment with a combination of an androgen receptor pathway inhibitor with androgen deprivation increases the risk of adverse events, including arterial hypertension and cardiovascular events. Patients with severe cardiac history were excluded from the pivotal trials. The patient's quality of life must be considered both in the indication but also in the continuation of such therapy. Side effects such as impaired cognition and memory may also affect the patient's autonomy. If necessary, relatives should also be involved in the recording of side effects and their evaluation. In patients with cardiac risk factors, close monitoring is advisable. In the case of the three androgen receptor pathway inhibitors, attention should be paid to drug interactions in terms of both attenuation and enhancement of the effect [834].

7.4.2 Metastatic castration-resistant prostate cancer (mCRPC)

7.4.2.1 Asymptomatic or mildly-symptomatic patients

7.34 · Evidence-based Recommendation · checked 2021

Patients with metastatic, castration-resistant, asymptomatic or mildly-symptomatic and progressive disease who are taking an androgen deprivation treatment can be offered a change of treatment provided the benefits and side effects are explained to them. The specific conditions and side effects of the therapies should be taken into account.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
4
Consensus
Strong consensus (97%)
7.35 · Evidence-based Recommendation · checked 2021

When a patient with metastatic, castration-resistant, asymptomatic, or low-symptomatic and progressive disease has decided against a wait-and-see approach, opting to switch treatment, one of the following options shall be offered:

(alphabetical order)

·       abiraterone (in combination with prednisone/prednisolone)

·       docetaxel

·       enzalutamide

For differential therapy, see Recommendations 7.36 and 7.37.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Strong consensus (100%)
References
[835] [836] [837] [838]
Evidence tables (3)Beer_NEJM_2014_Beer_EurUrol_2017; Berthold_JClinOncol_2008; Ryan_NEJM_2013
7.36 · Evidence-based Recommendation · checked 2021

Patients with metastatic, castration-resistant, asymptomatic, or mildly-symptomatic and progressive disease should be offered (in alphapetical order):

·         abiraterone (in combination with prednisone/prednisolone); or

·         enzalutamide

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
1+
Consensus
Strong consensus (100%)
References
[837] [838]
Evidence tables (2)Beer_NEJM_2014_Beer_EurUrol_2017; Ryan_NEJM_2013
7.37 · Evidence-based Recommendation · checked 2021

Patients with metastatic, castration-resistant, asymptomatic, or mildly-symptomatic and progressive disease can be offered docetaxel.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
1+
Consensus
Strong consensus (96%)
References
[835] [836]
Evidence tables (1)Berthold_JClinOncol_2008

Re: Recommendation 7.34 and 7.35

The group of patients not having undergone prior therapy with a new hormonal agent will become smaller in the coming years. The term new hormonal agent includes abiraterone, apalutamide, darolutamide or enzalutamide. As these patients continue to represent a significant population at this time, the recommendations from previous versions of this guideline are carried forward and supplemented with updated data as appropriate.

Progression of metastatic disease in castration-resistant prostate cancer is viewed as a continuum. A threshold for switching therapy has not been defined by studies. The decision regarding the timing of conversion of previously administered androgen deprivation to more advanced therapy measures must be made on an individual basis. Various forms of androgen deprivation are available for patients who choose a wait-and-see approach.

The basis for recommendations 7.34 and 7.35 in metastatic castration-resistant prostate cancer are studies conducted prior to the use of extended therapy in hormone-sensitive prostate cancer (see Chapter 7.3) and in non-metastatic castration-resistant prostate cancer (see Chapter 7. 4.1), respectively. Prior use of abiraterone, docetaxel, or androgen pathway inhibitors should be considered in the choice of follow-up therapies in metastatic stage.

Data from randomized trials on forms of antihormone therapy with abiraterone or enzalutamide, as well as chemotherapy with docetaxel, are available for the treatment of patients with metastatic, castration-resistant, asymptomatic or mildly-symptomatic and progressive disease. Immunotherapy with sipuleucel-T, listed in the guideline as of 2014, is no longer available in Europe as of mid-2015. Abiraterone and enzalutamide were tested against placebo, docetaxel against mitoxantrone. Abiraterone is given in combination with prednisone or prednisolone by default. Recommendations for this patient population are based on the inclusion criteria for  therapy with docetaxel [835][836], abiraterone [837], and enzalutamide [838] in patients without prior therapy at this disease stage. Progressive disease was defined as PSA progression according to Prostate Cancer Clinical Trial Working Group (PCWG2) criteria [839] or imaging progression in the abiraterone study, PSA and/or progression on imaging in the enzalutamide study, and PSA progression (increasing values in three consecutive measurements) or progression on imaging in the docetaxel study. Only patients with a Brief Pain Inventory-Short Form (BPI-SF) score of 0-3 as the most severe pain in the last 24 hours were included in the abiraterone trial as well as in the enzalutamide trial. Whether to initiate chemotherapy with docetaxel in asymptomatic patients with a PSA rise alone or with progression demonstrated by imaging is a matter of controversy. In subgroup analysis of TAX-327 [836][840], survival was also prolonged in patients without pain symptoms, in patients without visceral metastasis, and in patients with minimally symptomatic disease compared with mitoxantrone. To date, there has been no randomized-controlled trials on the benefit of early therapy in asymptomatic patients versus later therapy in symptomatic patients.

 

Re: Recommendation 7.36

The selective 7α-hydroxylase/C17,20-lyase (CYP17) inhibitor abiraterone causes extragonadal hormone suppression. The COU-AA-302 study (n = 1088) showed a survival benefit for abiraterone in combination with prednisone compared with placebo with prednisone (median overall survival: 34.7 vs. 30.3 months, HR: 0.81, 95% CI: 0.70-0.93, p = 0.0033) [841]. The predefined significance level (0.001) was not reached in this study. Since crossover from the placebo to the abiraterone arm was allowed in case of disease progression, the meaningfulness of the survival endpoint is limited. In addition, continuing therapy at progression after abiraterone was not defined. In the pivotal study [837], an increase in progression-free survival of approximately eight months was reported at the time of the second interim analysis. Compared to placebo, abiraterone (in combination with prednisone/prednisolone) showed a significant effect on several endpoints (progression-free survival, biochemical and imaging remission, symptomatology, and quality of life) in the interim analysis. In contrast, no data on progression-free survival and the other endpoints mentioned above are provided in the final analysis. Only patients with good general health (ECOG 0-1) were included. Patients with visceral metastases were not included in the study. In the pivotal study [837], 33% (n = 178) of patients on abiraterone plus prednisone and 26% (n = 142) of patients on placebo plus prednisone had at least one serious adverse event. In the final publication [841], the most common serious adverse events cited were heart disease (8 vs. 4%), elevated ALT levels (6 vs. <1%), and hypertension (5 vs. 3%) [841]. Very common side effects (> 10%) with abiraterone, according to the EMA document, are urinary tract infections, hypokalemia, hypertension, and peripheral edema; heart disease, hepatotoxicity, and fractures are other important side effects [842]. Abiraterone is approved in combination with prednisone/prednisolone for the treatment of metastatic castration-resistant prostate cancer in adult men with asymptomatic or mildly-symptomatic disease after failure of androgen deprivation therapy in whom chemotherapy is not yet clinically indicated [842].

Enzalutamide inhibits androgen receptor signaling at multiple levels. In addition to inhibiting the androgen receptor, enzalutamide prevents its translocation to the nucleus, DNA binding, and cofactor activation. The pivotal PREVAIL study included 1,717 chemotherapy-naïve patients with castration-resistant prostate cancer. Only patients in good general health (ECOG 0-1) were included. In contrast to the COU-AA302 trial of abiraterone, inclusion of patients with visceral metastasis was allowed in the PREVAIL trial. At baseline, 11.2% of patients in the enzalutamide arm and 12.5% of patients in the placebo arm had visceral metastasis with pulmonary and/or hepatic metastases [843]. Both the interim analysis [838] and the long-term analysis showed a survival benefit for enzalutamide over placebo [844] (median overall survival: 35.3 vs. 31.3 months, HR: 0.77, 95% CI: 0.67-0.87, p=0.0002). Radiographic progression-free survival, defined as the co-primary endpoint, was 20 months with enzalutamide vs. 5.4 months in the control arm (HR 0.32; p<0.00001) [844]. Among secondary endpoints, patient-relevant significant differences were found between enzalutamide and placebo arms (time to first SRE, duration to worsening quality of life, or initiation of opioid therapy as surrogate parameters for pain). Very common side effects with enzalutamide include flushing (HR = 2.29) and headache [844]. Common side effects include neutropenia, visual hallucinations, anxiety, cognitive impairment, memory impairment, hypertension, dry skin, pruritus, fractures, falls. Drug Information [845] also notes interactions with CYP2C8 and CYP3A4 inhibitors and inducers. A possible influence of enzalutamide on other drugs is listed for 14 drug groups. Enzalutamide is approved for the treatment of adult men with metastatic castration-resistant prostate cancer with asymptomatic or mildly-symptomatic progression after failure of androgen deprivation therapy in whom chemotherapy is not yet clinically indicated [846].

Uncertainty currently remains regarding sequence therapy [847][848]. Considerations and recommendations are summarized in section 7.4.2.3.

 

Re: Recommendation 7.37

The TAX-327 trial (n =1,006, Karnofsky performance status ≥ 60 %) showed a survival benefit of 2.9 months (range zero to seven months) with three-weekly docetaxel compared with the other two arms (weekly docetaxel lower dose, three-weekly mitoxantrone; randomization 1:1:1) for the overall group [835][836]. 45 % of patients were symptomatic at baseline (pain defined as a score ≥ 2 on the Present Pain Intensity (PPI) scale or a score ≥ 10 on the Analgesic Score). 13% of patients had a Karnofsky performance status ≤ 70%. Serious adverse events (Grade 3/4) that occurred in more than 20 % of patients in the pivotal studies were: alopecia (65 %), fatigue (53 %), nausea/vomiting (42 %), neutropenia (32 %), diarrhea (32 %), sensory neuropathy (30 %), onychodystrophy (30 %). The proportion of therapy-associated deaths was 0.3%. Significantly more patients receiving docetaxel reported improvement in quality of life compared with mitoxantrone, but median changes were small [835]. In the TAX-327 trial, weekly administration was associated with a median prolongation of overall survival (0.9 months, n. s.) compared with mitoxantrone in addition to three-week administration [835]. Another study showed a survival benefit of two-week versus three-week administration of docetaxel and a lower rate of serious adverse events, particularly neutropenia [849]. As the study demonstrated a relatively short survival for three-week docetaxel compared with other studies of docetaxel and also has methodological weaknesses (e.g., per protocol analysis), two-week and three-week administration are equally recommended. Docetaxel is approved in combination with prednisone/prednisolone for the treatment of metastatic castration-resistant prostate cancer [850].

The cautious recommendation for the use of docetaxel takes into account the higher rate of severe side effects with docetaxel.

7.4.2.2 Symptomatic patients

7.38 · Evidence-based Recommendation · checked 2021

Patients with metastatic, castration-resistant, symptomatic progressive disease who are in a good general condition shall be offered systemic therapy, if necessary in combination with symptom-related and supportive therapy. For differential therapy, see Recommendations 7.39, 7.40, 7.41.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Strong consensus (98%)
References
[835] [836] [837] [851]
Evidence tables (3)Parker_Lancet_2018; Berthold_JClinOncol_2008; Ryan_NEJM_2013
7.39 · Evidence-based Recommendation · checked 2021

Patients with metastatic, castration-resistant, symptomatic, and progressive disease can be offered docetaxel in two or three-week dosing regimens.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
1+
Consensus
Consent (95%)
References
[835] [836]
7.40 · Evidence-based Recommendation · checked 2021

Patients with metastatic, castration-resistant, symptomatic, and progressive disease can be offered:

(in alphabetical order)

•        abiraterone (in combination with prednisone/prednisolone); or

•        enzalutamide

(0)

 

Patients shall be informed that only patients with low symptomatic disease were treated in the pivotal study.

(A)

 

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Consent (95%)
References
[837] [838]
Evidence tables (2)Beer_NEJM_2014_Beer_EurUrol_2017; Ryan_NEJM_2013
7.41 · Evidence-based Recommendation · checked 2021

Patients with castration-resistant, symptomatic, progressive disease and reduced general condition (ECOG ≥ 2, Karnofsky index < 70) shall be offered symptom-based therapy.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Consent (95%)
7.42 · Evidence-based Recommendation · modified 2021

Patients with castration-resistant, progressive disease and reduced general condition (ECOG ≥ 2, Karnofsky index <70) can additionally be offered one of the following treatment options:

(in alphabetical order)

·         abiraterone (in combination with prednisone / prednisolone);

·         chemotherapy, if the reduced general condition is primarily due to the metastatic prostate cancer;

·         enzalutamide;

·         steroids (dexamethasone, prednisolone, prednisone).

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
4
Consensus
Consent (95%)
References
[835] [836] [837] [838]
Evidence tables (3)Beer_NEJM_2014_Beer_EurUrol_2017; Berthold_JClinOncol_2008; Ryan_NEJM_2013

Re: Recommendation 7.38

Even in symptomatic patients, the group of patients not having undergone prior therapy with a new hormonal agent will become smaller in the coming years. The term new hormonal agent includes abiraterone, apalutamide, darolutamide, or enzalutamide. As these patients continue to represent a significant population at this time, the recommendations from previous versions of this guideline are carried forward and supplemented with updated data.

Treatment options for patients with symptomatic disease who are in a good general condition are addressed in Recommendations 7.38 – 7.39. Good general condition is defined by the guideline group as ECOG < 2 or Karnofsky index ≥ 70%. The description of local therapy procedures for bone metastases can be found in Chapter 7.6 "Treatment of bone metastases". Different inclusion criteria were used in the studies investigating the other therapy options, therefore the results are difficult to compare. Comparative studies or studies on combinations of therapy options are not yet available.

Patients with having undergone an initial hormone chemotherapy or intensified hormone therapy with abiraterone (+ prednisone or prednisolone) or apalutamide (possibly still Enza depending on the approval status) in this context represent a group for which no data formally exist within the framework of studies, since this therapeutic approach did not exist at the time of recruitment of these studies. Nevertheless, a conclusion by way of analogy is favored here by the guideline group.

 

Re: Recommendation 7.39

The study situation on docetaxel in patients without prior therapy in this disease stage is presented in the background text to Recommendation 7.37.

 

Re: Recommendation 7.40

The state of the literature on abiraterone (in combination with prednisone/prednisolone) and enzalutamide in patients without having undergone prior treatment at this disease stage is presented in the background text to Recommendation 7.36. Both agents are approved for use only in asymptomatic or mildly-symptomatic patients [842][846]. Mildly-symptomatic or asymptomatic was defined in the pivotal studies as a BPI-SF of < 3 as the most severe pain in the last 24 hours.

 

Re: Recommendations 7.41 and 7.42

Patients in a poor general state of health (ECOG ≥ 2, Karnofsky index < 70%) and patients with limitations on their geriatric assessment are limited in their suitability to be treated. There are no randomized trials for the treatment of patients with progressive disease and reduced general condition (ECOG ≥ 2). Studies of abiraterone (ECOG: 0-1), docetaxel (Karnofsky index ≥ 60 %), enzalutamide (ECOG: 0-1) did not include any or only a few patients with reduced general condition. Therefore, symptom-based therapy is recommended for these patients. Furthermore, various additional therapy options may be offered. Notes or limitations on therapy options are provided in the background texts for Recommendations 7.38-7.40.

Only if the reduced general condition is mainly due to the metastatic prostate carcinoma, chemotherapy with docetaxel can be offered. The study situation on docetaxel as first-line therapy is presented in the background text to Recommendation 7.37.

7.4.2.3 Therapy sequence after pre-therapy with at least one new hormonal agent

7.43 · Evidence-based Recommendation · new 2021

Patients with progression under a new hormonal agent (NHA) should be offered a change in therapeutic strategy.

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
4
Consensus
Consent (95%)
References
[852]
Evidence tables (1)Wit_NEJM_2019
7.44 · Evidence-based Recommendation · new 2021

Patients with progression after pre-therapy that included a new hormonal agent shall be offered testing for BRCA 1/2 mutations.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1-
Consensus
Strong consensus (97%)
References
[853] [854]
Evidence tables (1)Bono_NEJM_2020_Hussain_NEJM_2020
7.45 · Evidence-based Recommendation · new 2021

If a BRCA1/2 mutation is detected, therapy with olaparib shall be offered.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1-
Consensus
Strong consensus (98%)
References
[853] [854]
Evidence tables (1)Bono_NEJM_2020_Hussain_NEJM_2020

Re: Recommendation 7.43

The demonstration of efficacy of new hormonal agents in hormone-sensitive metastatic prostate cancer as well as in high-risk nmCRPC (M0CRPC) – as well as the use of docetaxel in metastatic hormone-sensitive prostate cancer – influences the entire sequence of follow-up therapies. Abiraterone, apalutamide, darolutamide or enzalutamide are grouped under the term of a new hormonal substance.

The choice of the respective therapy option is based on the patient's individual therapy goal on the basis of shared decision-making between patient and physician, taking into account the previous therapy and carefully weighing up the benefits and potential harms.

In particular, the results of the mode-of-action studies with the use of cabazitaxel [852] or olaparib [853][854] after undergoing prior therapy with one of the newer androgen receptor-targeted therapies should also be considered.   A change in therapeutic principle is also supported by molecular biology studies of resistance mechanisms to androgen receptor-targeted therapy [855][856], so Recommendation 7.45 has been generally formulated as an expert consensus. A further discussion of subsequent treatment can be found in Recommendation 7.52. 

The recommendations are based on the inclusion criteria of the respective pivotal studies and, if applicable, subsequent data.

 

Re: Recommendation 7.44

To date, the indication for systemic antineoplastic therapy in advanced prostate cancer has been based on laboratory, radiological, and clinical criteria. This is currently changing. In up to 30% of patients with prostate cancer, genetic aberrations are found in tumor cells that affect the physiological mechanism of DNA repair [857]. The most common of these are alterations in the BRCA2 (BReast CAncer 2) gene, but the BRCA1 gene is also affected. In this case, the gene alterations can be hereditary in carriers of germline mutations or have a somatic origin with acquired alteration confined to tumor tissue. Patients with BRCA1 or BRCA2 mutations are at increased risk for prostate cancer; the prognosis of patients is considered to be less favorable [858][859]. At the same time, the biology of BRCA-mutated tumor cells provides an approach for targeted therapy with poly(ADP-ribose) polymerase (PARP) inhibitors. PARP inhibitors were first approved in advanced ovarian cancer, and then also in advanced breast or pancreatic cancer with evidence of BRCA1/2 mutations.

The basis for recommending testing for BRCA1/2 mutations is PROfound, an international, multicenter, randomized trial comparing the efficacy of olaparib versus placebo in patients with evidence of a DNA repair mechanism defect in the homologous recombination repair genes [853][854]. Given that clinically significant efficacy of olaparib was only demonstrated in the PROFOUND trial in patients with BRCA1/2 mutations, testing for the other genetic alterations was not included in this recommendation. This also applies to the ATM mutations in cohort A.

The recommendation for BRCA1/2 testing is aimed at patients with metastatic castration-resistant prostate cancer who have already received therapy with new hormonal agents. The term new hormonal therapy includes abiraterone, apalutamide, darolutamide, or enzalutamide. If such a therapy has not been used before, it is indicated on the basis of the inclusion criteria of the PROFOUND study and the approval text based on it before olaparib is potentially used, and thus also before BRCA1/2 testing [860].   

 

Re: Recommendation 7.45

Olaparib is a PARP inhibitor. It was approved in the EU in November 2020 as monotherapy for patients with metastatic castration-resistant prostate cancer and BRCA1/2 mutation (germline and/ or somatic) whose disease is progressive after having undergone prior treatment that included a new hormonal agent. Olaparib is taken orally.

The basis for approval was the PROfound study. PROfound included 387 patients with metastatic prostate cancer, following a progression when on abiraterone or enzalutamide, and with evidence of mutations in BRCA1, BRCA2, or ATM (cohort A) or in 12 rarer HRR mutations (cohort B) [853][854]. Among these, approximately 60 % of patients had already received taxane-containing chemotherapy at study inclusion, and approximately 20% of patients were pretreated with both androgen receptor-targeted drugs. Randomization was between olaparib or abiraterone/enzalutamide in a 2:1 ratio in favor of the olaparib arm [853][854]. The choice between abiraterone or enzalutamide was made by the treating physician. Olaparib, compared with abiraterone/enzalutamide in cohort A (BRCA1/2; ATM), resulted in an increase in response rate (33 vs. 2%; odds ratio (OR) 20.86; p<0.001)), significant prolongation of progression-free survival (7.4 vs. 3.6 months; hazard ratio (HR) 0.34; p< 0.001) and prolongation of median overall survival (19.1 vs. 14.7 months; HR 0.69; p=0.02) [853][854]. The time to progression of pain was also prolonged (HR 0.44; p=0.02). A subgroup analysis on individual gene alterations showed an advantage of olaparib, especially for patients with BRCA1/2 mutations. Accordingly, the approval of olaparib is limited to this group.

Adverse events of severity CTCAE 3/4 occurred in 51 vs. 38% in the control arm with olaparib [853][854]. Most common adverse events of all severities with greater severity than in the control arm were anemia (46%), nausea (41%), fatigue (41%), loss of appetite (30%), diarrhea (21%), vomiting (18%), constipation (18%), cough (11 %), and dyspnea (10%) [853][854]. Olaparib dosage was reduced in 22% of patients due to adverse events, and therapy was discontinued in 18% of patients [853][854].

7.4.2.4 Therapy sequences after docetaxel

7.46 · Evidence-based Recommendation · modified 2021

Patients with castration-resistant, progressive disease and good general health after chemotherapy with docetaxel shall be offered one of the following treatment options, in combination with symptom-based and supportive therapy if needed:

(in alphabetical order)

·         Abiraterone (in combination with prednisone/prednisolone).

·         Cabazitaxel

·         Enzalutamide

For differential therapy, see Recommendations 7.47 - 7.49.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Strong consensus (97%)
References
[861] [862] [863] [864] [865] [866] [867] [868]
Evidence tables (1)7.4.2_EvT_7.46
7.47 · Evidence-based Recommendation · checked 2021

Patients with castration-resistant, progressive disease and good general condition after chemotherapy with docetaxel can be offered:

(alphabetical order)

•        Abiraterone (in combination with prednisone/prednisolone) or

•        Enzalutamide

A prolongation of survival was shown in the respective pivotal trial.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
1+
Consensus
Strong consensus (100%)
References
[862] [863] [861]
Evidence tables (1)7.4.2_EvT_7.47
7.48 · Evidence-based Recommendation · checked 2021

Cabazitaxel can be offered to patients with castration-resistant, progressive disease and good general condition after chemotherapy with docetaxel. A prolongation of survival was shown in the pivotal trial.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Consensus
Strong consensus (100%)
References
[865]
Evidence tables (1)7.4.3.1 EvT 7.42
7.49 · Evidence-based Recommendation · modified 2021

a.     Radium-223 can be offered to patients who have castration-resistant, progressive prostate cancer with symptomatic osseous metastases (without known visceral metastases) and good general health, and who have received at least two prior systemic therapy options in this indication, or for whom no other available systemic mCRPC therapy is suitable.

(0)


b.      Radium-223 shall not be used in combination with abiraterone and prednisone/prednisolone.

(A)

Level of evidence
1+
Consensus
Strong consensus (a. 100 %, b. 100 %)
References
[851] [869] [870] [871]
Evidence tables (4)7.4.2 EvT 7.37; Parker_Lancet_2018; Vogelzang_ClinGenitourinCancer_2017; Smith_LancetOncol_2019
7.50 · Evidence-based Recommendation · modified 2021

Patients with castration-resistant, progressive disease after chemotherapy with docetaxel and reduced general condition (ECOG ≥ 2, Karnofsky <70) can be offered one of the following treatment options, in addition to a symptom-based therapy:

(in alphabetical order)

·         Abiraterone (in combination with prednisone/prednisolone).

·         Chemotherapy if the reduced general condition is primarily due to metastatic prostate cancer

·         Enzalutamide

·         Steroids (dexamethasone, prednisolone, prednisone).

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
4
Consensus
Strong consensus (97%)
References
[101] [190] [872]
Evidence tables (1)7.4.3.1 EvT 7.44
7.51 · Evidence-based Recommendation · checked 2021

For patients with castration-resistant, progressive disease in good general health, a trial of therapy with lutetium-177-PSMA can be offered based on the recommendation of an interdisciplinary tumor conference after other recommended therapeutic options (see Recommendation 7.46) are exhausted.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
3
Consensus
Consent (93%)
References
[873]
Evidence tables (1)Yadav_AJR_2019

Re: Recommendation 7.46

The recommendation is based on the inclusion criteria of the pivotal trials of abiraterone, cabazitaxel and enzalutamide, and radium-223. The pivotal trials are discussed in Recommendations 7.47 and 7.49. The guideline group defined good general health as ECOG 0-1 or Karnofsky ≥ 70.

For consideration of chemotherapy with cabazitaxel versus abiraterone/enzalutamide, please refer to the data from the CARD trial, see background text for Recommendation 7.48 [852]. Docetaxel retreatment is feasible in patients who have responded well to pretreatment with few side effects. Docetaxel retreatment is performed on an individual basis. There are no data available from randomized trials to establish selection criteria.

 

Re: Recommendation 7.47

Under therapy with abiraterone, after a median follow-up of approximately twelve months, an interim analysis showed that overall survival could be prolonged by a median of 3.9 months compared to placebo [862]. The randomized controlled trial (1,195 patients, 2:1 randomization) included asymptomatic and symptomatic patients with very good general condition (90 % ECOG 0-1) who had received at least one prior chemotherapy. The rates of side effects ocurring were lower compared to chemotherapy. The side effects are mainly based on the mineralocorticoid effect of the drug, notably hypokalemia, hypertension, and fluid retention/edema. In the follow-up evaluation [863], the benefit were even stronger with a prolongation of overall survival of a median of 4.8 months (15.8 months versus 11.2 months; HR: 0.74, 95% CI: 0.64–0.86; p < 0.001). Compared to placebo, abiraterone showed a significant effect on several endpoints (progression-free survival, biochemical and imaging remission, and symptomatology) [864]. In patients with low quality of life scores at baseline, the quality of life of more patients receiving abiraterone compared to placebo (defined as a 10-point improvement in FACT-P) improved. Abiraterone is approved in combination with prednisone/prednisolone for the treatment of metastatic castration-resistant prostate cancer in adult men whose disease has progressed during or after docetaxel-containing chemotherapy [842].

There are currently no data available on retreatment with abiraterone if it was used prior to docetaxel therapy.

The androgen receptor blocker enzalutamide showed a significant benefit in overall survival of a median of 4.8 months (18.4 months versus 13.6 months with placebo; HR: 0.63, 95% CI: 0.53-0.75; P < 0.001) in a randomized controlled trial of 1,199 patients (2:1 randomization) in good general health (EGOG 0-2) with progressive disease after therapy with docetaxel [874]. Among other factors, patients at increased risk for seizures were excluded from the study. Compared to placebo, enzalutamide prolonged progression-free survival (8.3 months vs. 2.9 months; HR: 0.40, 95 % CI: 0.35-0.47, p < 0.001) and time to PSA progression (8.3 months vs. 3.0 months; HR: 0.25, 95 % CI: 0.20-0.30, p < 0.001) and improved quality of life (defined as a 10-point improvement in FACT-P). Very common side effects in the phase-III clinical trial were headache and hot flushes. Seizures occurred only in patients in the enzalutamide arm (n = 6, 0.8%). Enzalutamide has been approved since June 2013 for the treatment of adult men with metastatic castration-resistant prostate cancer whose disease progresses during or after chemotherapy with docetaxel [846].

 

Re: Recommendation 7.48

There are data available on the use of cabazitaxel from three randomized trials: In the pivotal trial, cabazitaxel was tested in comparison to abiraterone and enzalutamide (each against placebo) versus mitoxantrone (each in combination with prednisone). Randomization was 1:1, the median age of patients was 67.5 years, and patients with ECOG 0-1 were predominantly (approximately 92%) included. Included patients all had extensive metastatic disease. Compared to mitoxantrone, cabazitaxel resulted in a median prolongation of life by 2.4 months (15.1 months vs. 12.7 months, HR: 0.70; 95 % CI: 0.59-0.83, p < 0.0001) and a prolongation of progression-free survival by 1.4 months (2.8 months vs. 1.4 months, HR: 0.74; 95 % CI: 0.64-0.86, p < 0.0001) [865]. Significant effects on additional endpoints (tumor response and PSA response) were demonstrated, whereas differences in symptomatology were not significant. Data on quality of life are not available. The guideline group wants to specifically emphasize the potential side effects of the drug, especially febrile neutropenia. This includes treatment-related deaths. For treatment practice in Germany, therapy-associated mortality is lower than in the pivotal study [875].

In the PROSELICA study of 1,200 patients, a lower dose of 20mg/m² was randomized to the previously recommended cabazitaxel dose of 25mg/m² . The study met its primary endpoint and demonstrated that the lower dosage was not inferior in terms of overall survival or progression-free survival. At the same time, the reduced dosage of 20mg/m² showed a significantly more favorable side effect profile than the previously used standard dosage. In particular, the rate of febrile neutropenia was significantly reduced (2.1% vs. 9.2%). Similarly, significantly less hematuria, diarrhea, and fatigue were observed [876]. When using cabazitaxel, careful monitoring and experience in the use of chemotherapies is essential. Cabazitaxel should only be administered by experienced physicians, and careful monitoring is essential. Cabazitaxel is approved in combination with prednisone or prednisolone for the treatment of patients with hormone-refractory metastatic prostate cancer who are pre-treated with a docetaxel-based regimen [877].

In the CARD study, a total of 255 patients were randomized to cabazitaxel 25mg/m² every three weeks or abiraterone (+ prednione/prednisolone) or enzalutamide [852]. Inclusion in the study was conditional on prior therapy with docetaxel and the other AR-targeted drug with a duration of response of less than 12 months [852]. Cabazitaxel therapy resulted in a prolongation of median overall survival with 13.6 months versus 11 months on the second AR-targeted drug (HR 0.64; p=0.008) [852]. Median time to radiographic progression (8.8 vs. 4.4 months; HR 0.54; p<0.001) and progression-free survival (4.4 vs. 2.7 months; HR 0.52; p<0.001) were also significantly prolonged by cabazitaxel [852]. An additional posthoc analysis was performed to examine the impact of the particular AR drug used on the study results. However, the benefit of cabazitaxel remained regardless of whether patients received abiraterone (HR 0.44) or enzalutamide (HR 0.57) as a second oral therapeutic agent.

 

Re: Recommendation 7.49

The radioactive nuclide radium-223 is an alpha emitter with a very short range. Radium-223 was approved in the European Union in November 2013 for the treatment of patients with castration-resistant, osseous metastatic prostate cancer without evidence of known visceral metastasis. Due to safety concerns, approval was limited in July 2018 to patients with metastatic prostate cancer (metastases to bone without known visceral metastases) who have received two other prior therapy options and patients who are unable to receive other systemic therapy options. Based on evidence of increased risk of fractures under combination therapy, it was recommended that radium-223 not be used in combination with abiraterone plus prednisone/prednisolone [878][879].

This restriction on approval is understandable because of the safety concerns. However, it now defines indications for use for which there is no explicit pivotal study. The basis of the 2013 approval was ALSYMPCA, an international, multicenter, randomized, placebo-controlled phase-III study of 921 patients [851]. Patients were randomized 2:1 in favor of the verum arm. Prolongation of overall survival was the primary study point of ALSYMPCA. Radium-223 resulted in a significant prolongation of median survival of 14.9 vs. 11.3 months (HR 0.70; p<0.001) [851]. When radium-223 was administered from third-line therapy, median survival was very similar in registry analyses, ranging from 10.9 – 11.1 months [880].

Radium-223 also resulted in prolongation of time to onerous complications in ALSYMPCA. The rate of severe CTCAE grade 3/4 adverse events was 58% under radium-223 vs 65% in the placebo arm. Only thrombocytopenias occurred more frequently under radium-223 than in the control, at 7% [870]. 

One of the reasons for the EMA approval restriction was data from ERA-223, a randomized, double-blind, placebo-controlled trial in 806 chemotherapy-naïve patients with progressive, castration-resistant, asymptomatic or mildly symptomatic prostate cancer. Abiraterone (plus prednisone/prednisolone) + radium-223 versus abiraterone (plus prednisone/prednisolone) was compared. The primary endpoint was "symptomatic skeletal event-free survival", defined as osseous events or death. The study objective was not met. Fractures occurred in 29% of patients in the radium-223 arm and in 11% in the control arm [878][879].

Radium-223 remains a treatment option in patients with symptomatic osseous metastases without evidence of visceral metastases. Explicit reference is made to the other options of supportive therapy for osseous metastases including local measures such as radiotherapy and systemic administration of bisphosphonates or denosumab [866][867][868]. Publication of further data on the combination of radium-223 with new forms of hormonal therapy are pending [881].

 

Re: Recommendation 7.50

There are no data available on the treatment of patients with poor general status (ECOG ≥ 2, Karnofsky < 70). In the pivotal trials (see Recommendation 7.47 - 7.49), patients with ECOG 0-2 were included but the number of included patients with ECOG = 2 lay only at about 10% of participants. Data on patients with ECOG > 2 is not available. 

Patients with castration-resistant prostate cancer are in an advanced stage of their disease. They are often affected by disease symptoms and psychosocial distress that can be positively influenced by low-threshold palliative medical co-care [882] (see also Recommendation 7.71 and https://www.leitlinienprogramm-onkologie.de/leitlinien/palliativmedizin/).  

 

Re: Recommendation 7.51

177Lu-PSMA ligands represent a new approach to radionuclide therapy in clinical trials [883]. A 2019 review summarized results of 671 patients from 16 studies. This showed a decrease in PSA in 75% of patients receiving 177Lu-PSMA [884]. This was also confirmed in the open-label, randomized phase-II TheraP trial comparing 177Lu-PSMA versus cabazitaxel. Only patients with a PSA level > 20ng/ml and PSMA-positive disease on PSMA-PET-CT were eligible for inclusion in the study. However, patients with FDG-positive metastases without PSMA positivity on an additional FDG-PET-CT were not included. In this selected group of patients, the rate of patients with a decrease in PSA by >50% was significantly higher under 177Lu-PSMA with 65 vs. 37 % than under cabazitaxel. Results on patient-relevant endpoints are pending [885].

Side effects of 177Lu-PSMA ligands are hematologic (anemia, thrombocytopenia, leukocytopenia), renal, and involve salivary glands. 177Lu-PSMA has not yet been approved as a drug.

Patients with castration-resistant prostate cancer are at an advanced stage of their disease. In particular, patients with reduced ECOG status due to the underlying disease, but also due to comorbidity, often suffer from symptoms and psychosocial distress that can be positively influenced by low-threshold palliative medical co-care [882] (see also Recommendation 7.71 as well as https://www.leitlinienprogramm-onkologie.de/leitlinien/palliativmedizin/).

7.4.2.5 Therapy sequence after androgen receptor-targeted treatment.

7.52 · Evidence-based Recommendation · checked 2021

Patients with castration-resistant, progressive disease and good general condition after androgen receptor-targeted therapy can be offered sequential therapy using one of the other effective drugs (see Recommendation 7.46).

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
4
Consensus
Strong consensus (100%)

Re: Recommendation 7.52

Sequential therapy after androgen receptor-targeted treatment has become the standard practice in a short time. Meanwhile, first data on the influence of sequential use of abiraterone, enzalutamide, cabazitaxel, and others are available [853][854][852]. At present, it cannot be conclusively assessed whether a second androgen receptor-targeted treatment after progression under first-line treatment with the other agent may be less effective than second-line chemotherapy. It should be noted, however, that in the currently available retrospective studies enzalutamide achieves a lower PSA response after abiraterone than in an earlier line of therapy [886]. The same appears to be true for abiraterone after enzalutamide. Causes for this possible cross-resistance are the subject of current investigations (cf. Recommendation 7.43).

In a crossover study with abiraterone followed by enzalutamide versus vice versa, the sequence abiraterone followed by enzalutamide showed significantly better efficacy in terms of PSA progression (HR 0.66), while enzalutamide followed by abiraterone showed little activity [847]. Also, a combination of enzalutamide plus abiraterone after enzalutamide is not effective [848]. According to the EAU guideline, these data may suggest that a sequence of abiraterone followed by enzalutamide would be preferable if therapy with agents targeting the androgen receptor alone is possible.

Among the various drug options for the androgen-independent or castration-resistant clinical situation, prednisolone has shown not only to address PSA progression and thus possibly clinical progression, but also to positively impact patient-relevant endpoints of pain, loss of appetite, fatigue, and overall quality of life, which are of paramount importance in the context of palliative therapy [887]. Therefore, prednisolone was given a prominent position in symptomatic patients within this guideline. This is consistent with the assessment of the Dutch guideline. For the low-dose administration of dexamethasone (0.5 mg/day) a decrease in PSA was described in approximately 50% (49/102) of the patients studied [888]. Since a comparative study for the efficacy of the different steroids is not available, all three currently used substances are referenced in the recommendation.

7.5 Local treatment for metastatic prostate cancer

7.5.1 Preamble

7.53 · Evidence-based Statement · new 2021

Oligometastatic prostate cancer is defined as a tumor with a maximum of 4 bone metastases detectable on conventional imaging (skeletal scintigraphy and CT or MRI) without extraosseous visceral metastases.

Level of evidence
1-
Consensus
Strong consensus (98%)
References
[889] [890]
Evidence tables (2)Parker_Lancet_2018; Burdett_EurUrol_2019

The definition of oligometastatic disease is not uniform across the literature. Furthermore, conventional imaging has been used in most cases. Classifications that are more recently collected using PSMA-PET hybrid imaging are therefore not directly comparable. The number up to and including 4 as the cutoff for defining oligometastatic prostate cancer in this recommendation was established for pragmatic reasons. The limits in the different studies were up to 2 (LATITUDE study) [891], up to 3 (CHAARTED and STAMPEDE study) [890] or up to 4 bone metastases (HORRAD study) [892], besides other heterogeneous criteria were used for definition [892][891][890]. In the context of this recommendation, extraosseous visceral metastases are understood as hematogenous metastases in liver, lung, brain, and other organs, but not lymphogenous distant metastases. Whether the latter are included in a possible local ablative therapy of oligometastases has to be decided on an individual basis.

7.5.2 Percutaneous radiotherapy and radical prostatectomy

7.54 · Evidence-based Recommendation · new 2021

a.     Patients with newly diagnosed oligometastatic prostate cancer should receive percutaneous radiotherapy of the prostate in addition to systemic therapy.


b.      External beam radiotherapy (EBRT) should be administered in a hypofractionated manner, and the radiation dose should not exceed an equivalent dose of 72 Gy in 2 Gy standard fractionation.

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
1-
Consensus
Strong consensus (100%)
References
[889] [890]
Evidence tables (2)Parker_Lancet_2018; Burdett_EurUrol_2019

Percutaneous radiotherapy of the prostate in addition to androgen deprivation (+/- docetaxel) significantly prolongs the median time to PSA progression in patients with newly diagnosed oligometastatic prostate cancer. Patients with "low-volume" disease according to CHAARTED criteria (or up to 3 bone metastases [890]) may further benefit in terms of overall survival [889][890]. The recommendation is based on a meta-analysis of several randomized trials that showed a survival benefit for this approach [889]. However, only one study reached a level of significance for the oligometastatic subgroup [890]. It remains unclear whether patients benefit equally from radiotherapy of the primary tumor even with a slightly higher number of bone metastases detected by PSMA hybrid imaging. The type and dose of radiotherapy recommended in this recommendation is analogous to the approach used in the STAMPEDE trial [890]. In this study, radiation was delivered in either a weekly regimen of 36 Gy in 6 fractions over 6 weeks or a daily regimen of 55 Gy in 20 fractions over 4 weeks [890]. In addition to the mostly used hypofractionated regimens, there were also patients with normal fractionation in the HORRAD study [892]. In sum, however, the preponderance of evidence to date has been developed using moderate or severe hypofractionated EBRT. However, the weakened level of recommendation ("should") takes into account the fact that normal fractionated radiation regimens are also possible.

7.55 · Evidence-based Recommendation · new 2021

Percutaneous radiotherapy of the prostate can be combined with extended systemic therapy (androgen deprivation (ADT) simultaneous, docetaxel sequential) in oligometastatic prostate cancer.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
1-
Consensus
Strong consensus (95%)
References
[890]
Evidence tables (1)Parker_Lancet_2018

In the STAMPEDE trial, a minority (18% of patients) received early systemic docetaxel treatment in addition to radiotherapy for the primary tumor [890]. If docetaxel treatment is given, sequential therapy is recommended; by contrast, in the STAMPEDE trial androgen deprivation was given "lifelong", i.e., also simultaneously with radiotherapy [890].

In addition to chemohormone therapy, abiraterone (high risk only) and apalutamide, and possibly enzalutamide in the future, are currently approved for hormone-sensitive metastatic prostate cancer. It is therefore possible that in the future the new hormone preparations mentioned above will also play a role in this indication in addition to radiotherapy of the prostate (or radical prostatectomy, see below) or in addition to metastasis-directed therapy (cf. 7.3).

7.56 · Evidence-based Recommendation · new 2021

a.     Radical prostatectomy is not adequately supported by evidence as a treatment option for oligometastatic prostate cancer.

(Statement) Expert consensus


b.      Radical prostatectomy can be offered for oligometastatic prostate cancer after discussion in an interdisciplinary tumor conference as part of multimodality therapy.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
2++
Consensus
Strong consensus (96%)
References
[893]
Evidence tables (1)Slaoui_WorldJUrol_2019

To date, there are no completed randomized studies on the use of radical prostatectomy for oligometastatic prostate carcinoma [894]. Retrospective comparisons are of limited value due to the high risk of bias. Therefore, it cannot be estimated with certainty whether radical prostatectomy is similarly effective as percutaneous radiotherapy in terms of mortality reduction in oligometastatic prostate carcinoma. Nevertheless, an open recommendation for radical prostatectomy was made by the guideline group, but this was subject to consultation in an interdisciplinary tumor board. In addition, the multimodal character (combination with androgen deprivation, radiotherapy, extended system therapy) of this therapeutic strategy is emphasized. Patients with extensive local tumor infiltration in oligometastatic prostate cancer or patients who are not willing to tolerate the possible morbidity associated with surgery (e.g., incontinence in combination with adjuvant radiotherapy) are not suitable for radical prostatectomy. Radical prostatectomy for oligometastatic prostate cancer is considered to prevent local progression-related complications in the lower urinary tract [894][893].

7.57 · Evidence-based Recommendation · new 2021

a.     Metastasis-directed local ablative therapy for oligometastatic prostate cancer is not adequately supported by evidence, particularly with regard to oncological outcomes.

(Statement) Expert Consensus


b.      Metastasis-directed local ablative therapy for oligometastatic prostate cancer can be used to delay androgen deprivation and/or tumor progression.

(0)


c.     External beam radiation therapy (EBRT) as ablative treatment should be hypofractionated, preferably as stereotactic radiation (SBRT).

(B)

Level of evidence
2++
Consensus
Consent (91%)
References
[230] [893] [895]
Evidence tables (2)Slaoui_WorldJUrol_2019; Connor_EurUrolOncol_2020

The oncological efficacy of metastasis-directed treatment in oligometastatic prostate cancer has not been adequately demonstrated to date. This is especially true for survival endpoints. Delay of androgen deprivation or tumor progression is possible with metastasis-directed treatment [893][895]. More studies, particularly randomized trials, are needed to determine whether these effects favorably affect disease progression or prolong survival [895]. In the ORIOLE study, the impact of metastasis-directed ablative radiation in patients with (metachronous) oligometastatic hormone-sensitive PCA was investigated in a randomized prospective phase-II setting. The initial diagnosis did not yet require PSMA imaging, so that, according to protocol, lesions that could only be visualized on optional PSMA PET may not have been treated. Metastasis-directed treatment significantly prolonged PSA progression in the entire intervention group, and higher-grade adverse events(≥ G3) were not observed. In addition, a subgroup analysis of a cohort in which all PSMA positive lesions received ablative radiation also showed longer metastasis-free survival [230].

However, in the case of metastasis-directed treatment for oligometastatic disease, it seems reasonable to additionally educate patients about the possibility of intensified systemic therapy. Until proven otherwise, it can be assumed that metastasis-directed treatment in oligometastatic prostate cancer cannot permanently replace intensified systemic therapy (i.e., with abiraterone, apalutamide, docetaxel, or enzalutamide).

7.6 Therapy of bone metastases

7.58 · Evidence-based Recommendation · modified 2018

The treatment of symptomatic osseous metastases is part of the overall oncological concept (see Recommendations 7.37, 7.39, 7.40, 7.43, 7.44). Patients with osseous metastases shall additionally be offered one or more of the following therapeutic options:

  • medicinal pain therapy
  • local radiotherapy, see Recommendation 7.48
  • surgical intervention (usually in combination with radiotherapy)
Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Consensus
Majority approval (86%)
References
[851] [896] [897]
Evidence tables (1)7.5 EvT 7.47
7.59 · Evidence-based Recommendation · checked 2018

Local percutaneous radiotherapy shall be used for bone metastases in the following situations:

  • persistent localized bone pain
  • impending spinal compression (possible after surgical intervention)
  • after surgical stabilization
  • increased risk of fracture
Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1++
Consensus
Strong consensus (97%)
References
[896]
Evidence tables (1)7.5 EvT 7.48
7.60 · Evidence-based Recommendation · modified 2021

Radionuclides can be used for pain treatment in multiple bone metastases in the castration-resistant stage. Please refer to Recommendation 7.49 for the indication of radium-223.

Recommendation grade
0: offene Empfehlung (Option) bzgl. einer Maßnahme - verwendete Ausdrucksweise = kann
Level of evidence
1+
Consensus
Strong consensus (100%)
References
[897]
Evidence tables (1)Bauman_RadiotherOncol_2005
7.61 · Evidence-based Recommendation · new 2018

Recommendation:

Bisphosphonates should not be used to prevent complications of bone metastases in the hormone-naïve stage.

Statement:

Currently the effect of denosumab at this stage cannot be assessed.

Recommendation grade
B: Empfehlung für oder gegen eine Maßnahme - verwendete Ausdrucksweise = sollte/sollte nicht
Level of evidence
1+

Zoledronic Acid


4

other bisphosphonates, denosumab

Consensus
Strong consensus (100%)
References
[794] [866] [898] [868] [899] [900] [901]
Evidence tables (1)7.5 EvT 7.50
7.62 · Evidence-based Recommendation · checked 2018

To prevent complications from bone metastases in a castration-resistant stage, the monoclonal antibody denosumab or bisphosphonate zoledronic acid shall be offered along with information on potential benefits and harms.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
1+
Consensus
Strong consensus (100%)
References
[866] [868]
Evidence tables (1)7.5 EvT 7.51
7.63 · Evidence-based Recommendation · checked 2018

The following steps shall be taken to prevent osteonecrosis of the jaw, prior to the administration of bisphosphonates or denosumab:

  • a dental examination and, if necessary, dental rehabilitation, and
  • instruction and motivation of the patient to maintain above-average oral hygiene
Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
3

+

Consensus
Strong consensus (98%)
References
[866] [902]
Evidence tables (1)7.5 EvT 7.52

Re: Recommendation 7.58

Bone metastases are present in over 80% of patients with metastatic prostate cancer [903], [904]. Typical predilection sites are the spine, pelvis, and chest [905]. For prostate cancer, osteoblastic metastases are predominant. The median survival time for known bone metastases is approximately three years [906]. Symptomatic complications predominantly include pain, hypercalcemia, pathologic fractures, and spinal cord compression in vertebral fractures.

Randomized controlled trials for the prevention of skeletal-related events in bone metastases are available for the mentioned treatment modalities (percutaneous radiotherapy, systemic application of radionuclides) [868], [896], [897]. The indications are explained further in the following recommendations. Side effects and contraindications need to be considered. Based on the available data, it must currently be concluded that a prolongation of life is generally not achieved with a specific treatment of bone metastases, even with treatment of singular metastases. Individual studies on the benefit of percutaneous radiotherapy or the administration of radionuclides, in which positive survival time effects were achieved are contrasted with other studies in which such effects were not demonstrable [897].

The standard of medicinal pain treatment for tumor patients is the WHO staging scheme. In summary, this regimen provides for a non-opioid analgesic and/or adjuvants in stage I, a weak-acting opioid (not under controlled substances law) combined with a non-opioid analgesic, coanalgesics and/or adjuvants in stage II, and a strong-acting opioid (controlled substance) combined with a non-opioid analgesic, coanalgesics and/or adjuvants in stage III [907].

 

Re: Recommendation 7.59

Local percutaneous radiotherapy of painful singular bone metastases is a palliative measure. The available randomized controlled trials show partial or complete pain relief after approximately three to eight days in more than 80% of patients. In at least 50% of patients, this pain relief lasts six months or longer. Assuming a single application of at least 6 Gy (usually 8 Gy), the degree and duration of pain relief in the studies do not correlate significantly with the radiation regimen used (single dose versus fractionated administration) [896], [908], [909]. However, the data show a significantly increased rate of subsequent retreatment after single-dose radiation compared with a fractionated regimen [896], [910].

Acute adverse effects of radiation therapy include nausea and vomiting in approximately one quarter of cases [897]. The incidence of spinal cord compression or pathologic fractures around the target volume are low later in the course. The incidence is 1-2% for spinal stenosis, and 1-4% for pathologic fractures in randomized controlled trials [896]. A twofold to threefold increased incidence of pathologic fractures compared with fractionated radiotherapy was reported for single-dose radiation when studies up until 2001 were analyzed [910]. A more recent meta-analysis with seven additional studies through July 2006 found no significant differences with respect to pathologic fractures [911]. Remineralization after six months is significantly better in patients receiving fractional radiotherapy compared to patients receiving single-dose radiotherapy (data from one study, [912]).

Data from one retrospective and four prospective case series (with a total of 545 patients) are available on the effect of radiotherapy in tumor-related spinal stenosis or spinal cord compression [896]. Signposts for a possible success of the therapy are the early diagnosis and the radiosensitivity of the tumor. The studies show an improvement of the presenting neurological symptoms in about 25-64% of patients ([896], but the definitions for this are heterogeneous.

For comparison of primary combined therapy with surgical decompression and percutaneous radiotherapy (10 x 3 Gy) vs. radiotherapy alone, a 2005 RCT is available [913]. A Cochrane review on the topic did not identify any other studies on surgical decompression combined with radiotherapy vs. radiotherapy alone until 2008 [914]. Searches through 3/2011 also yielded no new RCT on the topic. The RCT showed significantly better outcomes in patients (n = 103) with proven spinal canal compression and neurological symptoms < 48 hours in terms of maintaining or regaining walking ability with combined therapy (84% vs. 57% and 62% vs. 19%, respectively). The interventions were each performed within 24 hours. Patients whose symptomatology was due to unilocular compressive metastasis of a primary nonhigh-radiation tumor were included. Surgical intervention after unsuccessful radiotherapy showed far worse results than primary combined treatment in this RCT. Because of the significant result in favor of combination therapy, the RCT was discontinued after interim analysis. Among the patients included were 19 patients with prostate cancer. No differentiation is made whether this was hormone-naïve or castration-resistant.

In hormone-naïve patients with symptoms of spinal compression without pathologic fracture, there is also the possibility of (emergency) hormone-deprivation therapy.

The decision for surgical intervention should be made on an individual basis. Surgical stabilization should usually be considered in cases of impending fracture risk or after pathologic fracture, depending on the location. The indication should be determined on an interdisciplinary basis with the involvement of a specialist in radiological diagnostics, a radiation therapist, and a representative of a surgical specialty (neurosurgeon, orthopedist, or trauma surgeon). This is often an emergency situation, most of which requires immediate steroid medication.

Postoperative local radiotherapy (10 x 3 Gy) significantly reduces the rate of reoperation due to prosthesis loosening [897].

 

Re: Recommendation 7.60

In randomized controlled trials radionuclides have been shown to be effective in reducing pain in multiple diffuse bone metastases in the hormone refractory stage.

The radionuclides approved for the indication are strontium-89 (St-89), samarium-153 (Sm-153), and rhenium-186 (Re-186). The radionuclides Sr-89 at a dosage of 150-200 MBq and Sm-153 at a dosage of 1.0 mCi/kg have been best studied for intervention (several phase III RCTs available in each case). For Sr-89 application, pain relief sets in slightly later, at two to three weeks after initiation of treatment, than for Sm-153 [896] application, at one to two weeks. The occurrence of fewer new pain foci has been described for Sr-89 compared to Sm-153. A phase-III RCT is available for the efficacy of Re-186.

For the radionuclides Re-188 and Sn-117, only phase-II studies are available so far, but no phase-III studies.

The pain relief achieved in 60-80% lasts for a median of two to four months in the majority of patients. In addition, fewer new painful foci are reported compared to placebo [896], [897]. When determining the indication, the expected side effects should be considered. Nausea and/or vomiting occurs only in a minority of patients (up to 10%) [897]. However, treatment regularly results in mild to moderate myleosuppression ([896], [897]. Recovery of leukocyte counts to baseline values is observed with Sm-153 after approximately eight weeks [915]. This time period can also be expected with application of Re-186, whereas leukocytes do not return to baseline values after application of Sr-89 until after approximately twelve weeks. Patients should also be informed about a possible temporary increase in symptoms (flare phenomenon) before definitive pain relief (GCP).

Data on radium-223 is discussed in Recommendation 7.49 [851].

 

Re: Recommendation 7.61, Recommendation 7.62, and Recommendation 7.63

With regard to the treatment of bone metastases to prevent or delay symptomatic skeletal-related events, the guideline group decided to recommend both the RANKL antibody denosumab and the bisphosphonate zoledronic acid. This recommendation applies only to castration-resistant patients. By contrast, the administration of osteoprotective agents is not recommended in hormone-naïve patients with bone metastases: The majority of the identified studies show no advantage of using zoledronic acid in hormone-naïve patients with regard to time to skeletal-related events (SRE) and survival; no studies on other bisphosphonates and denosumab could be identified for this patient group. In addition to uncertain or unproven benefits, the agents are associated with potential risks: renal dysfunction (zoledronic acid [916]) or osteonecrosis of the jaw and hypocalcemia (denosumab [917], [918] (see also Recommendation 7.52), On this basis, the guideline authors advise against the use of osteoprotective agents in hormone-naïve patients with bone metastases. If treatment is indicated due to osteoporosis, this remains excluded from the negative recommendation. As generally recommended in the S3 guideline on supportive therapy (http://leitlinienprogramm-onkologie.de/Supportive-Therapie.95.0.html) [919], at least 500 mg calcium and 400 I.U. vitamin D should be given orally daily with both agents. The evidence for the bisphophonates or for the RANKL antibody in bone metastasis complications is presented below. No data are available on a "switch" of ongoing therapy with zoledronic acid to therapy with denosumab.

 

Evidence on bisphosphonates

In the combined analysis, the available studies show a borderline-significant effect with the use of different bisphosphonates with regard to the occurrence of skeletal-related events in bone metastases of castration-refractory prostate carcinomas (odds ratio: 0.79; 95% CI: 0.62-1.0; p = 0.05). Existing studies evaluating for this question used clodronate, pamidronate, and zoledronate [868]. Most bisphosphonates are effective only in bone metastases with osteolytic components; this explains the partial lack of efficacy in osteoblastic metastatic prostate carcinoma. For more information on the associated risks, see Recommendation 7.52 on osteonecrosis of the jaw.

In the single analysis, zoledronic acid in combination with vitamin D and calcium was the only substance studied to show a significant reduction for the occurrence of skeletal-related events compared to placebo administration (+ vitamin D and calcium), especially for pathologic fractures [867], [906], [920]. Therefore, the recommendation on zoledronic acid as a bisphosphonate was changed to a strong recommendation in the second guideline update in 2014. It should be noted that the evidence of the effect of zoledronic acid is based on a single study (n = 634). The absolute difference compared with placebo for the occurrence of skeletal-related events was 11% (44.2% in the placebo group, 33.2% in the zoledronic acid group). The time to occurrence of skeletal-related events was several months longer, this was statistically significant (placebo median 321 days, zoledronic acid median not yet reached after 15 months; p = 0.01). An update search on the third update identified four additional RCTs (three open label, one blinded) [794], [899], [900], [901]. Three studies find no significant effects compared to placebo in terms of time interval to skeletal-related events (SRE). The smallest study (n=188) [899] reports a significant prolongation of the SRE-free interval by 2.4 months (from 11.2 to 13.6 months, but the included patients in this study were already castration-resistant. Other endpoints including progression-free and overall survival again did not differ between zoledronic acid and placebo groups. Bisphosphonates including zoledronic acid are not approved for the indication of pain management, and their use constitutes an off-label use.

 

Evidence on denosumab

The human monoclonal antibody denosumab targets RANKL, thereby inhibiting osteoclast-mediated bone destruction.

Fizazi et al. published the results of a multicenter non-inferiority RCT (n = 1,904) in spring 2011 [866]. The primary endpoint of this study was to compare denosumab with zoledronic acid in terms of time to first skeletal-related event (defined as pathologic fracture, percutaneous osseous radiotherapy, surgical osseous intervention, or spinal cord compression) in patients with castration-resistant prostate cancer. Patients with creatinine clearance of < 0.5mL/s were not included because zoledronic acid cannot then be applied. In contrast to zoledronic acid (i.v.), denosumab is administered subcutaneously and can also be used in patients with impaired renal function. With denosumab, the first skeletal-related event occurred a median of 3.6 months later than with zoledronic acid (20.7 months versus 17.1 months), with an overall median survival of 22 months for both groups. The analysis was based on intention to treat. The difference was statistically significant in favor of denosumab in terms of hazard ratio – in terms of noninferiority, but also when tested for superiority. However, the confidence intervals of the absolute results overlapped in months. For denosumab (median 17.1 months), the lower limit of the 95% confidence interval is 18.8 months (upper limit 24.9 months); for zoledronic acid, the upper limit of the 95% confidence interval is 19.4 months (lower limit 15 months). This means that based on the lower and upper limits given in the confidence intervals for the "true value" there is uncertainty regarding superiority of denosumab. For this reason, the result is estimated that the equivalence of denosumab with zoledronic acid in terms of time to skeletal-related events can be confidently inferred from the study results, but not superiority.

The difference in absolute events over the entire observation period is 5% (36% denosumab, 41% zoledronic acid). The other study identified with the update search, [898], with the same study design, also reports an absolute risk reduction for skeletal-related events of 4.7% (35.9% denosumab, 40.6% zoledronic acid). It should be noted in this study that it has significant methodological flaws (patient characteristics not reported, statistics not transparent) and was supported by the manufacturer of the monoclonal antibody during manuscript preparation.

With regard to individual adverse effects, there were statistically significantly more hypocalcemias under denosumab (13% denosumab, 6% zoledronic acid). Significantly higher were grade 3+4 adverse events overall (72% vs. 66%) using the CTCAE (Common Terminology Criteria for Adverse Events version 3.0) classification. A Red Hand Letter from the manufacturer [907] reported severe symptomatic hypocalcemia, including cases with fatal outcomes, in patients treated with denosumab. To minimize the risk, monitoring of calcium levels was recommended by the manufacturing company in patients with severe renal impairment (creatinine clearance < 30ml/min) or in patients requiring dialysis. The S3 guideline on supportive therapy (http://leitlinienprogramm-onkologie.de/Supportive-Therapie.95.0.html) [919] recommends the simultaneous administration of at least 500 mg of oral calcium and 400 I.U. vitamin D daily as a preventive measure.

Under denosumab 2% and under zoledronic acid 1% of osteonecrosis of the jaw occurred, and secondary malignancies occurred just as frequently in each case. The differences are not statistically significant. The relatively short follow-up period should be considered. The risk of osteonecrosis of the jaw increases with longer treatment or follow-up duration (see Recommendation 7.51). For prevention of osteonecrosis of the jaw, please refer to the corresponding S3 guideline (http://www.awmf.org/leitlinien/detail/ll/007-091.html).

7.7 Supportive and palliative treatment

Palliative care is defined as an approach to improving the quality of life of patients and their families facing problems associated with a life-threatening illness. It does this by preventing and relieving suffering through early recognition, careful assessment, and treatment of pain and other problems of a physical, psychosocial, and spiritual nature. Palliative care is applied early in the course of disease, including in conjunction with other therapies aimed at prolonging life, such as chemotherapy or radiation, and includes investigations necessary to better understand and treat distressing complications [921]. Regarding palliative care aspects, regardless of the underlying diagnosis, please refer to the S3 Guideline Palliative Care of the Oncology Guideline Program https://www.leitlinienprogramm-onkologie.de/leitlinien/palliativmedizin/.

7.7.1 Prophylactic/supportive treatment of common adverse therapy-related effects

7.7.1.1 Operation

7.64 · Evidence-based Recommendation · checked 2018

The most common adverse outcomes after radical prostatectomy shall be treated as follows:

  • erectile dysfunction: see recommendations in the chapter on Rehabilitation;
  • incontinence: see recommendations in the chapter on Rehabilitation;
  • urethral structure: conservative or surgical treatment are possible
Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus
7.65 · Evidence-based Recommendation · checked 2018

a. After lymphadenectomy, an ultrasound examination - plus double-flow measurement if needed - shall be performed to diagnose lymphoceles.

b. Lymphoceles following lymphadenectomy shall be treated if they cause symptoms or vascular compression.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (98%)

Background information

The recommendations on supportive care were developed based in a consensus-based way. In addition to the source guidelines, other current guidelines on specific aspects were consulted and primary literature was contributed by the authors.

 

Re: Recommendation 7.64

The most common undesirable outcomes after radical prostatectomy are erectile dysfunction, incontinence, and urethral strictures. Treating erectile dysfunction and incontinence is discussed in detail in the Rehabilitation section of this guideline. The incidence of urethral strictures is reported in the literature to be between 0.5% and 9% [190], and in Germany the incidence is currently reported to be approximately 3%. There is currently no standard therapy for the treatment of urethral strictures. Treatment options are conservative via bougienage of the urethra or a surgical approach, for which different techniques are available. Other undesirable side effects such as increased stool frequency or passive fecal incontinence should be treated symptomatically.

In order to draw conclusions about the frequency of undesirable side effects after radical prostatectomy, it is useful to document occurring side effects in a uniform manner. The Clavien classification [922] is an available form of documenting side effects.

 

Re: Recommendation 7.65

Lymphoceles following lymphadenectomy are reported in the recent literature at a rate of approximately 10 to 18%, with a highly significant positive correlation between the occurrence and the performance of extended lymphadenectomy (ePLND) and the number of lymph nodes removed [923], [924]. In addition, a positive correlation of the occurrence of lymphoceles and the respective surgeon was found in one study [923]. Because lymphoceles can be hemodynamically relevant, they represent a proven risk factor for thromboembolic events (OR 2.8; [925]). Therefore, they should be diagnosed postoperatively and, if necessary, investigated for their hemodynamic relevance by Doppler flow measurement. In case of hemodynamic relevance or occurrence of other symptoms, the lymphocele should be relieved, for which sonographically assisted drainage is available as a first procedure [925]. Infected lymphocele should be treated locally with antibiotics.

7.7.1.2 Radiotherapy

7.66 · Evidence-based Statement · checked 2018

A drug-based prevention of radiotherapeutic side effects is currently not possible.

Level of evidence
4
Consensus
Strong consensus (100%)
References
[101]
Evidence tables (1)7.6.1.2 EvT 7.55
7.67 · Evidence-based Recommendation · checked 2018

a. Acute proctitis can be treated topically with sucralfate, butyrate, or hydrocortisone.

b. Treatment of diarrhea shall be symptomatic.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4

b


1+

a

Consensus
Strong consensus (97%)
References
[926] [927] [101]
Evidence tables (1)7.6.1.2 EvT 7.56

Re: Statement 7.66

The authors of the NICE guideline [101] carried out a systematic search for randomized controlled trials on the prevention of radiotherapy-related adverse effects using radioprotective agents. In the 15 studies identified, no significant effects of individual radioprotective substances could be demonstrated [101]. Radioprotective substances can therefore not be recommended at present.

In order to be able to draw conclusions on the frequency of undesirable side effects after radiotherapy, it is useful to document occurring side effects in a uniform manner. The CTCAE classification [928] is available for the documentation of side effects.

 

Re: Recommendation 7.67

Radiation-induced proctitis can be treated topically with the above-mentioned substances; two randomized studies with, however, small patient numbers of 20 [926] and 134 patients [927] are available. The risk of radiation-induced diarrhea depends on the size of the target volume. Clinically, the treatment of the sequelae of radiation-induced enteropathy with balancing of electrolytes and fluids is the main focus. For further treatment of acute radiation-induced diarrhea, RCTs are available showing clinical efficacy of several agents [101]. Accordingly, acute radiation-induced diarrhea can be treated with both loperamide [929] and octreotide [930].

For the treatment of forms of late proctopathy after radiotherapy, no standard therapy can be derived from the available studies. A systematic review [931] yielded six RCTs on nonoperative interventions for late radiation-related proctopathy. Each of these RCTs examined a different treatment. Although individual studies showed positive effects, they were not sufficient to derive recommendations for a standard approach to the treatment of radiation-induced proctopathy.

From another systematic review [932], there is little evidence to recommend specific dietary approaches for chronic gastrointestinal symptoms. This is also true for acute radiation-related toxicity. Based on this, the treatment of late radiation-induced proctopathy should be symptom-oriented and on an indivudal basis.

7.7.1.3 Androgen ablation therapy

7.68 · Consensus-based Recommendation · checked 2018

Patients shall be educated about treatment options for typical and common side effects of hormone ablation therapy.

Consensus
Strong consensus (98%)

Re: Recommendation 7.68

Hormonal therapy is mainly performed by castration (surgically or medicinally) or by administration of antiandrogens. If necessary, the combination of both concepts in terms of a complete or maximal androgen blockade is also performed (see 6.6 and 7.3). The adverse effects and their treatment options are summarized in Table 19 and are explained below.

  • Hot flushes
    Hot flushes are the most frequent side effect of castration and can be extremely stressful for the patient, and accordingly reduce the quality of life. There are several options for the treatment of hot flushes. Cyproterone acetate should be administered at a dosage of 50 mg 2 x 1 tbl. or 300 mg i.m. every two weeks. Other hormonal therapy options include administration of estrogens and progesterone. In addition, clonidine or antidepressants can be used. When determining the indication, the side effects or contraindications stated by the manufacturer must be observed in each case [933], [934].
  • Osteoporosis
    The diagnosis, prophylaxis, and therapy of osteoporosis are described in detail in the S3 guideline on supportive therapy (https://www.leitlinienprogramm-onkologie.de/leitlinien/supportive-therapie/).
    Bone density may be already decreased in "hormone-naïve" prostate cancer patients,  [935]. In particular, long-term use of LH-RH analogues carries a high risk of developing osteoporosis. The risk of fracture (fatigue fracture, low-trauma fracture) increases significantly after more than one year of treatment [935], [936], [937]. Risks and potential benefits to the patient should be weighed up when discussing drug treatment. Medication selection should take into account the approval for treatment [938], [939].
  • Weight gain, metabolic changes, cardiovascular disease
    Via castration-induced hypogonadism, there are changes in metabolism, a reduction in muscle mass, weight gain, lipid metabolic disturbances, insulin resistance, hyperglycemia and, on this basis, possibly an increased incidence of cardiovascular disease [940], [941] (papers arguing against this thesis: [942], [943]). The impact of these changes on the quality of life or life expectancy has not been adequately studied. However, as a consequence of these changes, patients undergoing androgen deprivation should be treated as a risk group for cardiovascular disease and diabetes.
  • Anemia
    Anemia leads to loss of performance and weakness (fatigue syndrome). It thus places a considerable burden on the patient's quality of life. Anemia as a side effect of androgen ablation treatment correlates with the duration of treatment as well as with the life expectancy of patients [944]. A treatment consists of transfusion and administration of erythropoietins depending on the symptoms. Erythropoietins have had an effect on anemia in prospective randomized trials [945], [946]. More recently, studies have shown evidence of tumor growth stimulation by erythropoietins. Although no evidence has been found for the association between erythropoietin administration and tumor progression in prostate carcinoma [947], the indication for the administration of erythropoietins should be made with caution. Further, diagnosis and therapy of tumor therapy-induced anemia are described in the S3 guideline on supportive therapy (https://www.leitlinienprogramm-onkologie.de/leitlinien/supportive-therapie/).
  • Gynecomastia/breast pain
    Breast pain and/or gynecomastia occur without prophylaxis in approximately one in two patients receiving antiandrogen monotherapy (bicalutamide 150 mg) [948]. They lead to discontinuation of therapy in a proportion of patients. Accordingly, supportive measures should be taken. Prophylactic radiation treatment prior to initiation of antiandrogenic therapy is considered an effective therapy [949], [950].
    In a prospective randomized comparison, treatment with tamoxifen was superior to radiotherapy [951]. However, tamoxifen is not approved for this indication.
    Hot flashes, libido decrease, and erectile dysfunction occur occasionally (< 10% compared with placebo) with androgen administration [948].

Table 20: Typical and common side effects of hormone ablative therapy and possibilities of prophylaxis and treatment to be communicated to the patient (based on Heidenreich 2007)

Adverse reactions (side effects)  Prophylaxis/Treatment
Castration
Loss of libido none
Erectile dysfunction Phosphodiesterase inhibitors (PDE5 inhibitors), intracavernosal injections (ICI, SKAT), MUSE vacuum pumps, penile implants
Hot flushes (55-80% of all patients during hormone ablative therapy) Cyproterone acetate (CPA), antidepressants (venlafaxine), clonidine
Gynecomastia and chest pain (50% during complete androgen blockade (CAB), 10-20% after castration) Prophylactic radiation, masectomy, tamoxifen (prescribing tamoxifen is equivalent to off-label use)
Increase in body fat Physical exercise/sports, nutrition counseling
Muscle loss Physical exercise/sport
Anemia (severe form in 13% of CAB patients) Transfusion in symptomatic anemia, erythropoiesis-stimulating substances (erythropoietin, etc.) with careful consideration of risks
Reduction of bone density Physical exercise/sports, calcium + vitamin D, bisphosphonates if osteoporosis is detected (see [952])
Decrease of cognitive abilities Brain training
Estrogens
Increased risk of cardiovascular events (acute myocardial infarction, chronic heart failure, cerebrovascular events, deep vein thrombosis, pulmonary embolism) Parenteral administration, anticoagulation if necessary
Steroidal antiandrogens
Main side effects of the drug group: loss of libido, erectile dysfunction, rarely gynecomastia Erectile dysfunction
Non-steroidal antiandrogens
Main side effects of the drug group: gynecomastia (49-66%), breast pain (40-72%), hot flashes (9-13%) Prophylactic radiation, mammectomy, tamoxifen or aromatase inhibitors (prescribing tamoxifen is equivalent to off-label-use).

7.7.1.4 Chemotherapy

7.69 · Consensus-based Recommendation · checked 2018

Supportive treatment of the adverse effects of chemotherapy shall be performed on the basis of the scientific evidence that is currently available.

Consensus
Strong consensus (98%)

Re: Recommendation 7.69

The most common adverse effects of chemotherapy are summarized in Table 20. For supportive management of adverse effects of chemotherapy, please refer to the respective S3 guideline [919].

Table 21: Prophylaxis and treatment of common side effects of chemotherapy in prostate cancer

Main side effects  Prophylaxis and/or treatment
Nausea/vomiting prophylactic administration of dexamethasone,
alternatively or in case of contraindications administration of metoclopramide,
in case of treatment failure of these drugs: prophylactic administration of 5HT3 receptor antagonists, if necessary also NK1 receptor antagonists
Allergy prophylactic administration of dexamethasone
Neutropenia/infections prophylactic and interventional administration of antibiotics
Thrombocytopenia in severe symptomatic thrombocytopenia: platelet concentrates
Anemia see Table 19 for treatment of AD side effects
Diarrhea symptomatic therapy, administration of loperamide, compensation of electrolyte and fluid losses
Mucositis oral hygiene, analgesics
Edema prophylactic administration of dexamethasone
Neuropathy symptom-oriented drug and physical therapy in case of sensory or motor polyneuropathy occurring
Extravasation stop infusion immediately, local therapy
Onychodystrophy and onycholysis prophylactic treatment for docetaxel possible (local cold application)
Alopecia No prophylactic treatment possible

Nausea and vomiting

Docetaxel and cabazitaxel belong to the group of chemotherapeutic agents with low emetogenic potential (occurrence of nausea and/or vomiting without prophylaxis in 10 to 30%). International guidelines [953], [954], [955] are consistent in recommending prophylactic administration of dexamethasone (8 mg/24 h) for these chemotherapeutic agents to prevent immediate onset nausea and vomiting. Alternatively, or in case of contraindications to dexamethasone, the administration of metoclopramide is indicated [953]. In case of treatment failure of dexamethasone, prophylactic administration of 5HT3 receptor antagonists is indicated in the next chemotherapy, and in case of further treatment failure, combination with NK1 receptor antagonists. Prophylaxis of delayed nausea or vomiting (occurrence >24 h after application of chemotherapy) is unanimously considered unnecessary in the three guidelines [956].

Allergy

Severe hypersensitivity reactions can occur with docetaxel. The risk can be greatly reduced by prophylactic administration of dexamethasone.

Neutropenia/infections/thrombocytopenia.

Docetaxel, mitoxantrone, and estramustine can cause reversible neutropenia with the risk of infectious complications up to sepsis. The risk is significantly increased with cabazitaxel. With it, grade 3/4 neutropenia after CTCAE occurred in 82% of patients in the pivotal study [865]. Regular blood count checks are required after therapy. If signs of an infection are present, diagnostics and antibiotic therapy must be initiated immediately. Prophylactic administration of antibiotics or hematopoietic growth factors is not required in the first round of chemotherapy [957], [958]. If severe symptomatic neutropenia occurs, dosage reduction or the use of prophylactic measures is indicated in the subsequent therapeutic cycle. Chemotherapy for prostate cancer can lead to reversible thrombocytopenia. Administration of platelet concentrates is usually not required [959].

Anemia

In patients with symptomatic anemia, the administration of red cell concentrates is indicated. In a recent meta-analysis, it was shown that the use of erythropoiesis-stimulating agents (epoetin alpha, epoetin beta, darbepoetin alpha) in cancer patients leads to increased mortality during treatment and shortened survival [960]. The use of erythroposis-stimulating agents is not indicated in patients with a hemoglobin level >11g/dl. The benefits of treatment must be carefully weighed up against the potential risks [958], [961].

Diarrhea

Chemotherapy-induced diarrhea can be treated according to the same principles as radiotherapy-induced diarrhea (see Recommendation 7.67).

Mucositis

The general recommendations on chemotherapy-induced mucositis emphasize the prophylactic value of careful oral hygiene [962]. When severe mucositis occurs, administration of analgesics including morphines is indicated. There is no specific evidence-based prophylaxis for docetaxel-induced mucositis. Analogous to data of 5FU-induced mucositis, ice cube sucking may be recommended.

Neuropathy

Polyneuropathy is one of the side effects of docetaxel. The toxicity is cumulative. There is no specific prophylaxis for docetaxel-induced polyneuropathy. Therapy is symptomatic.

Onychodystrophy/Onycholysis

Onychodystrophy and onycholysis is one of the side effects of doxetaxel. This side effect is more common with the weekly than with the tri-weekly administration. Two controlled studies – albeit small in number of cases – have shown for docetaxel that local cold application (hands and feet) can both decrease the number of events associated with nail changes and decrease the number of patients with onycholysis [963], [964], [965].

7.7.2 Palliative treatment

7.70 · Evidence-based Statement · checked 2018

The goal of palliative treatment is to improve quality of life through effective treatment of distressing symptoms, psychosocial support, and assistance with spiritual issues. Common symptoms in patients with advanced prostate cancer include pain, fatigue, weight loss, anxiety, depression, and organ-related symptoms (e.g., paralysis and urinary retention).

Level of evidence
4
Consensus
Strong consensus (97%)
References
[746] [907]
7.71 · Evidence-based Recommendation · checked 2018

The options for palliative therapy shall be comprehensively discussed with the patient and their relatives at an early stage. They include:

a. Information about all available care options (self-help groups, outpatient care, inpatient care, outpatient hospice services, inpatient hospices, specialized outpatient palliative care (SOPC), hospital palliative services, palliative care units).

b. Information about all treatment methods.

c. Preparation of a comprehensive treatment plan taking into account the patient's personal preferences.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (100%)
References
[938] [966] [967]
Evidence tables (1)7.6.2 EvT 7.60
7.72 · Evidence-based Recommendation · checked 2018

a)

Recommendation grade A

The medical treatment strategy shall be interdisciplinarily and multiprofessionally determined.

b)

Grade of recommendation: B

An interdisciplinary treatment team should be available to the patient (including professionals trained in psychosocial or psycho-oncological areas, as well as palliative care).

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (97%)
References
[101] [938]
Evidence tables (1)7.6.2 EvT 7.61
7.73 · Evidence-based Recommendation 2018

a. Drug treatment of tumor pain shall be based on the WHO staging scheme.

b. In the context of palliative pain therapy, non-drug physical (including positioning, lymphatic drainage, activating care) and psychosocial (psychological, if necessary spiritual support) measures shall also be considered.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (100%)
References
[907] [966]
Evidence tables (1)7.6.2 EvT 7.62
7.74 · Evidence-based Recommendation · checked 2018

Physical and psychological complaints such as anxiety, agitation, depression, dyspnea, weakness, and fatigue shall be regularly assessed and appropriate care or treatment shall be provided.

Recommendation grade
A: Starke Empfehlung für oder gegen ein Maßnahme - verwendete Ausdrucksweise = soll/soll nicht
Level of evidence
4
Consensus
Strong consensus (100%)
References
[938] [966]
Evidence tables (1)7.6.2 EvT 7.63

Background information

Re: Statement 7.70

In this guideline, palliative treatment in the narrower sense is understood as the care and treatment of the patient with symptomatic metastatic disease.

The primary therapy goal in palliative treatment is not to prolong survival, but to improve quality of life; this is also emphasized in the European guideline [746]. The individual needs of the patient are paramount. Priority is given to effective symptom-oriented treatment, particularly efficient pain reduction. The core documentation for palliative care units in 2004 shows that over 70% of patients with metastatic prostate cancer suffered from moderate or severe pain on admission.

Explanations for the treatment of tumor-related urinary retention

Acute symptomatic urinary retention can lead to fever, sepsis, renal insufficiency, and even renal failure. According to Oefelein et al. [968], the prognosis of such patients is worse than that of patients without urinary retention (median survival 42 vs. 59 months). Urinary diversion has not been comparatively tested with a Watch and Wait strategy for ethical reasons. Instrumental urinary diversion aims to remove obstruction to prolong life with good quality of life. The type of urinary diversion is subject to controversy across the literature. A comparison between internal and external urinary diversion showed no statistically significant differences with respect to inflammatory complications [969]. Specific criteria exist for the indication of percutaneous nephrostomy or retrograde and antegrade stent placement [970]. Causes of poor stent function include reduced urinary flow, ureteral peristalsis, and incrustations of the lateral holes [971]. Signs of stent malfunction include urinary retention, flank pain, pyelonephritis, and creatinine elevation [972]. Percutaneous nephrostomy is cited as a safe and effective method of urinary diversion for the majority of patients with existing ureteral obstruction in prostate cancer [972], [973].

In view of the palliative situation of the patients and with the high failure rate of retrograde probing, due to the frequent irritation originating from the lower urinary tract, and also due to of the non-optimal drainage and the risk of migration of the stent, it was decided by expert consensus to preferably recommend percutaneous drainage. However, the expected inconvenience of external urinary drainage should be made clear to each patient beforehand [974]. As with any therapeutic measure and especially with invasive procedures, a decision must be made together with the patient for one or the other therapeutic option, or a Watch and Wait approach, or a solely supportive approach (principles of good clinical practice (GCP)). Despite the preference for percutaneous derivation, information about alternatives should also be provided. A similar recommendation can be found in Ganatra et Loughin [975].

In the case of castration-resistant tumor, it must be clear to the affected that the situation is terminal. Therefore, at least in the case of bilateral obstruction, the indication for urinary diversion should be given only if the patient is in good general condition and values survival more than quality of life [976].

 

Re: Recommendation 7.71

The recommendations are fundamentally based on expert consensus. The patient and their relatives should be able to shape the period of palliative care according to their preferences and should be given comprehensive advice and information about this according to their wishes. This is in line with the recommendations in the NICE guideline [101] and the ACP guideline [938]. Certain possible events and how to deal with them should be anticipated and discussed. This concerns, for example, the termination of drug therapies in the event of the general condition deteriorating, the implementation of resuscitation measures, the possible referral to a hospital or a hospice, or the decision for outpatient care. It should be possible to revise the arrangements made if the clinical situation changes. Alternative treatment concepts should also be addressed if possible; this recommendation was adopted from the S3 guideline on the diagnosis, therapy, and aftercare of breast cancer [967].

 

Re: Recommendation 7.72

The NICE guideline [101] states that no comparative studies exist for evaluating specifications for palliative treatment. However, the need for an interdisciplinary approach is addressed in the available case series. The S3 guideline on breast cancer [967] also calls for an interdisciplinary treatment strategy. This includes good communication between the outpatient and inpatient teams. Patients should have specific contacts for pain management and psychosocial support.

 

Re: Recommendation 7.73

Both the treatment recommendations on tumor pain [907] and the general practitioner guideline of the Hesse guideline group on palliative care [966] recommend that pain treatment with analgesics be oriented according to the WHO's grading scheme.

The foundation for treatment is a careful diagnosis, in which non-tumor-related causes of pain always need to be taken into account. The extent of diagnostic imaging should depend on the stage of the disease and the patient's general condition. The pain history includes not only physical but also social and psychological aspects. Reversible causes of pain should be eliminated, see also explanations on the treatment of tumor-related urinary retention under Statement 7.59 and the chapter on Therapy of bone metastases.

Table 22: WHO pain management scheme

The WHO pain management scheme has three stages:  
Stage I  A nonopioid analgesic, e.g., acetaminophen or NSAIDs (ibuprofen, diclofenac), coanalgesics, and/or adjuvants.
Stage II A weak-acting opioid (not under controlled substance law), e.g., tramadol or tilidine/naloxone ± nonopioid analgesic, coanalgesics, and/or adjuvants.
Stage III A potent opioid (under controlled substance law), e.g., morphine ± nonopioid analgesic, coanalgesics, and/or adjuvants.

In addition to this stepwise regimen, the WHO recommends noninvasive oral therapy, if possible. This therapy should be individually tailored to the patient and given according to a fixed schedule [907].

For further information we refernce the two therapy recommendations on tumor pain and the GP guideline on palliative care [907], [966].

In any case, it should be examined which further non-drug measures can lead to additional pain relief.

 

Re: Recommendation 7.74

A progressive tumor disease brings physical and possibly psychological weakness and possibly also a fatigue syndrome with it. In order to deal with these limitations as a result of the advanced stage of the disease, the possibilities and limits of therapeutic interventions should be discussed with the patients and their relatives, as well as how to focus on the appropriate organization of everyday life. Reversible causes of physical or psychological complaints should be diagnosed and treated appropriately [966].

Other symptoms of the progressive tumor disease may also be of a physical or psychological nature. The nature and extent of the complaints should be regularly examined and enquired about. For treatmentappropriate to the respective situation, we reference the general practitioner's guideline on palliative care [966].

Evidence tables

7.1 EvT 7.1

Quelle

Fazit/Empfehlung

Empfehlungsgrad

Dutch Urological Association 2007

How is PSA recurrence diagnosed after radical prostatectomy with curative intent?

To diagnose PSA recurrence after radical prostatectomy requires two PSA tests of > 0.2 ng/mL with an interval of 2-3 months.

If the PSA doubling time is less than 10 months, it is likely that metastases or micrometastases are present.

Additional tests for PSA recurrence after radical prostatectomy

Biopsy of the prostate bed, ultrasound of the prostatic region, and DRE are not required to diagnose local recurrence after radical prostatectomy if the PSA level is low (< 2.0 ng/mL).
Requesting a bone scan is not necessary for asymptomatic patients with PSA recurrence after radical prostatectomy and PSA < 20 ng/mL.

Given its low sensitivity for detecting local recurrence in patients with PSA recurrence after radical prostatectomy, CT scan of the lower abdomen is not recommended.

MRI is not recommended for detecting local recurrence.

-

 

7.1 EvT 7.2

Quelle

Fazit/Empfehlung

Empfehlungsgrad

Dutch Urological Association 2007

How is PSA recurrence diagnosed after radical prostatectomy with curative intent?

To diagnose PSA recurrence after radical prostatectomy requires two PSA tests of > 0.2 ng/mL with an interval of 2-3 months.

If the PSA doubling time is less than 10 months, it is likely that metastases or micrometastases are present.

Additional tests for PSA recurrence after radical prostatectomy

Biopsy of the prostate bed, ultrasound of the prostatic region, and DRE are not required to diagnose local recurrence after radical prostatectomy if the PSA level is low (< 2.0 ng/mL).
Requesting a bone scan is not necessary for asymptomatic patients with PSA recurrence after radical prostatectomy and PSA < 20 ng/mL.

Given its low sensitivity for detecting local recurrence in patients with PSA recurrence after radical prostatectomy, CT scan of the lower abdomen is not recommended.

MRI is not recommended for detecting local recurrence.

-

 

7.1 EvT 7.3

Quelle

Fazit/Empfehlung

Empfehlungsgrad

EAU 2007

Definition of recurrence

  • Following radical prostatectomy, PSA values > 0.4 ng/mL represent recurrent cancer.
  • Following radiation therapy, a PSA value 2 ng/mL above the nadir after radiation therapy represent recurrent cancer.

Definition of local and systemic failure

  • Local failure following radical prostatectomy is predicted with an 80% probability by PSA increase > 3 years after radical prostatectomy, PSADT > 11 months, Gleason score < 6, stage < pT3a pN0, pTx R1.
  • Systemic failure following radical prostatectomy is predicted with > 80% accuracy by a PSA increase < 1 year after radical prostatectomy, PSADT of 4-6 months, Gleason score 8-10, stage pT3b, pTxpN1.
  • Local failure after radiation therapy is documented by a positive prostatic biopsy and negative imaging studies.
  • Prostatic biopsy after radiation therapy is only necessary if local procedures such as salvage prostatectomy are indicated in an individual patient.

Diagnostic procedures in patients with PSA relapse

1. Following radical prostatectomy, CT scans of the pelvis and abdomen are of low sensitivity and specificity in patients with PSA levels < 20 ng/mL or a PSA velocity of < 20 ng/mL/year.

2. Endorectal MRI or PET scans may help to detect local recurrences if PSA is > 1 - 2.0 ng/mL but is not yet part of routine clinical use.

3. If available, the capromab pendetide scan shows a diagnostic yield of 60% to 80% independent of the PSA serum level.

4. Following radiation therapy, local recurrence is documented by a positive biopsy > 18 months after the procedure.

 

 

7.1 EvT 7.4

Quelle

Fazit/Empfehlung

Empfehlungsgrad

NICE 2008

Analyse serial prostate specific antigen (PSA) levels after radical treatment using the same assay technique.

Biopsy of the prostatic bed should not be performed in men with prostate cancer who have had a radical prostatectomy.

Biopsy of the prostate after radiotherapy should only be performed in men with prostate cancer who are being considered for local salvage therapy in the context of a clinical trial.

For men with evidence of biochemical relapse following radical treatment and who are considering radical salvage therapy:

– Routine MRI scanning should not be performed prior to salvage radiotherapy in men with prostate cancer

– Perform an isotope bone scan if symptoms or PSA trends are suggestive of metastases.

Biochemical relapse (a rising PSA) alone should not necessarily prompt an immediate change in treatment.

Biochemical relapse should trigger an estimate of PSA doubling time, based on a minimum of 3 measurements over at least a 6 month period.

 

 

7.1 EvT 7.5

Quelle

Fazit/Empfehlung

Empfehlungsgrad

NICE 2008

Analyse serial prostate specific antigen (PSA) levels after radical treatment using the same assay technique.

Biopsy of the prostatic bed should not be performed in men with prostate cancer who have had a radical prostatectomy.

Biopsy of the prostate after radiotherapy should only be performed in men with prostate cancer who are being considered for local salvage therapy in the context of a clinical trial.

For men with evidence of biochemical relapse following radical treatment and who are considering radical salvage therapy:

– Routine MRI scanning should not be performed prior to salvage radiotherapy in men with prostate cancer

– Perform an isotope bone scan if symptoms or PSA trends are suggestive of metastases.

Biochemical relapse (a rising PSA) alone should not necessarily prompt an immediate change in treatment.

Biochemical relapse should trigger an estimate of PSA doubling time, based on a minimum of 3 measurements over at least a 6 month period.

 

 

Studien-typ

Autoren, Jahr

Untersuchte Studien/Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

LoE

Literaturbelege

syst. Review

Nguyen, 2007

syst. Recherche in Pubmed 1990-2005           (+Handsuche in Referenzlisten)

Suchbegriffe: salvage therapy, radiation failure, radiation recurrence, prostate cancer, salvage brachytherapy, salvage prostatectomy, salvage cryotherapy, reirradiation

 

Diagnostik, Therapie, Nachsorge, Komplikationen bei lokaler Rezidivtherapie bei PSA-Rezidiv nach RT

RPE:

- insgesamt 14 Studien seit 1990 (n=531)

- 5J-Raten für RFS (biochemical) nach RPE zwischen 50% und 60%

- bei PSA<10ng/ml z.Zt. der RPE signifikant geringere PSA-Rezidive als bei PSA≥10ng/ml

k.A.

1. Bianco FJ Jr, et al. Int J Radiat Oncol Biol Phys. 2005

2. Ward JF, et al. J Urol 2005

3. Cheng L, et al. Cancer 1998

Zystoprostatektomie (CP):

- 5J-Raten für RFS (biochemical) schlechter im Vergleich zu RPE

- (lokal ausgedehnte Karzinome) cave: systematischer Fehler durch Patientenauswahl nicht auszuschließen!

-Schlussfolgerung der Autoren:  stellen CP als Monotherapie in Frage

k.A.

- siehe 2.

4. Gheiler EL, et al. Urology 1998

neoadjuvante Androgendeprivation:

- nur retrospektive Daten verfügbar:

SM+-Raten nicht geringer, auch keine weiteren positiven Resultate

- geringster Anteil SM+ = 21% (n=24), gleichzeitig - 5J-Raten für RFS (biochemical) = 80%

-Schlussfolgerung der Autoren:  insgesamt keine Empfehlung für Routineanwendung neoadjuvanter ADT vor Rezidiv-OP

k.A.

- siehe 1. und 2.

5. van der Poel HG, et al. Eur Urol 2007

6. Garzotto M, Wajsman Z. J Urol 1998

7. Sanderson KM, et al. J Urol 2006

Komplikationen bei operativer Therapie:

- Risiko durch Veränderungen nach RT insgesamt größer als bei Primärtherapie

- wichtigste unerwünschte Folgen (Mittelwerte): Inkontinenz (41%), rektale Verletzungen (4,7%), BlasenhalSRTrikturen (24%), Tod (1 Ereignis))

- im Laufe der Jahre weniger rektale Verletzungen (bis 1992 15% nach 1992 2%)

- Reduktion der Nebenwirkungen (Grad 2 bis 4) insgesamt von 33% auf 13% vor bzw. nach 1993

- keine Veringerungen bei Inkontinenz und BlasenhalSRTrikturen

k.A:

- Gunderson LL, Tepper JE. Clinical Radiation Oncology. 2. ed. Philadelphia: Elsevier-Churchill Livingstone; 2007

- siehe 1. und 2./4. bis 7.

- Dall’Oglio MF, et al. Int Braz J Urol 2006

- Darras J, et al. Eur J Surg Oncol 2006

- Link P, Freiha FS. Urology 1991

- Moul JW, Paulson DF. Cancer 1991

- Pisters LL, et al. J Urol 2000

- Stein A, et al. Urology 1992

- Vaidya A, Soloway MS. J Urol 2000

- Vallancien G, et al. J Urol 2003

- Stephenson AJ, et al. J Urol 2004

Kryotherapie (KT):

- insgesamt 8 Studien seit 1990 (n=510)

- Vergleich erschwert durch verschiedene Definitionen von PSA-Rezidiv und verschieden langes Followup

- Prediktoren für sign. Abnahme der PSA-Werte: PSA-Niveau >10ng/ml vor Kryotherapie, klinisch T3 o. T4 (vor KT bzw. nach RT) und GS >8 (vor RT bzw. ≥8 vor KT)

- PSA-DT <16Mon. mit schlechteren PSA-Werten nach Therapie verbunden

- im Allgemeinen RFS (biochemical) nach KT etwas geringer als nach RP

- mögliche Erklärung: unvollständige Entfernung der Prostata

- aber weniger unerwünschte Folgen

Komplikationen bei KT:

- Inkontinenz bei 36% (besonders bei vorangegangener TURP+RT)

- fehlender urethraler Wärmenkatheter anderer wichtiger Risikofaktor für Incontinenz

- Schleimhautablösungen bei 11%

- BlasenhalSRTrikturen oder Harnverhalt bei 17%

- perianale Schmerzen bei 36% (zum Teil so stark, dass Alltagsleben beeinträchtigt)

- auch hier Zusammenhang mit fehlendem Wärmenkatheter wahrscheinlich

- Fisteln bei 2,6% (besonders bei T3/T4)

- Tod sehr selten

k.A:

- Izawa JI, et al. J Clin Oncol 2002

- Chin JL, et al. J Urol 2001

- Spiess PE, et al. Cancer 2006

- Bahn DK, et al. Clin Prostate Cancer 2003

- Bales GT, et al. Urology 1995

- Donnelly BJ, et al. Prostate Cancer Prostatic Dis 2005

- Ghafar MA, et al. J Urol  2001

- Han KR, et al. J Urol 2003

- Miller RJ Jr, et al. Cancer 1996

. Izawa JIet al. J Urol 2000

- Long JP, et al. J Urol 1998

- Perrotte P, et al. J Urol 1999

- Pisters LL, et al. J Urol 1997

Brachytherapie (BT):

- insgesamt 10 Studien seit 1990

- Vergleich erschwert durch verschiedene Definitionen von PSA-Rezidiv und verschieden langes Followup

- keine stat. signifikanten Prediktoren für die Verbesserung der PSA-Werte berichtet

- die Senkung der PSA-Werte durch BT scheint vergleichbar mit Effekt von RPE - etwas besser als durch KT

Komplikationen bei BT:

- kontraindiziert bei Pat. mit proctopathy oder Blasenentzündungen nach vorangegangener RT (hohes Risiko für rektale oder Blasenverletzungen)

- Inkontinenz bei 6%

- Nebenwirkungen (Grad 3 o. 4) bei 5,6%

- Nebenwirkungen des Urogenitaltraktes (Grad 3 o. 4) bei 17%

- besonders Fisteln (Prostata-Rektum) bei 3,4%

- Risiko der Nebenwirkungen (Grad 3 o. 4) steigt mit kürzerem Abstand zwischen der primären RT und der Rezidiv-RT (<4,5J Abstand = Vorteil)

- Anzahl der Rektalverletzungen nach BT ähnlich KT und OP

- Anzahl der Inkontinenzfolgen geringer als bei KT und OP

- Häufigkeit von Fisteln gering, aber mit bedeutendem Einfluss auf die LQ

k.A:

- Grado GL, et al. Urology 1999

- Koutrouvelis P, et al. Technol Cancer Res Treat 2003

- Lee B, et al. Int J Radiat Oncol Biol Phys 2007

- Lo K, et al. Int J Radiat Oncol Biol Phys 2005

- Nguyen PL, et al. Cancer 2007

- Wong WW, et al. J Urol 2006

- Beyer DC. Urology 1999

- Wallner KE, et al. J Urol 1990

- LoEning SA, Turner JW. Prostate 1993

- Dattoli M, et al. Proc Am Soc Clin Oncol 1997

Hochintensiv focussierter Ultraschall (HIFU):

- nach primärer RT

- 30Mon.-Rate für RFS bei 38% (n=71)

- Nebenwirkungen: Inkontinenz (Grad 3) bei 7%, BlasenhalSRTrikturen bei 17%, Rektalfisteln bei 6% (Followup 14,8Mon.)

weitere experimentelle Therapieansätze:

- ferromagnetische Thermoablation (ferromagnetic thermal ablation)

- interstitielle Hyperthermie (interstitial microwave thermal therapy)

- werden zurzeit in Studien überprüft

k.A:

- Gelet A, et al. Urology 2004

- Kadoch C, et al. Oncologist 2005

- Master VA, et al. J Urol 2004

- McCann C, et al. Phys Med Biol 2003

 

7.2.2 EvT 7.12

Studien-typ

Autoren, Jahr

Untersuchte Studien/Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

LoE

Literaturbelege

syst. Review

Nguyen, 2007

syst. Recherche in Pubmed 1990-2005           (+Handsuche in Referenzlisten)

Suchbegriffe: salvage therapy, radiation failure, radiation recurrence, prostate cancer, salvage brachytherapy, salvage prostatectomy, salvage cryotherapy, reirradiation

 

Diagnostik, Therapie, Nachsorge, Komplikationen bei lokaler Rezidivtherapie bei PSA-Rezidiv nach RT

RPE:

- insgesamt 14 Studien seit 1990 (n=531)

- 5J-Raten für RFS (biochemical) nach RPE zwischen 50% und 60%

- bei PSA<10ng/ml z.Zt. der RPE signifikant geringere PSA-Rezidive als bei PSA≥10ng/ml

k.A.

1. Bianco FJ Jr, et al. Int J Radiat Oncol Biol Phys. 2005

2. Ward JF, et al. J Urol 2005

3. Cheng L, et al. Cancer 1998

Zystoprostatektomie (CP):

- 5J-Raten für RFS (biochemical) schlechter im Vergleich zu RPE

- (lokal ausgedehnte Karzinome) cave: systematischer Fehler durch Patientenauswahl nicht auszuschließen!

-Schlussfolgerung der Autoren:  stellen CP als Monotherapie in Frage

k.A.

- siehe 2.

4. Gheiler EL, et al. Urology 1998

neoadjuvante Androgendeprivation:

- nur retrospektive Daten verfügbar:

SM+-Raten nicht geringer, auch keine weiteren positiven Resultate

- geringster Anteil SM+ = 21% (n=24), gleichzeitig - 5J-Raten für RFS (biochemical) = 80%

-Schlussfolgerung der Autoren:  insgesamt keine Empfehlung für Routineanwendung neoadjuvanter ADT vor Rezidiv-OP

k.A.

- siehe 1. und 2.

5. van der Poel HG, et al. Eur Urol 2007

6. Garzotto M, Wajsman Z. J Urol 1998

7. Sanderson KM, et al. J Urol 2006

Komplikationen bei operativer Therapie:

- Risiko durch Veränderungen nach RT insgesamt größer als bei Primärtherapie

- wichtigste unerwünschte Folgen (Mittelwerte): Inkontinenz (41%), rektale Verletzungen (4,7%), BlasenhalSRTrikturen (24%), Tod (1 Ereignis))

- im Laufe der Jahre weniger rektale Verletzungen (bis 1992 15% nach 1992 2%)

- Reduktion der Nebenwirkungen (Grad 2 bis 4) insgesamt von 33% auf 13% vor bzw. nach 1993

- keine Veringerungen bei Inkontinenz und BlasenhalSRTrikturen

k.A:

- Gunderson LL, Tepper JE. Clinical Radiation Oncology. 2. ed. Philadelphia: Elsevier-Churchill Livingstone; 2007

- siehe 1. und 2./4. bis 7.

- Dall’Oglio MF, et al. Int Braz J Urol 2006

- Darras J, et al. Eur J Surg Oncol 2006

- Link P, Freiha FS. Urology 1991

- Moul JW, Paulson DF. Cancer 1991

- Pisters LL, et al. J Urol 2000

- Stein A, et al. Urology 1992

- Vaidya A, Soloway MS. J Urol 2000

- Vallancien G, et al. J Urol 2003

- Stephenson AJ, et al. J Urol 2004

Kryotherapie (KT):

- insgesamt 8 Studien seit 1990 (n=510)

- Vergleich erschwert durch verschiedene Definitionen von PSA-Rezidiv und verschieden langes Followup

- Prediktoren für sign. Abnahme der PSA-Werte: PSA-Niveau >10ng/ml vor Kryotherapie, klinisch T3 o. T4 (vor KT bzw. nach RT) und GS >8 (vor RT bzw. ≥8 vor KT)

- PSA-DT <16Mon. mit schlechteren PSA-Werten nach Therapie verbunden

- im Allgemeinen RFS (biochemical) nach KT etwas geringer als nach RP

- mögliche Erklärung: unvollständige Entfernung der Prostata

- aber weniger unerwünschte Folgen

Komplikationen bei KT:

- Inkontinenz bei 36% (besonders bei vorangegangener TURP+RT)

- fehlender urethraler Wärmenkatheter anderer wichtiger Risikofaktor für Incontinenz

- Schleimhautablösungen bei 11%

- BlasenhalSRTrikturen oder Harnverhalt bei 17%

- perianale Schmerzen bei 36% (zum Teil so stark, dass Alltagsleben beeinträchtigt)

- auch hier Zusammenhang mit fehlendem Wärmenkatheter wahrscheinlich

- Fisteln bei 2,6% (besonders bei T3/T4)

- Tod sehr selten

k.A:

- Izawa JI, et al. J Clin Oncol 2002

- Chin JL, et al. J Urol 2001

- Spiess PE, et al. Cancer 2006

- Bahn DK, et al. Clin Prostate Cancer 2003

- Bales GT, et al. Urology 1995

- Donnelly BJ, et al. Prostate Cancer Prostatic Dis 2005

- Ghafar MA, et al. J Urol  2001

- Han KR, et al. J Urol 2003

- Miller RJ Jr, et al. Cancer 1996

. Izawa JIet al. J Urol 2000

- Long JP, et al. J Urol 1998

- Perrotte P, et al. J Urol 1999

- Pisters LL, et al. J Urol 1997

Brachytherapie (BT):

- insgesamt 10 Studien seit 1990

- Vergleich erschwert durch verschiedene Definitionen von PSA-Rezidiv und verschieden langes Followup

- keine stat. signifikanten Prediktoren für die Verbesserung der PSA-Werte berichtet

- die Senkung der PSA-Werte durch BT scheint vergleichbar mit Effekt von RPE - etwas besser als durch KT

Komplikationen bei BT:

- kontraindiziert bei Pat. mit proctopathy oder Blasenentzündungen nach vorangegangener RT (hohes Risiko für rektale oder Blasenverletzungen)

- Inkontinenz bei 6%

- Nebenwirkungen (Grad 3 o. 4) bei 5,6%

- Nebenwirkungen des Urogenitaltraktes (Grad 3 o. 4) bei 17%

- besonders Fisteln (Prostata-Rektum) bei 3,4%

- Risiko der Nebenwirkungen (Grad 3 o. 4) steigt mit kürzerem Abstand zwischen der primären RT und der Rezidiv-RT (<4,5J Abstand = Vorteil)

- Anzahl der Rektalverletzungen nach BT ähnlich KT und OP

- Anzahl der Inkontinenzfolgen geringer als bei KT und OP

- Häufigkeit von Fisteln gering, aber mit bedeutendem Einfluss auf die LQ

k.A:

- Grado GL, et al. Urology 1999

- Koutrouvelis P, et al. Technol Cancer Res Treat 2003

- Lee B, et al. Int J Radiat Oncol Biol Phys 2007

- Lo K, et al. Int J Radiat Oncol Biol Phys 2005

- Nguyen PL, et al. Cancer 2007

- Wong WW, et al. J Urol 2006

- Beyer DC. Urology 1999

- Wallner KE, et al. J Urol 1990

- LoEning SA, Turner JW. Prostate 1993

- Dattoli M, et al. Proc Am Soc Clin Oncol 1997

Hochintensiv focussierter Ultraschall (HIFU):

- nach primärer RT

- 30Mon.-Rate für RFS bei 38% (n=71)

- Nebenwirkungen: Inkontinenz (Grad 3) bei 7%, BlasenhalSRTrikturen bei 17%, Rektalfisteln bei 6% (Followup 14,8Mon.)

weitere experimentelle Therapieansätze:

- ferromagnetische Thermoablation (ferromagnetic thermal ablation)

- interstitielle Hyperthermie (interstitial microwave thermal therapy)

- werden zurzeit in Studien überprüft

k.A:

- Gelet A, et al. Urology 2004

- Kadoch C, et al. Oncologist 2005

- Master VA, et al. J Urol 2004

- McCann C, et al. Phys Med Biol 2003

 

 

 

Artikel (Autor, Jahrl)

Studientyp

Anzahl der Patienten

Patientenmerkmale

Intervention

Vergleichs-Intervention

Nachverfol­gung

Outcomes

Ergebnisse

Bemerkungen

Evidenz-Niveau (SIGN)

Heidenreich, 2010

retrospektive Fallserie

n=55

Pat mit durch Biopsie bestätigtem Rezidiv eines PCa nach EBRT oder Brachytherapie

Salvage Prostatektomie (SRP), +ePNLD

keine Vergleichsintervention, Stratifizierung nach prognostischen Faktoren für ein Lokalrezidiv

23 MO nach SRP

 

68 MO nach RT

Rezidiv organbegrenzt

LK-Status

Schnittrand

multivariate Analyse von Prädiktoren eines in sano zu operierenden Rezidivs

 

OP-Daten

n=40 (72.7%):  organbegrenztes PCa

n=15 (27.3%) local fortgeschr.     PCa

n=11 (20%) LK+

n=7 (12.7%) R1 (pos. Schnittrand)

Multivariate Analyse :

signifikante Prädiktoren eines organbeschränkten Rezidivs mit negativen Schnitträndern:

1. Gleason Score aus der Biopsie vor SRP ( p=0.02),

2. <50% positive Stanzen (p=0.001),

3. PSA-Verdopplungszeit >12 mo ( p=0.001),

4. Z.n. LDR ( p=0.001)

Med. OP-Zeit: 120 Min

Mde. Intraop. Blutverlust: 360ml

 

3

 

7.2.2 EvT 7.13

Studientyp

Autoren, Jahr

Untersuchte Studien/Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

LoE

Literaturbelege

syst. Review

Nguyen, 2007

syst. Recherche in Pubmed 1990-2005           (+Handsuche in Referenzlisten)

Suchbegriffe: salvage therapy, radiation failure, radiation recurrence, prostate cancer, salvage brachytherapy, salvage prostatectomy, salvage cryotherapy, reirradiation

 

Diagnostik, Therapie, Nachsorge, Komplikationen bei lokaler Rezidivtherapie bei PSA-Rezidiv nach RT

RPE:

- insgesamt 14 Studien seit 1990 (n=531)

- 5J-Raten für RFS (biochemical) nach RPE zwischen 50% und 60%

- bei PSA<10ng/ml z.Zt. der RPE signifikant geringere PSA-Rezidive als bei PSA≥10ng/ml

k.A.

1. Bianco FJ Jr, et al. Int J Radiat Oncol Biol Phys. 2005

2. Ward JF, et al. J Urol 2005

3. Cheng L, et al. Cancer 1998

Zystoprostatektomie (CP):

- 5J-Raten für RFS (biochemical) schlechter im Vergleich zu RPE

- (lokal ausgedehnte Karzinome) cave: systematischer Fehler durch Patientenauswahl nicht auszuschließen!

-Schlussfolgerung der Autoren:  stellen CP als Monotherapie in Frage

k.A.

- siehe 2.

4. Gheiler EL, et al. Urology 1998

neoadjuvante Androgendeprivation:

- nur retrospektive Daten verfügbar:

SM+-Raten nicht geringer, auch keine weiteren positiven Resultate

- geringster Anteil SM+ = 21% (n=24), gleichzeitig - 5J-Raten für RFS (biochemical) = 80%

-Schlussfolgerung der Autoren:  insgesamt keine Empfehlung für Routineanwendung neoadjuvanter ADT vor Rezidiv-OP

k.A.

- siehe 1. und 2.

5. van der Poel HG, et al. Eur Urol 2007

6. Garzotto M, Wajsman Z. J Urol 1998

7. Sanderson KM, et al. J Urol 2006

Komplikationen bei operativer Therapie:

- Risiko durch Veränderungen nach RT insgesamt größer als bei Primärtherapie

- wichtigste unerwünschte Folgen (Mittelwerte): Inkontinenz (41%), rektale Verletzungen (4,7%), BlasenhalSRTrikturen (24%), Tod (1 Ereignis))

- im Laufe der Jahre weniger rektale Verletzungen (bis 1992 15% nach 1992 2%)

- Reduktion der Nebenwirkungen (Grad 2 bis 4) insgesamt von 33% auf 13% vor bzw. nach 1993

- keine Veringerungen bei Inkontinenz und BlasenhalSRTrikturen

k.A:

- Gunderson LL, Tepper JE. Clinical Radiation Oncology. 2. ed. Philadelphia: Elsevier-Churchill Livingstone; 2007

- siehe 1. und 2./4. bis 7.

- Dall’Oglio MF, et al. Int Braz J Urol 2006

- Darras J, et al. Eur J Surg Oncol 2006

- Link P, Freiha FS. Urology 1991

- Moul JW, Paulson DF. Cancer 1991

- Pisters LL, et al. J Urol 2000

- Stein A, et al. Urology 1992

- Vaidya A, Soloway MS. J Urol 2000

- Vallancien G, et al. J Urol 2003

- Stephenson AJ, et al. J Urol 2004

Kryotherapie (KT):

- insgesamt 8 Studien seit 1990 (n=510)

- Vergleich erschwert durch verschiedene Definitionen von PSA-Rezidiv und verschieden langes Followup

- Prediktoren für sign. Abnahme der PSA-Werte: PSA-Niveau >10ng/ml vor Kryotherapie, klinisch T3 o. T4 (vor KT bzw. nach RT) und GS >8 (vor RT bzw. ≥8 vor KT)

- PSA-DT <16Mon. mit schlechteren PSA-Werten nach Therapie verbunden

- im Allgemeinen RFS (biochemical) nach KT etwas geringer als nach RP

- mögliche Erklärung: unvollständige Entfernung der Prostata

- aber weniger unerwünschte Folgen

Komplikationen bei KT:

- Inkontinenz bei 36% (besonders bei vorangegangener TURP+RT)

- fehlender urethraler Wärmenkatheter anderer wichtiger Risikofaktor für Incontinenz

- Schleimhautablösungen bei 11%

- BlasenhalSRTrikturen oder Harnverhalt bei 17%

- perianale Schmerzen bei 36% (zum Teil so stark, dass Alltagsleben beeinträchtigt)

- auch hier Zusammenhang mit fehlendem Wärmenkatheter wahrscheinlich

- Fisteln bei 2,6% (besonders bei T3/T4)

- Tod sehr selten

k.A:

- Izawa JI, et al. J Clin Oncol 2002

- Chin JL, et al. J Urol 2001

- Spiess PE, et al. Cancer 2006

- Bahn DK, et al. Clin Prostate Cancer 2003

- Bales GT, et al. Urology 1995

- Donnelly BJ, et al. Prostate Cancer Prostatic Dis 2005

- Ghafar MA, et al. J Urol  2001

- Han KR, et al. J Urol 2003

- Miller RJ Jr, et al. Cancer 1996

. Izawa JIet al. J Urol 2000

- Long JP, et al. J Urol 1998

- Perrotte P, et al. J Urol 1999

- Pisters LL, et al. J Urol 1997

Brachytherapie (BT):

- insgesamt 10 Studien seit 1990

- Vergleich erschwert durch verschiedene Definitionen von PSA-Rezidiv und verschieden langes Followup

- keine stat. signifikanten Prediktoren für die Verbesserung der PSA-Werte berichtet

- die Senkung der PSA-Werte durch BT scheint vergleichbar mit Effekt von RPE - etwas besser als durch KT

Komplikationen bei BT:

- kontraindiziert bei Pat. mit proctopathy oder Blasenentzündungen nach vorangegangener RT (hohes Risiko für rektale oder Blasenverletzungen)

- Inkontinenz bei 6%

- Nebenwirkungen (Grad 3 o. 4) bei 5,6%

- Nebenwirkungen des Urogenitaltraktes (Grad 3 o. 4) bei 17%

- besonders Fisteln (Prostata-Rektum) bei 3,4%

- Risiko der Nebenwirkungen (Grad 3 o. 4) steigt mit kürzerem Abstand zwischen der primären RT und der Rezidiv-RT (<4,5J Abstand = Vorteil)

- Anzahl der Rektalverletzungen nach BT ähnlich KT und OP

- Anzahl der Inkontinenzfolgen geringer als bei KT und OP

- Häufigkeit von Fisteln gering, aber mit bedeutendem Einfluss auf die LQ

k.A:

- Grado GL, et al. Urology 1999

- Koutrouvelis P, et al. Technol Cancer Res Treat 2003

- Lee B, et al. Int J Radiat Oncol Biol Phys 2007

- Lo K, et al. Int J Radiat Oncol Biol Phys 2005

- Nguyen PL, et al. Cancer 2007

- Wong WW, et al. J Urol 2006

- Beyer DC. Urology 1999

- Wallner KE, et al. J Urol 1990

- LoEning SA, Turner JW. Prostate 1993

- Dattoli M, et al. Proc Am Soc Clin Oncol 1997

Hochintensiv focussierter Ultraschall (HIFU):

- nach primärer RT

- 30Mon.-Rate für RFS bei 38% (n=71)

- Nebenwirkungen: Inkontinenz (Grad 3) bei 7%, BlasenhalSRTrikturen bei 17%, Rektalfisteln bei 6% (Followup 14,8Mon.)

weitere experimentelle Therapieansätze:

- ferromagnetische Thermoablation (ferromagnetic thermal ablation)

- interstitielle Hyperthermie (interstitial microwave thermal therapy)

- werden zurzeit in Studien überprüft

k.A:

- Gelet A, et al. Urology 2004

- Kadoch C, et al. Oncologist 2005

- Master VA, et al. J Urol 2004

- McCann C, et al. Phys Med Biol 2003

 

 

 

7.2.2 EvT 7.15

Zitat

Population

Intervention

Suchstrategie

eingeschlossene Studien

Ergebnisse

Methodische Validität

LoE

Parekh A.
Cancer control and complications of salvage local therapy after failure of radiotherapy for prostate cancer: a systematic review.
Semin Radiat Oncol 2013

pat.s with PCa recurrence

body of evidence: 7 studies on HIFU (n=4; 19; 22; 31; 46; 71; 167 pat.s)

HIFU as salvage treatment post-RT for PCa

Pubmed search, reference lists
up to Sept.2011

studies reporting either cancer control and/or toxicity outcomes

survival outcomes (individual studies):
[n=167 pat.s; mean follow-up 18mo] 3y progression-free survival rate 53%
[n=22 pat.s] 2y biochemical disease-free survival by risk group: low 100%, intermediate 84%, high 14%
bDFS rate in other studies: range 39-73% (follow-up in studies: 6mo-5y)

adverse events:
incontinence 37%
rectal injury 2%
bladder neck stricture 15%
fistula 4%
urinary retention 7.8%

1. a priori Design - nein
2. Selektion und Extraktion durch 2 unabh. Bearbeiter - ja
3. Literaturrecherche min 2 Datenbanken + 1 Zusatz - nein
4. graue Literatur eingeschl. - nein
5. Vollständige Liste ein- und ausgeschl. Studien - nein
6. Details der eingeschlossenen Studien - ja
7. Qualität der eingeschlossenen Studien bewertet - nein
8. In Conclusion Hinweise auf methodische Qualität - nein
9. Heterogenität erhoben?- nein
10. Publication bias (funnel plot) - nein
11. CoI / funding für Review UND für alle Studien berichtet - nein

AMSTAR-Score 2/11

3

 

 

Zitat

Population

Intervention

Studiendesign

Ergebnisse

Methodische Qualität

LoE

Crouzet S.
Salvage high-intensity focused ultrasound (HIFU) for locally recurrent prostate cancer after failed radiation therapy: Multi-institutional analysis of 418 patients.
BJU Int 2017.

n = 418 pat.s with recurrent PCa after EBRT

total-gland salvage HIFU (Ablatherm)

tx 1995.2009

retrospective cohort analysis
no comparator

median follow-up 3.3y (1.5-5.2y)

outcomes: biochemical failure-free survival; progression to metastasis, cancer-specific death

oncological outcomes
7-y overall survival: 72%
7-y cancer-specific survival: 82%
7-y metastasis-free survival: 81%

5-y biochemical failure-free survival: 49%
-  by risk group - 58% for low, 51% for intermediate, 36% for high (similar for PSA and GSC categories)

salvage treatment-free survival: 37%
-  by risk group - 54% for low, 37% for intermediate, 23% for high (similar for PSA and GSC categories)

Studientyp - retrospective case series
Patientencharakteristika beschrieben (Tabelle)? - ja
ausbalanciert zwischen den Gruppen? - n.a.
Drop-out beschrieben/Consort-Diagramm? - nein/n.a.
Statistische Methoden beschrieben? - ja
angewendete Methoden und Definitionen beschrieben (z. B. Schmerzskala, Definition von Schmerz, Messmethoden etc.) - ja
ITT-Analyse? - n.a.
Interessenkonflikte/Sponsoring beschrieben? - ja (vorhanden)

3

Kanthabalan A.
Focal salvage high-intensity focused ultrasound in radiorecurrent prostate cancer.
BJU Int 2017

n = 150 pat.s with recurrent PCa after EBRT

focal salvage HIFU (Sonablate)
tx 2006-2015

retrospective cohort analysis
no comparator

mean follow-up

primary outcome: composite failure = biochemical failure and/or positive imaging results and/or bx and/or syst.tx and/or metastases and/or PCa-specific death
secondary outcome: biochemical failure by Phoenix-Astro definition (PSA nadir +2 ng/ml)

oncological outcomes
3-y composite endpoint-free survival: 40% (95%CI 31-50)
- by risk group before salvage tx: 100% for low, 49% for intermediate, 24% for high)
- in PSA responders (nadir ≤0.5ng/ml): 67% (95%CI 53-82%)

3-y biochemical disease-free survival: 48% (95%CI 39-59)
- by risk group before salvage tx: 100% for low, 61% for intermediate, 32% for high)
- in PSA responders (nadir ≤0.5ng/ml): 78% (95%CI 67-92%)

complications:
UTI in 11.3%; epididymitis in 1.3%, bladder neck strictures in 8%, rectourethral fistula after first HIFU in 2%, osteitis pubis in 0.7%

Studientyp - retrospective case series
Patientencharakteristika beschrieben (Tabelle)? - ja
ausbalanciert zwischen den Gruppen? - n.a.
Drop-out beschrieben/Consort-Diagramm? - nein/n.a.
Statistische Methoden beschrieben? - ja
angewendete Methoden und Definitionen beschrieben (z. B. Schmerzskala, Definition von Schmerz, Messmethoden etc.) - ja
ITT-Analyse? - n.a.
Interessenkonflikte/Sponsoring beschrieben? - nein

3

 

 

 

7.2.3 EvT 7.16

Quelle

Fazit/Empfehlung

Empfehlungs- grad

NICE 2008

It is not known whether treating biochemical relapse, rather than waiting until there are clinical signs of disease, will influence survival. Biochemical relapse after radical treatment, in many cases, does not lead to metastases or death from prostate cancer. Whether men with biochemical relapse should be treated depends in part on the timing and rate of rise of PSA as a predictor of clinical progression. Management options can be divided into local salvage therapies and systemic therapies.

Recommendations

  • Biochemical relapse (a rising PSA) alone should not necessarily prompt an immediate change in treatment.
  • Biochemical relapse should trigger an estimate of PSA doubling time, based on a minimum of 3 measurements over at least a 6 month period.

Qualifying statement: There is evidence from longitudinal studies and clinical trials to support making these recommendations.

Local Salvage Therapy

For men with biochemical relapse following radical prostatectomy

Surveys of current practice in the UK have shown a large variation in the selection of men for salvage radiotherapy: whether to give radiotherapy as soon as relapse is confirmed or when a PSA threshold is reached; whether to treat just the prostate bed or surrounding tissues as well; and whether or not to use adjuvant hormonal therapy in addition.

Recommendations

  • Men with biochemical relapse after radical prostatectomy, with no known metastases, should be offered early radical radiotherapy to the prostatic bed. Qualifying statement: There is a range of evidence to support this recommendation.
  • Men with biochemical relapse should be considered for entry to appropriate clinical trials.Qualifying statement: This recommendation is based on GDG consensus.

For men with biochemical relapse following radical radiotherapy (external beam or brachytherapy)

Salvage local therapies for biochemical relapse after radiotherapy (external beam or brachytherapy) include radical prostatectomy, cryotherapy and high intensity focused ultrasound. Radical prostatectomy as salvage has been shown to produce biochemical control in highly selected men but carries a higher risk of incontinence, impotence and rectal damage than when used as primary treatment.

Systemic Therapy

Hormonal therapy may control symptomatic, progressive or metastatic disease following either surgery or radiation. There are variations in practice with regard to the indications for, and the timings of, hormonal therapy in these situations. Other systemic therapies such as chemotherapy, bisphosphonates and celecoxib are being investigated in continuing clinical trials.

Recommendation

- Hormonal therapy is not routinely recommended for men with prostate cancer who have a biochemical relapse unless they have: – symptomatic local disease progression, or – any proven metastases, or – a PSA doubling time of < 3months.

Qualifying statement: There is evidence from randomised controlled trials to support this recommendation.

n.a.

 

 

 

Sweeney_NEJM_2015

Zitat

Studiencharakteristika

Studienergebnisse

Methodische Bewertung

LoE

Sweeney CJ. Chemohormonal Therapy in Metastatic Hormone-Sensitive Prostate Cancer. N Engl J Med 2015;373(8):737-46.

• multicenter RCT
• Studiendesign: Randomisierung ADT + Docetaxel 75 mg/m2 alle 3 Wo, 6 Zyklen + Prednison vs. ADT alone
• Follow-up  median 28.9 mo
• Keine Verblindung, eingangs unterschieldiche Monitorings für intervention arm (alle 3 Wo während Dsosianpassung)
• Population: (n=790, 1:1 Randomisierung), m mit PCa Diagnose und raiolog. nachgewiesenen Metastasen ECOG 0-2
• primary Endpoint: OS, secondary EP: Time to castration resistant PCa; time to clinical progression
• enrollment: 2006 - 2012; interimanalyse: 12/2013
• USA/ ECOG/ACRIN, Southwest Oncology Group, Alliance for clinical trials in Oncology, NRG oncology
• high volume metastases: visceral Metastases or ≥4 bone lesions with ≥ 1 beyond vertebral bodies and pelvis

• Baseline characteristics balanced between groups: age median 64, 85% white; 69% ECOG 0; high volume 66% (intervention) vs. 63% (control); 61% Gleason 8-10; PSA median 51 ng/ml; 78% no local therapy
• treatment effects:
o OS: 57,6 mo (intervention group) vs. 44 mo (control), HR 0,61, 95%CI 0,47 - 0,80; p< 0,001);
o OS signifikant für high volume metastases: 49,2 mo vs. 32,2 mo (HR 0,60, 95%CI 0,45 - 0,81, p<0,001), nicht signifikant für low volume disaese
o time to castration-resistant PCa: 20,2 mo vs. 11,7 mo (HR 0,61, 95%CI 0,0,51 - 0,72, p<0,001);
o Time to clinical progression 33,2 mo vs. 19,8 mo (HR 0,61, 95%CI 0,50 - 0,75, p=0,001)
• Adverse Events in intervention group: 2% Grad 3/4 allergic reaction; 4% grad 3/4 fatigue; 12,1% neutropenia grad 3/4;  6,1% febrile neutropenia; 2,3% infection with neutropenia; 1,3 % anemia ; 1 sudden death

• Randomisierung
• Allocation concealment nicht beschrieben
• nicht verblindet; engeres monitoring für interventiongroup während Dosisanpassung
• Patientenbcharakterisika ausbalanciert zwischen den Gruppen, aber größtenteils sehr fitte Patienten
• Statistische Methoden beschrieben
• relevante Methoden und Definitionen beschrieben, Endpunkte definiert
• primärer Endpunkt OS / patientenrelevant/ Fazit auf Basis OS,
• ITT-Analyse
• Interimanalyse

• Interessenkonflikte nicht beschrieben/ Finanzierung durch NIH, Sponsoring durch Sanofi

Da keine Verblindung, ggf. Bias zugunsten Intervention, kurzes follow up,
gesamt-Ereignisraten: 85 Todesfälle interventiongroup vs. 114 Todesfälle control group (Unterschied: 29/790)

1 (+/-)

 

 

Fizazi_NEJM_2017_Fizazi_LancetOncol_2019

Fizazi K, Tran NP, Fein L et al. Abiraterone plus Prednisone in Metastatic, Castration-Sensitive Prostate Cancer. N Engl J Med. 2017 Jul 27;377(4):352-360. doi: 10.1056/NEJMoa1704174.

Fizazi K, Tran NP, Fein L et al. Abiraterone acetate plus prednisone in patients with newly diagnosed high-risk metastatic castration-sensitive prostate cancer (LATITUDE): final overall survival analysis of a randomised, double-blind, phase 3 trial. Lancet Oncol. 2019 May;20(5):686-700. doi: 10.1016/S1470-2045(19)30082-8.

Link

https://pubmed.ncbi.nlm.nih.gov/28578607/

https://pubmed.ncbi.nlm.nih.gov/30987939/

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: low

Attrition bias

lost to follow-up: low

ITT-analysis: low

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the trial was designed by the senior academic authors and employees of the sponsor, data analyses were performed by statisticians employed by the sponsor

Design

  randomized, double-blind placebo-controlled, phase 3 trial

  LATITUDE, NCT01715285

- Cut-off: October 31, 2016 (1. interim analysis), August 15, 2018 (final analysis)

Population

newly diagnosed, metastatic, castration-sensitive prostate cancer

235 sites in 34 countries (Europe, Asia-Pacific region, Latin America, Canada)

February 12, 2013 – December, 11, 2014

Inclusion criteria:

- to be at least 18 years of age

- Eastern Cooperative Oncology Group (ECOG) performance-status score of 0 to 2 (on a 5-point scale, with higher numbers indicating greater disability)

- newly diagnosed (≤3 months before randomization)

- pathologically confirmed prostate cancer without neuroendocrine differentiation or small-cell histologic features

- high-risk, metastatic, castration-sensitive prostate cancer (mHSCP) as documented by a positive bone scan or metastatic lesions at the time of diagnosis on computed tomography (CT) or magnetic resonance imaging (MRI) (Response Evaluation Criteria in Solid Tumors (RECIST), version 1.1)

- have at least two of the three following high-risk factors (poor prognosis):

- a Gleason score of 8 or more (on a scale of 2 to 10, with higher scores indicating more aggressive disease),

- at least three bone lesions, and

- the presence of measurable visceral metastasis

Exclusion criteria:

- received previous chemotherapy, radiation therapy, or surgery for metastatic prostate cancer, with the exception of 3 months or less of androgen-deprivation therapy with luteinizing hormone–releasing hormone analogues or orchiectomy with or without concurrent first-generation androgen-receptor antagonists before baseline or one course of palliative radiation or surgical therapy to treat symptoms associated with metastatic disease

Intervention

androgen-deprivation therapy + abiraterone and prednisone

Control

androgen-deprivation therapy and dual placebos

Outcome(s)

primary: overall survival (OS), radiographic progression-free survival (rPFS)

secondary:

- time to the next “skeletal-related event” (described here as a “symptomatic skeletal event,” which was defined as a clinical or pathological fracture, spinal-cord compression, palliative radiation to bone, or surgery involving bone),

- time to progression with respect to prostate-specific antigen (PSA) level on the basis of Prostate Cancer Working Group 2 criteria,

- time to the next therapy for prostate cancer,

- time to initiation of chemotherapy,  

- time to pain progression

Results

ITT n=1199 (n(I)=597 vs. n(C)=602); at the final analysis, 72 patients had crossed over  

mean age (standard deviation) (I) 67.3 (8.5) vs. (C) 66.8 (8.7)

Gleason score at initial diagnosis ≥8: (I) n=584 (98%) vs. (C) n=586 (97%)

bone metastasis at screening ≥3: (I) n= 586 (98%) vs. (C) n=585 (97%)

- the interim analysis resulted in a recommendation on January 12, 2017, that the trial be unblinded to allow crossover among the patients in the placebo group to receive abiraterone

- protocol amendment (Feb 15, 2017) – open-label extension phase of the study (up to 18 months), patients who crossed over from placebo had not had cancer progression and consented to receive active treatment after unmasking

outcome

LATITUDE 1. interim analysis     I vs. C (cut-off 31.10.2016)

LATITUDE final analysis I vs. C (cut-off 15.08.2018)

death

n=406 (169 (28%) vs. 237 (39%))

HR 0.62 (0.51-0.76) P<0.001

n=618 (275 (46%) vs. 343 (57%))

median follow-up

30.4 months (IQR 18.4-32.2)

51.8 months (IQR 47.2-57.0)

median treatment duration

24 months vs. 14 months

25.8 (IQR 12.3-49.0) months vs. 14.4 (IQR 7.3-25.8) months vs.

cr-ov 11.9 (IQR 9.2-12.9) months

overall rate of survival at 3 years

66% vs. 49%

 

OS (median, 95% CI)

 

53.3 (48.2-NR) months vs.

36.5 (33.5-40.0) months

HR 0.66 (0.56-0.78) P<0,0001

rPFS

33.0 months vs. 14.8 months

239 events vs. 354 events

HR 0.47 (0.39-0.55) P<0.001

was not reanalysed after crossover (crossover was not considered as an event)

AE n (%)

grade 3-4

any serious

due to dose: discontinuation:

led to death:

558 (93) vs. 557 (93)*

374 (63) vs. 287 (48)

165 (28) vs. 146 (24)

32% vs. 17%

73 (12) vs. 61 (10)

28 (5) vs. 24 (4)

569 (95) vs. 561 (93), cr-ov 44 (71)**

403 (68) vs. 299 (50), cr-ov 14 (19)

192 (32) vs. 151 (25), cr-ov 4 (6)

209 (35) vs. 108 (18), cr-ov 7 (10)

93 (16) vs. 63 (10), cr-ov 3 (4)

38 (6) vs. 25 (4), cr-ov 2 (3)

secondary:

median time to pain progress. (months)

NR vs. 16.6

HR 0.70 (0.58–0.83) P<0.001

47.4 (33.2-NR) vs. 16.6 (11.1-24.0)

HR 0.72 (0.61-0.86) P=0.00024

n=245 (42%) vs. n=292 (49%)

median time to PSA progress. (months)

33.2 months vs. 7.4 months

HR 0.30 (0.26–0.35) P<0.001

33.3 (29.4-46.1) vs. 7.4 (7.2-9.2)

HR 0.31 (0.27-0.36) P <0.0001

n=273 (46%) vs. n=448 (74%)

median time to next symptomatic skeletal event

NR vs. NR

HR 0.70 (0.54–0.92) P=0.009

NR vs. NR

HR 0.75 (0.60-0.95) P=0.0181

n=132 (22%) vs. n=150 (25%)

median time to chemotherapy (months)

NR vs. 38.9

HR 0.44 (0.35–0.56) P<0.001

NR (62.6-NR) vs. 57.6 (38.2-NR)

HR 0.51 (0.41-0.63) P<0.0001

n=150 (25% vs. n=218 (36%)

median time to subsequent prostate cancer therapy

NR vs. 21.6

HR 0.42 (0.35–0.50) P<0.001

54.9 (45.4-NR) vs. 21.2 (18.6-23.5)

HR 0.45 (0.38-0.53) P<0.0001

n=248 (42%) vs. n=355 (59%)

AE=adverse event, CI=confidence interval, cr-ov=cross-over, HR: hazard ratio (95% CI), IQR=interquartile range), NR=not reached, OS=overall survival, progress.=progression, PSA=prostate-specific antigen, rPFS=radiographic progression-free survival

*most common: hypertension (219 (37) vs. 133 (22)), hypokalaemia (122(20) vs. 22(4)), back pain (110 (18) vs. 123 (20), bone pain (74 (12) vs. 88 (15)) (see also Table 2 within the publication)

**most common: hypertension (I) 125 (21) vs. (C) 60 (10), cr-ov 3 (4), back pain (I) 123 (21) vs. (C) 128 (21), cr-ov 5 (7), hypokalaemia (I) 70 (12) vs. (C) 10 (2), cr-ov 2 (3)

Comments

- patients were stratified according to the presence or absence of measurable visceral disease and ECOG performance-status score (0 or 1 vs. 2)

- potential bias favoring the abiraterone group in the assessment of radiographic progression by investigators was audited with the use of the method of the Pharmaceutical Research and Manufacturers Association in a sample of 202 randomly selected patients with blinded central review

- patients with life-extending subsequent therapy for PCa (ITT): (I) n=176 (30%) vs. (C) n=345 (57%), most common: docetaxel (see Table 2 of Fizazi et al. 2019)

- the analysis was confounded by patients in the control group crossing over

 

7.3 EvT 7.18

Artikel (Autor, Jahr, Journal)

Studientyp

Anzahl der Patienten

Patientenmerkmale

Intervention

Vergleichs-Intervention

Nachverfolgung

Outcomes

Ergebnisse

Bemerkungen

Evidenz-Niveau (SIGN)

Iversen 2006

RCT

1.218

cT1-4 Nx M0; 59,5% T1/2, 40,4% T3/4; „standard care“: 12,7% RPE, 6,4% RT/Brachy, 80,1% WW

Standard care + HT (Bicalutamide) (607 Pat)

Standard care ohne HT (611 Pat)

Median 7,1 J

HR für Progression (PFS) und Tod (OS), NW

HR für PFS HT vs keine HT:

T1-4: 0,65 (signifikant)

T1-2: 0,85 (n.s.)

T3-4: 0,47 (signifikant)

HR für OS HT vs. keine HT:

T1-4: 0,91 (n.s.)

T1-2: 1,23 (n.s.)

T3-4: 0,65 (signifikant)

HR für OS HT vs. keine HT in WW-Subgruppe:

T1-2: 1,18 (n.s.)

T3-4: 0,67 (signifikant)

NW Bicalutamide: Brustschmerz (63,6%), Gynäkomastie (58,5%), Impotenz (17,4%), Libidoverminderung (3,8%)

 

für viele Patienten wurde HT „open label“ unverblindet eingesetzt

1(+)

Studer 2006

RCT

985

cT0-4 N0-2 M0, lokale Therapie abgelehnt oder technisch nicht möglich

Sofortige AD

 (Buserelin oder Orchiektomie) n=493

verzögerte AD (Buserelin oder Orchiektomie) n=492

7,8 J

OS, prostate-cancer mortality, symptom-free survival

OS significant höher für sofortige HT, wobei PCa-Mortalität und symptomfreies Überleben nicht unterschiedlich waren; OS-Vorteil durch weniger nicht-PCa-assoziierte Todesfälle in der sofort therapierten Gruppe

Methodisch gute Studie; Ergebnisse in Meta-analyse von Boustead enthalten

1++

MRC 1997

RCT

938

cT2-4 „too advanced for curative treatment“ 500 M0 (54%), 173 Mx (19%), 261 M1 (28%)

Sofortige HT (Orchiektomie oder LHRHa) (n=469);

M0 n=256)

Verzögerte HT (n=465);

M0 n=244

?

TTDDP, TTD overall, TTD from prostate cancer

für M0-Patienten:

signifikanter Unterschied in TTDDP: höher für sofortige HT

signifikanter Unterschied in TTD overall und TTD prostate cancer: höher für sofortige HT

für alle Patienten (M0+M1):

signifikanter Unterschied TTD overall und TTD prostate cancer: höher für sofortige HT

wenn Mx- und M1-Patienten separat analysiert werden, ergibt sich kein signifikanter Unterschied

Spärliche Randomisationsangaben, TURP und Bestrahlung ebenfalls eingeschlossen, Vermisch­ung cT2 und cT3/4; medi­anes Follow-up nicht an­gegeben; Er­gebnisse in Metaanalyse von Boustead enthalten

1(-)

 

Autoren, Jahr

Studientyp

Untersuchte Studien/
Materialien

Welche Behandlungen wurden geprüft

Welche Outcomes?

Befunde in Bezug auf Therapie-Wirkungen und Überleben

Bemerkungen

LoE

Literaturbelege

Boustead 2007

Syst. Review/ Meta-analyse

7 RCTs aus systematischer Suche in CENTRAL, BIOS, DARE, EMBASE, MEDLINE bis Juli 2006

Sofortige vs verzögerte HT bei „Standard treatment“ (RPE, RT, WW) bei lokal fortgeschrittenem nichtmetastasiertem PCa

Sterblichkeit (overall mortality), PCSM, OP, LP, DP

Subgruppen-Analyse für WW-Patienten: RR für Sterblichkeit geringer bei sofortiger vs. verzögerter HT: 0,88 (95%CI 0,81-0,96)

Keine Subgruppen-Analysen für übrige Outcome-Parameter

Subgruppen-Analyse nicht graphisch dargestellt; MRC- und Studer-Studie (s.u.) mit eingeschlossen

1+

23-30

Wilt 2001

Syst. Review/ Meta-analyse

4 RCTs (2.167 Pat) aus systemat. Suche in MEDLINE, EMBASE, CancerLit, Cochrane u.a. von 1966 bis August 2001

Frühe vs. verzögerte AS bei lokal fortgeschrittenen und metastasierten PCas

OS, PCSS, PFS

10-Jahres-OS signifikant besser für frühe Therapie, 1-, 2-, 5-Jahres OS etwas besser für frühe Therapie, aber nicht signifikant

PCSS etwas besser für frühe Therapie, aber nicht signifikant

PFS in allen Studien besser für frühe Therapie, aber kein Daten-Pooling möglich auf Grund variierender Definitionen von PFS

Sowohl Patienten mit lokal fortgeschrittenem als auch metastasiertem PCa, keine Subgruppen-Analysen

1++

Messing 1999, MRC 1997, Jordan 1977, Byar 1973

                     

 

 

 

Agarwal_LancetOncol_2019_Chi_NEJM_2019

Chi KN, Agarwal N, Bjartell A. Apalutamide for Metastatic, Castration-Sensitive Prostate Cancer. N Engl J Med. 2019 Jul 4;381(1):13-24. doi: 10.1056/NEJMoa1903307.

Agarwal N, McQuarrie K, Bjartell A. Health-related quality of life after apalutamide treatment in patients with metastatic castration-sensitive prostate cancer (TITAN): a randomised, placebo-controlled, phase 3 study. Lancet Oncol. 2019 Nov;20(11):1518-1530. doi: 10.1016/S1470-2045(19)30620-5.

Link

https://pubmed.ncbi.nlm.nih.gov/31150574/ 

https://pubmed.ncbi.nlm.nih.gov/31578173/

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: low

Attrition bias

lost to follow-up: low

ITT-analysis: unclear

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the Trial was designed by the sponsor

one patient of the intervention group (I) did not receive study drug; 45 patients across the two groups withdrew consent for the trial intervention (patients were followed for survival and secondary end points, data were not missing); 39 patients were either lost to

follow-up or withdrew from all further data collection (not shown in the figure S1)

Design

  randomized, double-blind placebo-controlled, phase 3 trial

  TITAN, NCT02489318

- Cut-off: November 23, 2018 (1. interim analysis)

Population

metastatic, castrationsensitive prostate cancer (mHSPC)

260 sites in 23 countries

randomization between December, 15 2015 – July 25, 2017

Inclusion criteria:

- documented adenocarcinoma of the prostate

- distant metastatic disease (at least one lesion on bone scanning), with or without visceral or lymph-node involvement

- Eastern Cooperative Oncology Group performance-status score of 0 or 1 (on a scale of 0 to 5, with higher scores reflecting greater disability)

- castration sensitive (i.e., not receiving ADT at the time of disease progression)

- previous treatment for prostate cancer was limited to previous

- docetaxel use (for a maximum of six cycles, with no evidence of progression during treatment or before randomization),

- ADT for no more than 6 months for metastatic, castration-sensitive prostate cancer or no more than 3 years for localized prostate cancer,

- one course of radiation or surgical therapy for symptoms associated with metastatic disease, and

- other localized treatments (e.g., radiation therapy or prostatectomy) completed at least 1 year before randomization

- patients receiving a gonadotropin-releasing hormone agonist within 28 days before randomization were required to take a first-generation antiandrogen (i.e., bicalutamide, flutamide, or nilutamide) for 14 or more days before randomization

- antiandrogen therapy must have been discontinued before randomization

Exclusion criteria:

- severe angina, myocardial infarction, congestive heart failure, arterial or venous thromboembolic events,

- a history of or predisposition to seizure,

- recent ventricular arrhythmias

Intervention

androgen-deprivation therapy (ADT) + apalutamide

Control

ADT + placebo

Outcome(s)

primary: radiographic progression-free survival (rPFS)#, overall survival (OS)

secondary:

- time to cytotoxic chemotherapy,

- time to pain progression (BPI-SF; worst pain [item 3]; scores range from 0 to 10),

- time to chronic opioid use,

- time to skeletal-related event

# defined as the time from randomization to first imaging based documentation of progressive disease or death, whichever occurred first

patient related outcomes:

- Brief Pain Inventory-Short Form (BPI-SF),

- Brief Fatigue Inventory (BFI),

- Functional Assessment of Cancer Therapy-Prostate (FACT-P) and the EQ-5D-5L (will be collected throughout the study as well as during the Follow-up Phase (up to 12 months after treatment discontinuation)

Results

n=1052, n(I)=525 vs. n(C)=527

median age (I) 69 (range 45-94) vs. (C) 68 (range 43-90)

Gleason score at initial diagnosis >7: n(I)=351 (66.9%) vs. n(C)=358 (67.9%)

1. Interim analysis: (cut-off 23.11.2018): 66.2% of the patients in the intervention group and 46.1% in the placebo group were receiving the trial intervention

- 45 patients across the two groups withdrew consent for the trial intervention (followed for survival and secondary end points)

- 39 patients were either lost to follow-up or withdrew from all further data collection

outcome

TITAN 1. interim analysis    

I vs. C (cut-off 23.11.2018)

death

n=200, n(I)=83 vs. n(C)=117

median follow-up

22.7 months (IQR 19.4-25.8)

median treatment duration

20.5 months (IQR 14.9-24.7) vs.

18.3 months (IQR 19.3-22.9)

rPFS

median months

at 24 months, % (95% CI)

n=365 (n(I)=134 vs. n(C)=231)

NE vs. 22.1 (18.5-32.9)

68.2 (62.9-72.9) vs. 47.5 (42.1-52.8)

HR 0.48 (0.39-0.60) P<0.001

OS (median, 95% CI)

at 24 months, % (95% CI)

NE vs. NE

82.4 (78.4-85.8) vs. 73.5 (68.7-77.8)

HR 0.67 (0.51-0.89) P=0.005

AE n (%)

grade 3-4

any serious

discontinuation:

led to death:

507 (96.8) vs. 506 (96.6) (*)

221 (42.2) vs. 215 (40.8)

104 (19.8) vs. 107 (20.3)

42 (8.0) vs. 28 (5.3)

10 (1.9) vs. 16 (3.0)

secondary:

median time to chemotherapy (months)

NE vs. NE

HR 0.39 (0.27-0.56) P<0.001

median time to pain progress. (months)

NE vs. NE

HR 0.83 (0.65-1.05) P=0.12

median time to chronic opioid use (months)

NE vs. NE

HR 0.77 (0.54-1.11) (-)

median time to skeletal event

NE vs. NE

HR 0.80 (0.56-1.15) (-)

patient-related outcomes

BPI-SF, pain, median (IQR)

 

time to pain progression (see secondary outcome)

baseline 1.14 (0-3.17) vs. 1.00 (0-2.86)

n=503 (96%) vs. 513 (97%)

NR vs. NR

HR 0.83 (0.65-1.05) P=0.12

BFI, fatigue, median (IQR)

 

time to worst fatigue

baseline 1.29 (0-3.29) vs. 1.43 (0.14-3.14)

n=503 (96%) vs. 513 (97%)

NR vs. NR

HR 1.09 (0.88-1.35) P=0.44

FACT-P, median (IQR)

time to deterioration, median (QR)

EQ-5D-5L

baseline 113 (98-128) vs. 113 (99-127)

8.87 months (1.87-NR) vs. 9.23 months (2.79-24.77)

HR 1.02 (0.85-1.22) P=0,85

(-)

CI=confidence interval, HR=Hazard ratio (95 % CI), IQR=interquartile range, NE could not be estimated, NR=not reached, OS=overall survival, rPFS=radiographic progression-free survival

* most common I vs. C (n (%)): rash (142 (27.1) vs. 45 (8.5)), hot flush (119 (22.7) vs. 86 (16.3)), fatigue (103 (19.7) vs. 88 (16.7)), hypertension (93 (17.7) vs. 82 (15.6), back pain (91 (17.4) vs. 102 (19.4)), arthralgia (91 (17.4) vs. 78 (14.8))

Comments

-patients were stratified according to Gleason score at diagnosis (≤7 vs. >7, on a scale of 2 to 10, with higher scores indicating higher-grade cancer that may be more aggressive), geographic region (North America and European Union vs. all other countries), and previous treatment with docetaxel (yes vs. no)

 

Armstrong_JClinOncol_2019

 

Armstrong AJ, Szmulewitz RZ, Petrylak DP. ARCHES: A Randomized, Phase III Study of Androgen Deprivation Therapy With Enzalutamide or Placebo in Men With Metastatic Hormone-Sensitive Prostate Cancer. J Clin Oncol. 2019 Nov 10;37(32):2974-2986. doi: 10.1200/JCO.19.00799.

Link

https://pubmed.ncbi.nlm.nih.gov/31329516/  

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: low

Attrition bias

lost to follow-up: low

ITT-analysis: low

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the trial was designed by the sponsor and the analyses were performed by the study sponsor

Design

randomized, double-blind placebo-controlled, phase 3 trial

ARCHES, NCT02677896

Cut-off: October 14, 2018 (1. interim analysis)

Population

patients with metastatic hormone-sensitive prostate cancer (mHSPC)

202 centers in North and Latin America, Europe, and Asia

randomization between March 21, 2016 an January 12, 2018

Inclusion criteria:

- adult (defined according to local regulation)

- males with pathologically confirmed prostate adenocarcinoma

- without neuroendocrine differentiation, signet-cell, or small-cell features

- Eastern Cooperative Oncology Group performance status score of 0 or 1

- hormone-sensitive metastatic disease

- de novo or after recurrence after prior local therapy

- prior ADT and up to six cycles of prior docetaxel chemotherapy were permitted

Exclusion criteria:

- disease progression prior to randomization while receiving ADT and/or docetaxel

- previous pharmacotherapy, radiation therapy, or surgery for metastatic prostate cancer, with the exception of:

- ≤ 3 months, or ≤ 6 months (docetaxel), of ADT with LHRH analogs or orchiectomy, with or without concurrent antiandrogens, with no radiographic evidence of disease progression or rising PSA levels

- one course of palliative radiation or surgical therapy to treat symptoms

- 6 cycles of docetaxel therapy completed within 2 months of day 1 and no evidence of disease progression during or after completion

- prior ADT given for < 39 months in duration and > 9 months before randomization as neoadjuvant/adjuvant therapy

Intervention

enzalutamide plus ADT

Control

placebo plus ADT

Outcome(s)

primary: radiographic progression-free survival (rPFS)#

secondary:

- time to PSA progression

- time to initiation of new antineoplastic therapy (including cytotoxic and hormone therapies)

- PSA undetectable rate

- objective response rate

- time to deterioration in urinary symptoms

- overall survival (OS)

- time to first symptomatic skeletal event

- time to castration resistance

- patient-reported outcomes (PROs)

- time to deterioration of quality of life (QoL)

- ime to pain progression

# defined as the time from randomization to the first objective evidence of radiographic disease progression, as assessed by independent central review or death (defined as death from any cause within 24 weeks from study drug discontinuation), whichever occurred first

Results

n=1150 (n(I)=574 vs. n(C)=576)

median age (I) 70.0 (range 46-92) vs. 70.0 (range 42-92)

Gleason score ≥8 n(I)=386 (67.2%) vs. n(C)=373 (64.8%)

- n(I)=25 (4.4%) vs. n(C)=30 (5.2%) patients withdrawal

outcome

ARCHES 1. interim analysis    

I vs. C (cut-off 14.10.2018)

death

n=84 (n(I)=39 vs. n(C)=45)

median follow-up

14.4 months

rPFS

median months (95% CI)

n=292 (n(I)=91 (15.9%) vs. n(C)=201 (34.9%))

NR (NR-NR) vs. 19.0 (16.6-22.2)

HR 0.39 (0.30-0.50) P<0.001

AE n (%)*

grade ≥3

any serious

discontinuation:

led to death:

487 (85.1) vs. 493 (85.9)

139 (24.3) vs. 147 (25.6)

104 (18.2) vs. 112 (19.5)

41 (7.2) vs. 30 (5.2)

14 (2.4) vs. 10 (1.7)

secondary:

time to PSA progression, median months

NR vs. NR

HR 0.19 (0.13-0.26) P<0.001

median time to antineoplastic therapy (months)

30.2 vs. NR

HR 0.28 (0.20-0.40) P<0.001

PSA undetectable rate

348 (68.1) vs. 89 (17.6)

P<0.001

objective response rate

147 (83.1) vs. 116 (63.7)

P<0.001

time to deterioration in urinary symptoms

NR vs. 16.8

HR 0.88 (0.72-1.08) P=0.2162

OS, median months

NR vs. NR

HR 0.81 (0.53-1.25) P=0.3361

time to skeletal event, median months

NR vs. NR

HR 0.52 (0.33-0.80) P=0.0026

time to castration resistance, median months

NR vs. 13.8

HR 0.28 (0.22-0.36) P<0.001

time to deterioration of quality of life (QoL), median months

11.3 vs. 11.1

HR 0.96 (0.81-1.14) P=0.6548

time to pain progression, median months

8.3 vs. 8.3

HR 0.92 (0.78-1.07) P=0.2715

CI=confidence interval, HR=Hazard ratio (95 % CI), IQR=interquartile rande, NE could not be estimated, NR=not reached, OS=overall survival, rPFS=radiographic progression-free survival

* most common I vs. C (n (%)): hot flash 155 (27.1) vs. 128 (22.3), fatigue 112 (19.6) vs. 88 (15.3), Arthralgia 70 (12.2) vs. 61 (10.6), back pain 43 (7.5) vs. 62 (10.8)

also relevant: hypertension 49 (8.6) vs. 36 (6.3), cognitive impairment 26 (4.5) vs. 12 (2.1), fractures 37 (6.5) vs. 24 (4.2), musculoskeletal events 151 (26.4) vs. 159 (27.7)

Comments

- randomization was stratified (disease volume, prior docetaxel use)

 

Gravis_LancetOncol_2013

Zitat

Studiencharakteristika

Studienergebnisse

Methodische Bewertung

LoE

Gravis G. Androgen-deprivation therapy alone or with docetaxel in non-castrate metastatic prostate cancer (GETUG-AFU 15): a randomised, open-label, phase 3 trial. Lancet Oncol 2013;14(2):149-58.

• Studientyp: RCT, multicenter open label,
• ADT + Doce (75mg/m2 21d/ 9 cycles) vs. ADT alone
•median follow up 50 mo
• nicht verblindet, unterschiedliche Monitorings,
• Population: (n=385; 1:1 Randomisierung ), m >18J; N+ oder M+ (radiologisch bestätigt); Karnowsky >70%; adäquate hämatolog., Leber- & Nierenfunktion
• primärer Endpunkt OS, sekundäre Endpunkte radiographic progression, biochemical progression;
• 2004 - 2008 (recruitment),
• Ort: Frankreich & Belgien

• Baseline-Pat.charakteristika ausgewogen Age 63,5 Y; ≥ 95% ECOG 0;
o Pat. in intervention group younger (63y. vs. 64 y.), mehr Gleason 8-10 in control group (59% vs. 55%), beides nicht signifikant
• benefit:
o OS not  significantly different (176 deaths =46% per group, HR 1,01, 95%CI 0,75 - 1,36, p=0.955)
o bPFS significantly longer in intervention group (22.9 mo vs. 12.9 mo, HR 0.67, 95%CI 0.54 - 0.84, p=<0,001)
o rPFS signifcantly longer in intervention group (22.9 mo vs. 15.3 mo; HR 0.69,95%CI 0.55 - 0.87, p=0.002
• Adverse events: grade 3/4: 32% neutropenia; 7% febrile neutropenia; 2% infections with neutropenia; abnormal liver function test 2%; 4 treatment related deaths in intervention Group (2 due to neutropenia)
- after amendment: granulocyte colony stimulating factor (5
μg/kg/day from day 5-10 after each Doce treatment) rate fell to 15% neutropenia and 6% febrile neutropenia)
• QoL Scores significantly lower in intervention group (p=0,005);  no differences in mean global and functional scores

• Randomisierung
• Allocation concealment nicht beschrieben
• nicht verblindet, unterschiedliche Monitorings (Intervention group engmaschigere Kontrolle)
• Patientencharakteristika beschrieben; körperlich sehr fitte Patienten (>95% ECOG 0); Patienten im Interventionsarm etwas jünger
• Drop-out beschrieben
• Statistische Methoden beschrieben
• OS beschrieben (kein Benefit); sekundäre outcomes rPFS und bPFS nicht patientenrelevant;
• Intention-to-treat-Analyse
• Finanzierung durch frz. Ministerium, Unterstützung durch Sanofi-Aventis; Astra-Zeneca-Amgen

relativ kleine Patientenzahl, aber langes follow up und hohe Ereignisrate für primären Endpunkt (46% deaths per intervention group)
Kommentar: Design birgt Risk of bias für Effekt in der Interventionsgruppe, da dieser trotz Verzerrungsrisiko nicht signifikant, upgrading der Qualität (analog GRADE)

1+

 

 

James_Lancet_2016

Zitat

Studiencharakteristika

Studienergebnisse

Methodische Bewertung

LoE

James ND. Addition of docetaxel, zoledronic acid, or both to first-line long-term hormone therapy in prostate cancer (STAMPEDE): survival results from an adaptive, multiarm, multistage, platform randomised controlled trial. Lancet 2016;387(10024):1163-77.

• RCT, vierarmig, multizentrisch
• ADT vs. ADT+Zoledronsäure vs. ADT+Docetaxel vs. ADT+Zoledronsäure+Docetaxel; Randomisation 2:1:1:1 (Regime Docetaxel: 6 3-wöchige Zyklen 75mg/m2)
• Follow-up median 43 mo
• keine Verblindung
• Population: n=2962, PCa M+, N+ oder M0N0 high risk; "fit for chemotherapy", keine kardiovaskulären Ereignisse in der Vorgeschichte
• Primary Endpoints: OS and failure-free survival
  secondary: prostate-cancer-speciic survival and adverse events
• Studienzeitraum: 2005 - 2013 (recruitment)
• UK & Schweiz

• Baseline-Patientencharakteristika ausgewogen zwischen den Armen:; 62% N+M+; age median 65; 75% WHO performance status 0

• Effektivität
o OS: kein Effekt für Zoledronsäure+ADT (HR 0,94; 95%CI 0,79 - 1,11);
          Effekt für Docetaxel+ADT (Überleben median 81 mo vs. 71mo nur ADT;
          HR 0,78; 95%CI 0,66 - 0,93, p=0,006); (175 Todesfälle, davon 143 PCa);
          Effekt für Doce+Zol.+ADT (Überleben median 76mo vs. 71 mo nur ADT;
          HR 0,82; 95%CI 0,69-0,97; p=0,022); (187 Todesfälle, davon 150 PCa)
o failure-free-survival: kein Effekt für Zoledronsäure + ADT
          Effekt für Doce+ADT: 315 Ereignisse; HR 0,61; 95%CI 0,54-0,70
          Effekt für Doce+Zoledr.+ADT: 316 Ereignisse; HR 0,62; 95%CI 0,54- 
          0,70
o pre-planned analysis OS for metastatic disease only:
      Doce+ADT: 60 mo vs. 45 mo ADT only; HR 0,76; 95%CI 0,62-0,92;
      p=0,005;
      Doce+Zole+ADT: 55mo vs.45 ADT only; HR 0,79; 95%CI 0,66-0,96;
      p=0,015
 
• Adverse Events Grad 3/4/5:
      Doce+ ADT 52% vs. 15% ADT allein
      Doce+Zoledr. + ADT 52%  vs. 15% ADT allein

      darunter febrile Neutropenie: 15%
      Neutropenie: 12%

• Randomisierung
• Allocation concealment beschrieben
• keine Verblindung, aber gleiches Monitoring für alle Gruppen
• Patientencharakteristika ausbalanciert zwischen den Gruppen, aber nur sehr fitte Patienten eingeschlossen (WHO performance status 0 > 75%?
• Drop-out beschrieben, nicht relevant
• Statistische Methoden beschrieben
•  Effekt für OS als primären, pat.rel. Endpunkt
• ITT-Analyse ab Randomisierung?
• Interessenkonflikte angegeben und massiv vorhanden
• Finanzierung durch Medical Research Council UK, keine finanzielle Pharmabeteiligung

große Patientenzahl (~ 600 pro Arm, 1.200 für control), mittleres follow-up (31/2 Jahre), mittlere Ereignisrate

1+

 

 

7.3 EvT 7.21

Quelle

Fazit/Empfehlung

Empfehlungsgrad

Evidenzgraduierung

Literaturbelege

ASCO 2004 und 2007

What are the standard initial treatment options?

Bilateral orchiectomy or medical castration with luteinizing hormone–releasing hormone (LHRH) agonists are the recom.mended initial treatments for metastatic prostate cancer. A full discussion between practitioner and patient should occur to determine which is best for the patient. Diethylstibestrol should not be considered as a standard first-line treatment option and currently is no longer commercially available in North America.

 

 

Robinson 1995, Byar 1973, Byar 1988, VACURG 1967

ASCO 2004 und 2007

Are Antiandrogens As Effective As Other Castration Therapies?

Nonsteroidal antiandrogen (NSAA) monotherapy may be discussed as an alternative, but steroidal antiandrogen (AA) monotherapy should not be offered.

 

 

Seidenfeld 2000

ASCO 2004 und 2007

Is Combined Androgen Blockade Better Than Castration Alone?

Combined androgen blockade (CAB) should be considered.

 

 

 

Akaza 2004, Klotz 2004, Samson 2002, Schmitt 2001, PCTCG 2000, Eisenberger 1998, Bayoumi 2000

ASCO 2004 und 2007

Does Early ADT Improve Outcomes Over Deferred Therapy?

For patients with metastatic or progressive prostate cancer, there is a moderate decrease (17%) in relative risk (RR) for prostate cancer–specific mortality, a moderate increase (15%) in RRfor non–prostate cancer–specific mortality, and no overall survival advantage for immediate institution ofADTversus waiting until symptom onset for patients. Therefore, the Panel cannot make a strong recommendation for the early use of ADT. PSA kinetics and other metrics allow the identification of populations at high risk for prostate cancer–specific and overall mortality. Further studies must be completed to assess whether patients with adverse prognostic factors gain a survival advantage from immediate ADT. If a patient decides to wait until symptoms for ADT, he should have regular visits for monitoring. For patients with recurrent disease, clinical trials should be considered if available.

 

 

Wilt 2003, McLeod 2005, Schroder 2004, Studer 2004, Kirk 2004, Studer 2006, Messing 1999, Byar 1988, Bayoumi 2000, Byar 1973, Wirth 2001, Borkowski 2003, Jordan 1977, MRC 2000

ASCO 2004 und 2007

What Is the Role of Intermittent Androgen Blockade?

2007 recommendation. Currently, data are insufficient to support the use of intermittent androgen blockade outside of clinical trials.

 

 

de Leval 2002

 

Artikel (Autor, Jahr, Journal)

Studientyp

Anzahl der Patienten

Patientenmerkmale

Intervention

Vergleichs-Intervention

Nachverfolgung

Outcomes

Ergebnisse

Bemerkungen

Evidenz-Niveau (SIGN)

Iversen 2006

RCT

1.218

cT1-4 Nx M0; 59,5% T1/2, 40,4% T3/4; „standard care“: 12,7% RPE, 6,4% RT/Brachy, 80,1% WW

Standard care + HT (Bicalutamide) (607 Pat)

Standard care ohne HT (611 Pat)

Median 7,1 J

HR für Progression (PFS) und Tod (OS), NW

HR für PFS HT vs keine HT:

T1-4: 0,65 (signifikant)

T1-2: 0,85 (n.s.)

T3-4: 0,47 (signifikant)

HR für OS HT vs. keine HT:

T1-4: 0,91 (n.s.)

T1-2: 1,23 (n.s.)

T3-4: 0,65 (signifikant)

HR für OS HT vs. keine HT in WW-Subgruppe:

T1-2: 1,18 (n.s.)

T3-4: 0,67 (signifikant)

NW Bicalutamide: Brustschmerz (63,6%), Gynäkomastie (58,5%), Impotenz (17,4%), Libidoverminderung (3,8%)

 

für viele Patienten wurde HT „open label“ unverblindet eingesetzt

1(+)

Studer 2006

RCT

985

cT0-4 N0-2 M0, lokale Therapie abgelehnt oder technisch nicht möglich

Sofortige AD

 (Buserelin oder Orchiektomie) n=493

verzögerte AD (Buserelin oder Orchiektomie) n=492

7,8 J

OS, prostate-cancer mortality, symptom-free survival

OS significant höher für sofortige HT, wobei PCa-Mortalität und symptomfreies Überleben nicht unterschiedlich waren; OS-Vorteil durch weniger nicht-PCa-assoziierte Todesfälle in der sofort therapierten Gruppe

Methodisch gute Studie; Ergebnisse in Meta-analyse von Boustead enthalten

1++

 

Autoren, Jahr

Studientyp

untersuchte Studien/ Materialien

Welche Behandlungen wurden geprüft?

Welche Outcomes?

Ergebnisse

 

Bemerkungen

LoE

Literaturbelege

Wilt 2001

Syst. Review/ Meta-analyse

4 RCTs (2.167 Pat) aus systemat. Suche in MEDLINE, EMBASE, CancerLit, Cochrane u.a. von 1966 bis August 2001

Frühe vs. verzögerte AS bei lokal fortgeschrittenen und metasta­sierten PCas

OS, PCSS, PFS

10-Jahres-OS signifikant besser für frühe Therapie, 1-, 2-, 5-Jahres OS etwas besser für frühe Therapie, aber nicht signifikant

PCSS etwas besser für frühe Therapie, aber nicht signifikant

PFS in allen Studien besser für frühe Therapie, aber kein Daten-Pooling möglich auf Grund variierender Definitionen von PFS

Sowohl Patienten mit lokal fortgeschrittenem als auch metastasiertem PCa, keine Subgruppen-Analysen

1++

Messing 1999, MRC 1997, Jordan 1977, Byar 1973

 

 

7.3 EvT 7.22

Quelle

Fazit/Empfehlung

Empfehlungsgrad

Evidenzgraduierung

Literaturbelege

ASCO 2004 und 2007

What are the standard initial treatment options?

Bilateral orchiectomy or medical castration with luteinizing hormone–releasing hormone (LHRH) agonists are the recom.mended initial treatments for metastatic prostate cancer. A full discussion between practitioner and patient should occur to determine which is best for the patient. Diethylstibestrol should not be considered as a standard first-line treatment option and currently is no longer commercially available in North America.

 

 

Robinson 1995, Byar 1973, Byar 1988, VACURG 1967

ASCO 2004 und 2007

Are Antiandrogens As Effective As Other Castration Therapies?

Nonsteroidal antiandrogen (NSAA) monotherapy may be discussed as an alternative, but steroidal antiandrogen (AA) monotherapy should not be offered.

 

 

Seidenfeld 2000

ASCO 2004 und 2007

Is Combined Androgen Blockade Better Than Castration Alone?

Combined androgen blockade (CAB) should be considered.

 

 

 

Akaza 2004, Klotz 2004, Samson 2002, Schmitt 2001, PCTCG 2000, Eisenberger 1998, Bayoumi 2000

ASCO 2004 und 2007

Does Early ADT Improve Outcomes Over Deferred Therapy?

For patients with metastatic or progressive prostate cancer, there is a moderate decrease (17%) in relative risk (RR) for prostate cancer–specific mortality, a moderate increase (15%) in RRfor non–prostate cancer–specific mortality, and no overall survival advantage for immediate institution ofADTversus waiting until symptom onset for patients. Therefore, the Panel cannot make a strong recommendation for the early use of ADT. PSA kinetics and other metrics allow the identification of populations at high risk for prostate cancer–specific and overall mortality. Further studies must be completed to assess whether patients with adverse prognostic factors gain a survival advantage from immediate ADT. If a patient decides to wait until symptoms for ADT, he should have regular visits for monitoring. For patients with recurrent disease, clinical trials should be considered if available.

 

 

Wilt 2003, McLeod 2005, Schroder 2004, Studer 2004, Kirk 2004, Studer 2006, Messing 1999, Byar 1988, Bayoumi 2000, Byar 1973, Wirth 2001, Borkowski 2003, Jordan 1977, MRC 2000

ASCO 2004 und 2007

What Is the Role of Intermittent Androgen Blockade?

2007 recommendation. Currently, data are insufficient to support the use of intermittent androgen blockade outside of clinical trials.

 

 

de Leval 2002

EAU 2007

1. In advanced CaP, ADT delays progression, prevents potentially catastrophic complications and effectively palliates symptoms, but does not prolong survival (level of evidence: 1b)

2. In advanced CaP, all forms of castration as monotherapy (orchiectomy, LHRH and DES) have equivalent therapeutic efficacy (level of evidence: 1b)

3. Non-steroidal antiandrogen monotherapy (e.g. bicalutamide) is an effective alternative to castration in patients with locally advanced disease (level of evidence: 1b)

4. In advanced CaP, the addition of a non-steroidal antiandrogen to castration (CAB) results in a small advantage in OS over castration alone but is associated with increased adverse events, reduced QoL and high costs (level of evidence: 1a)

5. Intermittent and ‘minimal’ ADT should still be regarded as experimental therapies (level of evidence: 3)

6. In advanced CaP, immediate (given at diagnosis) androgen suppression significantly reduces disease progression and complication rate due to progression itself compared to deferred (delivered at symptomatic progression) androgen deprivation (level of evidence: 1b)

7. Bilateral orchiectomy may be the most cost-effective form of ADT, especially if initiated after    occurrence of symptoms from metastatic disease (level of evidence: 3).

 

-

 

NICE 2008

Healthcare professionals should offer bilateral orchidectomy to all men with metastatic prostate cancer as an alternative to continuous LHRHa therapy.

Combined androgen blockade is not recommended as a first-line treatment for men with metastatic prostate cancer.

For men with metastatic prostate cancer who are willing to accept the adverse impact on overall survival and gynaecomastia in the hope of retaining sexual function, antiandrogen monotherapy with bicalutamide (150 mg)1 is appropriate.Healthcare professionals should begin androgen withdrawal and stop bicalutamide treatment in men with metastatic prostate cancer who are taking bicalutamide monotherapy and who do not maintain satisfactory sexual function.

 

Intermittent androgen withdrawal may be offered to men with metastatic prostate cancer providing they are informed that there is no long-term evidence of its effectiveness.

 

-

-

Dutch Urological Association 2007

Hormone therapy

Hormone therapy is the treatment of choice for patients with metastatic prostate cancer. The preferred methods are bilateral orchiectomy, LHRH analogue, or parenteral oestrogen. Maximal androgen blockade is not recommended as first-line therapy.

Optimal timing of hormone therapy

The time to start hormone therapy is determined on an individual basis.

Optimal schedule of hormone therapy:

Intermittent hormone therapy for patients with metastatic prostate cancer is an experimental approach that is preferably given in the context of a clinical trial only.

 

-

 

 

Artikel (Autor, Jahr, Journal)

Studientyp

Anzahl der Patienten

Patientenmerkmale

Intervention

Vergleichs-Intervention

Nachverfolgung

Outcomes

Ergebnisse

Bemerkungen

Evidenz-Niveau (SIGN)

Iversen 2006

RCT

1.218

cT1-4 Nx M0; 59,5% T1/2, 40,4% T3/4; „standard care“: 12,7% RPE, 6,4% RT/Brachy, 80,1% WW

Standard care + HT (Bicalutamide) (607 Pat)

Standard care ohne HT (611 Pat)

Median 7,1 J

HR für Progression (PFS) und Tod (OS), NW

HR für PFS HT vs keine HT:

T1-4: 0,65 (signifikant)

T1-2: 0,85 (n.s.)

T3-4: 0,47 (signifikant)

HR für OS HT vs. keine HT:

T1-4: 0,91 (n.s.)

T1-2: 1,23 (n.s.)

T3-4: 0,65 (signifikant)

HR für OS HT vs. keine HT in WW-Subgruppe:

T1-2: 1,18 (n.s.)

T3-4: 0,67 (signifikant)

NW Bicalutamide: Brustschmerz (63,6%), Gynäkomastie (58,5%), Impotenz (17,4%), Libidoverminderung (3,8%)

 

für viele Patienten wurde HT „open label“ unverblindet eingesetzt

1(+)

Studer 2006

RCT

985

cT0-4 N0-2 M0, lokale Therapie abgelehnt oder technisch nicht möglich

Sofortige AD

 (Buserelin oder Orchiektomie) n=493

verzögerte AD (Buserelin oder Orchiektomie) n=492

7,8 J

OS, prostate-cancer mortality, symptom-free survival

OS significant höher für sofortige HT, wobei PCa-Mortalität und symptomfreies Überleben nicht unterschiedlich waren; OS-Vorteil durch weniger nicht-PCa-assoziierte Todesfälle in der sofort therapierten Gruppe

Methodisch gute Studie; Ergebnisse in Meta-analyse von Boustead enthalten

1++

MRC 1997

RCT

938

cT2-4 „too advanced for curative treatment“ 500 M0 (54%), 173 Mx (19%), 261 M1 (28%)

Sofortige HT (Orchiektomie oder LHRHa) (n=469);

M0 n=256)

Verzögerte HT (n=465);

M0 n=244

?

TTDDP, TTD overall, TTD from prostate cancer

für M0-Patienten:

signifikanter Unterschied in TTDDP: höher für sofortige HT

signifikanter Unterschied in TTD overall und TTD prostate cancer: höher für sofortige HT

für alle Patienten (M0+M1):

signifikanter Unterschied TTD overall und TTD prostate cancer: höher für sofortige HT

wenn Mx- und M1-Patienten separat analysiert werden, ergibt sich kein signifikanter Unterschied

Spärliche Randomisationsangaben, TURP und Bestrahlung ebenfalls eingeschlossen, Vermisch­ung cT2 und cT3/4; medi­anes Follow-up nicht an­gegeben; Er­gebnisse in Metaanalyse von Boustead enthalten

1(-)

 

Autoren, Jahr

Studientyp

untersuchte Studien/ Materialien

Welche Behandlungen wurden geprüft?

Welche Outcomes?

Ergebnisse

 

Bemerkungen

LoE

Literaturbelege

Wilt 2001

Syst. Review/ Meta-analyse

4 RCTs (2.167 Pat) aus systemat. Suche in MEDLINE, EMBASE, CancerLit, Cochrane u.a. von 1966 bis August 2001

Frühe vs. verzögerte AS bei lokal fortgeschrittenen und metasta­sierten PCas

OS, PCSS, PFS

10-Jahres-OS signifikant besser für frühe Therapie, 1-, 2-, 5-Jahres OS etwas besser für frühe Therapie, aber nicht signifikant

PCSS etwas besser für frühe Therapie, aber nicht signifikant

PFS in allen Studien besser für frühe Therapie, aber kein Daten-Pooling möglich auf Grund variierender Definitionen von PFS

Sowohl Patienten mit lokal fortgeschrittenem als auch metastasiertem PCa, keine Subgruppen-Analysen

1++

Messing 1999, MRC 1997, Jordan 1977, Byar 1973


Autoren, Jahr

Studientyp

Untersuchte Studien/
Materialien

Welche Behandlungen wurden geprüft

Welche Outcomes?

Befunde in Bezug auf Therapie-Wirkungen und Überleben

Bemerkungen

LoE

Literaturbelege

Boustead 2007

Syst. Review/ Meta-analyse

7 RCTs aus systematischer Suche in CENTRAL, BIOS, DARE, EMBASE, MEDLINE bis Juli 2006

Sofortige vs verzögerte HT bei „Standard treatment“ (RPE, RT, WW) bei lokal fortgeschrittenem nichtmetastasiertem PCa

Sterblichkeit (overall mortality), PCSM, OP, LP, DP

Subgruppen-Analyse für WW-Patienten: RR für Sterblichkeit geringer bei sofortiger vs. verzögerter HT: 0,88 (95%CI 0,81-0,96)

Keine Subgruppen-Analysen für übrige Outcome-Parameter

Subgruppen-Analyse nicht graphisch dargestellt; MRC- und Studer-Studie (s.u.) mit eingeschlossen

1+

23-30

PCTCG 2000

Syst. Review/ Meta-analyse

27 RCTs aus systematischer Suche, 8.275 Patienten

MAB vs AS bei Pat. mit M1 (88%) und T3/4 M0 („locally advanced“)

Sterblichkeit (all cause mortality)

alle Patientengruppen:

Unterschied im 5-J-Überleben von 25,4% (MAB) vs. 23,6% (AS) (n.s.)

Trend für höhere Tox in MAB- vs AS-Gruppe (n.s.)

Subgruppen Nilutamide+Flutamide zusammen analysiert: Unterschied im 5-J-Überleben von 27,6% (MAB) vs 24,7% (AS) (p=0,005)

Subgruppen Cyproteron allein analysiert: Unterschied im 5-J-Überleben von 15,4% (MAB) vs 18,1% (AS) (p=0,04)

Subgruppen-Analyse Orchiektomie vs. medikamentöse Blockade: kein Unterschied

Subgruppen-Analyse für alle M0-Patienten: ebenfalls kein signifikanter Unterschied in Sterblichkeit

Suchstrategie sehr spärlich

1+

10-35

 

Quelle

Population

Populationen - detailliert

Intervention

Suchstrategie, Ein- und Ausschlusskriterien

eingeschlossene Studien

Ergebnisse

Methodische Bewertung AMSTAR

LoE

Magnan S. Intermittent vs Continuous Androgen Deprivation Therapy for Prostate Cancer: A Systematic Review and Meta-analysis. JAMA Oncol 2015;1(9):1261-9.

patients with prostate cancer (any stage)

n=6856 patients from 15 trials

2x locally advanced, metastatic, or BCR;

2x locally advanced or metastatic;

2x BCR (after ?);

6x metastatic;

1x not reported

intermittent vs continuous androgen deprivation therapy ADT (any regimen) (detailed in Table 1)

search of Cochrane CENTRAL, Medline, Embase, and Web of Science; abstracts and conference proceedings in BiosisPreviews; ongoing trials at clinicaltrials.gov and controlled-trials.com; gray-literature sources: National Technical Information Service (NTIS),OpenSIGLE, and Google Scholar; references from published guidelines, narrative and systematic reviews identified in the systematic search

- searches from inception of databases through March 2014
- search for randomized clinical trials

22 articles from 15 RCTs (6856 pat.s)
published between 2000 and 2013

primary outcomes: overall survival,  quality of life
secondary outcomes: cancer-specific survival, progression-free survival
(biochemical and/or clinical)
[time to castration resistance, skeletal-related events, drug-related adverse effects]

noninferiority analysis for primary
outcome overall survival: absolute difference of 5% at median survival was considered as clinically relevant (difference equivalent to an upper boundary of the HR for death of less than 1.15)

[all but 1 study had an unclear or high risk of bias]

overall survival: no significant difference between intermittent and continuous therapy
HR for death 1.02 (95%CI, 0.93-1.11; I2 = 23%)
(based on pooled results of 8 trials, 5352 pat.s)

quality of life: disparity of instruments used and unavailability of quantitative data did not allow meta-analysis

cancer-specific survival: no significant difference between intermittent and continuous therapy
HR for death 1.02 (95%CI,0.87-1.19; I2 = 14%)
(based on pooled results of 5 trials, 3613 pat.s)

progression-free survival: no significant difference between intermittent and continuous ADT - HR for progression 0.96 (95% CI, 0.76-1.21; I2 = 75%) (based on pooled results for time to progression: 5 trials, 3523 pat.s) / HR for progression 0.94 (95% CI, 0.84-1.05; I2 = 0%) (based on pooled results for progression-free survival: 4 trials, 1774 pat.s)

1 Protokoll/Suchstrategie/ Fragestellung vorhanden - ja
2 Selektion und Extraktion durch 2 unabh. Bearbeiter - ja
3 Literaturrecherche min 2 Datenbanken + 1 Zusatz - ja
4 graue Literatur - ja
5 Vollständige Liste der ein- und ausgeschlossenen Studien - nein
6 Details der eingeschlossenen Studien - nein
7 Qualität der eingeschlossenen Studien bewertet - ja
8 In Conclusion Hinweise auf methodische Qualität - ja
9 Heterogenität erhoben? - ja
10 Publication bias berichtet (funnel plot/verbal) - ja
11 CoI/funding für Review UND für alle Studien berichtet - nein

--> AMSTAR-Score 8-9/11

1(+)

Niraula S. Treatment of prostate cancer with intermittent versus continuous androgen deprivation: a systematic review of randomized trials. J Clin Oncol 2013;31(16):2029-36.

patients with rising PSA after radical treatment for localized disease,
advanced, or metastatic disease

n=5508 pat.s from 9 trials

1x locally advanced, metastatic, or BCR after RP;

4x locally advanced or metastatic;

1x BCR after RP;

1x BCR after RT;

2x metastatic

intermittent vs continuous androgen deprivation therapy ADT
(GnRH agonist + / - concurr.  peripheral antiandrogen; pat.s with PSA decline to <4ng/mL after initial ADT randomized to IAD or CAD)

search of Medline, Embase, Cochrane library; references provided in review articles and proceedings of major oncology / genitourinary conferences
- searches from inception of databases through September 2012
- search for randomized controlled trials comparing IAD and CAD if reporting overall survival or biochemical / radiologic time to disease progression

Nine RCTs (5508 patients)
published between 2002 and 2013

outcomes: overall survival, time to progression, cancer-specific survival, quality of life, adverse events

overall survival: not significantly different between IAD and CAD. HR 1.02 (95% CI 0.93-1.11, I2 10%)
(based on pooled HRs from 4 studies, 4,101 pat.s)

time to progression: not significantly different between IAD and CAD. HR 0.96 (95% CI 0.76-1.20, I2 74%)
(based on pooled HRs from 3 studies, 2,596 pat.s)

cancer-specific survival:  not significantly different between IAD and CAD. HR 0.98 (95% CI 0.85-1.38, I2 62%)
(based on pooled HRs from 3 studies, 2,566 pat.s)
more PCa-specific deaths in IAD groups while more non-cancer related deaths in CAD groups

quality of life: better scores in favor of IAD in some domains of QoL and less treatment-related AEs in 6/9 studies, significant superiority of overall QoL in 1 study

1 Protokoll/Suchstrategie/ Fragestellung vorhanden - ?
2 Selektion und Extraktion durch 2 unabh. Bearbeiter - ja
3 Literaturrecherche min 2 Datenbanken + 1 Zusatz - ja
4 graue Literatur - nein
5 Vollständige Liste der ein- und ausgeschlossenen Studien - nein
6 Details der eingeschlossenen Studien - ja
7 Qualität der eingeschlossenen Studien bewertet - ja
8 In Conclusion Hinweise auf methodische Qualität - ja
9 Heterogenität erhoben? - ja
10 Publication bias berichtet (funnel plot/verbal) - ja
11 CoI/funding für Review UND für alle Studien berichtet - nein

--> AMSTAR-Score 7-8/11

1(+)

 

Saad_LancetOncol_2018_Small_AnnOncol_2019_Smith_NEJM_2018_Smith_EurUrol_2020

 

Smith MR, Saad F, Chowdhury S et al. Apalutamide Treatment and Metastasis-free Survival in Prostate Cancer. N Engl J Med. 2018 Apr 12;378(15):1408-1418. doi: 10.1056/NEJMoa1715546.

Saad F, Cella D, Basch E. Effect of apalutamide on health-related quality of life in patients with non-metastatic castration-resistant prostate cancer: an analysis of the SPARTAN randomised, placebo-controlled, phase 3 trial. Lancet Oncol. 2018 Oct;19(10):1404-1416. doi: 10.1016/S1470-2045(18)30456-X.

Small EJ, Saad F, Chowdhury S et al. Apalutamide and overall survival in non-metastatic castration-resistant prostate cancer. Ann Oncol. 2019 Nov 1;30(11):1813-1820. doi: 10.1093/annonc/mdz397. 

Smith MR, Saad F, Chowdhury S et al. Apalutamide and Overall Survival in Prostate Cancer. Eur Urol. 2020 Sep 6:S0302-2838(20)30628-X. doi: 10.1016/j.eururo.2020.08.011.

Link

https://pubmed.ncbi.nlm.nih.gov/29420164/  

https://pubmed.ncbi.nlm.nih.gov/30213449/

https://pubmed.ncbi.nlm.nih.gov/31560066/

https://pubmed.ncbi.nlm.nih.gov/32907777/

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: low

Attrition bias

lost to follow-up: low

ITT-analysis: low

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the trial was designed by two of the academic authors (the first and last authors) and representatives of the sponsor

(within the placebo group also older patients were included (age range 52-97 vs. 48-94 (median age was 74 years in both groups))

Design

  randomized, double-blind placebo-controlled, phase 3 trial

  SPARTAN, NCT01946204

1. interim analysis after 378 events (cut-off May 19,2017)

2. interim analysis after 285 events (overall survival) (cut-off Feb 01,2019)

3. analysis, final analysis (clinical cut-off Feb 01, 2020)

Population

nonmetastatic castration-resistant prostate cancer (nmCRPC) with a prostate-specific antigen (PSA) doubling time of 10 months or less

- 332 sites in 26 countries (North America, Europe, and the Asia–Pacific Region)

- October 14, 2013 to December 15, 2016

Inclusion criteria:

- 18 years of age or older

- histologically or cytologically confirmed adenocarcinoma of the prostate

- castrationresistant

- high risk for the development of metastasis (defined as a PSA doubling time of 10 months or less during continuous androgen-deprivation therapy (bilateral orchiectomy or treatment with gonadotropin-releasing hormone analogue agonists or antagonists)

- no local or regional nodal disease (classified as N0 on the tumor–node–metastasis staging system) or

- malignant pelvic lymph nodes that measured less than 2 cm in the short axis (classified as N1) and were located below the aortic bifurcation

Exclusion criteria:

- (all the patients underwent a technetium-99m bone scan and computed tomography (CT) of the pelvis, abdomen, chest, and head)

- distant metastasis was detected, either on these imaging studies or with the use of other information sources

Intervention

androgen-deprivation therapy + apalutamide

Control

androgen-deprivation therapy + placebo

Outcome(s)

primary: metastasis-free survival (defined as the time from randomization to the first detection of distant metastasis on imaging or death from any cause)

secondary:

- time to metastasis involving the bone or soft tissue

- progression-free survival (PFS)

- time to symptomatic progression

- overall survival (OS)

- time to the initiation of cytotoxic chemotherapy

exploratory analysis of health-related quality of life (HRQOL) – Functional Assessment of Cancer Therapy-Prostate treatment (FACT.P) and EQ.5D.3L questionnaires (Saad et al. Lancet Oncol. 2018)

Results

n=1207 (n(I)=806 vs. n(C)=401)

median age (I) 74 (range 48-94) vs. (C) 74 (range 52-97)

PSA doubling time ≤6 months (n (%)): (I) 576 (71.5) vs. (C) 284 (70.8)

local or regional nodal disease N0 (n (%)): (I) 673 (83.5) vs. (C) 336 (83.8)

Gleason Score at initial diagnosis >7 (n (%)): (I) 341 (43.5) vs. (C) 169 (43.7)

- three patients in each group did not receive study drug

- one patient in each group lost to follow-up

- safety population n=1201 (n(I)=803 vs. n(C)=398)

- at the 1. interim analysis 60.9% (I) vs. 29.9% (C) were still receiving the assigned regimen

- at the 2. interim analysis 69% (I) vs. 40% (C) had received subsequent life-prolonging therapy for mCRPC

- at the 3. analysis the trial was unblinded in July 2017, patients receiving placebo who had not yet developed metastases were offered open-label apalutamide; at unblinding 398 patients (C), 322 had already discontinued therapy, 238 (74%) for progressive disease; the remaining 76 (19%) received open-label apalutamide (crossover group)

- at final analysis data cut, 237 of patients originally randomised to apalutamide and 46 (61%) of 76 crossover patients continued treatment with apalutamide

outcome

SPARTAN 1. interim analysis I vs. C (cut-off 19.05.2017)

SPARTAN 2. interim analysis I vs. C vs. cross-over (cut-off 01.02.2019)

SPARTAN (3.) final analysis I vs. C vs. cross-over (cut-off 01.02.2020)

death

10 (1.2) vs.

1 (0.3)

178 (22%) vs.

107 (27%)

274 (of 806) vs.

154 (of 401)

median follow-up (months)

20.3 (IQR 14.8-26.6)

41

52.0

median treatment duration (months)

 

31.4 (0.1-62.5) vs.

11.5 (0.1-37.2) vs.

15.0 (1.0-16.9)

32.9 (0.1–74.5) vs.

11.5 (0.1–37.2) vs.

26.1 (1.0–28.9)

metastasis-free survival (months)

n=378

n(I)=184 (22.8%) vs. n(C)=194 (48,4%)

40.5 vs. 16.2

HR 0.28 (0.23-0.35) P<0.001

-

-

AE n (%)

 

 

grade 3-4

 

 

any serious

 

 

discontinuat.:

 

 

led to death:

775 (96.5) vs.

371 (93.2) (*)

 

362 (45.1) vs.

136 (34.2)

 

24.8% vs.

23.1 %

 

85 (10.6) vs.

28 (7.0)

 

10 (1.2) vs.

1 (0.3)

781 (97.3) vs.

373 (93.7) vs.  

65 (85.5)

426 (53.1) vs.

146 (36.7) vs.

24 (31.6)

269 (33.5) vs.

99 (24.9) vs.

12 (15.8)

109 (13.6) vs.

29 (7.3) vs.

8 (10.5)

17 (2.1) vs.

2 (0.5) vs.

2 (2.6)

781 (97) vs.

373 (94) vs.

68 (89)

449 (56) vs.

145 (36) vs.

29 (38)

290 (36) vs.

99 (25) vs.

19 (25)

120 (15) vs.

29 (7.3) vs.

8 (11)

24 (3.0) vs.

2 (0.5) vs.

2 (2.6)

secondary: (months)

median time to metastasis

40.5 vs. 16.6

HR 0.27 (0.22-0.34) P<0.001

-

-

median PFS

40.5 vs. 14.7

HR 0.29 (0.24-0.36) P<0.001

55.6 vs. 43.8

HR 0.55 (0.45-0.68) P<0.0001

55.6 vs. 41.2            HR 0.55 (0.46–0.66)          P<0.0001

median time to symptomatic progression

NR vs. NR

HR 0.45 (0.32-0.63) P<0,001

-

n= 264

n(I)=156 vs. n(C)=108

HR 0.57 (0.44–0.73)    

P < 0.0001

median OS, months (95% CI)

NR vs.

39.0

HR 0.70 (0.47-1.04)

P=0.07

n=285

n(I)=178 vs. n(C)=107

HR 0.75 (0.59-0.96) P=0.0197

73.9 (61.2–NR) vs.

59.9 (52.8–NR)

HR 0.78 (0.64–0.96) P=0.016

median time to chemotherapy

NR vs. NR

HR 0.44 (0.29-0.66)    P (-)

 HR 0.60 (0.45-0.80)    P (-)

HR 0.63 (0.49–0.81)    P=0.0002

HRQOL, median (IQR) or mean (SE); (total n(I)=803 vs. n(C)=398) safety population

Baseline FACT-P score

n=797 (99%) vs.

n=395 (99%)

md 117.2 (19.2) vs. 116.6 (19.3)

mn 115.9 (1.5) vs. 119.3 (1.3)

 

before metastasis FACT-P score

n=175 vs. n=191

mn 117.9 (1.4) vs. 117.9 (1.3)

 

after metastasis FACTP

n=157 vs. n=184

mn 112.5 (1.9) vs. 114.5 (1.6)

 

Baseline FACT-General

n=175 vs. 190

md 84.1 (14.4) vs.

83.4 (14.2)

mn 83.5 (1.1) vs.

84.8 (1.0)

 

before metastasis FACT-General

n=175 vs. n=191

mn 84.4 (1.0) vs.

84.1 (1.0)

 

after metastasis FACT-General

n=157 vs. n=184

mn 80.7 (1.3) vs.

81.8 (1.1)

-

-

Baseline EQ-5D-3L score

n=782 (97%) vs.391 (98%)

mean health utility score

0.85 (0.17) vs. 0.86 (0.17)

mean health status score

76.2 (17.3) vs. 76.8 (16.9)

 

 

AE=adverse event, CI=confidence interval, cr-ov=cross-over, HR: hazard ratio (95% CI), HRQOL=health-related quality of life, IQR=interquartile range, NR=denotes not reached, md=median, mn=mean, OS=overall survival, PFS=progression-free survival, SE=standard error

*most common: fatigue n(I)=244 (30.4%) vs. n(C)=84 (21.1%), hypertension n(I)=199 (24.8%) vs. n(C)=79 (19.8%), rash n(I)=191 (23.8%) vs. n(C)=22 (5.5%), diarrhea n(I)=163 (20.3) vs. n(C)=60 (15.1), nausea n(I)=145 (18.1) vs. n(C)=63 (15.8)

additionally e.g.: weight loss 129 (16.1) vs. 25 (6.3), falls 125 (15.6) vs. 36 (9.0), fracture 94 (11.7) vs. 26 (6.5)

Comments

- patients were stratified according to PSA doubling time (>6 months vs. ≤6 months), use of bonesparing agents (yes vs. no), and classification of local or regional nodal disease (N0 vs. N1) at the time of trial entry

- interventions for the management of local or regional symptoms were allowed

- after the first detection of distant metastasis or after the trial regimen was discontinued, patients were eligible to receive treatment with sponsorprovided abiraterone acetate plus prednisone or any treatment for mCRPC

- a total of 7.0 % of patient within the intervention group and 10.6 % of the control group withdrew consent from the trial

- protocol information (V1 Nov 2012): “Health-related quality of life response will be defined as a 16-point or higher improvement in the global FACT-P score as compared to baseline. The proportion of patients with a quality of life response will be summarized by treatment arm. The response rates between treatment arms will be compared using a Mantel-Haenszel test, stratified by PSADT (> 6 months vs. ≤ 6 months), the use of a bone-sparing agent (Yes vs. No), and the presence of loco-regional disease (N0 vs. N1) at a two-sided 0.05 significance level. The EQ-5D data will be summarized descriptively by treatment group and study visit.”

- 779 patients were censored without event, with a median follow-up time of 50.4 months

- median OS at the final analysis was estimated with limitation since only a small number of patients were at risk and the CI for the median was not estimable

 

Fizazi_NEJM_2019_Fizazi_NEJM_2020

 

Fizazi K, Shore N, Tammela TL et al. Darolutamide in Nonmetastatic, Castration-Resistant Prostate Cancer. N Engl J Med. 2019 Mar 28;380(13):1235-1246. doi: 10.1056/NEJMoa1815671.

Fizazi K, Shore N, Tammela TL et al. Nonmetastatic, Castration-Resistant Prostate Cancer and Survival with Darolutamide. N Engl J Med. 2020 Sep 10;383(11):1040-1049. doi: 10.1056/NEJMoa2001342.

Link

https://pubmed.ncbi.nlm.nih.gov/30763142/

https://pubmed.ncbi.nlm.nih.gov/32905676/  

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: low

Attrition bias

lost to follow-up: low

ITT-analysis: unclear

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: sponsors developed the trial design with the first and last authors, data were collected by the investigators, analyzed by statisticians who were employed by the sponsors, and interpreted by the authors, including employees of the sponsors

baseline characteristics: presence of lymph nodes on central imaging review (n(I)=163 (17%) vs. n(C)=158 (29%)

Design

  randomized, double-blind placebo-controlled, phase 3 trial

  ARAMIS, NCT02200614

  patients were enrolled from Sep 2014 through March 2018

- Cut-off: September 3, 2018 (1. interim analysis), after 437 end-point events

- Final analysis, cut-off 15 Nov 2019 (planned after 240 deaths)

Population

men with nonmetastatic, castration-resistant prostate cancer (nmCRPC) and a prostate-specific antigen doubling time of 10 months or less

36 countries worldwide, 409 centers

enrolment between September 2014 and March 2018

Inclusion criteria:

- 18 years of age or older

- histologically or cytologically confirmed adenocarcinoma of the prostate

- without neuroendocrine differentiation or small cell features

- castration-resistant prostate cancer

- castrate level of serum testosterone (<1.7 nmol/l [50 ng/dl]) on gonadotrophinreleasing hormone (GnRH) agonist or antagonist therapy or after bilateral orchiectomy at screening or day 1 visit

- baseline PSA level of at least 2 ng per milliliter

- PSA doubling time of 10 months or less

- Eastern Cooperative Oncology Group (ECOG) performance status of 0 or 1 (scores range from 0 to 5, with higher numbers reflecting greater disability)

- blood counts at screening: hemoglobin ≥9.0 g/dl, absolute neutrophil count ≥1500/μl (1.5×109/l), platelet count ≥100,000/μl (100×109/l)

- screening values of serum alanine transaminase (ALT) and aspartate transaminase (AST) ≤2.5 x upper limit of normal (ULN), total bilirubin ≤1.5 x ULN (except patients with a diagnosis of Gilbert’s disease), creatinine ≤2.0 x ULN

- the presence of pelvic lymph nodes less than 2 cm in diameter in the short axis below the aortic bifurcation was allowed

Exclusion criteria:

- detectable metastases or a history of metastatic disease

- symptomatic local-regional disease that requires medical intervention

- acute toxicities of prior treatments and procedures not resolved to grade ≤1

- prior treatment with: (1) second-generation androgen receptor (AR) antagonists (e.g. enzalutamide and apalutamide, or darolutamide); (2) CYP17 enzyme inhibitors (e.g. abiraterone acetate, TAK-700; (3) oral ketoconazole for longer than 28 days)

- use of estrogens or 5-α reductase inhibitors (finasteride, dutasteride) within 28 days before randomization and AR antagonists (bicalutamide, flutamide, nilutamide, cyproterone acetate) at least 28 days before screening

- prior chemotherapy or immunotherapy for prostate cancer, except adjuvant/neoadjuvant treatment completed >2 years before randomization

- use of systemic corticosteroid

- radiation therapy (external beam radiation therapy [EBRT], brachytherapy, or

radiopharmaceuticals) within 12 weeks before randomization

- severe or uncontrolled concurrent disease, infection or comorbidity that

would make the patient inappropriate for enrollment

- treatment with an osteoclast-targeted therapy (bisphosphonate or denosumab)

- any of the following within 6 months before randomization: stroke, myocardial

infarction, severe/unstable angina pectoris, coronary/peripheral artery bypass graft; congestive heart failure New York Heart Association (NYHA) Class III or IV

- uncontrolled hypertension as indicated by a systolic blood pressure ≥160 mmHg

or diastolic blood pressure ≥100 mmHg at screening.

- prior malignancy

Intervention

androgen-deprivation therapy (ADT) + darolutamide

Control

ADT + placebo

Outcome(s)

primary:

- metastasis-free survival (MFS)#

secondary:

- overall survival (OS)

- time to pain progression (BPI-SF questionnaire or initiation of opioid treatment for cancer pain)

- time to first symptomatic skeletal event

- time to first cytotoxic chemotherapy

# defined as the time from randomization to confirmed evidence of distant metastasis on imaging or death from any cause, whichever occurred first

Results

n=1509 (n(I)=955 vs. n(C)=554)

- median age (I) 74 (range 48-95) vs. (C) 74 (range 50-92)

- one patient (intervention group) did not start the treatment

- at primary analysis, 64% of patients in the intervention group and 36% in the control group were still receiving the assigned trial regimen

- at the time of unblinding, all 170 patients who were still receiving placebo crossed over to receive darolutamide (unblinding occurred after the primary analysis, 20 Nov 2018)

outcome

ARAMIS 1. interim analysis    

I vs. C (cut-off 23.11.2018)

ARAMIS 2. analysis 

I vs. C vs. cross-over (cut-off 15.11.2019)

death

n=136 (n(I)=78 vs. n(C)=58)

n=254 (n(I)=148 (15%) vs. n(C)=106 (19%))

median follow-up

17.9 months

29.0 months

median treatment duration (months)

14.8 vs. 11.0

25.8 vs. 11.6 vs. 11.0

MFS, n

median (months (95% CI))

437

40.4 (34.3-NR) vs. 18.4 (15.5-22.3)

HR 0.41 (0.34-0.50) P<0.001

-

AE n (%)

 

grade 3-4

 

any serious

 

discontinuation:

led to death:

794 (83.2) vs. 426 (76.9) (*)

 

236 (24.7) vs. 108 (19.5)

 

237 (24.8) vs. 111 (20.0)

 

85 (8.9) vs. 48 (8.7)

1 vs. 2

818 (85.7) vs. 439 (79.2)

vs. 119 (70.0)

251 (26.3) vs. 120 (21.7)

vs. 27 (15.9)

249 (26.1) vs. 121 (21.8)

vs. 26 (15.3)

85 (8.9) vs. 48 (8.7) vs. 8 (4.7)

secondary:

OS, n

median (months (95% CI))

78 vs. 58

NR (44.5–NR) vs. NR (NR–NR)

HR 0.71 (0.50–0.99) P=0.045

83% (80-86) vs. 77% (72-81)      at 3 years HR 0.69 (0.53-0.88) P=0.003

time to pain progress., n

median (months)

251 vs. 178

40.3 vs. 25.4

HR 0.65 (0.53–0.79) P<0.001

251 vs. 178

53 (47–60) vs. 32 (22–43)  

HR 0.65 (0.53–0.79) P<0.001

time to chemotherapy, n

median (months)

73 vs. 79

NR vs. 38.2

HR 0.43 (0.31–0.60) P<0.001

127 vs. 98

83 (80–86) vs. 75 (69–80)         HR 0.58 (0.44–0.76) P<0.001

time to skeletal event, n

median (months)

16 vs. 18

NR vs. NR

HR 0.43 (0.22–0.84) P=0.01

29 vs. 28

96 (95–98) vs. 92 (89–96)         HR 0.48 (0.29–0.82) P=0.005

CI=confidence interval, HR=Hazard ratio (95 % CI), IQR=interquartile range, MFS=metastasis-free survival, NE could not be estimated, NR=not reached, OS=overall survival

* most common I vs. C (n (%)): fatigue 115 (12.1) vs. 48 (8.7), back pain 84 (8.8) vs. 50 (9.0), arthralgia 77 (8.1) vs. 51 (9.2)

also important: bone fracture 40 (4.2) vs. 20 (3.6), falls 40 (4.2) vs. 26 (4.7), rash 28 (2.9) vs. 5 (0.9), weight decrease 34 (3.6) vs. 12 (2.2)

Comments

- randomization was stratified according to PSA doubling time (≤6 months or >6 months) and the use of osteoclast-targeted therapy at randomization (yes or no)

 

Hussain_NEJM_2018_Sternberg_NEJM_2020_Tombal_LancetOncol_2019

 

Hussain M, Fizazi K, Saad F. Enzalutamide in Men with Nonmetastatic, Castration-Resistant Prostate Cancer. N Engl J Med. 2018 Jun 28;378(26):2465-2474. doi: 10.1056/NEJMoa1800536.

Tombal B, Saad F, Penson D. Patient-reported outcomes following enzalutamide or placebo in men with non-metastatic, castration-resistant prostate cancer (PROSPER): a multicentre, randomised, double-blind, phase 3 trial. Lancet Oncol. 2019 Apr;20(4):556-569. doi: 10.1016/S1470-2045(18)30898-2.

Sternberg CN, Fizazi K, Saad F. Enzalutamide and Survival in Nonmetastatic, Castration-Resistant Prostate Cancer. N Engl J Med. 2020 Jun 4;382(23):2197-2206. doi: 10.1056/NEJMoa2003892.

Link

https://pubmed.ncbi.nlm.nih.gov/29949494/

https://pubmed.ncbi.nlm.nih.gov/30770294/

https://pubmed.ncbi.nlm.nih.gov/32469184/

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: low

Attrition bias

lost to follow-up: low

ITT-analysis: low

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the sponsor designed the study and wrote the protocol, analyses were performed by the sponsor, the manuscript was prepared by a professional medical writer, paid by the sponsor  

comment: baseline serum PSA value (ng/ml, range) median 11.1 (0.8-1071.1) vs. 10.2 (0.2-467.5), PSA doubling time (months, range) median 3.8 (0.4-37.4) vs. 3.6 (0.5-71.8)

Design

  randomized, double-blind placebo-controlled, phase 3 trial

  PROSPER, NCT02003924

- cut-off: June 28, 2017 (1. interim analysis), after 447 primary end-point events

- cut-off: October 15, 2019 (2. interim analysis)

Population

nonmetastatic, castration-resistant prostate cancer (nmCRPC) and a prostate-specific antigen (PSA) doubling time of 10 months or less (high risk for metastasis)

300 sites in 32 countries

randomization between November 26, 2013 and June 28, 2017

Inclusion criteria:

- 18 years or older

- histologically or cytologically confirmed prostate adenocarcinoma without neuroendocrine differentiation, signet-cell features, or small-cell features

- a rising PSA level despite castration-associated testosterone levels (serum testosterone level, ≤1.73 nmol per liter [0.50 ng per milliliter])

- receiving androgen-deprivation therapy with a gonadotropinreleasing hormone agonist or antagonist or bilateral orchiectomy

- a minimum of three rising PSA values at an interval of at least 1 week apart

- a baseline PSA level of 2 ng per milliliter or greater

- a PSA doubling time of 10 months or less

- no previous or current evidence of metastatic disease

- asymptomatic prostate cancer

- Eastern Cooperative Oncology Group performance-status score of 0 or 1

- estimated life expectancy ≥ 12 months

Exclusion criteria:

- suspected brain metastases or active leptomeningeal disease or with a history of seizure or a condition that may confer a predisposition to seizure

- prior cytotoxic chemotherapy

- treatment with hormonal therapy

- known or suspected brain metastasis or active leptomeningeal disease

- history of another invasive cancer within 3 years of randomization

- absolute neutrophil count < 1000/μL, platelet count < 100,000/μL, or hemoglobin < 10 g/dL (6.2 mmol/L) at screening

- total bilirubin ≥ 1.5 times the upper limit of normal (ULN); alanine aminotransferase (ALT) or aspartate aminotransferase (AST) ≥ 2.5 times ULN

- Creatinine > 2 mg/dL (177 μmol/L) at screening

- Albumin < 3.0 g/dL (30 g/L) at screening

- clinically significant cardiovascular disease

Intervention

androgendeprivation therapy (ADT) + enzalutamide

Control

ADT + placebo

Outcome(s)

primary: metastasis-free survival (MFS)#

secondary:

- Overall survival (OS)

- Time to pain progression

- Time to opiate use for prostate cancer pain

- Time to pain progression or opiate use for prostate cancer pain

- Time to first use of cytotoxic chemotherapy

- Time to first use of new antineoplastic therapy

- Time to prostate-specific antigen (PSA) progression

- PSA response rates

- Time to functional status deterioration as assessed by the Functional Assessment of Cancer Therapy-Prostate (FACT-P) global score

- Quality of life as assessed by the European Quality of Life-5 Dimensions-5 Levels (EQ-5D-5L) health questionnaire and Quality of Life Questionnaire-Prostate 25 (QLQ-PR25) module

# defined as the time from randomization to radiographic progression, as determined by central review at any time, or as the time to death from any cause during the period from randomization to 112 days after the discontinuation of the trial regimen without evidence of radiographic progression, whichever occurred first

Results

n=1401, n(I)= 933 vs. n(C)=468

(median PSA doubling time 3.7 months)

median age (I) 74 (range 50-95) vs. 73 (53-92)

- at the first data-cutoff date, 810 patients were receiving the trial regimen (634 patients in the enzalutamide group and 176 in the placebo group)

- after the primary analysis, the trial data were unblinded, patients in the placebo group were given the opportunity to receive enzalutamide (crossover 12.02.2018)

- 87 of 114 patients in the placebo group who were still receiving treatment when crossover began, received enzalutamide in the open-label extension

- baseline BPI-SF, EORTC QLQPR25, FACT-P, and EQ-5D-5L were completed by (I) 887 (95%) vs. (C) 439 (94%) – “more than 85% of patients completed all patient-reported outcome questionnaires in both treatment groups”(Tombal et al. 2019)

outcome

PROSPER 1. interim analysis    

I vs. C (cut-off 28.06.2017)

PROSPER 2. interim analysis    

I vs. C (cut-off 15.10.2019)

death

n(I)=103 (11%) vs. n(C)=62 (13%)

n(I)=288 (31%) vs. n(C)=178 (38%)

follow-up, median months (IQR)

18.5 (10.7-29.2) vs. 15.1 (7.4-25.9)

48 months

treatment duration, median months

18.4 (IQR 9.7-27.2) vs. 11.1 (IQR 6.4-18.7)

33.9 (95 % CI 0.2-68.8) vs. 14.2 (95 % CI 0.1-51.3)

MFS

median months (95% CI)

n=447, n(I)=219 (23%) vs. n(C)=228 (49%)

36.6 (33.1–NR) vs. 14.7 (14.2–15.0)

HR 0.29 (0.24-0.35) P<0.001

-

AE n (%)

grade ≥3

any serious

discontinu.:

led to death:

808 (87) vs. 360 (77)*

292 (31) vs. 109 (23)

226 (24) vs. 85 (18)

87 (9) vs. 28 (6)

32 (3) vs. 3 (1)

876 (94) vs. 380 (82)§

446 (48) vs. 126 (27)

372 (40) vs. 100 (22)

158 (17) vs. 41 (9)

51 (5) vs. 3 (1)

secondary: median, months (95 % CI)

OS

n(I)=103 (11%) vs. n=(C)=62 (13%)

NR vs. NR

HR 0.80 (0.58–1.09) P=0.15

n(I)=288 (31%) vs. n(C)=178 (38%)

67.0 (64.0-NR) vs. 56.3 (54.4-63.0)

HR 0.73 (0.61-0.89) P=0.001

time to pain progression$

36.83 (34.69-NR) vs. NR

HR 0.75 (0.57-0.97) P=0.028

-

time to opioid use

-

-

time to pain progression or opiate use

-

-

time to chemotherapy

-

NR (66.0-NR) vs. NR (54.9-NR)

HR 0.54 (0.44-0.67)

time to first use of new antineoplastic therapy

n(I)=142 (15%) vs. n(C)=226 (48%)

39.6 vs. 17.7

HR 0.21 (0.17–0.26) P<0.001

 

66.7 (56.4-NR) vs. 19.1 (17.4-22.1)

HR 0.29 (0.25-0.34)

time to prostate-specific antigen (PSA) progression

n(I)=208 (22%) vs. n(C)=324 (69%)

37.2 vs. 3.9

HR 0.07 (0.05–0.08) P<0.001

-

PSA response ≥50%

n(I)=712 (76%) vs. n(C)=11 (2%)

-

time to** functional status deterioration

n(I)=506 (54%) vs. n(C)=239 (51%)

11.1 vs. 11.1

HR 0.92 (0.79–1.08)

-

Quality of life***

bowel symptoms

 

33.15 (29.50-NR) vs. 25.89 (18.43-29.67)

HR 0.72 (0.59-0.89) P=0.0018

-

hormonal treatment-rel. symptoms

33.15 (29.60-NR) vs. 36.83 (29.47-NR)

HR 1.29 (1.02-1.63) P=0.035

-

urinary symptoms

36.86 (33.35-NR) vs. 25.86 (18.53-29.47)

HR 0.58 (0.46-0.72) P<0.0001

-

EQ-VAS

22.11 (18.46-25.66) vs. 14.75 (11.07-18.17)

HR 0.75 (0.63-0.90) P=0.0013

-

CI=confidence interval, HR=Hazard ratio (95 % CI), IQR=interquartile rande, MFS=metastasis-free survival, NE could not be estimated, NR=not reached, OS=overall survival, rPFS=radiographic progression-free survival

$ Brief Pain Inventory Short Form (BPI-SF) questionnaire

*** European Quality of Life-5 Dimensions-5 Levels (EQ-5D-5L) health questionnaire and Quality of Life Questionnaire-Prostate 25 (QLQ-PR25) module

** Functional Assessment of Cancer Therapy-Prostate (FACT-P) global score

* most common I vs. C (n (%)): fatigue 303 (33) vs. 64 (14), hot flush 121 (13) vs. 36 (8), hypertension 114 (12) vs. 25 (5), nausea 106 (11) vs. 40 (9), fall 106 (11) vs. 19 (4)

§ most common: fatigue 424 (46) vs. 103 (22), musculoskeletal events 315 (34) vs. 107 (23), fracture 168 (18) vs. 29 (6), hypertension 167 (18) vs. 28 (6), fall 167 (18) vs. 25 (5)

Comments

- randomization was stratified by PSA doubling time (< 6 months vs ≥ 6 months) and baseline use of a bone-targeting agent (yes vs no)

 

Beer_NEJM_2014_Beer_EurUrol_2017

 

Beer TM, Armstrong AJ, Rathkopf DE. Enzalutamide in metastatic prostate cancer before chemotherapy. N Engl J Med. 2014 Jul 31;371(5):424-33. doi: 10.1056/NEJMoa1405095.

Beer TM, Armstrong AJ , Rathkopf D. Enzalutamide in Men with Chemotherapy-naïve Metastatic Castration-resistant Prostate Cancer: Extended Analysis of the Phase 3 PREVAIL Study. Eur Urol. 2017 Feb;71(2):151-154. doi: 10.1016/j.eururo.2016.07.032. 

Link

https://pubmed.ncbi.nlm.nih.gov/24881730/  

https://pubmed.ncbi.nlm.nih.gov/27477525/

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: low

Attrition bias

lost to follow-up: low

ITT-analysis: low

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the sponsor influenced study design, data analyses were conducted by the sponsor, a professional writer was paid by the sponsors to prepare the manuscript,

comment: study protocol refers to OS as primary outcome followed by rPFS, the first publication refers to PFS followed by OS

Design

  randomized, double-blind placebo-controlled, phase 3 trial

  PREVAIL, NCT01212991

- Cut-off: September 16, 2013 (1. Interim analysis), January 15, 2014 (2. interim analysis – updated survival analysis), June 1, 2014 (3. interim analysis)

Population

men with metastatic prostate cancer who have not received chemotherapy, in whom the disease has progressed despite androgen-deprivation therapy

207 sites globally

randomization between September 2010 and September 2012

Inclusion criteria:

- histologically or cytologically confirmed adenocarcinoma of the prostate without neuroendocrine differentiation or small cell features

- documented metastases

- PSA progression, radiographic progression, or both in bone or soft tissue, despite receiving LHRH analogue therapy or undergoing orchiectomy,

- serum testosterone level of 1.73 nmol per liter (50 ng per deciliter) or less

- continued androgen-deprivation therapy

- previous antiandrogen therapy and concurrent use of glucocorticoids were permitted but not required

- Eastern Cooperative Oncology Group performance status grade of 0 or 1

- no symptoms or ambulatory but restricted in strenuous activities and were either asymptomatic (score of 0 to 1) or mildly symptomatic (score of 2 to 3) (Brief Pain Inventory Short Form question)

- estimated life expectancy of ≥ 6 months

Exclusion criteria:

- severe concurrent disease, infection, or co-morbidity

- known or suspected brain metastasis or active leptomeningeal disease

- history of another malignancy within the previous 5 years

- absolute neutrophil count < 1,500/μL, or platelet count < 100,000/μL, or hemoglobin < 5.6 mmol/L (9 g/dL) at the Screening visit

- total bilirubin, alanine aminotransferase (ALT) or aspartate aminotransferase (AST) > 2.5 times the upper limit of normal

- Creatinine > 177 μmol/L (2 mg/dL) at the Screening visit;

- Albumin < 30 g/L (3.0 g/dL) at the Screening visit;

- history of seizure or any condition that may predispose to seizure

- history of loss of consciousness or transient ischemic attack within 12 months of enrollment  

- clinically significant cardiovascular disease

- use of opiate analgesics for pain from prostate cancer within 4 weeks of enrollment

- radiation therapy for treatment of the primary tumor within 3 weeks of enrollment

- radiation or radionuclide therapy for treatment of metastasis

- reatment with flutamide, bicalutamide or nilutamide, with 5-α reductase inhibitors (finasteride, dutasteride), estrogens, cytproterone, systemic biologic therapy for prostate cance or other agents with anti-tumor activity

- history of prostate cancer progression on ketoconazole

- prior use, or participation in a clinical trial, of an investigational agent that blocks

- androgen synthesis (e.g., abiraterone acetate, TAK-700, TAK-683, TAK-448) or blocks the androgen receptor (e.g., BMS 641988)

- participation in a previous clinical trial of MDV3100

- use of an investigational agent

Intervention

androgen deprivation therapy (ADT) + enzalutamide

Control

ADT + placebo

Outcome(s)

primary: radiographic progression-free survival (rPFS) and overall survival (OS)

secondary:

- time to first skeletal-related event

- time to initiation of cytotoxic chemotherapy

- time to PSA progression

- quality of life (Functional Assessment of Cancer Therapy-Prostate (FACT-P) and the European Quality of Life 5-Domain Scale (EQ-5D)

- emergence of pain, (Brief Pain Inventory)

- safety

Results

n=1717, n(I)=872 vs. n(C)=845

- one patient (I) vs. one patient (C) did not receive allocated treatment, one patient (I) vs. 0 was lost-to-follow-up, 21 (I) vs. 40 (C) withdrew consent - safety follow-up

- at least 12 months of treatment: 68% vs. 18%

- continued to receive treatment as of the data-cutoff date: 42% vs. 7%

- the study was stopped at the first interim analysis (final analysis)

- crossover began January 1, 2014

- at the time of the 3. interim analysis 167 patients in the placebo arm had crossed over to receive enzalutamide

outcome

PREVAIL 1. interim analysis    

I vs. C (cut-off 16.09.2013)

PREVAIL 3. interim analysis    

I vs. C (cut-off 01.06.2014)

death

n(I)=241 (28%) vs. n(C)=299 (35%)

 

median follow-up

22 months

22 months vs. 11 months

median treatment duration

16.6 months vs. 4.6 months

18.2 months vs. 5.4 months

rPFS (May 6, 2012)

median months

 

at 12 months, % (95% CI)

118 of 832 patients (14%) vs. 321 of 801 patients (40%)

NR vs. 3.9

HR 0.19 (0.15-0.23) P<0.001

65 % vs. 14 %

 

                                                     20.0 (18.9–22.1) vs. 5.4 (4.1–5.6)

HR 0.32 (0.28–0.37) P<0.0001

OS (median, months)

Update 15.01.2014

32.4 vs. 30.2

HR 0.71 (0.60 to 0.84) P<0.001

26.2 vs. 26.5

HR 0.73 (0.63-0.85) P<0.001

n(I)=299 (34%) vs. n(C)=357 (42%)

35.3 (32.2–NR) vs. 31.3 (28.8–34.2)

HR 0.77 (0.67–0.88) P=0.0002

AE n (%)

grade ≥3

any serious

discontinuation

led to death:

844 (97) vs. 787 (93) (*)

374 (43) vs. 313 (37)

279 (32) vs. 226 (27)

49 (6) vs. 51 (6)

37 (4) vs. 32 (4)

849 (97.5) vs. 788 (93.4) (**)

398 (45.7) vs. 316 (37.4)

314 (36.1) vs. 229 (27.1)

54 (6.2) vs. 51 (6.0)

43 (4.9) vs. 32 (3.8)

secondary:

median time to skeletal event (months)

n(I)=278 (32%) vs. n(C)=309 (37%)

31.1 vs. 31.3

HR 0.72 (0.61–0.84) P<0.001

-

median time to chemotherapy (months)

28.0 vs. 10.8

HR 0.35 (0.30–0.40) P<0.001

-

time to PSA progression (months)

11.2 vs. 2.8

HR 0.17 (0.15–0.20) P<0.001

-

quality of life, decline in FACT-P (months)

11.3 vs. 5.6

HR 0.63 (0.54–0.72) P<0.001

-

emergence of pain

-

-

CI=confidence interval, FACT-P=Functional Assessment of Cancer Therapy-Prostate, HR=Hazard ratio (95 % CI), IQR=interquartile rande, NE could not be estimated, NR=not reached, OS=overall survival, rPFS=radiographic progression-free survival

* most common I vs. C (n (%)): fatigue 310 (36) vs. 218 (26), back pain 235 (27) vs. 187 (22), constipation 193 (22) vs. 145 (17), arthralgia 177 (20) vs. 135 (16)decreased appetite 158 (18) vs. 136 (16), hot flush 157 (18) vs. 65 (8)

**most common: fatigue 312 (35.8) vs. 219 (25.9), back pain 249 (28.6) vs. 189 (22.4), constipation 2020 (23.2) vs. 146 (17.3), arthralgia 186 (21.4) vs. 136 (16.1), decreased appetite 165 (18.9) vs. 138 (16.4), hot flush 157 (18.0) vs. 66 (7.8), diarrhea 146 (16.8) vs. 121 (14.3), hypertension 124 (14.2) vs. 35 (4.1)

Comments

-

 

Berthold_JClinOncol_2008

Quelle/
Studientyp

Population

(verglichene) Interventionen/
ggf. Dosierung/
ggf.
Follow-up

Outcomes

Ergebnisse

Bemerkungen

Evidenzniveau (SIGN)

Berthold 2008

(RCT)

 

TAX327:

n=1006 men

recruitment from 03/2000 till 06/2001

patients with metastatic prostatic carcinoma and progression despite androgen-deprivation therapy

3-weekly docetaxel-prednisone 75mg/m2 (D3P) vs. weekly docetaxel-prednisone 30mg/m2 (D1P) vs.

3-weekly mitoxantrone-prednisone (MP) 12mg/m2

Overall survival

Overall survival:

D3P: 19.2m (range 17.5-21.3) vs.

D1P: 17.8m (range: 16.2-19.2) vs.

MP: 16.3m (range: 14.3-17.9)

HR (D3P vs. MP): 0,79 (95% CI: 0.67-0.93), p=0.004

HR (D1P vs. MP): 0.87(95% CI: 0.74-1.02), p=0.086

3-J-OS

D3P: 18.6%

D1P: 16.8%

MP: 13.5%

Update from Tannock et al NEJM 2004

Already included in Evidenztabellen  S3 Leitlinie Prostatkarzinom

1+

 

Ryan_NEJM_2013
 

Quelle/
Studientyp

Population

(verglichene) Interventionen/
ggf. Dosierung/
ggf.
Follow-up

Outcomes

Ergebnisse

Bemerkungen

Evidenzniveau (SIGN)

Ryan 2013

(COU-AA-302)

N=1088 (1:1 ratio, Intervention n=546, Control n=542) 

Enrollment from 04/2009 - 06/2010

Inclusion criteria (selection)

Adenocarcinoma of the prostate

PSA progression (PCWG2)

ECOG performance: 0-1

Ongoing androgen deprivation, testosterone level<50 ng/dL

Previous antiandrogen therapy

BPI-SF: 0-3

Exclusion criteria (selection):

Treatment with ketoconazol

 

1g abiraterone acetate + (2 x 5mg) prednisone vs. 1x1g placebo + (2 x 5mg) prednisone

Co-primary endpoints:

1. overall survival time

2. radiographic progression-free survival (composite endpoint, blinded evaluation)

Secondary pre-specified endpoints

3. Median time to PSA progression (PCWG2 criteria)

Other endpoints:

4. PSA response (≥50% decline)

5. BPI-SF (+ 30%) 

6. FACT-P (-10 points)

 

1a) Median overall survival:

not reached vs. 27.2 months

(HR: 0.75, 95% CI, 0.61-0.93, p=0.01)

1b) Death:

27% (147/546) vs. 34% (186/542)

2a) At 1st  interim analysis (blinded evaluation): 

Median not reached vs.  8.3 months

(HR: 0.43, 95% CI: 0.35-0.52, p<0.001)

2b) At 2nd interim analysis (investigator assessed):

Median: 16.5 months vs. 8.3 months

(HR 0.53, 95% CI: 0.45-0.62, p<0.001)

3) PSA progression: 11.1 months vs. 5.6 months

(HR: 0.49, 95% CI 0.42-0.57, p<0.001)

4) PSA response:

62% vs. 24%

(RR: 2.59, 95% CI: 2.19-3.05, p<0.001)

5) BPI-SF:

 26.7 months vs. 18.4 months

(HR: 0.82, 95% CI 0.67-1.00, p=0.049)

6) FACT-P:

12.7 months vs. 8.3 months

(HR:0.78 (95% CI 0.66-0.92, p=0.003)

Median follow-up duration at 2nd interim-analysis: 22.2 months

Study stopped after 2nd interim-analysis

Baseline criteria (‘well balanced’), but not reported in publication

No data about location of centers (multinational study) in publication

ITT, Double-blinded, randomized

Stratification based on ECOG performance status score (0 vs. 1)

Adverse events:

higher incidence of  hypertension, hypokalemia, fluid retention/edema in the abiraterone acetate group

more serious adverse events in abiraterone acetate group

4% (n=20) adverse events leading to death in abiraterone acetate group vs. 2% (n=12) 

Conflict of Interest in supplement

No data on Lost-to-follow-up/ Discontinuation of intervention in publication

Sponsor: Janssen Research and Development;  Analysis conducted by employees of sponsor

1+

 

Parker_Lancet_2018

 

Parker CC, James ND, Brawley CD et al. Radiotherapy to the primary tumour for newly diagnosed, metastatic prostate cancer (STAMPEDE): a randomised controlled phase 3 trial Lancet. 2018 Dec 1;392(10162):2353-2366. doi: 10.1016/S0140-6736(18)32486-3.

Link

https://pubmed.ncbi.nlm.nih.gov/30355464/ 

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: high*

Detection bias

blinding of evaluation: unclear*

Attrition bias

lost to follow-up: low

ITT-analysis: low

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: unclear

comment: the research was funded, employees of the Medical Research Council (MRC), one of the funder, contributed to the design of the study, the data collection as well as the analysis and interpretation

*patients and clinical and study staff were aware of the treatment allocation for practical reasons, and the key efficacy outcome measures were objective

Design

  randomised controlled trial,  phase 3

  (STAMPEDE, NCT00268476), randomisation between Jan 2013, and Sept 2016

Population

Inclusion criteria:

- prostate cancer (newly diagnosed)

- no previous radical treatment

- metastatic disease confirmed on a bone scintigraphic scan and soft-tissue imaging done within 12 weeks of starting androgen deprivation therapy

- no contraindications to radiotherapy

- no clinically significant cardiovascular history

117 hospitals in Switzerland and the UK

Intervention

standard of care and radiotherapy (external-beam radiotherapy to the prostate)#

# either 36 Gy in six consecutive weekly fractions of 6 Gy,or 55 Gy in 20 daily fractions of 2·75 Gy over 4 week

Control

standard of care

comment: all patients in both groups received lifelong androgen deprivation therapy (ADT) as either gonadotrophin-releasing hormone agonists or antagonists or orchidectomy (Docetaxel was permitted in addition to hormone therapy after its approval in the UK on Dec 17, 2015, with or without prednisolone)

Outcome(s)

primary:

- overall survival (OS) (measured as the number of deaths)

secondary:

- failure-free survival (defined as time from randomisation to first evidence of at least one of: biochemical failure; progression either locally, in lymph nodes, or in distant metastases; or death from prostate cancer)

- progression-free survival (defined as failure-free survival but without biochemical events)

- metastatic progression-free survival (defined as time from randomization to new metastases or progression of existing metastases or death)

- prostate cancer-specific survival

- symptomatic local event-free survival

Results

n=2061 patients (n(I)=1032 vs. n(C)=1029)

- median age: 68 years (IQR 63–73) (age range (I) 45-87 vs. (C) 37-86)

- median amount of prostate-specific antigen: 97 ng/mL (33–315)

- safety population: n(I)=985 vs. n(C)=1050

- median follow-up: 37 months (interquartile range (IQR) 24–48 months)

- death: n(I)=370 (median survival 48 months (IQR 27–not reached)) vs. n(C)=391 (median survival 46 months (IQR 27–not reached))

primary: (I vs. C)

- OS:

- survival at 3 years: 65% vs. 62%

- mean survival time (months): 42.5 vs. 41.6

- Hazard Ratio (HR) adjusted 0.92 (95% confidence interval (CI) 0.80–1.06; p=0.266

- adverse events (AE):

- at 6 months: n=2028

- comment: AE were reported selective and not systematically, an overview was missing

secondary: (I vs. C)

- failure-free survival:

- at 3 years: 32% vs. 23%

- mean time (months): 26.2 vs. 21.4

- HR adjusted 0.76 (95% CI 0.68–0.84; p<0.0001)

- progression-free survival:

- at 3 years: 44% vs. 44%

- mean time (months): 33.1 vs. 32.4

- HR adjusted 0.96 (95% CI 0.85–1.08)

- metastatic progression-free survival:

- at 3 years: 47% vs. 47%

- mean time (months): 34.4 vs. 33.9

- HR adjusted 0.97 (95% CI 0.86–1.10)

- prostate cancer-specific survival:

- at 3 years: 69% vs. 66%

- mean time (months): 44.6 vs. 43.9

- HR adjusted 0.93 (95% CI 0.80–1.09)

- symptomatic local event-free survival:

- at 3 years: 55% vs. 57%

- mean time (months): 37.2 vs. 38.2

- HR adjusted 1.07 (95% CI 0.93–1.22)

Comments

- authors documented numerous subgroup analysis

- a discussion of limitations was missing

 

Wit_NEJM_2019

 

De Wit R, de Bono J, Sternberg CN et al. Cabazitaxel versus Abiraterone or Enzalutamide in Metastatic Prostate Cancer. N Engl J Med. 2019 Dec 26;381(26):2506-2518. doi: 10.1056/NEJMoa1911206.

Link

https://pubmed.ncbi.nlm.nih.gov/31566937/

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: high*

Detection bias

blinding of evaluation: unclear*

Attrition bias

lost to follow-up: low

ITT-analysis: low

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the sponsor and the steering committee contributed to the trial design, data analysis and interpretation

a baseline imbalance could not be excluded (e.g. median age: (I) 70.0 years (46–85) vs. (C) 71.0 years (45–88), patients ≥75 years: (I) n=45 (34.9%) vs. n=34 (27.0%)

*the study design was open-label

Design

 randomized, open-label, clinical trial

(NCT02485691, CARD) (62 sites across 13 European countries)

randomization between Nov 2015 and Nov 2018

Population

Inclusion criteria:

1. Histologically confirmed prostate cancer

2. Metastatic disease

3. Castrate levels of serum testosterone (< 0.5 ng/mL); treatment with luteinizing hormone-releasing hormone agonists or antagonists

4. Disease progression (Response Evaluation Criteria in Solid Tumors) and/or appearance of ≥ 2 new bone lesions and/or rising prostate-specific antigen (PSA

5. Having received prior docetaxel for at least three cycles; docetaxel in newly diagnosed metastatic setting and docetaxel rechallenge allowed

6. Progression within 12 months of treatment with a prior androgen-signaling-targeted inhibitor (abiraterone or enzalutamide, before or after docetaxel); abiraterone in metastatic hormone-sensitive setting allowed

7. Eastern Cooperative Oncology Group performance status ≤ 1 (patients with an Eastern Cooperative Oncology Group performance status of 2 were allowed if related to prostate cancer)

8. PSA value of at least 2 ng/mL at study entry

9. Prior androgen-signaling-targeted inhibitor must be stopped at least 2 weeks before study treatment

Exclusion criteria:

1. Prior chemotherapy other than docetaxel for prostate cancer, except estramustine and except adjuvant/neoadjuvant treatment completed > 3 years ago

2. Less than 28 days elapsed from prior treatment with chemotherapy, immunotherapy, radiotherapy or surgery to the time of randomization

3. Adverse events from any prior anticancer therapy of Grade > 1 at the time of randomization

4. Less than 18 years (or country’s legal age of majority if the legal age is > 18 years)

5. Eastern Cooperative Oncology Group performance status > 2 (related to PCa)

6. Prior malignancy; adequately treated basal cell or squamous cell skin or superficial (pTis, pTa, and pT1) bladder cancer are allowed, as well as any other cancer for which treatment has been completed ≥ 5 years ago and from which the patient has been disease-free for ≥ 5 years

7. Participation in another clinical trial and any concurrent treatment with any investigational drug within 30 days prior to randomization

and other (see also supplement)

Intervention

 cabazitaxel

(at a dose of 25 mg per square meter of body-surface area intravenously every 3 weeks, plus prednisone daily and granulocyte colony-stimulating factor)

15. Contraindications to the use of corticosteroid treatment

16. Symptomatic peripheral neuropathy Grade ≥ 2 (National Cancer Institute Common

Terminology Criteria v4.0)

17. Uncontrolled severe illness or medical condition including uncontrolled diabetes mellitus,

history of cardiovascular disease (uncontrolled hypertension, arterial thrombotic events in

the past 6 months, congestive heart failure, severe or unstable angina pectoris, recent

myocardial infraction within last 6 months or uncontrolled cardiac arrhythmia)

18. Concomitant vaccination with yellow fever vaccine

Control

 abiraterone or enzalutamide

(either 1000 mg of abiraterone plus prednisone daily or 160 mg of enzalutamide daily)

Outcome(s)

primary: imaging-based progression-free survival*

secondary:

- overall survival,

- progression-free survival

- PSA response

- tumour response

- pain response

- time to symptomatic skeletal event

- safety

*defined as the time from randomization until the occurrence of one of the following events: objective tumor progression (Response Evaluation Criteria in Solid Tumors, v1.1), progression of bone lesions (Prostate Cancer Working Group 2) or death due to any cause

Results

n=255, n(I)=129 vs. n(C)=126

- n=250 were treated (n=58 received abiraterone, n=66 received enzalutamide)

- n=2 within the intervention group were classified as lost-to-follow-up

- median age: 70 years, with 31.0% of the patients being 75 years of age or older

- median duration of treatment was longer in patients receiving cabazitaxel than in those receiving an androgen-signaling–targeted inhibitor (22.0 weeks vs. 12.5 weeks)

- median follow-up: 9.2 months

- death: n=153, n(I)=70 (54.3%) vs. n(C)=83 (65.9%)

- imaging-based progression or death: (I vs. C)

- 95 of 129 patients (73.6%) vs. 101 of 126 patients (80.2%)

- hazard ratio (HR) 0.54 (95% confidence interval (CI) 0.40-0.73) P<0.001

- median imaging-based progression-free survival: (I vs. C)

- 8.0 months vs. 3.7 months

- median overall survival: (I vs. C)

- 13.6 months vs. 11.0 months

- HR 0.64 (95% CI, 0.46-0.89) P = 0.008)

- median progression-free survival: (I vs. C)

- 4.4 months vs. 2.7 months

- HR 0.52 (95% CI, 0.40-0.68) P<0.001

- prostate-specific antigen response: (I vs. C)

- 35.7% vs. 13.5% P<0.001

- tumor response: (I vs. C)

- 36.5% vs. 11.5% P = 0.004

- adverse event of any grade: (I vs. C)

- n=124 (98.4%) vs. n=117 (94.4%)

- serious adverse events of any grade: (I vs. C)

- n=49 (38.9%) vs. n=48 (39.7%)

- adverse events of grade 3 or higher: (I vs. C)

- n=71 (56.3%) vs. n=65 (52.4%)

Comments

- his was an open-label trial with no central review of the standard imaging, although a previous study has suggested little variance between local and central imaging review in this population

- no preplanned analysis of the influence of the sequence of abiraterone–enzalutamide (or vice versa) was undertaken

 

Bono_NEJM_2020_Hussain_NEJM_2020

 

de Bono J, Mateo J, Fizazi K, Saad F, Shore N, Sandhu S, et al. Olaparib for Metastatic Castration-Resistant Prostate Cancer. New England Journal of Medicine. 2020;382(22):2091-102.

Link

https://pubmed.ncbi.nlm.nih.gov/32343890/

Assessment

(Cochrane Risk of bias Tool Bewertung)

high

Design

Prospective randomized, open-label, phase 3 trial, multicenter, April 2017-November 2018

randomization was stratified according to previous taxane use (yes or no) and measurable disease (yes or no).

Eligible patients were included in one of two cohorts (A or B) depending on their qualifying gene alteration. In each cohort, patients received either olaparib or enzalutamide or abiraterone (randomly allocated).

Population

men (≥18 years of age) with confirmed metastatic castration-resistant prostate cancer whose disease had progressed during treatment with enzalutamide or abiraterone, administered for metastatic or nonmetastatic castration-resistant prostate cancer or for metastatic hormone-sensitive prostate cancer

Olaparib (n=256)

Control (n=130)

Cohort A (n=245) had at least one alteration in BRCA1, BRCA2, or ATM

Cohort B (n=142) had alterations in any of 12 other prespecified genes, prospectively and centrally determined from tumor tissue

Intervention

olaparib tablets (300 mg twice daily)

Control

enzalutamide (160 mg once daily) or abiraterone (1000 mg once daily, plus prednisone at a dose of 5 mg twice daily) (physician’s choice)

Outcome(s)

primary endpoint: imaging-based progression-free survival in Cohort A

secondary end points: objective response rate, time to pain progression, overall survival (interim analysis), a reduction of at least 50% in the concentration of prostate-specific antigen (PSA), circulating-tumor-cell conversion rate, adverse events (only assessed in the overall cohort)

Results

Cohort A (n=162 olaparib, n=83 control):

Median progression-free survival (at data maturity 71%)

Olaparib: 7.4 month

Control: 3.6 month

Hazard Ratio (HR): 0.34 (95% Confidence Interval (CI) 0.25 to 0.47) p<0.001

 

Median overall survival (interim)

Olaparib: 18.5 month

Control: 15.1 month

Hazard Ratio (HR): 0.64 (95% CI 0.43 to 0.97) p<0.02

 

Objective response rate

Olaparib: 28/84 patients (33%)

Control: 1/43 patients (2%)

Odds Ratio: 20.86 (95% CI, 4.18 to 379.18) p<0.001

 

Median time to pain progression

in favor of olaparib (HR 0.44 (95% CI, 0.22 to 0.91), p= 0.02)

Overall population (Cohort A+B, n=256 olaparib, n=131 control):

Median progression-free survival

Olaparib: 5.8 month

Control: 3.5 month

HR: 0.49 (95% CI 0.38 to 0.63) p<0.001

 

Objective response rate (among selected patients)

Olaparib: 30/138 patients (22%)

Control: 3/67 patients (2%)

Odds Ratio: 5.93 (95% CI, 2.01 to 25.40)

 

pain progression at 6 months

in favor of olaparib: HR: 0.64 (95% CI not reported)

 

Median overall survival (interim, at data maturity 41%)

Olaparib: 17.5 month

Control: 14.3 month

HR: 0.67 (95% CI 0.49 to 0.93)

 

Among patients in the control group with independent review–confirmed imaging-based progression, 82% crossed over to olaparib treatment.

 

Adverse events: werden nur zusammen, nicht getrennt nach Kohorte, berichtet:

Interruption of intervention due to adverse event: olaparib 45%, control 18%

Discontinuation of intervention due to adverse event: olaparib 18%, control 8%

Death due to adverse event: olaparib 4%, control 4%

Comments

Open label, but blinded independent central review for some of the endpoints

At the time of PFS analyses, 10 patients (4%) in the olaparib group and 8 (6%) in the control group had withdrawn consent, and their data were censored

Selective outcome reporting possible

Strong and diverse influence of pharmaceutical industries in all study aspects

 

Hussain M, Mateo J, Fizazi K, Saad F, Shore N, Sandhu S, et al. Survival with Olaparib in Metastatic Castration-Resistant Prostate Cancer. New England Journal of Medicine. 2020.

Link

https://pubmed.ncbi.nlm.nih.gov/32955174/

Assessment

(Cochrane Risk of bias Tool Bewertung)

high

Design

(see above)

Population

(see above)

(see above)

Intervention

(see above)

Control

(see above)

Outcome(s)

Overall survival in cohort A

time from randomization to a second progression (after a first event) or death

adverse events

Results

Cohort A (n=162 olaparib, n=83 control)

Median duration of overall survival

Olaparib: 19.1 month (95% CI 17.4-23.4)

Control: 14.7 month (95% CI 11.9-18.8)

HR 0.69 (95% CI 0.50 to 0.97) p=0.02

 

Sensitivity analysis (adjusted for crossover from control therapy to

olaparib)

HR 0.42 (95% CI 0.19 to 0.91)

 

Time to second progression or death

Olaparib: 15.5 month

Control: 10.6 month

HR 0.64 (95% CI 0.45-0.93)

Cohort B (n=94 olaparib, n=48 control)

Median duration of overall survival

Olaparib: 14.1 month (95% CI 11.1-15.9)

Control: 11.5 month (95% CI 8.2-17.1)

HR 0.96 (95% CI 0.63 to 1.49)

 

Sensitivity analysis (adjusted for crossover from control therapy to

olaparib)

HR 0.83 (95% CI 0.11 to 5.98)

 

Time to second progression or death

Olaparib: 9.9 month

Control: 7.9 month

HR 0.77 (95% CI 0.50-1.21)

Overall population (Cohort A+B, n=256 olaparib, n=131 control))

Median duration of overall survival

Olaparib: 17.3 month (95% CI 15.5-18.6)

Control: 14.0 month (95% CI 11.5-17.1)

HR 0.79 (95% CI 0.61 to 1.03)

 

Sensitivity analysis (adjusted for crossover from control therapy to

olaparib)

HR 0.55 (95% CI 0.29 to 1.06)

 

Time to second progression or death

Olaparib: 13.4 month

Control: 9.7 month

HR 0.68 (95% CI 0.51-0.90)

 

adverse events

Any serious adverse event: olaparib 37%, control 30%

Interruption of treatment due to adverse event: olaparib 46%, control 19%

Discontinuation of treatment due to adverse event: olaparib 20%, control 8%

Death due to adverse event: olaparib 4%, control 5%

Comments

label, but blinded independent central review for some of the endpoints

The crossover-adjusted analysis included 86 of 131 patients (66%) in the control group who had crossed over to receive olaparib; this subgroup included 83 of 99 patients (84%) who had disease progression and chose to cross over in accordance with the protocol and 3 additional patients who did not meet the crossover eligibility criteria and received olaparib outside of the trial.

Discontinuation of treatment (due to adverse events) is higher in the olaparid and crossed over from control group

Strong and diverse influence of pharmaceutical industries in all study aspects

 

7.4.2_EvT_7.46

Quelle/
Studientyp

Population

(verglichene) Interventionen/
ggf. Dosierung/
ggf.
Follow-up

Outcomes

Ergebnisse

Bemerkungen

Evidenzniveau (SIGN)

Fizazi 2012

N = 1195 (2:1 ratio, Intervention n=797, Control n=398) 

Enrollment from 05/2008 - 07/2009

Inclusion criteria (selection)

Adenocarcinoma of the prostate

Castrate level of testosterone

Progressive disease (according to PSAWG)

Max 2 chemotherapy regimens, min 1 with docetaxel

Ongoing androgen deprivation therapy

ECOG performance: 0-2

Exclusion criteria (selection):

Abnormal aminotransferase level

-Serious coexisting nonmalignant disease

Active, symptomatic hepatitis, chronic liver disease

Treatment with ketoconazol

1g abiraterone acetate + (2 x 5mg) prednisone vs. 1x1g placebo + (2 x 5mg) prednisone

Primary endpoints:

1. overall survival

Secondary outcomes (pre-specified):

2. PSA response (≥50% decline)

3. Time to PSA progression (+25% over nadir PSA value)

4. Radiographic progression free survival

5. Time to pain intensity progression

6. Time to 1st skeletal-related event

7. QoL total score and each subscale score as assessed by FACT-P

1a) Overall Survival

15.8 months (95% CI, 14.8 to 17.0)

vs. 11.2 months (95% CI, 10.4 to 13.1)

HR,  0.74 (95% CI, 0.64-0.86, p<0.001)

1b) Deaths:

61% (n=501) vs. 69% (n=274)

2) PSA response: 235 (29.5%) vs. 22 (5.5%), p<0.001

3) PSA progression: 8.5 months (95% CI, 8.3 to 11.1) vs. 6.6 months (95% CI, 5.6-8.3)

HR,  0.63 (95% CI, 0.52-0.78, p<0.001)

4) Radiographic progression free survival: 5.6 months (95% CI: 5.6-6.5 vs. 3.6 months (2.9-5.5)

HR, 0.66 (95% CI, 0.58-0.76, p<0.0001)

5+ 6) reported in Logothesis et al. Lancet Oncol 2012 [9315] see below

7) not (yet) reported

 

Study stopped (crossover) based on interim analyses after 775 events as recommended by DMC

At study stop: 16% (n=125) received abiraterone acetate  vs. 5% (n=18) placebo

147 centers in 13 countries, enrollment procedures not specified.

Time delay ( ?) between interim-analyses (after 520 events) and study stop unclear

Proportional Hazard requirements for primary endpoint not met, hazard ratio should be interpreted with caution

Median duration of drug exposure 7.4 months vs. 3.6 months,

Median duration of follow-up: 20.2 months

ITT, Double-blinded, randomized

Stratification based on ECOG performance status score (0 vs. 1 vs. 2), BPI-SF (question 3, pain), number of previous chemotherapy regimen, type of PSA progression

Adverse events:

higher rate of mineralocorticoid-related events, fluid retention, edema, hypokalemia, cardiac events 

Conflict of Interest: reported (ICMJE forms attached)

Flowchart ; Lost-to-follow-up: n=0, patients without intervention: n=6 vs. n=4

At baseline (and at initial diagnosis): slightly higher median PSA

Sponsor: Janssen Research & Development Employees of Janssen R&D participated in all parts of the study; corresponding author had full access to all data and final responsibility for the publication.

1+

De Bono 2011

COU-AA-301

Is interim analysis of Fizazi et al  2012 Lancet Oncology  [9316] (see above)

 

 

 

 

 

Logothesis 2012

Is exploratory analysis of Fizazi et al  2012 Lancet Oncology  [9316] (see above)

 

 

5)a) Time to pain intensity progression

5)b) Time to pain interference progression

6) Occurrence of first skeletal-related event:

25.0 months (95% CI 25.0-not estimable) vs.

20.3 months (95%CI 16.9-not estimable)

HR: 0.615 (95% CI 0.478-0.791

Exploratory endpoints, Time to pain intensity progression (n=12 data missing (intervention group), Time to pain interference progression (n=37 (22+15) data missing.

Data as a proxy-outcome for QoL (evaluated, but not (yet) reported) or for morbidity

1+

Scher 2012

AFFIRM

N = 1299 (2:1 ratio, Intervention n=800, Control n=399) 

Enrollment from 09/2009-11/2010

Inclusion criteria (selection)

Adenocarcinoma of the prostate

Castrate level of testosterone

Progressive disease (according to PCWG2)

Max 2 chemotherapy regimens, min 1 with docetaxel

Ongoing androgen deprivation therapy or orchiectomy

ECOG performance: 0-2

Estimated life expectancy ≥ 6 months

Exclusion criteria (selection):

Severe concurrent disease

Metastases in brain

Treatment with 5-α reductase inhibitors, androgen receptor agonist, therapeutic immunization

Treatment with ketoconazole

160 mg enzalutamide vs. placebo (once daily)

Primary endpoint:

1. overall survival

Secondary endpoints (pre-specified):

[Rank-prioritized]

2. Time to PSA progression

3. Radiographic progression free survival

4. Time to first skeletal-related event

[no ranking]

5. Quality of life (FACT-P-response: 10-point improvement compared to baseline)

Secondary endpoints (exploratory)

6. PSA response (≥50% decline, ≥90% decline)

1a) Overall Survival

18.4 months (95% CI, 17.3 to not yet reached)

vs. 13.6 months (95% CI, 11.3 to 15.8)

HR,  0.63 (95% CI, 0.53-0.75, p<0.001)

1b) Deaths

39% (n=308) vs. 53% (n=212)

2) PSA Progression

8.3 months (95% CI, 5.8-8.3, p<0.001) vs.  3.0 months (95% CI, 2.9-3.7, p<0.001),

(HR, 0.25 (95% CI, 0.20-0.30, p<0.001)

3) Radiographic progression free survival 8.3 months (8.2-9.4) vs. 2.9 months (2.8-3.4) (HR, 0.40 (95% CI, 0.35-0.47, p<0.001)

4) Time to first skeletal-related event

16.7 months (14.6-19.1) vs. 13.3 months (9.9-not yet reached)

5) QoL 43% vs. 18% (data available 81% vs. 64%)

6) PSA response: 54% vs. 2%, 25% vs.  1%

Study stopped (crossover) after interim analyses (520 events) as recommended by Data Monitoring Committee (DMC)

156 centers in 15 countries (no specification regarding sites or countries, No differences between geographic region (North America vs. Others) ; enrollment procedures not specified.

Median time on treatment: 8.3 months vs. 3.0 months, median duration of follow-up: 14.4 months

ITT, Double-blinded, randomized

Stratification based on ECOG performance status score (0 vs. 1 vs. 2) and BPI-SF (question 3, pain)

Adverse events:

higher incidence of  all grades of fatigue, diarrhea, hot flashes, musculoskeletal pain, and headache in the enzalutamide group;

higher rate of hypertension or increased blood pressure.

n= 5 (0.6%) with seizures compared to n=0 in placebo

Conflict of Interest: reported (ICMJE forms attached)

Flowchart in supplement: Lost-to-follow-up: n=0, Discontinuation of intervention: 71% vs. 95% (mainly due to disease progression)

At baseline: control group is slightly older, slightly more patients with Gleason>7, slightly higher median PSA

At baseline patients with orchiectomy  5.5% (n=44) vs. 5.8% (n=23)

Sponsor: Medivation and Astellas Pharma Global Development

1+

De Bono 2010

TROPIC

EFC6193

N=755 (1:1 ratio, cabazitaxel n=378, mitoxantrone n=377) 

Enrollment from 01/2007 - 10/2008

Inclusion criteria (selection)

Pathologically proven prostate cancer

Documented disease progression during or after docetaxel treatment

ECOG: 0-2

Previous or ongoing castration

Exclusion criteria (selection):

Previous treatment with mitoxantrone, radiotherapy to ≥ 40% of bone marrow, cancer therapy (other than LHRH analogues) within 4 weeks of enrolment

Serious illnesses

 

12 mg/m2 cabazitaxel (i.v., 3-weekly) + (10 mg) prednisone (daily)

vs. 25mg/m2 mitoxantrone (i.v., 3-weekly) + (10 mg) prednisone (daily)

Primary endpoint:

1. overall survival

Secondary endpoints:

2. progression-free survival (composite endpoint)

3. PSA response (≥50% decline, only for pts with baseline –PSA ≥20µg/L)

4. PSA progression (≥25% increase)

5. Pain progression (only for pts with PPI≥2 or analgesic score≥10

(..)

 

1a) Overall survival:

15.1 months (95% CI 14.1-16.3) vs.

12.7 months  (95% CI, 11.6-13.7)

(HR: 0.70, 95% CI, 0.59-0.83, p<0.001)

1b) Death:

62% (234/378) vs.74% (279/377)

2) Progression-free survival:  2.8 months (95% CI 2.4-3.0) vs. 1.4 (95% CI 1.4-1.7)

(HR 0.74, 95% CI: 0.64-0.86, p<0.001)

3) PSA response rate: 39.2% (33.9-44.5; n=329(87%))

vs. 17.8% (13.7-22.0, n=325(86%))

(p=0.0002)

4) median time to PSA progression:

6.4 months (2.2-10.1)  vs. 3.1 months (0.9-9.1)

(HR 0.75, 95% CI:0.63-0.90, p=0.001)

5) median time to pain progression: 11.1 (2.9- not reached) vs. not reached

HR 0.91 (95% CI 0.69-1.19, p=0.52)

Baseline: mitoxantrone group more patients with bone metastases, lower median PSA, more patients with only 1 previous docetaxel regimen, lower total previous docetaxel dose.

Study conducted in 146 centers in 26 countries

Median follow-up duration: 12.8 months

ITT, open-label, study team masked to data analysis, randomized

Stratification based on ECOG performance status score (0-1 vs. 2) and disease measurability

Adverse events:

Higher rates of most adverse events in the cabazitaxel group (e.g. neutropenia (94%), anemia (97%), and diarrhea (47%)

18% (n=67) discontinued with study due to adverse event (mitoxantrone: 8% (n=32))

Conflict of Interest reports relatively limited

28% (n=105) completed study treatment in cabazitaxel group, 12% (n=46) in mitoxantrone group; reason for discontinuation:  disease progression (mitoxantrone 71%,n=267 vs. cabazitaxel 48%, n=180)

Sponsor: Sanofi-Aventis

1+

 

 

Autor, Jahr/
Studientyp

Anzahl der Patienten/
Patientenmerkmale

Intervention/
ggf. Nachverfolgung

Vergleichs-intervention

Outcomes

Ergebnisse

Bemerkungen

Evidenzniveau
SIGN

Fizazi K. et al, 2011

RCT

Multizentrisch

N=1904

Pat. mit  kastrationsresistenem PCa und Knochenmetastasen ohne vorausgegangene Bisphosphonattherapie

950 Pat mit

Denosumab

120mg s.c. +i.v. Placebo

Med. Follow up : 12,2Mo

951 Pat. mit Zoledronsäure i.v.4mg + s.c. Placebo

Med. Follow up :

11,2 Mo

 

Zeit bis zum Auftreten des 1. Skeletal Related Events (SRE)

Rate an Progression

Gesamtüberleben

Nebenwirkungen

Lebensqualität

Anteil Patienten mit SRE

Denosumab: 36% (n=341)

Zoledronsäure: 41% (n=386)

1. Zeit bis zum Auftreten des 1. skeletal Related Events (Monate)

Denosumab-Gruppe: 20,7 [95%KI 18,8-24,9%]

 Zoledronsäure-Gruppe: 17,1 [95%KI 15-19,4]

Cave: bei Betrachtung des absoluten Unterschieds überlappen die Konfidenzintervalle!

Nichtunterlegenheit Denosumab: HR 0,82 [95%KI 0,71-0,92] p=0,0002

Überlegenheit Denosumab: s.o. p=0,008

2. Rate an Progression

Denosumab + Zoledronsäure im Median 8,4 Mo n.s.

3. Gesamtüberleben
Denosumab im Median 19,4 Mo

Zoledronsäure im Median 19,8 Mo n.s.

4. Nebenwirkungen

Viele NW n.s., hier nur aufgeführt die mit Unterschieden

a. Grade 3 o. 4 adverse events nach CTCAE (Common Terminology Criteria for Adverse Events )

Denosumab Gruppe: 72%

Zoledronsäure Gruppe 66% p< 0,01

b. Hypokalzämie

Denosumab Gruppe: 13%

Zoledronsäure Gruppe:  6% p< 0,0001

c. Kiefernekrose

Denosumab Gruppe: 2% (n=22)

Zoledronsäure Gruppe:  1% (n=8) p = 0,09

d. Auftreten eines weiteren Malignoms

Denosumab Gruppe: 2% (n=18)

Zoledronsäure Gruppe:  1% (n=10) p = 0,13

5. Lebensqualität: nicht erfasst.

Randomisierung ok.

in Gruppe mit Zoledronsäure: 4% mehr Patienten>65J , 3,7Mo länger von ED bis Randomisierung., 2,5 Mo länger von ED Knochenmet. bis Randomisierung.

Studie ist von AMGEN gesponsert. (AMGEN sammelte die Daten und machte die Auswertung)

Studie als Non-inferioeritätsstudie angelegt, sekundär auch Auswertung auf Superiorität

1+

 

 

Artikel (Autor, Jahr, Journal)

Studientyp

Anzahl der Patienten

Patientenmerkmale

Intervention

Vergleichs-Intervention

Nachverfolgung

Outcomes

Ergebnisse

Bemerkungen

Evidenz-Niveau (SIGN)

Saad 2002

RCT, dreiarmig

n=643

Pat. mit fortgeschrittenem Pca im hormonrefraktärem Stadium und Knochenmetastasen

 

1.n=214

Zoledronsäure 4 mg alle 3 Wo

 

2. n=221

Zoledronsäure 8mg dann reduziert auf 4mg alle 3 Wochen

 

3. n= 208

Placebo

 

15 Mo

primär: Skelettale Kom  plikationen (pro 3 Wochen wurde 1 Ereignis gezählt)

sekundär: Zeit bis zum Auftreten der SREs, Lebensqualität

1. Skelettale Komplikationen

Placebo:                      44,2%

Zoledronsäure 4mg:    33,2%   p=0,02

[95%KI -20,8% bis -1,8%]

statistisch signifikanter Unterschied von 11% relativ zugunsten von Zoledronsäure

 

2. Zeit bis zum Auftreten von skelettalen Komplikationen

Placebo: median 321 Tage

Zoledronsäure: nach 15 Mo Median noch nicht erreicht  p=0,01

 

Unterschied im Anstieg des SchmerzScores (BPI) nicht signifikant

 

Studie wurde von 38% mit Zoledronsäure 4mg und 31% mit Placebo beendet

in der Anlage dreiarmige Studie , der Arm mit Zoledronsäure 8mg wurde wegen der Nebenwirkungen (Nierenfunktion) auf 4mg reduziert

1+

           

 

Autoren Jahr

Literaturtyp

untersuchte Studien/
Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

Methodische Besonder­
heiten/
Bemerkungen

LoE

Literaturbelege

Yuen 2006

Syst. Review/ Meta-analyse

10 RCTs aus syst. Suche in Medline, Embase, DARE, AMED, CENTRAL, LILACS bis April 2005

Bisphosphonate vs. Placebo o. keine Therapie bei Knochen-Metastasen durch PCa

Signifikante Ergebnisse:

bessere Schmerzreduktion in Bisphosphonat-Gruppe (OR 1,54, 95%CI 0,97-2,44, p=0,07 bzw. nach Re-Analyse OR 1,64, 95%CI 1,02-2,61, p=0,04)

weniger „skeletal events“ (path. Fraktur, Hyperkalzämie, Indikation zur Radiotherapie, Rückenmarkkompression, Indikation zur Chirurgie) in Bisphosphonat-Gruppe (OR 0,79, 95%CI 0,62-1,0, p=0,05)

mehr Übelkeit in Bisphosphonat-Gruppe (OR 1,35, 95%CI 1,02-1,77, p=0,03)

nicht signifikant:

mittlerer Schmerz-Score, Analgetika-Verbrauch, Frakturen, Rückenmarkkompression, Knochenbestrahlung, Knochenchirurgie, OS, DP, radiologisches Ansprechen, QoL, Erbrechen, Anämie, Knochenschmerz, Nierentoxizität

untersch. Bisphosphonate, wenige signifikante Ergebnisse, unterschiedl. Definitionen der Studien, signifikante Schmerzreduktion signifikant in Abhängigkeit von Analyse-Technik

1+

Adami 1989, Dearnaley 2003, Elomma 1992, Ernst 2003, Kylmala 1993, Kylmala 1997, Saad 2002, Small 2003, Smith 1989, Strang 1997

 

7.4.2_EvT_7.47

Quelle/
Studientyp

Population

(verglichene) Interventionen/
ggf. Dosierung/
ggf.
Follow-up

Outcomes

Ergebnisse

Bemerkungen

Evidenzniveau (SIGN)

Fizazi 2012

N = 1195 (2:1 ratio, Intervention n=797, Control n=398) 

Enrollment from 05/2008 - 07/2009

Inclusion criteria (selection)

Adenocarcinoma of the prostate

Castrate level of testosterone

Progressive disease (according to PSAWG)

Max 2 chemotherapy regimens, min 1 with docetaxel

Ongoing androgen deprivation therapy

ECOG performance: 0-2

Exclusion criteria (selection):

Abnormal aminotransferase level

-Serious coexisting nonmalignant disease

Active, symptomatic hepatitis, chronic liver disease

Treatment with ketoconazol

1g abiraterone acetate + (2 x 5mg) prednisone vs. 1x1g placebo + (2 x 5mg) prednisone

Primary endpoints:

1. overall survival

Secondary outcomes (pre-specified):

2. PSA response (≥50% decline)

3. Time to PSA progression (+25% over nadir PSA value)

4. Radiographic progression free survival

5. Time to pain intensity progression

6. Time to 1st skeletal-related event

7. QoL total score and each subscale score as assessed by FACT-P

1a) Overall Survival

15.8 months (95% CI, 14.8 to 17.0)

vs. 11.2 months (95% CI, 10.4 to 13.1)

HR,  0.74 (95% CI, 0.64-0.86, p<0.001)

1b) Deaths:

61% (n=501) vs. 69% (n=274)

2) PSA response: 235 (29.5%) vs. 22 (5.5%), p<0.001

3) PSA progression: 8.5 months (95% CI, 8.3 to 11.1) vs. 6.6 months (95% CI, 5.6-8.3)

HR,  0.63 (95% CI, 0.52-0.78, p<0.001)

4) Radiographic progression free survival: 5.6 months (95% CI: 5.6-6.5 vs. 3.6 months (2.9-5.5)

HR, 0.66 (95% CI, 0.58-0.76, p<0.0001)

5+ 6) reported in Logothesis et al. Lancet Oncol 2012 [9315] see below

7) not (yet) reported

 

Study stopped (crossover) based on interim analyses after 775 events as recommended by DMC

At study stop: 16% (n=125) received abiraterone acetate  vs. 5% (n=18) placebo

147 centers in 13 countries, enrollment procedures not specified.

Time delay ( ?) between interim-analyses (after 520 events) and study stop unclear

Proportional Hazard requirements for primary endpoint not met, hazard ratio should be interpreted with caution

Median duration of drug exposure 7.4 months vs. 3.6 months,

Median duration of follow-up: 20.2 months

ITT, Double-blinded, randomized

Stratification based on ECOG performance status score (0 vs. 1 vs. 2), BPI-SF (question 3, pain), number of previous chemotherapy regimen, type of PSA progression

Adverse events:

higher rate of mineralocorticoid-related events, fluid retention, edema, hypokalemia, cardiac events 

Conflict of Interest: reported (ICMJE forms attached)

Flowchart ; Lost-to-follow-up: n=0, patients without intervention: n=6 vs. n=4

At baseline (and at initial diagnosis): slightly higher median PSA

Sponsor: Janssen Research & Development Employees of Janssen R&D participated in all parts of the study; corresponding author had full access to all data and final responsibility for the publication.

1+

De Bono 2011

COU-AA-301

Is interim analysis of Fizazi et al  2012 Lancet Oncology  [9316] (see above)

 

 

 

 

 

Scher 2012

AFFIRM

N = 1299 (2:1 ratio, Intervention n=800, Control n=399) 

Enrollment from 09/2009-11/2010

Inclusion criteria (selection)

Adenocarcinoma of the prostate

Castrate level of testosterone

Progressive disease (according to PCWG2)

Max 2 chemotherapy regimens, min 1 with docetaxel

Ongoing androgen deprivation therapy or orchiectomy

ECOG performance: 0-2

Estimated life expectancy ≥ 6 months

Exclusion criteria (selection):

Severe concurrent disease

Metastases in brain

Treatment with 5-α reductase inhibitors, androgen receptor agonist, therapeutic immunization

Treatment with ketoconazole

160 mg enzalutamide vs. placebo (once daily)

Primary endpoint:

1. overall survival

Secondary endpoints (pre-specified):

[Rank-prioritized]

2. Time to PSA progression

3. Radiographic progression free survival

4. Time to first skeletal-related event

[no ranking]

5. Quality of life (FACT-P-response: 10-point improvement compared to baseline)

Secondary endpoints (exploratory)

6. PSA response (≥50% decline, ≥90% decline)

1a) Overall Survival

18.4 months (95% CI, 17.3 to not yet reached)

vs. 13.6 months (95% CI, 11.3 to 15.8)

HR,  0.63 (95% CI, 0.53-0.75, p<0.001)

1b) Deaths

39% (n=308) vs. 53% (n=212)

2) PSA Progression

8.3 months (95% CI, 5.8-8.3, p<0.001) vs.  3.0 months (95% CI, 2.9-3.7, p<0.001),

(HR, 0.25 (95% CI, 0.20-0.30, p<0.001)

3) Radiographic progression free survival 8.3 months (8.2-9.4) vs. 2.9 months (2.8-3.4) (HR, 0.40 (95% CI, 0.35-0.47, p<0.001)

4) Time to first skeletal-related event

16.7 months (14.6-19.1) vs. 13.3 months (9.9-not yet reached)

5) QoL 43% vs. 18% (data available 81% vs. 64%)

6) PSA response: 54% vs. 2%, 25% vs.  1%

Study stopped (crossover) after interim analyses (520 events) as recommended by Data Monitoring Committee (DMC)

156 centers in 15 countries (no specification regarding sites or countries, No differences between geographic region (North America vs. Others) ; enrollment procedures not specified.

Median time on treatment: 8.3 months vs. 3.0 months, median duration of follow-up: 14.4 months

ITT, Double-blinded, randomized

Stratification based on ECOG performance status score (0 vs. 1 vs. 2) and BPI-SF (question 3, pain)

Adverse events:

higher incidence of  all grades of fatigue, diarrhea, hot flashes, musculoskeletal pain, and headache in the enzalutamide group;

higher rate of hypertension or increased blood pressure.

n= 5 (0.6%) with seizures compared to n=0 in placebo

Conflict of Interest: reported (ICMJE forms attached)

Flowchart in supplement: Lost-to-follow-up: n=0, Discontinuation of intervention: 71% vs. 95% (mainly due to disease progression)

At baseline: control group is slightly older, slightly more patients with Gleason>7, slightly higher median PSA

At baseline patients with orchiectomy  5.5% (n=44) vs. 5.8% (n=23)

Sponsor: Medivation and Astellas Pharma Global Development

1+

 

7.4.3.1 EvT 7.42

Quelle/
Studientyp

Population

(verglichene) Interventionen/
ggf. Dosierung/
ggf.
Follow-up

Outcomes

Ergebnisse

Bemerkungen

Evidenzniveau (SIGN)

De Bono 2010

TROPIC

EFC6193

N=755 (1:1 ratio, cabazitaxel n=378, mitoxantrone n=377) 

Enrollment from 01/2007 - 10/2008

Inclusion criteria (selection)

Pathologically proven prostate cancer

Documented disease progression during or after docetaxel treatment

ECOG: 0-2

Previous or ongoing castration

Exclusion criteria (selection):

Previous treatment with mitoxantrone, radiotherapy to ≥ 40% of bone marrow, cancer therapy (other than LHRH analogues) within 4 weeks of enrolment

Serious illnesses

 

12 mg/m2 cabazitaxel (i.v., 3-weekly) + (10 mg) prednisone (daily)

vs. 25mg/m2 mitoxantrone (i.v., 3-weekly) + (10 mg) prednisone (daily)

Primary endpoint:

1. overall survival

Secondary endpoints:

2. progression-free survival (composite endpoint)

3. PSA response (≥50% decline, only for pts with baseline –PSA ≥20µg/L)

4. PSA progression (≥25% increase)

5. Pain progression (only for pts with PPI≥2 or analgesic score≥10

(..)

 

1a) Overall survival:

15.1 months (95% CI 14.1-16.3) vs.

12.7 months  (95% CI, 11.6-13.7)

(HR: 0.70, 95% CI, 0.59-0.83, p<0.001)

1b) Death:

62% (234/378) vs.74% (279/377)

2) Progression-free survival:  2.8 months (95% CI 2.4-3.0) vs. 1.4 (95% CI 1.4-1.7)

(HR 0.74, 95% CI: 0.64-0.86, p<0.001)

3) PSA response rate: 39.2% (33.9-44.5; n=329(87%))

vs. 17.8% (13.7-22.0, n=325(86%))

(p=0.0002)

4) median time to PSA progression:

6.4 months (2.2-10.1)  vs. 3.1 months (0.9-9.1)

(HR 0.75, 95% CI:0.63-0.90, p=0.001)

5) median time to pain progression: 11.1 (2.9- not reached) vs. not reached

HR 0.91 (95% CI 0.69-1.19, p=0.52)

Baseline: mitoxantrone group more patients with bone metastases, lower median PSA, more patients with only 1 previous docetaxel regimen, lower total previous docetaxel dose.

Study conducted in 146 centers in 26 countries

Median follow-up duration: 12.8 months

ITT, open-label, study team masked to data analysis, randomized

Stratification based on ECOG performance status score (0-1 vs. 2) and disease measurability

Adverse events:

Higher rates of most adverse events in the cabazitaxel group (e.g. neutropenia (94%), anemia (97%), and diarrhea (47%)

18% (n=67) discontinued with study due to adverse event (mitoxantrone: 8% (n=32))

Conflict of Interest reports relatively limited

28% (n=105) completed study treatment in cabazitaxel group, 12% (n=46) in mitoxantrone group; reason for discontinuation:  disease progression (mitoxantrone 71%,n=267 vs. cabazitaxel 48%, n=180)

Sponsor: Sanofi-Aventis

1+

 

7.4.2 EvT 7.37

Quelle/
Studientyp

Population

(verglichene) Interventionen/
ggf. Dosierung/
ggf.
Follow-up

Outcomes

Ergebnisse

Bemerkungen

Evidenzniveau (SIGN)

Parker 2013

 

N = 921 (2:1 ratio, Intervention n=614, Control n=307) 

Enrollment from 06/2008-02/2011

Inclusion criteria (selection)

Progressive castration-resistant PCa (defined as a serum testosterone level of 50 ng per deciliter or lower (≤1.7 nmol per liter) after bilateral orchiectomy or during maintenance treatment

two or more bone metastases detected on skeletal scintigraphy

no known visceral metastases;

had received docetaxel, were not healthy enough or declined to receive it, or it was not available.

symptomatic disease with regular use of analgesic medication or treatment with external-beam radiation therapy required for cancer related bone pain within the previous 12 weeks.

ECOG performance: 0-2

Baseline PSA≥5 ng/mL with two consecutive increases of PSA values over the previous reference value

Life expectancy ≥ 6 months

Exclusion criteria (selection):

History of presence of visceral metastases

Chemotherapy within the previous 4 weeks

6 injections of radium-223 (at a dose of 50 kBq per kilogram of body weight intravenously) vs. matching placebo;

One injection was administered every 4 weeks.

Additionally patients in both groups received “best standard of care” defined as routine care provided at each centre (e.g. local external-beam radiation therapy, treatment with glucoccorticoids, antiandrogens, ketoconazole, estrogens, estramustine)

 

Primary endpoint:

1. overall survival (defined as time to death)

Secondary endpoints (pre-specified):

2. Time to increase in PSA level

3. Time to first symptomatic skeletal event (clinically defined)

4. QoL (FACT-P): increase of score of ≥ 10 (during period of study-drug administration)

 

1) Overall Survival

1a) interim analyses (n=809):

14.0 months (95% CI not reported in publication)

vs. 11.2 months (95% CI not reported in publication)

HR,  0.70 (95% CI, 0.55-0.88, p=0.002)

Death: 35% (191/541) vs. 64% (123/268)

1b) updated analyses (n=921)

14.9 months (95% CI not reported in publication)

vs. 11.3 months (95% CI not reported in publication)

HR,  0.70 (95% CI, 0.58-0.83, p<0.001)

Death: 54% (333/614) vs. 64% (195/307)

2) Time to increase in PSA level (Updated analyses, n=921)

3.6 months (95% CI not reported in publication)

vs. 3.4 months (95% CI not reported in publication)

HR, 0.64 (95% CI, 0.54-0.77), p<0.001

3) Time to first symptomatic skeletal event (Updated analyses, n=921)

15.6 months (95% CI not reported in publication)

vs. 9.8 months (95% CI not reported in publication)

HR,  0.66 (95% CI, 0.52-0.83, p<0.001)

4) QoL (FACT-P increase of score of ≥ 10)

25% vs. 12% (p=0.02)

(no numbers of pts available)

 

 

Study stopped after interim analyses for efficacy as recommended by Data Monitoring Committee (DMC)

Interim analyses conducted in n=809 patients (n=541 in the radium-223 group, n=268 in the placebo group)

ITT, Double-blinded, randomized

136 centers in 19 countries (no specification regarding sites or countries in publication)

Stratification according to previous use or nonuse of docetaxel, baseline alkaline phosphatase level, current use or nonuse of bisphosphonate

57% with prior docetaxel therapy, 43% without prior docetaxel therapy

Numbers, reasons and distribution of patients between groups without prior docetaxel therapy not described

In the exploratory subgroup analyses: no significant benefit for radium for patients with ECOG ≥ 2 (n=118 patients), for patients with <6 metastases (n=138) and for patients with ALP<220 U/liter (n=517)

Adverse events: more patients in the radium group had thrombocytopenia (12% vs. 6%), neutropenia (5% vs. 1%) and diarrhea (25% vs. 15%)

Sponsor: Algeta and Bayer Health Care

1+

 

 

 

Vogelzang_ClinGenitourinCancer_2017

 

Vogelzang, Nicholas J.; Coleman, Robert E.; Michalski, Jeff M.; Nilsson, Sten; O'Sullivan, Joe M.; Parker, Christopher et al. (2017): Hematologic Safety of Radium-223 Dichloride. Baseline Prognostic Factors Associated With Myelosuppression in the ALSYMPCA Trial. Clin Genitourin Cancer. 2017 Feb;15(1):42-52.e8.doi: 10.1016/j.clgc.2016.07.027.

Link

https://pubmed.ncbi.nlm.nih.gov/27613490/

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: low

Attrition bias

lost to follow-up: unclear

ITT-Analyse: unclear

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the study was designed, conducted, and analyzed by employees of the sponsors, in collaboration with the investigator

Design

randomized, double-blind, placebo-controlled phase 3 study

ALSYMPCA, NCT00699751, Parker N Engl J Med 2013, Sartor Lancet Oncol 2014*

radium-223 hematologic safety in ALSYMPCA

Population

ALSYMPCA, n=921, n(I)=614 vs. n(C)=307

safety population n(I)=600, n(C)=301 (patients who received ≥ 1 study-drug injection)

planned follow-up: 3 years

comment: data were not collected on the number of previous docetaxel doses or the cumulative docetaxel dose received; concomitant chemotherapy was not permitted during treatment

Inclusion criteria: (see also Parker N Engl J Med 2013)

- symptomatic and progressing CRPC and ≥2 bone metastases

- no visceral metastases

- elevated and rising prostate-specific antigen (PSA)

- Eastern Cooperative Oncology Group performance status ≤2

- life expectancy ≥6 months

- adequate baseline hematologic#, renal, and liver functions

- patients either had previous docetaxel treatment or were unsuitable for or declined docetaxel

Exclusion criteria: (see also Parker N Engl J Med 2013)

# comment: adequate hematologic function was defined as absolute neutrophil count ≥ 1.5 x 109/L, platelet count ≥ 100 x 109/L, and hemoglobin ≥ 10.0 g/dL.

Intervention

radium-223 50 kBq/kg (55 kBq/kg following the National Institute of Standards and Technology [NIST] update) plus BSoC* every 4 weeks for 24 weeks

Control

placebo plus BSoC* every 4 weeks for 24 weeks

* BSoC: routine care provided at each center (including external beam radiation therapy for bone pain as indicated)

Outcome(s)

primary end point: overall survival (OS)

safety was a secondary end point

incidence and severity of hematologic and nonhematologic treatment-emergent AEs, clinical chemistry, electrocardiogram, and physical examination; relationship to treatment was reported as judged by the investigator

all AEs occurring after randomization and within 12 weeks after the last study-drug injection were reported and evaluated for potential relationship to study drug

AEs were graded per Common Terminology Criteria for Adverse Events (CTCAE), version 3.0. Grade 3/4 thrombocytopenia was based on investigator’s reporting of AEs, which included relevant symptoms, and not based solely on platelet laboratory values

blood samples for hematology were to be analyzed in the local laboratory and evaluated within 24 hours before each study-drug administration

Results

baseline factors significantly associated with increased risk for grade 2-4 hematologic AE

for radium-223 (anemia):

- greater extent of disease (6-20 vs. < 6 metastases; odds ratio (OR) 2.76; P = .022)

- higher PSA (OR 1.65; P = .006)

for radium-223 (neutropenia):

- prior docetaxel therapy (OR 3.44; P = .020)

- World Health Organization (WHO) ladder cancer pain score (2 vs. 3) (OR 3.58; P = .042)

for radium-223 (thrombopenia):       

- prior docetaxel therapy (OR 2.16; P = .035)

- decreases in baseline hemoglobin (OR 1.35; P = .008)

- decreases in baseline platelets (OR 1.44; P = .030)

radium-223:

- median hemoglobin decreased from 12.2 g/dL at baseline to 11.4 g/dL at treatment end (week 24) and 10.8 g/dL at follow-up visit 2 (week 44)

- median absolute neutrophil counts (ANCs): baseline 4.5 x 109/L; week 24, 3.3 x 109/L; and week 44, 3.9 x 109/L

- platelet count: baseline 244 x 109/L, week 202 x 109/L, week 44 216 x 109/L

placebo:

- hemoglobin decreased from 12.1 g/dL at baseline to 11.6 g/dL and 11.2 g/dL at weeks 24 and 44, respectively

- median absolute neutrophil counts (ANCs): 4.6 x 109/L, 4.5 x 109/L, and 4.3 x 109/L, respectively

- platelet count: 240 x 109/L, 232 x 109/L, and 218 x 109/L, respectively


outcome*

ALSYMPCA (I) n=600, safety population

ALSYMPCA (C) n=301, safety population

AE

 

all

Grade 2

Grade 3

Grade 4

Grade 5

 

all

Grade 2

Grade 3

Grade 4

Grade 5

                 all

Grade 2

Grade 3

Grade 4

Grade 5

AE**

Anemia

187 (31)

97 (16)

66 (11)

11 (2)

0

Neutropenia

30 (5)

12 (2)

9 (2)

4 (1)

0

Thrombocytopenia

69 (12)

19 (3)

20 (3)

18 (3)

1 (<1)

AE***

Leukopenia

25 ( (4)

14 (2)

7 (1)

1 (<1)

0

Lymphopenia

5 (1)

2 (<1)

3 (1)

0

0   Pancytopenia

12 (2)

4 (1)

4 (1)

3 (1)

0 

AE**

Anemia

92 (31)

44 (15)

37 (12)

2 (1)

1 (<1)

Neutropenia

3 (1)

1 (<1)

2 (1)

0

0

Thrombocytopenia

17 (6)

7 (2)

5 (2)

1 (<1)

0

AE***

Leukopenia

1 (<1)

0

1 (<1)

0

0

Lymphopenia

1 (<1)

0

1 (<1)

0

0

 Pancytopenia

0

0

0

0

0

         

** hematologic AE (adverse events) occurring in ≥5% of patients, n (%)

*** additional hematologic AE occurring in ≥1 Radium-223 patient, n (%)

P value (all grade AE):

anemia not significant, neutropenia 0.002, thrombocytopenia 0.005

Comments

publication of ALSYMPCA (also included in the 2 included systematic reviews above):

*Parker C, Nilsson S, Heinrich D et al. Alpha emitter radium-223 and survival in metastatic prostate cancer. N Engl J Med 2013; 369: 213–23

Sartor O, Coleman R, Nilsson S et al. Effect of radium-223 dichloride on symptomatic skeletal events in patients with castration-resistant prostate cancer and bone metastases: results from a phase 3, double-blind, randomised trial. Lancet Oncol 2014; 15: 738–46

 

Smith_LancetOncol_2019

 

Smith M, Parker C, Saad, F, Miller K; Tombal B, Ng QS et al. (2019): Addition of radium-223 to abiraterone acetate and prednisone or prednisolone in patients with castration-resistant prostate cancer and bone metastases (ERA 223). A randomised, double-blind, placebo-controlled, phase 3 trial. Lancet Oncol. 2019 Mar;20(3):408-419. doi: 10.1016/S1470-2045(18)30860-X.

Link

https://pubmed.ncbi.nlm.nih.gov/30738780/

Assessment

(Risk of bias Tool)

Selection bias

randomization: low

concealment and unpredictability: low

Performance bias

blinding of participants and staff: low

Detection bias

blinding of evaluation: unclear

Attrition bias

lost to follow-up: unclear

ITT-analysis: unclear

Reporting bias

selective result presentation: low

Other bias

conflict of interest/ sponsoring: high

comment: the study funder had roles in study design, data collection, data analysis, and data interpretation, and writing of the report

Design

  randomized, double-blind placebo-controlled, phase 3 trial

  ERA 223, NCT02043678

Population

ITT: n=806 (n(I)=401 vs. n(C)=405) (enrolled between March 2014 and Aug 2016)

median age (I) 71 (65-77) vs (C) 71 (66-77)

Gleason score at diagnosis n (%) I vs. C:

  <8 149 (35) vs. 154 (38)

  ≥8 246 (61) vs. 233 (58)

  missing 15 (4) vs. 18 (4)

Inclusion criteria:

- medically or surgically castrated patients aged ≥18 years           

- histologically confirmed, progressive, asymptomatic or mildly symptomatic, castration-resistant adenocarcinoma of the prostate (mCRPC)

- at least two bone metastases on bone scan

- no known visceral or brain metastasis

- blood testosterone concentration < 50 ng/dL (1.7 nmol/L)

- patients not undergone orchiectomy had to have received luteinizing-hormone-releasing hormone agonists or antagonists for at least 4 weeks before randomization (continue throughout the study)

- patients received anti-androgen had to have shown PSA progression after discontinuation of the anti-androgen before enrolement

- ECOG performance status of 0 or 1, life expectancy of at least 6 months, adaequate haematological, renal, and liver function

Exclusion criteria:

- previous treatment with cytotoxic chemotherapy for CRPC or abiraterone+prednisone/prednisolone

- uncontrolled hypertension or clinically significant heart disease

- previous hemibody external radiotherapy

Intervention

radium-223 dichloride (six intravenous injections at a dose of 55 kBq/kg, interval of 4 weeks) in combination with abiraterone + prednisone/prednisolone

Control

abiraterone + prednisone/prednisolone + placebo

Outcome(s)

primary: symptomatic skeletal event-free survival (defined as the time from randomization to first symptomatic skeletal event or death from any cause)

secondary: overall survival (OS), time to opiate use for cancer (OuC), to cytotoxic chemotherapy (CC), to radiological-progression-free survival (rPFS), to pain, and safety

Results

ITT n=806 (n(I)=401 vs. n(C)=405)  

safety population n(I)=392 vs. n(C)=394 (results see also Table 5 within the publication)

study was unblinded prematurely by Nov 2017

median follow-up: 21.2 months (IQR 17.0-25.8) vs. for primary outcome

death n(%): (I) 155 (39) of 401 patients vs. (C) 141 (35) of 405 patients (cutoff Feb 2018)

outcome

ERA 223 ITT (I vs. C)

primary:

SSE-free survival (months)

 22.3 (20.4-24.8) vs. 26.0 (21.8-28.3), HR 1.122 (0.917-1.374)*

 

 patients with at least one SSE or death (n (%)): 196 (49) vs. 190 (47)

secondary:

OS (months)

 30.7 (25.8-ne) vs. 33.3 (30.2-41.1), HR 1.195 (0.950-1.505)

rPFS (months)

central review

investigator review

 

 11.2 (9.1-11.8) vs. 12.4 (10.8-14.5), HR 1.152 (0.960-1.383)

 20.0 (16.4-22.3) vs. 19.2 (16.6-22.8), HR 0.971 (0.784-1.201)

CC (months)

 29.5 (26.5-35.7) vs. 28.5 (23.7-ne), HR 1.033 (0.816-1.308)

OuC (months)

19.0 (14.4-23.2) vs. 22.6 (18.0-25.7), HR 1.126 (0.921-1.378)

AE n (%)

grade 3-4

grade 5

any serious

due to dose,

to discontinuation:

ra-223 or placebo

abiraterone

prednisone/-olone

381 (97) vs. 386 (98)

219 (56) vs. 206 (52) (most common: hypertension 43 (11), 52 (13))

26 (7) vs. 20 (5)

160 (41) vs. 155 (39)**

141 (36) vs. 123 (31)

 

14 (4) vs. 13 (3)

57 (15) vs. 47 (12)

55 (14) vs. 47 (12)

AE=adverse event, ne= not estimated, values: median (95% confidence interval (CI))

*Hazard ratio (95% CI; two-sided p value)

**n(I) vs. n(C): urinary tract infection 14 vs. 9, back pain 8 vs. 10, pneumonia 8 vs. 10, traumatic fracture 11 vs. 3, treatment-related deaths 2 (acute myocardial infarction and interstitial lung disease) vs. 1 (arrhythmia)

Comments

- patients who did not receive study drug: 11 (I) and 9 (C)

- patients who stopped treatment after at least one dose: 304 (I) and 291 (C) (e.g. 53 (I) and 45 (C) for protocol-driven decisions, 11 (I) and 12 (C) withdrew, 1 lost to follow-up, respectively)

- patients with ongoing treatment: 88 (I) and 103 (C)

- authors report that the greatest difference in AE between groups was in the incidence of any fractures (103 (26%) of 392 patients for radium-223 and 38 (10%) of 394 patients in the placebo group (Table 5 within the publication)

 

7.4.3.1 EvT 7.44

Quelle

Fazit/Empfehlung

Empfehlungsgrad

Evidenzgraduierung

Literaturbelege

Dutch Urological Association 2007

Hormone therapy

Hormone therapy is the treatment of choice for patients with metastatic prostate cancer. The preferred methods are bilateral orchiectomy, LHRH analogue, or parenteral oestrogen. Maximal androgen blockade is not recommended as first-line therapy.

Optimal timing of hormone therapy

The time to start hormone therapy is determined on an individual basis.

Optimal schedule of hormone therapy:

Intermittent hormone therapy for patients with metastatic prostate cancer is an experimental approach that is preferably given in the context of a clinical trial only.

 

-

 

NICE 2008

When men with prostate cancer develop biochemical evidence of hormone-refractory disease, their treatment options should be discussed by the urological cancer multidisciplinary team (MDT) with a view to seeking an oncological and/or specialist palliative care opinion as appropriate.

Docetaxel is recommended, within its licensed indications, as a treatment option for men with hormone-refractory metastatic prostate cancer only if their Karnofsky performance-status score is 60% or more.

·       It is recommended that treatment with docetaxel should be stopped:

·       at the completion of planned treatment of up to 10 cycles, or

·       if severe adverse events occur, or

·       in the presence of progression of disease as evidenced by clinical or laboratory criteria, or by imaging studies.

Repeat cycles of treatment with docetaxel are not recommended if the disease recurs after completion of the planned course of chemotherapy.

A corticosteroid such as dexamethasone (0.5 mg daily) daily is recommended as thirdline hormonal therapy after androgen withdrawal and anti-androgen therapy for men with hormone-refractory prostate.

n.a.

n.a.

Leitlinie basiert auf systematischen Literaturrecherche,  transparente Aufarbeitung der Evidenz in Evidenztabellen 

 

 

Artikel (Autor, Jahr, Journal)

Studientyp

Anzahl der Patienten

Patientenmerkmale

Intervention

Vergleichs-Intervention

Nachverfolgung

Outcomes

Ergebnisse

Bemerkungen

Evidenz-Niveau (SIGN)

Fossa 2007

RCT

109

cTxNxM1, kastrationsresistent, Alter median 70-72 J

Docetaxel wöchentlich + Prednisolon

Prednisolon

?

OS, PFS

                   DP               P

PFS            11 M            4 M   

OS             27 M             18 M  

1-J-OS       82%              67%

2-J-OS       61%              29%

diese Publikation liegt der Fossa 2008 Zugrunde

1+

 

 

           

 

 

Yadav_AJR_2019

 

Yadav MP, Ballal S, Sahoo RK et al. Radioligand Therapy With 177 Lu-PSMA for Metastatic Castration-Resistant Prostate Cancer: A Systematic Review and Meta-Analysis. AJR Am J Roentgenol. 2019 Aug;213(2):275-285. doi: 10.2214/AJR.18.20845.

Link

https://pubmed.ncbi.nlm.nih.gov/30995089/     

AMSTAR 2 rating

low

Design

systematic review (and meta-analysis)

Sources

MEDLINE, Embase, PubMed, and Cochrane Central Register of Controlled Trials, ClinicalTrials.gov, systematic reviews and meta-analyses were used for cross verification

(up to August 2018)

Selection criteria

Inclusion criteria:

- study of PSMA radionuclide therapy;

- original article (clinical study or trial);

- single arm;

- patient number 10 or greater;

- pilot results from ongoing clinical trials;

- biochemical PSA response; objective response; overall survival (OS); progression-free survival (PFS); and toxicity

comment: if multiple articles originating from the same author had greater than 50% overlapping data, the study with the highest number of patients was included

Exclusion criteria:

- languages other than English,

- short communications, abstracts, letters to the editor, case reports, review articles,

- articles on radiochemistry,

- preclinical studies, biodistribution, or dosimetry

Outcome(s)

primary: PSA response

secondary:

- objective response according to RECIST (Response Evaluation Criteria in Solid Tumors),

- molecular response according to PERCIST (PET (positron emission tomography (?)) Response Criteria in Solid Tumors),

- overall survival (OS),

- progression-free survival (PFS),

- safety

Quality of evidence

quality of the included studies was assessed according to the Newcastle-Ottawa scale for nonrandomized cohort studies (graded as a star system with a score of 7 or greater for good quality)

grade ranging from 7 to 9

Results

n=23 articles were classified as eligible, n=6 were excluded due to overlapping data

n=17 studies were included (n=744 patients, range 10-119 patients)

- n=14 prospective design, n=3 retrospective design

- n=15 177Lu-PSMA radioligand therapy (RLT), n=2 177Lu-PSMA-I&T (I&T indicating imaging and therapy), n=1 both interventions

- median age 72 years (interquartile range (IQR) 71-73 years)

- median baseline PSA level 298.5 ng/mL (IQR, 190.9–348.5 ng/mL)

- n=17 included patients who had undergone multiple lines of treatment (median, 3 lines of treatment; range, 1–7), end-stage mCRPC

- n=1 study was classified as phase 2 study

prostate-specific antigen (PSA) decline: n=16 studies, n=671 patients

- PSA-decline: n=493 patients (overall 75% (95% confidence interval (CI), 70–79%)

- 0.75 (95% CI 0.70–0.79), I2=47.43%, p=0.02

PSA progression: n=12 studies, n=234 patients

- PSA progression: n= 87 patients (37.1%; 95% CI, 33.9–40.3%)

objective response: n=8 studies, n=175 patients

- partial remission: n=65 (37.2%) patients

- stable disease: n=67 (38.3%) patients

- progressive disease: n=43 (24.5%) patients

molecular response: n=8 studies, n=167 patients

- partial molecular response: n=74 patients

- stable disease: n=39 patients

- disease progression: n= 54 patients

survival; n=10 studies, n=577 patients

- n=189 (32.6%) died

overall survival (OS): n=8 studies

- median OS was not reached in 2 studies

- median OS of the other 6 studies: 13.7 months (IQR, 8–14 months)

- pooled proportion: 46% (95% CI, 32–60%),

- 0.46 (95% CI 0.32–0.60), I2=85.51% (p=0.000)

progression free survival (PFS): n=5 studies

- median PFS: 11 months (IQR, 7.6–13.7 months)

- pooled proportion: 19% (95% CI, 11–27%)

- 0.19 (95% CI 0.11–0.27), I2=61.06%, p=0.04

safety:

- various toxicities were reported,

- most common: haemoglobin toxicities (n = 14 studies, 23% (IQR, 7–35%) of patients)

- leukopenia (n=12 studies, 14.2% (IQR, 8–25%) of patients)

- thrombocytopenia (n=12 studies, 15% (IQR, 6–24%) of patients)

- nephrotoxicity (n=10 studies, 9.5% (IQR, 0–20%) of patients)

- salivary gland toxicities (pain, swelling, and dry mouth) (n=12 studies, 14.5% (IQR, 5.5–33.5%) of patients)

Comments

- eight of 17 studies assessed objective or molecular response

- eight of 17 studies assessed data on OS

- five of 17 studies assessed PFS

- detailed results of the single studies were reported within the publication (Table 3/4)

- heterogeneity was reported

- all studies were single armed studies (no control group)

Articles included in this publication:

12. Ahmadzadehfar H, Rahbar K, Kurpig S, et al. Early side effects and first results of radioligand therapy with 177Lu-DKFZ-617 PSMA of castration-resistant metastatic prostate cancer: a twocentre study. EJNMMI Res 2015; 5:114

13. Ahmadzadehfar H, Eppard E, Kürpig S, et al. Therapeutic response and side effects of repeated radioligand therapy with 177Lu-PSMA-DKFZ-617 of castrate-resistant metastatic prostate cancer. Oncotarget 2016; 7:12477–12488

14. Baum RP, Kulkarni HR, Schuchardt C, et al. 177Lu-labeled prostate-specific membrane antigen radioligand therapy of metastatic castration-resistant prostate cancer: safety and efficacy. J Nucl Med 2016; 57:1006–1013

15. Heck MM, Retz M, D’Alessandria C, et al. Systemic radioligand therapy with 177Lu labeled prostate specific membrane antigen ligand for imaging and therapy in patients with metastatic castration resistant prostate cancer. J Urol 2016; 196:382–391

16. Kratochwil C, Giesel FL, Stefanova M, et al. PSMA-targeted radionuclide therapy of metastatic castration-resistant prostate cancer with 177Lu-labeled PSMA-617. J Nucl Med 2016; 57:1170–1176

17. Kulkarni HR, Singh A, Schuchardt C, et al. PSMAbased radioligand therapy for metastatic castration-resistant prostate cancer: the Bad Berka experience since 2013. J Nucl Med 2016; 57(suppl 3):97S–104S

18. Rahbar K, Schmidt M, Heinzel A, et al. Response and tolerability of a single dose of 177Lu-PSMA-617 in patients with metastatic castration-resistant prostate cancer: a multicenter retrospective analysis. J Nucl Med 2016; 57:1334–1338

19. Rahbar K, Bode A, Weckesser M, et al. Radioligand therapy with 177Lu-PSMA-617 as a novel therapeutic option in patients with metastatic castration resistant prostate cancer. Clin Nucl Med 2016; 41:522–528

20. Ahmadzadehfar H, Wegen S, Yordanova A, et al. Overall survival and response pattern of castration-resistant metastatic prostate cancer to multiple cycles of radioligand therapy using [177Lu]Lu-PSMA-617. Eur J Nucl Med Mol Imaging 2017; 44:1448–1454

21. Bräuer A, Grubert LS, Roll W, et al. 177Lu-PSMA-617 radioligand therapy and outcome in patients with metastasized castration-resistant prostate cancer. Eur J Nucl Med Mol Imaging 2017; 44:1663–1670

22. Fendler WP, Reinhardt S, Ilhan H, et al. Preliminary experience with dosimetry, response and patient reported outcome after 177Lu-PSMA-617 therapy for metastatic castration-resistant prostate cancer. Oncotarget 2017; 8:3581–3590

23. Ferdinandus J, Eppard E, Gaertner FC, et al. Predictors of response to radioligand therapy of metastatic castrate-resistant prostate cancer with 177Lu-PSMA-617. J Nucl Med 2017; 58:312–319

24. Rahbar K, Boegemann M, Yordanova A, et al. PSMA targeted radioligand therapy in metastatic castration resistant prostate cancer after chemotherapy, abiraterone and/or enzalutamide: a retrospective analysis of overall survival. Eur J Nucl Med Mol Imaging 2018; 45:12–19

25. Scarpa L, Buxbaum S, Kendler D, et al. The 68Ga/177Lu theragnostic concept in PSMA targeting of castration-resistant prostate cancer: correlation of SUVmax values and absorbed dose estimates. Eur J Nucl Med Mol Imaging 2017; 44:788–800

26. Yadav MP, Ballal S, Tripathi M, et al. 177Lu-DKFZPSMA-617 therapy in metastatic castration resistant prostate cancer: safety, efficacy, and quality of life assessment. Eur J Nucl Med Mol Imaging 2017; 44:81–91

27. Rathke H, Giesel FL, Flechsig P, et al. Repeated 177Lu-labeled PSMA-617 radioligand therapy using treatment activities of up to 9.3 GBq. J Nucl Med 2018; 59:459–465

31. Hofman MS, Violet J, Hicks RJ, et al. [177Lu]-PSMA-617 radionuclide treatment in patients with metastatic castration-resistant prostate cancer (LuPSMA trial): a single-centre, single-arm, phase 2 study. Lancet Oncol 2018; 19:825–833

     

 

Burdett_EurUrol_2019

 

Burdett S, Boevé LM, Ingleby FC et a.l. Prostate Radiotherapy for Metastatic Hormone-sensitive Prostate Cancer: A STOPCAP Systematic Review and Meta-analysis. Eur Urol. 2019 Jul;76(1):115-124. doi: 10.1016/j.eururo.2019.02.003.

Link

https://pubmed.ncbi.nlm.nih.gov/30826218/    

AMSTAR 2 rating

critically low

Design

systematic review and meta-analysis

FAME principles:

(1) start the systematic review process whilst trials are ongoing or yet to report

(2) search comprehensively for all eligible trials

(3) liaise with trial teams to develop a detailed picture of these trials

(4) predict when sufficient results will be available for reliable meta-analysis

(5) conduct meta-analysis and interpret results taking account of any unavailable data

(6) assess the value of updating

Sources

MEDLINE, EMBASE, clinicaltrials.gov, and Cochrane CENTRAL, conference proceedings, reference lists

(up to June 2018)

Selection criteria

Inclusion criteria:

- randomized controlled trial (RCT) (all published, unpublished, and ongoing trials)

- men with mHSPC

- starting or responding to first-line hormone therapy

- compared prostate radiotherapy plus ADT versus ADT

- comment: trials including additional agents (eg, docetaxel, abiraterone) were also eligible, provided that the same additional agents were used in both groups

Outcome(s)

primary:

- survival (defined as the time from randomisation to death from any cause)

secondary:

- progression-free survival (PFS) (defined as the time from randomisation to first symptomatic clinical or radiological progression or death, excluding biochemical progression)

- biochemical progression (defined as the time from randomisation to first biochemical (prostate-specific antigen [PSA]) progression)

- failure-free survival (FFS) (defined as the time from randomisation to first biochemical, clinical, or radiological progression)

- acute toxicity on the radiotherapy arm

Quality of evidence

Cochrane Risk of Bias Tool

both trials judged to be at a low risk of bias

Results

n=3 studies

- one ongoing (PEACE-1, NCT01957436) trial

- two completed (HORRAD and STAMPEDE) trials, n=2126 patients

- newly diagnosed mHSPC

- receiving long-term ADT for the first time

- intervention: radiotherapy+ADT vs. control: androgen deprivation therapy (ADT)

- randomized men:

- HORRAD n=432 (between 2004 and 2014)

- STAMPEDE n=1694 (between 2013 and 2016)

- median age:

- HORRAD 67 years

- STAMPEDE 68 years

- median follow-up:

- HORRAD 47 months (interquartile range (IQR) 36–68 months)

- STAMPEDE 41.9 months (IQR 31–49 months)

- death: n=969 of 2126 men (in HORRAD and STAMPEDE)

- survival: hazard ratio (HR) 0.92 (95% confidence interval (CI) 0.81–1.04) p = 0.195; heterogeneity chi-square = 0.08, degree of freedom = 1, p = 0.78

- PFS (1305 events): HR 0.94 (95% CI 0.84–1.05) p = 0.238

- PSA progression (1533 events): HR 0.74 (95% CI 0.67–0.82) p = 0.94 x 10-8

- FFS (1662 events): HR 0.76 (95% CI 0.69–0.84) p = 0.64 x 10-7

- oxicity (were not available for HORRAD, based on the results from STAMPEDE):

- 4% of men who received prostate radiotherapy had severe acute bladder toxicity,

- 1% of men had severe acute bowel toxicity (RTOG scale),

- 4% of men had severe late effects

Comments

PROSPERO registration: CRD42018096108

Articles included in this study:

[11] Parker CC, James ND, Brawley CD, et al. Radiotherapy to the primary tumour for newly-diagnosed, metastatic prostate cancer: a randomised controlledphase III trial (STAMPEDE). Lancet 2018;392:2353–66.

[12] Boeve LMS, Hulshof MCCM, Vis AN, et al. Effect on survival of androgen deprivation therapy alone compared to androgen deprivation therapy combined with concurrent radiation therapy to the prostate in patients with primary bone metastatic prostate cancer in a prospective randomised clinical trial: data from the HORRAD trial. Eur Urol 2019;75:410–8.

     

 

Slaoui_WorldJUrol_2019

 

Slaoui A, Albisinni S, Aoun F et al. A systematic review of contemporary management of oligometastatic prostate cancer: fighting a challenge or tilting at windmills? World J Urol. 2019 Nov;37(11):2343-2353. doi: 10.1007/s00345-019-02652-7.

Link

https://pubmed.ncbi.nlm.nih.gov/30706122/    

AMSTAR 2 rating

critically low

Design

systematic review

Sources

PubMed, EMBASE, Cochrane Library, CINHAL, Google Scholar, Ovid, conference abstracts

(up to Nov, 1 2018)

Selection criteria

Inclusion criteria:

- published in English

- oligometastatic prostate cancer

- local and metastasis-directed therapy

- comparator: standard of care (ADT with taxane chemotherapy or abiraterone)

- translational science studies, cohort studies, case-control studies, prospective studies

Exclusion criteria:

- case reports, editorials, letters

Outcome(s)

oncologic outcomes (metastasis-free survival, cancer specific survival, overall survival)

progression of local symptoms, need for surgery due to local symptoms

Quality of evidence

-

Results

n=36 articles

- the authors described narrative:

- the definition of oligometastatic disease

- the rationale for local and metastatic directed therapy

- the role of cytoreductive prostatectomy (feasibility, retrospective studies, explorative studies)

- the role of radiation (retrospective analysis; HORRAD randomized controlled trial, phase II n=432, bone-metastatic PCa; STAMPEDE randomized controlled trial phase 3, n=1032 (overall survival: hazard ratio (HR) 0.92  (95% confidence interval (CI) 0.80-1.06) P=0.2266, survival without failure: HR 0.76 (95% CI 0.68-0.84) P<0.0001))

- the role of metastasis-directed therapy (MDT, retrospective studies, prospective studies (STOMP, ORIOLE) phase II)

- ongoing trials

Comments

- authors documented that the term oligometastatic disease was described first in 1995 (defined as a limited number of clinically detectable metastases, and its prognostic and therapeutic value was considered to be in-between localized and metastatic diseases)

- authors documented that the limitation of imaging techniques limited the detection

- authors added that currently, no test is available to differentiate between the two clinical scenarios, and no consensus on the definition of oligometastatic disease

- they concluded that literature suggested to report five variables in the description of oligometastatic disease:

- the distinction between synchronous and metachronous metastases

- the number and site of lesions

- castrate-resistant status

- the imaging modality used to define oligometastatic disease

- they added three categories of oligometastatic PCa:

- de novo oligometastases (synchronous)

- recurrent oligometastases (metachronous)

- progressive oligometastases (induced)

     

 

Connor_EurUrolOncol_2020

 

Connor MJ, Smith A, Miah S et al. Targeting Oligometastasis with Stereotactic Ablative Radiation Therapy or Surgery in Metastatic Hormone-sensitive Prostate Cancer: A Systematic Review of Prospective Clinical Trials. Eur Urol Oncol. 2020 Oct;3(5):582-593. doi: 10.1016/j.euo.2020.07.004.

Link

https://pubmed.ncbi.nlm.nih.gov/32891600/    

AMSTAR 2 rating

critically low

Design

systematic review

Sources

Medline, Embase, Cochrane Review Database, and clinical trial databases

(up to March, 28 2020)

Selection criteria

Inclusion criteria:

- reporting on oncological outcomes (progression-free survival [PFS], OS, and ADT-free survival [ADT-FS]), complications of surgical metastasectomy, and toxicity of SABR (Stereotactic Ablative Radiation) or surgery

- men with metastatic (M1a-c) prostate cancer (recurrent or oligometastatic)

- treatment of primary prostate gland had occurred

- burden of up to 10 metastases

Comment: surgical metastasectomy was defined as any surgical excision of M1 (a-c) disease; radiotherapy was considered SABR if radiotherapy delivery fell within American Association of Physicists in Medicine (AAPM) Task Group 101 and UK SABR Consortium (version 6.1) guidelines

Exclusion criteria:

- review articles, cost-analysis studies, letters, bulletins, comments, and conference abstracts

- nonclinical trials

- exclusively castrate-resistant disease,

- the primary tumour was not treated (absence of radical prostatectomy, prostate radiotherapy, or brachytherapy)

- no separate analysis of outcomes in mixed primary studies

- involved non-SABR radiotherapy

Outcome(s)

oncological outcomes and safety

Quality of evidence

risk of bias: Cochrane 2.0 or ROBINS-I tool

- authors rated that the included RCT had some concerns in two of four domains, with no concerns in the remaining domains

GRADE:

- authors graded: PFS, very low; LC, low; ADT-FS, low; SABR toxicity, low; and surgical complications, very low

Results

n=4 studies, n=169 patients

- n=2 randomised (phase II, ORIOLE, STOMP) and n=2 nonrandomised single arm clinical trials (POPSTAR, MSKCC)

- no randomised controlled clinical trial, phase 3 was identified

- hormone-sensitive recurrent (metachronous) disease (one reporting on de novo (synchronous) disease)

- SABR (n = 2) or surgical metastasectomy with or without SABR (n = 2)

- median age: range 43 to 75 years

- median follow-up: range 18.8 months to 40 months

- progression free survival (PFS):

- median PFS: (I) not reached vs (C) 31 months table, 5.8 months text (?) (1 study, ORIOLE, n=54)

- at 6 months: (I) 19% (95% CI 9.6–35.4) vs. 61% (95% CI 38.5–79.6)

- Hazard ration (HR) 0.30 (95% confidence interval (CI) 0.11–0.81) p = 0.02

- ADT free survival:

- median: (I) 21 months vs. 13 months (1 study, STOMP, n=62)

- HR 0.60 (95% CI 0.40–0.90) p = 0.11

- comment: feasibility was the primary outcome

- complications for SABR:

- grade I: 80.5% (n=29 of n= 36) (1 study, ORIOLE)

- grade I: 19,4% (n=6 of n=31) (1 study, STOMP)

- grade II: 8.3% (n=3 of n=36) (1 study, ORIOLE)

- grade II: 0% n=0 of n=31) (1 study, STOMP)

- grade III: 0% (2 studies, ORIOLE and STOMP)

Comments

- the study was prospectively registered on PROSPERO (CRD42019127071)

- authors documented that the definitions of each outcome were heterogeneous

- small cohorts

- lacked comparative primary end points

- significant heterogeneity in the dose/fractionation and surgical approaches

- lack of standardised definitions of treatment failure

- MDT in de novo synchronous disease is experimental and, findings presented are based on a single nonrandomised pilot study

Articles included in this study:

[20] Siva S, Bressel M, Murphy DG, et al. Stereotactic ablative body radiotherapy (SABR) for oligometastatic prostate cancer: a prospective clinical trial. Eur Urol 2018;74:455–62.

[21] Ost P, Reynders D, Decaestecker K, et al. Surveillance or metastasisdirected therapy for oligometastatic prostate cancer recurrence: a prospective, randomized, multicenter phase II trial. J Clin Oncol 2017;36:446–53.

[22] O'Shaughnessy MJ, McBride SM, Vargas HA, et al. A pilot study of a multimodal treatment paradigm to accelerate drug evaluations in early-stage metastatic prostate cancer. Urology 2017;102:164–72.

[23] Phillips R, Shi WY, Deek M, et al. Outcomes of observation vs stereotactic ablative radiation for oligometastatic prostate cancer. The ORIOLE phase 2 randomized clinical trial. JAMA Oncol 2020;6:650–9.

     

 

7.5 EvT 7.47

Quelle/
Studientyp

Population

(verglichene) Interventionen/
ggf. Dosierung/
ggf.
Follow-up

Outcomes

Ergebnisse

Bemerkungen

Evidenzniveau (SIGN)

Parker 2013

 

N = 921 (2:1 ratio, Intervention n=614, Control n=307) 

Enrollment from 06/2008-02/2011

Inclusion criteria (selection)

Progressive castration-resistant PCa (defined as a serum testosterone level of 50 ng per deciliter or lower (≤1.7 nmol per liter) after bilateral orchiectomy or during maintenance treatment

two or more bone metastases detected on skeletal scintigraphy

no known visceral metastases;

had received docetaxel, were not healthy enough or declined to receive it, or it was not available.

symptomatic disease with regular use of analgesic medication or treatment with external-beam radiation therapy required for cancer related bone pain within the previous 12 weeks.

ECOG performance: 0-2

Baseline PSA≥5 ng/mL with two consecutive increases of PSA values over the previous reference value

Life expectancy ≥ 6 months

Exclusion criteria (selection):

History of presence of visceral metastases

Chemotherapy within the previous 4 weeks

6 injections of radium-223 (at a dose of 50 kBq per kilogram of body weight intravenously) vs. matching placebo;

One injection was administered every 4 weeks.

Additionally patients in both groups received “best standard of care” defined as routine care provided at each centre (e.g. local external-beam radiation therapy, treatment with glucoccorticoids, antiandrogens, ketoconazole, estrogens, estramustine)

 

Primary endpoint:

1. overall survival (defined as time to death)

Secondary endpoints (pre-specified):

2. Time to increase in PSA level

3. Time to first symptomatic skeletal event (clinically defined)

4. QoL (FACT-P): increase of score of ≥ 10 (during period of study-drug administration)

 

1) Overall Survival

1a) interim analyses (n=809):

14.0 months (95% CI not reported in publication)

vs. 11.2 months (95% CI not reported in publication)

HR,  0.70 (95% CI, 0.55-0.88, p=0.002)

Death: 35% (191/541) vs. 64% (123/268)

1b) updated analyses (n=921)

14.9 months (95% CI not reported in publication)

vs. 11.3 months (95% CI not reported in publication)

HR,  0.70 (95% CI, 0.58-0.83, p<0.001)

Death: 54% (333/614) vs. 64% (195/307)

2) Time to increase in PSA level (Updated analyses, n=921)

3.6 months (95% CI not reported in publication)

vs. 3.4 months (95% CI not reported in publication)

HR, 0.64 (95% CI, 0.54-0.77), p<0.001

3) Time to first symptomatic skeletal event (Updated analyses, n=921)

15.6 months (95% CI not reported in publication)

vs. 9.8 months (95% CI not reported in publication)

HR,  0.66 (95% CI, 0.52-0.83, p<0.001)

4) QoL (FACT-P increase of score of ≥ 10)

25% vs. 12% (p=0.02)

(no numbers of pts available)

 

 

Study stopped after interim analyses for efficacy as recommended by Data Monitoring Committee (DMC)

Interim analyses conducted in n=809 patients (n=541 in the radium-223 group, n=268 in the placebo group)

ITT, Double-blinded, randomized

136 centers in 19 countries (no specification regarding sites or countries in publication)

Stratification according to previous use or nonuse of docetaxel, baseline alkaline phosphatase level, current use or nonuse of bisphosphonate

57% with prior docetaxel therapy, 43% without prior docetaxel therapy

Numbers, reasons and distribution of patients between groups without prior docetaxel therapy not described

In the exploratory subgroup analyses: no significant benefit for radium for patients with ECOG ≥ 2 (n=118 patients), for patients with <6 metastases (n=138) and for patients with ALP<220 U/liter (n=517)

Adverse events: more patients in the radium group had thrombocytopenia (12% vs. 6%), neutropenia (5% vs. 1%) and diarrhea (25% vs. 15%)

Sponsor: Algeta and Bayer Health Care

1+

 

Autoren Jahr

Literaturtyp

untersuchte Studien/
Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

Methodische Besonder­
heiten/
Bemerkungen

LoE

Literaturbelege

Falkmer, 2003

Systema­tischer Review

8 RCTs, 10 prospektive Studien,3 retrospektive Studien und 3 Reviews und andere Artikel aus systematischer Recherche in Medline und Cochrane library (01.01.94- 01.10.2001)

Effekt von EBRT auf Schmerzen durch Knochenmetas­tasen bei versch. Primärtumorarten

lokale RT bei Knochenschmerzen (8 RCTs, 5 prospektive Studien):

- niedrigste Einzeldosis 4Gy, höchste Einzeldosis 10Gy

- Mehrfachbstrahlungen von 1,8 bis 5Gy in 4 bis 23 Sitzungen

- gut dokumentierte Effekte bei Knochenschmerzen

- Ergebnisse zur kompletten Schmerzremission aufgrund unterschiedlicher Erhebung der Schmerzen und der Effekte schwer evaluierbar

- Gesamtschmerzentlastung bei 59 bis 90% berichtet

- Effekt bei Einzeldosis mit 4Gy signifikant geringer als bei anderen Fraktionierungsrhythmen

- niedrigste wirkungsvolle Einzeldosis 6Gy

- Gesamtschmerzentlastung und Schmerzrezidiv scheinbar unabhängig von Fraktionierungsrhythmen

- im Mittel 5-6Mon. Schmerzlinderung nach RT, Anstieg auf Schmerzskala nach 1J bei 40% der Pat.

- Bedarf erneuter RT in Gruppen mit Einzelbestrahlungen signifikant höher als bei Mehrfachbestrahlungen

- Gründe für die Unterschiede nicht eindeutig

- Nebenwirkungen insgsamt leicht

- Spinalkanalstenosen in 4 Studien berichtet (nicht eindeutig, ob im Bestrahlungsfeld aufgetreten)

- Inzidenz 1-2% in großen RCTs und 8-9% in kleineren Studien

- jeweils keine signifikanten Unterschiede zwischen Einzel- oder Mehrfachbestrahlungen

- pathologische Frakturen in 6 Studien berichtet - Inzidenz 1-10% (in großen RCTs 1-4%)

- bei Einzelbestrahlungen 2-3mal höhere Inzidenz als bei Mehrfachbstrahlungen

- keine QoL-Unterschiede zwischen den Behandlungsgruppen

- Sensitivität von Schmerztagebüchern zu gering um Effekte zu messen

- Remineralisierung nach 6Mon. bei Mehrfachbstrahlten signifikant besser als bei Einfachbestrahlten (Koswig et al.)

 

1++

- Gaze MN, et al. Radiother Oncol 1997

- Jeremic B, et al. Int J Radiat Oncol Biol Phys 1998

- Koswig S, et al. Strahlenther Onkol 1999

- Nielson OS, et al. Radiother Oncol 1998

- Niewald M, et al. Int J Radiat Oncol Biol Phys 1996

- Rasmusson B, et al. Radiat Oncol 1995

- Steenland E, et al. Radiother Oncol 1999

- The Bone Pain Trail working Party. Radiother Oncol 1999

- Arcangeli G, et al. Int J Radiat Oncol Biol Phys 1998

- Bremer M, et al. Strahlenther Onkol 1999

- Huguenin PI, et al. Int J Radiat Oncol Biol Phys 1998

- Tomboloini V, et al. Tumouri 1994

- Uppelschoten JM, et al. Radiother Oncol 1995

Bauman, 2005

syst. Review

RCTs und Metaanalysen, randomisierte Phase I und II-Trials, evidenzbasierte clinical practice-Guidelines

Syst. Recherche in Medline (1966-2004), Embase (1980-2004), Cancerlit (1975-2002), Cochrane Library (2003)

Einschluss Outcomes:

-Schmerzevaluation

-Analgetikaeinnahme

-QoL

-OS

-Nebenwirkungen

Ausschlusskriterien:

-n<20

-nicht englisch-sprachig publiziert

Radionuklide (Sr-89, Sm-153, Re-186 o. -188, Sn-117, P-32) bei schmerzhaften Knochenmetastasen, die nicht auf Analgetika ansprechen

Sr-89

Porter 1993:

-40% mit Sr-89 vs. 23% mit Placebo nach 3 Monaten schmerzfrei

-17% vs. 2% ohne weitere Schmerzmedikation, p<0,05

-weniger neue schmerzende Stellen: 0,59 neue vs. 1,21 neue Stellen, p<0,002

Oosterhof 2003:

-n=203, Sr-89 vs. lokale R

-Schmerzfreiheit und time to progression in beiden Gruppen gleich

-Verbesserung des Überlebens mit RT, 11 vs. 7,2 Monate, p=0,0457, Ein-Jahres- Überleben: 45% vs.34%

-keine Unterschiede bei Nebenwirkungen

-Sr-89- Therapie 25% teurer

Quilty 1994:

Lokale/ Halbseiten-RT vs. Sr-89

-Schmerzlinderung und Analgetikareduktion in allen Gruppen gleich

-signifikant mehr Pat. mit Sr-89 vs. lokale/Halbseiten-RT frei von neuen schmerzenden Stellen, 64% vs. 42%, p<0,05; 73% vs.51%, p<0,05

Buchali 1988

-n=41, Sr-89 vs. Placebo

-kein sign. Unterschied bei Schmerzlinderung nach 1-3J

-sign. besseres 2-J-Überleben mit Sr-89 vs. Placebo, 46% vs. 4%, p<0,05 (CAVE: 10Pat. starben in ersten 3Monaten, wurden aus Analyse ausgeschlossen)

Sciuto 2001:

Brustkrebs-Pat.

Tu 2001 (Phase II Trial)

-n=72 mit PCa, Doxorubicin+Sr-89 vs. Doxorubicin

-time-to-progression 13,9 vs. 7,0 Monate, p<0,0001

-OS: HR 2,76, [95% CI 1,44-5,29], p=0,0014

Brundage 1998 (Practice guideline)

-Empfehlung für Sr-89 bei kastrationsresistentem PCa, das für RT nicht geeignet ist

-keine Empfehlung als adjuvante Therapie zu RT

Sm-153

Olea 2000

-73% mit Schmerzfreiheit, dosisunabhängig

Serafini 1998

-2 Dosen Sm-153 vs. Placebo

-besseres Ansprechen bei höherer Dosis vs. Placebo (31% vs. 14%, p<0,016)

Sartor 1997

-Schmerzlinderung in Sm-153-Gruppe, keine Werte angegeben

Tian 1999 und Resche 1997

-versch. Primärtumoren, keine Unterschiede

Re-186 o. -188

Han 2002

79 v. 111 auswertbar (72%)

-Re-186 vs. Placebo

-Schmerzintensität: Zunahme in der Placebogruppe vs. Abnahme in der 186-Re-Gruppe, p=0,039

-tägliche Aktivitäten: Abnahme in der Placebogruppe vs. gleichbleibend/ Zunahme in 186-Re-Gruppe, p=0,019

- Tage mit Schmerzlinderung in 186-Re-Gruppe größer, 23/84 Tagen, 27%, [95% CI 16,6-37,1] vs. 11/84 Tagen, 13%, [95% CI 5,8-20,0], p<0,05

Responder: n=28/43 (65%) vs. 13/36 (36%), p=0,01

- Palmedo 2003 (Phase II Trial)

-n=64 mit hormonresist. PCa, 1 o. 2 Re-188-Injektionen

-sign. Schmerzlinderung, besseres OS und DFS nach 2 Injektionen

-CAVE: nicht genug power

Sciuto 2001

Brustkrebs

Sn-117

Srivastava 1998 (Phase I Trial)

-n=30 mit metastasiertem PCa, Sn-117 2,64-10,58MBq/kg

-75% Responder, Schmerzfreiheit nach 5+-3 Tagen bei Dosen≥462MBQ vs. Schmerzfreiheit nach 19+-15 Tagen bei Dosen≤370MBq, p<0,05

P-32

Shah Syed 1999 (Phase II Trial)

-n=20 mit metastasiertem PCa mit P-32 (185MBq) behandelt

-Analgetika-Score-Abnahme von 4,2+-1,4 (baseline) auf 1,8+-1,5 (8 Wochen nach Behandlung)

Nebenwirkungen

-Thrombozytopenie bei 30-50% der Pat. mit Radionuklidtherapie (Grad 2 o. weniger)

-seltener: Neutropenie (häufigeres Auftreten bei Kombination mit Chemotherapie)

-weniger Übelkeit und Erbrechen im Vergleich zu RT

Schlussfolge-rung der Autoren:

Radionuklide (Sr-89 und Sm-153) können bei Pat. mit PCa  und multiplen schmerzhaften Knochenmetasta-sen, bei denen eine Analgetika-therapie nicht erfolgreich und eine externe RT nicht indiziert ist, in Betracht gezo-gen werden. Die Knochenmarks-funktion, der ge-sundheitliche Sta-tus, andere Kno-chenmarks supprimierende Therapien (ChT, RT), die Lebens-erwartung und Eignung des Pat. für eine palliative Therapie müssen berücksichtigt werden.

1+

Sciuto 2002, Porter 1993, Smeland 2003, Oosterhof 2003, Quilty 1994, Buchali 1988, Sciuto 2001, Tu 2001, Lewington 1991

Olea 2000, Serafini 1998, Sartor 1997, Tian 1999, Resche 1997,

Brundage 1998, Han 2002, Srivastava 1998, Shah Syed 1999

7.5 EvT 7.48

Autoren Jahr

Literaturtyp

untersuchte Studien/
Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

Methodische Besonder­
heiten/
Bemerkungen

LoE

Literaturbelege

Falkmer, 2003

Systema­tischer Review

8 RCTs, 10 prospektive Studien,3 retrospektive Studien und 3 Reviews und andere Artikel aus systematischer Recherche in Medline und Cochrane library (01.01.94- 01.10.2001)

Effekt von EBRT auf Schmerzen durch Knochenmetas­tasen bei versch. Primärtumorarten

lokale RT bei Knochenschmerzen (8 RCTs, 5 prospektive Studien):

- niedrigste Einzeldosis 4Gy, höchste Einzeldosis 10Gy

- Mehrfachbstrahlungen von 1,8 bis 5Gy in 4 bis 23 Sitzungen

- gut dokumentierte Effekte bei Knochenschmerzen

- Ergebnisse zur kompletten Schmerzremission aufgrund unterschiedlicher Erhebung der Schmerzen und der Effekte schwer evaluierbar

- Gesamtschmerzentlastung bei 59 bis 90% berichtet

- Effekt bei Einzeldosis mit 4Gy signifikant geringer als bei anderen Fraktionierungsrhythmen

- niedrigste wirkungsvolle Einzeldosis 6Gy

- Gesamtschmerzentlastung und Schmerzrezidiv scheinbar unabhängig von Fraktionierungsrhythmen

- im Mittel 5-6Mon. Schmerzlinderung nach RT, Anstieg auf Schmerzskala nach 1J bei 40% der Pat.

- Bedarf erneuter RT in Gruppen mit Einzelbestrahlungen signifikant höher als bei Mehrfachbestrahlungen

- Gründe für die Unterschiede nicht eindeutig

- Nebenwirkungen insgsamt leicht

- Spinalkanalstenosen in 4 Studien berichtet (nicht eindeutig, ob im Bestrahlungsfeld aufgetreten)

- Inzidenz 1-2% in großen RCTs und 8-9% in kleineren Studien

- jeweils keine signifikanten Unterschiede zwischen Einzel- oder Mehrfachbestrahlungen

- pathologische Frakturen in 6 Studien berichtet - Inzidenz 1-10% (in großen RCTs 1-4%)

- bei Einzelbestrahlungen 2-3mal höhere Inzidenz als bei Mehrfachbstrahlungen

- keine QoL-Unterschiede zwischen den Behandlungsgruppen

- Sensitivität von Schmerztagebüchern zu gering um Effekte zu messen

- Remineralisierung nach 6Mon. bei Mehrfachbstrahlten signifikant besser als bei Einfachbestrahlten (Koswig et al.)

 

1++

- Gaze MN, et al. Radiother Oncol 1997

- Jeremic B, et al. Int J Radiat Oncol Biol Phys 1998

- Koswig S, et al. Strahlenther Onkol 1999

- Nielson OS, et al. Radiother Oncol 1998

- Niewald M, et al. Int J Radiat Oncol Biol Phys 1996

- Rasmusson B, et al. Radiat Oncol 1995

- Steenland E, et al. Radiother Oncol 1999

- The Bone Pain Trail working Party. Radiother Oncol 1999

- Arcangeli G, et al. Int J Radiat Oncol Biol Phys 1998

- Bremer M, et al. Strahlenther Onkol 1999

- Huguenin PI, et al. Int J Radiat Oncol Biol Phys 1998

- Tomboloini V, et al. Tumouri 1994

- Uppelschoten JM, et al. Radiother Oncol 1995 

Bauman_RadiotherOncol_2005

Autoren Jahr

Literaturtyp

untersuchte Studien/
Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

Methodische Besonder­
heiten/
Bemerkungen

LoE

Literaturbelege

Bauman, 2005

syst. Review

RCTs und Metaanalysen, randomisierte Phase I und II-Trials, evidenzbasierte clinical practice-Guidelines

Syst. Recherche in Medline (1966-2004), Embase (1980-2004), Cancerlit (1975-2002), Cochrane Library (2003)

Einschluss Outcomes:

-Schmerzevaluation

-Analgetikaeinnahme

-QoL

-OS

-Nebenwirkungen

Ausschlusskriterien:

-n<20

-nicht englisch-sprachig publiziert

Radionuklide (Sr-89, Sm-153, Re-186 o. -188, Sn-117, P-32) bei schmerzhaften Knochenmetastasen, die nicht auf Analgetika ansprechen

Sr-89

Porter 1993:

-40% mit Sr-89 vs. 23% mit Placebo nach 3 Monaten schmerzfrei

-17% vs. 2% ohne weitere Schmerzmedikation, p<0,05

-weniger neue schmerzende Stellen: 0,59 neue vs. 1,21 neue Stellen, p<0,002

Oosterhof 2003:

-n=203, Sr-89 vs. lokale R

-Schmerzfreiheit und time to progression in beiden Gruppen gleich

-Verbesserung des Überlebens mit RT, 11 vs. 7,2 Monate, p=0,0457, Ein-Jahres- Überleben: 45% vs.34%

-keine Unterschiede bei Nebenwirkungen

-Sr-89- Therapie 25% teurer

Quilty 1994:

Lokale/ Halbseiten-RT vs. Sr-89

-Schmerzlinderung und Analgetikareduktion in allen Gruppen gleich

-signifikant mehr Pat. mit Sr-89 vs. lokale/Halbseiten-RT frei von neuen schmerzenden Stellen, 64% vs. 42%, p<0,05; 73% vs.51%, p<0,05

Buchali 1988

-n=41, Sr-89 vs. Placebo

-kein sign. Unterschied bei Schmerzlinderung nach 1-3J

-sign. besseres 2-J-Überleben mit Sr-89 vs. Placebo, 46% vs. 4%, p<0,05 (CAVE: 10Pat. starben in ersten 3Monaten, wurden aus Analyse ausgeschlossen)

Sciuto 2001:

Brustkrebs-Pat.

Tu 2001 (Phase II Trial)

-n=72 mit PCa, Doxorubicin+Sr-89 vs. Doxorubicin

-time-to-progression 13,9 vs. 7,0 Monate, p<0,0001

-OS: HR 2,76, [95% CI 1,44-5,29], p=0,0014

Brundage 1998 (Practice guideline)

-Empfehlung für Sr-89 bei kastrationsresistentem PCa, das für RT nicht geeignet ist

-keine Empfehlung als adjuvante Therapie zu RT

Sm-153

Olea 2000

-73% mit Schmerzfreiheit, dosisunabhängig

Serafini 1998

-2 Dosen Sm-153 vs. Placebo

-besseres Ansprechen bei höherer Dosis vs. Placebo (31% vs. 14%, p<0,016)

Sartor 1997

-Schmerzlinderung in Sm-153-Gruppe, keine Werte angegeben

Tian 1999 und Resche 1997

-versch. Primärtumoren, keine Unterschiede

Re-186 o. -188

Han 2002

79 v. 111 auswertbar (72%)

-Re-186 vs. Placebo

-Schmerzintensität: Zunahme in der Placebogruppe vs. Abnahme in der 186-Re-Gruppe, p=0,039

-tägliche Aktivitäten: Abnahme in der Placebogruppe vs. gleichbleibend/ Zunahme in 186-Re-Gruppe, p=0,019

- Tage mit Schmerzlinderung in 186-Re-Gruppe größer, 23/84 Tagen, 27%, [95% CI 16,6-37,1] vs. 11/84 Tagen, 13%, [95% CI 5,8-20,0], p<0,05

Responder: n=28/43 (65%) vs. 13/36 (36%), p=0,01

- Palmedo 2003 (Phase II Trial)

-n=64 mit hormonresist. PCa, 1 o. 2 Re-188-Injektionen

-sign. Schmerzlinderung, besseres OS und DFS nach 2 Injektionen

-CAVE: nicht genug power

Sciuto 2001

Brustkrebs

Sn-117

Srivastava 1998 (Phase I Trial)

-n=30 mit metastasiertem PCa, Sn-117 2,64-10,58MBq/kg

-75% Responder, Schmerzfreiheit nach 5+-3 Tagen bei Dosen≥462MBQ vs. Schmerzfreiheit nach 19+-15 Tagen bei Dosen≤370MBq, p<0,05

P-32

Shah Syed 1999 (Phase II Trial)

-n=20 mit metastasiertem PCa mit P-32 (185MBq) behandelt

-Analgetika-Score-Abnahme von 4,2+-1,4 (baseline) auf 1,8+-1,5 (8 Wochen nach Behandlung)

Nebenwirkungen

-Thrombozytopenie bei 30-50% der Pat. mit Radionuklidtherapie (Grad 2 o. weniger)

-seltener: Neutropenie (häufigeres Auftreten bei Kombination mit Chemotherapie)

-weniger Übelkeit und Erbrechen im Vergleich zu RT

Schlussfolge-rung der Autoren:

Radionuklide (Sr-89 und Sm-153) können bei Pat. mit PCa  und multiplen schmerzhaften Knochenmetasta-sen, bei denen eine Analgetika-therapie nicht erfolgreich und eine externe RT nicht indiziert ist, in Betracht gezo-gen werden. Die Knochenmarks-funktion, der ge-sundheitliche Sta-tus, andere Kno-chenmarks supprimierende Therapien (ChT, RT), die Lebens-erwartung und Eignung des Pat. für eine palliative Therapie müssen berücksichtigt werden.

1+

Sciuto 2002, Porter 1993, Smeland 2003, Oosterhof 2003, Quilty 1994, Buchali 1988, Sciuto 2001, Tu 2001, Lewington 1991

Olea 2000, Serafini 1998, Sartor 1997, Tian 1999, Resche 1997,

Brundage 1998, Han 2002, Srivastava 1998, Shah Syed 1999

 

7.5 EvT 7.50

Zitat

Studiencharakteristika

Studienergebnisse

Methodische Bewertung

LoE

James ND. Addition of docetaxel, zoledronic acid, or both to first-line long-term hormone therapy in prostate cancer (STAMPEDE): survival results from an adaptive, multiarm, multistage, platform randomised controlled trial. Lancet 2016;387(10024):1163-77.

• RCT, vierarmig, multizentrisch
• ADT vs. ADT+Zoledronsäure vs. ADT+Docetaxel vs. ADT+Zoledronsäure+Docetaxel; Randomisation 2:1:1:1 (Regime Docetaxel: 6 3-wöchige Zyklen 75mg/m2)
• Follow-up median 43 mo
• keine Verblindung
• Population: n=2962, PCa M+, N+ oder M0N0 high risk; "fit for chemotherapy", keine kardiovaskulären Ereignisse in der Vorgeschichte
• Primary Endpoints: OS and failure-free survival
  secondary: prostate-cancer-speciic survival and adverse events
• Studienzeitraum: 2005 - 2013 (recruitment)
• UK & Schweiz

• Baseline-Patientencharakteristika ausgewogen zwischen den Armen:; 62% N+M+; age median 65; 75% WHO performance status 0

• Effektivität
o OS: kein Effekt für Zoledronsäure+ADT (HR 0,94; 95%CI 0,79 - 1,11);
          Effekt für Docetaxel+ADT (Überleben median 81 mo vs. 71mo nur ADT;
          HR 0,78; 95%CI 0,66 - 0,93, p=0,006); (175 Todesfälle, davon 143 PCa);
          Effekt für Doce+Zol.+ADT (Überleben median 76mo vs. 71 mo nur ADT;
          HR 0,82; 95%CI 0,69-0,97; p=0,022); (187 Todesfälle, davon 150 PCa)
o failure-free-survival: kein Effekt für Zoledronsäure + ADT
          Effekt für Doce+ADT: 315 Ereignisse; HR 0,61; 95%CI 0,54-0,70
          Effekt für Doce+Zoledr.+ADT: 316 Ereignisse; HR 0,62; 95%CI 0,54- 
          0,70
o pre-planned analysis OS for metastatic disease only:
      Doce+ADT: 60 mo vs. 45 mo ADT only; HR 0,76; 95%CI 0,62-0,92;
      p=0,005;
      Doce+Zole+ADT: 55mo vs.45 ADT only; HR 0,79; 95%CI 0,66-0,96;
      p=0,015
 
• Adverse Events Grad 3/4/5:
      Doce+ ADT 52% vs. 15% ADT allein
      Doce+Zoledr.
+ ADT 52%  vs. 15% ADT allein

      darunter febrile Neutropenie: 15%
      Neutropenie: 12%

• Randomisierung
• Allocation concealment beschrieben
• keine Verblindung, aber gleiches Monitoring für alle Gruppen
• Patientencharakteristika ausbalanciert zwischen den Gruppen, aber nur sehr fitte Patienten eingeschlossen (WHO performance status 0 > 75%?
• Drop-out beschrieben, nicht relevant
• Statistische Methoden beschrieben
•  Effekt für OS als primären, pat.rel. Endpunkt
• ITT-Analyse ab Randomisierung?
• Interessenkonflikte angegeben und massiv vorhanden
• Finanzierung durch Medical Research Council UK, keine finanzielle Pharmabeteiligung

große Patientenzahl (~ 600 pro Arm, 1.200 für control), mittleres follow-up (31/2 Jahre), mittlere Ereignisrate

1+

Autor, Jahr/
Studientyp

Anzahl der Patienten/
Patientenmerkmale

Intervention/
ggf. Nachverfolgung

Vergleichs-intervention

Outcomes

Ergebnisse

Bemerkungen

Evidenz-niveau
SIGN

Fizazi K. et al, 2011

RCT

Multizentrisch

N=1904

Pat. mit  kastrationsresistenem PCa und Knochenmetastasen ohne vorausgegangene Bisphosphonattherapie

950 Pat mit

Denosumab

120mg s.c. +i.v. Placebo

Med. Follow up : 12,2Mo

951 Pat. mit Zoledronsäure i.v.4mg + s.c. Placebo

Med. Follow up :

11,2 Mo

 

Zeit bis zum Auftreten des 1. Skeletal Related Events (SRE)

Rate an Progression

Gesamtüberleben

Nebenwirkungen

Lebensqualität

Anteil Patienten mit SRE

Denosumab: 36% (n=341)

Zoledronsäure: 41% (n=386)

1. Zeit bis zum Auftreten des 1. skeletal Related Events (Monate)

Denosumab-Gruppe: 20,7 [95%KI 18,8-24,9%]

 Zoledronsäure-Gruppe: 17,1 [95%KI 15-19,4]

Cave: bei Betrachtung des absoluten Unterschieds überlappen die Konfidenzintervalle!

Nichtunterlegenheit Denosumab: HR 0,82 [95%KI 0,71-0,92] p=0,0002

Überlegenheit Denosumab: s.o. p=0,008

2. Rate an Progression

Denosumab + Zoledronsäure im Median 8,4 Mo n.s.

3. Gesamtüberleben
Denosumab im Median 19,4 Mo

Zoledronsäure im Median 19,8 Mo n.s.

4. Nebenwirkungen

Viele NW n.s., hier nur aufgeführt die mit Unterschieden

a. Grade 3 o. 4 adverse events nach CTCAE (Common Terminology Criteria for Adverse Events )

Denosumab Gruppe: 72%

Zoledronsäure Gruppe 66% p< 0,01

b. Hypokalzämie

Denosumab Gruppe: 13%

Zoledronsäure Gruppe:  6% p< 0,0001

c. Kiefernekrose

Denosumab Gruppe: 2% (n=22)

Zoledronsäure Gruppe:  1% (n=8) p = 0,09

d. Auftreten eines weiteren Malignoms

Denosumab Gruppe: 2% (n=18)

Zoledronsäure Gruppe:  1% (n=10) p = 0,13

5. Lebensqualität: nicht erfasst.

Randomisierung ok.

in Gruppe mit Zoledronsäure: 4% mehr Patienten>65J , 3,7Mo länger von ED bis Randomisierung., 2,5 Mo länger von ED Knochenmet. bis Randomisierung.

Studie ist von AMGEN gesponsert. (AMGEN sammelte die Daten und machte die Auswertung)

Studie als Non-inferioeritätsstudie angelegt, sekundär auch Auswertung auf Superiorität

1+

 

 

           

 

Autoren Jahr

Literaturtyp

untersuchte Studien/
Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

Methodische Besonder­
heiten/
Bemerkungen

LoE

Literaturbelege

Yuen 2006

Syst. Review/ Meta-analyse

10 RCTs aus syst. Suche in Medline, Embase, DARE, AMED, CENTRAL, LILACS bis April 2005

Bisphosphonate vs. Placebo o. keine Therapie bei Knochen-Metastasen durch PCa

Signifikante Ergebnisse:

bessere Schmerzreduktion in Bisphosphonat-Gruppe (OR 1,54, 95%CI 0,97-2,44, p=0,07 bzw. nach Re-Analyse OR 1,64, 95%CI 1,02-2,61, p=0,04)

weniger „skeletal events“ (path. Fraktur, Hyperkalzämie, Indikation zur Radiotherapie, Rückenmarkkompression, Indikation zur Chirurgie) in Bisphosphonat-Gruppe (OR 0,79, 95%CI 0,62-1,0, p=0,05)

mehr Übelkeit in Bisphosphonat-Gruppe (OR 1,35, 95%CI 1,02-1,77, p=0,03)

nicht signifikant:

mittlerer Schmerz-Score, Analgetika-Verbrauch, Frakturen, Rückenmarkkompression, Knochenbestrahlung, Knochenchirurgie, OS, DP, radiologisches Ansprechen, QoL, Erbrechen, Anämie, Knochenschmerz, Nierentoxizität

untersch. Bisphosphonate, wenige signifikante Ergebnisse, unterschiedl. Definitionen der Studien, signifikante Schmerzreduktion signifikant in Abhängigkeit von Analyse-Technik

1+

Adami 1989, Dearnaley 2003, Elomma 1992, Ernst 2003, Kylmala 1993, Kylmala 1997, Saad 2002, Small 2003, Smith 1989, Strang 1997

 

 

Zitat

Population

Intervention

Studiendesign

Ergebnisse

Methodische Qualität

LoE

N. D. James. Clinical Outcomes and Survival Following Treatment of Metastatic Castrate-Refractory Prostate Cancer With Docetaxel Alone or With Strontium-89, Zoledronic Acid, or Both: The TRAPEZE Randomized Clinical Trial. JAMA Oncol 2 (4):493-499, 2016.

pat.s with progressive
metastatic CRPC, with 1 o rmore sclerotic bone metastases
n = 188 in ZA arm (+ docetaxel)
n = 191 docetaxel
(ITT analysis)

ECOG 0-2 (mostly 0-1)

docetaxel alone or with ZA, Sr89, or both
- ZA: 3 i.v. doses of docetaxel plus zoledronic acid (DZA) /wk  during chemotx, then 4 doses /wk until disease progression
- comparison:
3 i.v. doses of docetaxel /wk up to 10 cycles

randomized, open-label, phase 3 trial
ZA (+Docetaxel) vs. placebo (+Docetaxel)

ZA vs. no ZA
(stratified for Sr89 use)

primary outcome: CPFS (= number of whole days from date of randomization to first occurrence of a symptomatic SRE, pain progression, or death)

secondary outcomes:
symptomatic SRE-free interval
pain rogression–free interval
overall survival (OS)
numbers of SREs (clinically detected)

median follow-up: 22mo

Clinical Progression-Free Survival:
addition of ZA had no significant impact on CPFS (HR, 0.98; 95%CI,0.85-1.14; p=0.81)

OS and Pain Progression Free Interval:
no significant differences in pain progression free interval (ZA: HR=0.91; 95%CI 0.75-1.10; p=0.31)
Median OS with and without ZA was not significantly different: 16.99 vs. 17.06mo (HR=0.99; 95%CI 0.84-1.16; p=0.91)

SRE-Free Interval:
ZA prolonged the median SRE-free interval from 11.2 to 13.6mo(HR=0.78; 95%CI 0.65-0.95; p=0.01); decrease of 30% in overall SREs in pat.s allocated to ZA therapy (424 vs 605); number of pat.s experiencing 2 or more SREs in ZA arms decreased from 154 (40%) to 103 (27%) and pat.s experiencing no SREs increased from 147 (39%) to 173 (46%)

safety:
overall, 493 known grade 3 or 4 adverse events reported relating to 237 pat.s, equally distributed between treatment arms
in total, 583 SAEs were reported, relating to 373 (49%) pat.s. Of these 286 (49%) were unrelated SAEs  and 276 (47%) were serious adverse reactions

Randomisierung? - ja
Allocation concealment beschrieben? - ja
Details der Verblindung beschrieben? - n.a.
Patientencharakteristika beschrieben (Tabelle)? - ja
ausbalanciert zwischen den Gruppen? - ja
Drop-out beschrieben/Consort-Diagramm? - ja
Statistische Methoden beschrieben? - ja
angewendete Methoden und Definitionen beschrieben (z. B. Schmerzskala, Definition von Schmerz, Messmethoden etc.) - ja
ITT-Analyse? - ja
Gab es ein Cross-over? - nein
Interessenkonflikte/Sponsoring beschrieben? - ja (Pharma-gesponsert aber "kein Einfluss auf Studiendesign und -Durchführung")

1+

T. Kamba. A phase III multicenter, randomized, controlled study of combined androgen blockade with versus without zoledronic acid in prostate cancer patients with metastatic bone disease: results of the ZAPCA trial. Int J Clin Oncol, 2016.

n = 227 men with treatment-naive
prostate cancer and bone metastasis
- ZA group: n=115
- control: n= 112

mostly ECOG 0-1

ZA w/ combined
androgen blockade (CZ group) or
combined androgen
blockade alone (CAB group)

ZA every 4w for up to 2y,
treatment was initiated 4w after first LH–RH agonist
injection

randomized, open-label, phase 3 trial
ZA (+combined ADT) vs. placebo (+combined ADT)

outcomes:
time to treatment failure (TTTF)
time to the first skeletal-related event (TTfSRE)
overall survival (OS) rates

median follow-up duration: 41.5mo

median TTTF:
ZA group 12.4mo (95%CI 10.6–16.6) vs. control 9.7 mo [95% 8.9–12.6]
HR = 0.75 (95%CI 0.57–1.00)

median TTf-SRE:
ZA group 64.7mo (95%CI 48.5- not yet estimable) vs. control 45.9mo (95%CI 34.1–55.0)

median OS:
not yet estimable for ZA group vs. control 60.2mo (95%CI 20.5- not yet estimable)

safety:
AEs of grade ≥3 in ZA group 33.0% vs. control 25.9% (p = 0.30)

Randomisierung? - ja
Allocation concealment beschrieben? - ja
Details der Verblindung beschrieben? - n.a.
Patientencharakteristika beschrieben (Tabelle)? - ja
ausbalanciert zwischen den Gruppen? - ja
Drop-out beschrieben/Consort-Diagramm? - ja
Statistische Methoden beschrieben? - ja
angewendete Methoden und Definitionen beschrieben (z. B. Schmerzskala, Definition von Schmerz, Messmethoden etc.) - ja
ITT-Analyse? - ? nein
Gab es ein Cross-over? - nein / n.a.
Interessenkonflikte/Sponsoring beschrieben? - ja

1(+)

M. R. Smith. Randomized controlled trial of early zoledronic acid in men with castration-sensitive prostate cancer and bone metastases: results of CALGB 90202 (alliance). J Clin Oncol 32 (11):1143-1150, 2014.

n = 645 patients
(ZA group: n=323 vs. n=322 placebo) with castration-sensitive PCa and bone metastases

mostly ECOG 0-1

ZA or placebo every 4w
(+ standard ADT)

treatment until first SRE

randomized, blinded, phase 3 trial
ZA (+standard ADT) vs. placebo (+standard ADT)

primary endpoint: time to first SRE
(interval between date of randomization to either RT to bone, clinical fracture, spinal cord compression, surgery to bone, or death as a result of PCa)

secondary endpoints: overall survival,
progression-free survival (PFS), safety


median time on study: 11.8mo in ZA group and 13.6mo in placebo group

time to first SRE:
ZA group 31.9mo (95%CI 24.2-40.3) vs placebo 28.8mo (95%CI 25.3-37.2)  stratified hazard ratio (HR) = 0.97 (95%CI 0-1.174) - not significant
subgroup analysis:
in men with an SRE before study randomization (n=82), median time to first SRE was 31.9mo in ZA group vs. 17.6mo in placebo group (HR=0.56; 95%CI 0.31-1.02); no-prior-SRE subgroup: median time to first SRE was similar

progression-free survival:
ZA group 10.6mo (95%CI 8.5-15.4) vs placebo 9.2mo (95%CI 8.0-11.9)
adjusted HR = 0.89 (95%CI 0.74-1.07)
(majority of the events were PSA progression (70%), followed by bone progression (22%), and patient deaths (8%))

overall survival:
ZA group 37.9mo (95%CI 34.2-49.2) vs placebo 36.0mo (95%CI 30.2-41.6)
adjusted HR = 0.88 (95%CI 0.70-1.12)

safety:
most common treatment-related grade 3 or higher events included pain (ZA group 3%; placebo 3%), hypophosphatemia (ZA group 3%; placebo 2%),
fatigue (ZA 3%; placebo 2%), and hypocalcemia (ZA 3%; placebo 1%)

Randomisierung? - ja
Allocation concealment beschrieben? - ?
Details der Verblindung beschrieben? - ja
Patientencharakteristika beschrieben (Tabelle)? - ja
ausbalanciert zwischen den Gruppen? - ja
Drop-out beschrieben/Consort-Diagramm? - ja
Statistische Methoden beschrieben? - ja
angewendete Methoden und Definitionen beschrieben (z. B. Schmerzskala, Definition von Schmerz, Messmethoden etc.) - ja
ITT-Analyse? - ja
Gab es ein Cross-over? - nein / n.a.
Interessenkonflikte/Sponsoring beschrieben? - ja

1+

M. R. Smith. Denosumab for the prevention of skeletal complications in metastatic castration-resistant prostate cancer: comparison of skeletal-related events and symptomatic skeletal events. Ann Oncol 26 (2):368-374, 2015.

pat.s with bone metastasis, ECOG status ≤2
ZA arm: n = 951
denosumab: n = 950

ECOG≤2
prior hormonal tx

s.c. denosumab
120 mg plus i.v. placebo every 4w or
s.c. placebo plus i.v. ZA 4 mg every 4w

randomized, blinded, phase 3 trial
denosumab vs. zoledronic acid

primary endpoint: skeletal-related events (SREs; radiation to bone, pathologic fracture, surgery to bone, or spinal cord compression)

preplanned exploratory end point: SSEs, defined as a subset of SREs considered symptomatic by the investigators at the time the event was first noted
(in contrast with SREs, ascertainment of SSEs does not include scheduled radiographic assessments)

first on-study SSE: fewer pat.s in denosumab arm (n = 241) vs. ZA arm (n = 289); similar results for first on-study SRE
(fewer first on-study SSEs than first on-study SREs)
first and subsequent on-study SSEs: fewer in denosumab arm (n = 329; 0.35 mean events/pat.) vs. ZA arm (n = 409; 0.43 mean events/pat.)
- compared with zoledronic acid, denosumab reduced the risk of first on-study SSE by 22% (HR=0.78; 95%CI 0.66–0.93; p=0.005)
- denosumab reduced the risk of first and subsequent on-study SSE by 22%
vs. ZA (rate ratio=0.78; 95%CI 0.65–0.92; p=0.004)
- median time to first on-study SSE was not reached in the denosumab arm; 24.2mo in ZA arm
absolute risks:
first SSE - 25,3% (Denosumab) vs. 30,3% (ZA) -- ARR = 5,0%
first SRE - 35,9% (Denosumab) vs. 40,6% (ZA) -- ARR = 4,7%
first and subsequent SSE - 34,6% (Denosumab) vs. 43% (ZA) -- ARR = 8,4%
first and subsequent SRE - 52% (Denosumab) vs. 61,4% (ZA) -- ARR = 9,4%


comparison Fizazi et al. 2011:
risk of first on-study SRE: denosumab reduced the risk of first on-study SRE, as well as the risk of first and subsequent SRE, by 18% vs. ZA

Randomisierung? - ja
Allocation concealment beschrieben? - ?
Details der Verblindung beschrieben? - ja
Patientencharakteristika beschrieben (Tabelle)? - nein
ausbalanciert zwischen den Gruppen? - ja
Drop-out beschrieben/Consort-Diagramm? - ja
Statistische Methoden beschrieben? - nein / kaum
angewendete Methoden und Definitionen beschrieben (z. B. Schmerzskala, Definition von Schmerz, Messmethoden etc.) - ja
ITT-Analyse? - ja
Gab es ein Cross-over? - nein / n.a.
Interessenkonflikte/Sponsoring beschrieben? - ja: funded by Amgen (mit Beteiligung an der Manuskripterstellung)

1-

--> Studie ausschließen?

 

7.5 EvT 7.51

Autor, Jahr/
Studientyp

Anzahl der Patienten/
Patientenmerkmale

Intervention/
ggf. Nachverfolgung

Vergleichs-intervention

Outcomes

Ergebnisse

Bemerkungen

Evidenz-niveau
SIGN

Fizazi K. et al, 2011

RCT

Multizentrisch

N=1904

Pat. mit  kastrationsresistenem PCa und Knochenmetastasen ohne vorausgegangene Bisphosphonattherapie

950 Pat mit

Denosumab

120mg s.c. +i.v. Placebo

Med. Follow up : 12,2Mo

951 Pat. mit Zoledronsäure i.v.4mg + s.c. Placebo

Med. Follow up :

11,2 Mo

 

Zeit bis zum Auftreten des 1. Skeletal Related Events (SRE)

Rate an Progression

Gesamtüberleben

Nebenwirkungen

Lebensqualität

Anteil Patienten mit SRE

Denosumab: 36% (n=341)

Zoledronsäure: 41% (n=386)

1. Zeit bis zum Auftreten des 1. skeletal Related Events (Monate)

Denosumab-Gruppe: 20,7 [95%KI 18,8-24,9%]

 Zoledronsäure-Gruppe: 17,1 [95%KI 15-19,4]

Cave: bei Betrachtung des absoluten Unterschieds überlappen die Konfidenzintervalle!

Nichtunterlegenheit Denosumab: HR 0,82 [95%KI 0,71-0,92] p=0,0002

Überlegenheit Denosumab: s.o. p=0,008

2. Rate an Progression

Denosumab + Zoledronsäure im Median 8,4 Mo n.s.

3. Gesamtüberleben
Denosumab im Median 19,4 Mo

Zoledronsäure im Median 19,8 Mo n.s.

4. Nebenwirkungen

Viele NW n.s., hier nur aufgeführt die mit Unterschieden

a. Grade 3 o. 4 adverse events nach CTCAE (Common Terminology Criteria for Adverse Events )

Denosumab Gruppe: 72%

Zoledronsäure Gruppe 66% p< 0,01

b. Hypokalzämie

Denosumab Gruppe: 13%

Zoledronsäure Gruppe:  6% p< 0,0001

c. Kiefernekrose

Denosumab Gruppe: 2% (n=22)

Zoledronsäure Gruppe:  1% (n=8) p = 0,09

d. Auftreten eines weiteren Malignoms

Denosumab Gruppe: 2% (n=18)

Zoledronsäure Gruppe:  1% (n=10) p = 0,13

5. Lebensqualität: nicht erfasst.

Randomisierung ok.

in Gruppe mit Zoledronsäure: 4% mehr Patienten>65J , 3,7Mo länger von ED bis Randomisierung., 2,5 Mo länger von ED Knochenmet. bis Randomisierung.

Studie ist von AMGEN gesponsert. (AMGEN sammelte die Daten und machte die Auswertung)

Studie als Non-inferioeritätsstudie angelegt, sekundär auch Auswertung auf Superiorität

1+

 

 

           

 

Autoren Jahr

Literaturtyp

untersuchte Studien/
Materialien

Welche Behandlungen wurden geprüft

Befunde in Bezug auf Therapie-Wirkungen und Überleben

Methodische Besonder­
heiten/
Bemerkungen

LoE

Literaturbelege

Yuen 2006

Syst. Review/ Meta-analyse

10 RCTs aus syst. Suche in Medline, Embase, DARE, AMED, CENTRAL, LILACS bis April 2005

Bisphosphonate vs. Placebo o. keine Therapie bei Knochen-Metastasen durch PCa

Signifikante Ergebnisse:

bessere Schmerzreduktion in Bisphosphonat-Gruppe (OR 1,54, 95%CI 0,97-2,44, p=0,07 bzw. nach Re-Analyse OR 1,64, 95%CI 1,02-2,61, p=0,04)

weniger „skeletal events“ (path. Fraktur, Hyperkalzämie, Indikation zur Radiotherapie, Rückenmarkkompression, Indikation zur Chirurgie) in Bisphosphonat-Gruppe (OR 0,79, 95%CI 0,62-1,0, p=0,05)

mehr Übelkeit in Bisphosphonat-Gruppe (OR 1,35, 95%CI 1,02-1,77, p=0,03)

nicht signifikant:

mittlerer Schmerz-Score, Analgetika-Verbrauch, Frakturen, Rückenmarkkompression, Knochenbestrahlung, Knochenchirurgie, OS, DP, radiologisches Ansprechen, QoL, Erbrechen, Anämie, Knochenschmerz, Nierentoxizität

untersch. Bisphosphonate, wenige signifikante Ergebnisse, unterschiedl. Definitionen der Studien, signifikante Schmerzreduktion signifikant in Abhängigkeit von Analyse-Technik

1+

Adami 1989, Dearnaley 2003, Elomma 1992, Ernst 2003, Kylmala 1993, Kylmala 1997, Saad 2002, Small 2003, Smith 1989, Strang 1997

 

 

 

7.5 EvT 7.52

Autor, Jahr/
Studientyp

Anzahl der Patienten/
Patientenmerkmale

Intervention/
ggf. Nachverfolgung

Vergleichs-intervention

Outcomes

Ergebnisse

Bemerkungen

Evidenz-niveau
SIGN

Fizazi K. et al, 2011

RCT

Multizentrisch

N=1904

Pat. mit  kastrationsresistenem PCa und Knochenmetastasen ohne vorausgegangene Bisphosphonattherapie

950 Pat mit

Denosumab

120mg s.c. +i.v. Placebo

Med. Follow up : 12,2Mo

951 Pat. mit Zoledronsäure i.v.4mg + s.c. Placebo

Med. Follow up :

11,2 Mo

 

Zeit bis zum Auftreten des 1. Skeletal Related Events (SRE)

Rate an Progression

Gesamtüberleben

Nebenwirkungen

Lebensqualität

Anteil Patienten mit SRE

Denosumab: 36% (n=341)

Zoledronsäure: 41% (n=386)

1. Zeit bis zum Auftreten des 1. skeletal Related Events (Monate)

Denosumab-Gruppe: 20,7 [95%KI 18,8-24,9%]

 Zoledronsäure-Gruppe: 17,1 [95%KI 15-19,4]

Cave: bei Betrachtung des absoluten Unterschieds überlappen die Konfidenzintervalle!

Nichtunterlegenheit Denosumab: HR 0,82 [95%KI 0,71-0,92] p=0,0002

Überlegenheit Denosumab: s.o. p=0,008

2. Rate an Progression

Denosumab + Zoledronsäure im Median 8,4 Mo n.s.

3. Gesamtüberleben
Denosumab im Median 19,4 Mo

Zoledronsäure im Median 19,8 Mo n.s.

4. Nebenwirkungen

Viele NW n.s., hier nur aufgeführt die mit Unterschieden

a. Grade 3 o. 4 adverse events nach CTCAE (Common Terminology Criteria for Adverse Events )

Denosumab Gruppe: 72%

Zoledronsäure Gruppe 66% p< 0,01

b. Hypokalzämie

Denosumab Gruppe: 13%

Zoledronsäure Gruppe:  6% p< 0,0001

c. Kiefernekrose

Denosumab Gruppe: 2% (n=22)

Zoledronsäure Gruppe:  1% (n=8) p = 0,09

d. Auftreten eines weiteren Malignoms

Denosumab Gruppe: 2% (n=18)

Zoledronsäure Gruppe:  1% (n=10) p = 0,13

5. Lebensqualität: nicht erfasst.

Randomisierung ok.

in Gruppe mit Zoledronsäure: 4% mehr Patienten>65J , 3,7Mo länger von ED bis Randomisierung., 2,5 Mo länger von ED Knochenmet. bis Randomisierung.

Studie ist von AMGEN gesponsert. (AMGEN sammelte die Daten und machte die Auswertung)

Studie als Non-inferioeritätsstudie angelegt, sekundär auch Auswertung auf Superiorität

1+

 

 

           

 

 

 

 

7.6.1.2 EvT 7.55

Quelle

Fazit/Empfehlung

Empfehlungsgrad

NICE 2008

 

Rectal Problems after Radiotherapy

Radiotherapy for prostate cancer may lead to a range of adverse effects on the bowel. Men receiving radiotherapy to pelvic lymph nodes may experience problems from irradiation of the small bowel. More commonly, radiotherapy is targeted at the prostate alone (and not the lymph nodes) and it is the rectum that is at risk of radiation effects.

Acute and late stage toxicity in the bowel is an important complication of radiotherapy for prostate cancer.

Radiation-induced injury to the bowel may be functional without underlying anatomical disturbance, and symptoms and signs may well be due to treatable causes or intercurrent pathology. There is an increased risk of rectal cancer after pelvic radiation but faecal occult blood testing is a poor discriminator due to telangiectasis and the emerging National Screening Programe for bowel cancer is inappropriate for these men.

There is a relative lack of research and specialisation by oncologists and gastroenterologists in radiation-induced gastrointestinal (GI) tract injury. In consequence, there is no structured way for patients with GI toxicity to be assessed and potential protective treatments have not been tested adequately in man.

Recommendations:

- Men presenting with symptoms consistent with radiation-induced enteropathy should be fully investigated (including using flexible sigmoidoscopy) to exclude inflammatory bowel disease or malignancy of the large bowel and to ascertain the nature of the radiation injury. Particular caution should be taken with anterior wall rectal biopsy following brachytherapy because of the risk of fistulation.

-Men treated with radical radiotherapy for prostate cancer should be offered flexible sigmoidoscopy every 5 years.

- Steroid enemas should not be used for treating men with radiation proctopathy.

- The nature and treatment of radiation-induced injury to the gastrointestinal (GI) tract should be included in the training programmes for oncologists and gastroenterologists. Qualifying statement: These recommendations are based on expert opinion and GDG consensus.

Clinical Evidence

Many of the trials were not restricted to prostate cancer but included any patients with any malignancy requiring pelvic EBRT. There was inconsistent evidence for the use of aminosalicylatessucralfate and misoprostol for the prevention of acute bowel toxicity during pelvic radiotherapy. Other trials reported effective interventions for treatment of acute bowel toxicity but each intervention was only tested in a single trial.

There was no evidence, from fifteen randomised trials in patients receiving pelvic radiotherapy, to support the use of radioprotective agents (see evidence review). Other randomised trials demonstrated clinical effectiveness of loperamide (Sherman et al. 1989), octreotide (Yavuz et al. 2002) and butyrate (Vernia et al. 2000) for acute radiation-induced diarrhoea.

A systematic review of non-surgical interventions for late radiation proctopathy (Denton et al. 2002) identified six randomised trials. Although some of studies reported positive results, the trials were small and each examined a different intervention. There was insufficient evidence,therefore, to recommend any specific intervention.

A systematic review (McGough et al. 2004) concluded there was little evidence to support the use of nutritional interventions for acute or chronic gastrointestinal symptoms. Due to the lack of good evidence for this question the GDG commissioned an expert position paper (see Appendix B of the evidence review).

Recommendation:

Men with metastatic prostate cancer should be offered tailored information and access to specialist urology and palliative care teams to address the specific needs of men with metastatic cancer. They should have the opportunity to discuss any significant changes in their disease status or symptoms as these occur.

The regular assessment of needs should be applied systematically to men with metastatic prostate cancer.

Palliative interventions at any stage should be integrated into coordinated care, and any transitions between care settings should be facilitated as smoothly as possible.

Healthcare professionals should discuss personal preferences for palliative care as early as possible with men with metastatic prostate cancer, their partners and carers. Treatment/care plans should be tailored accordingly and the preferred place of care should be identified.

Healthcare professionals should ensure that palliative care is available when needed and is not limited to the end of life. It should not be restricted to being associated with hospice care.

Qualifying statement: There is evidence from qualitative studies and GDG (group) consensus to support these recommendations

Evidence:

Literature searches did not find any studies that compared palliative care settings or models in prostate cancer. Several observational studies described experiences with palliative care in particular settings. Although this shows that care is possible in such settings, without comparative studies there was no evidence about which palliative care model or setting was best.Several themes emerged: the need for multidisciplinary delivery of palliative care (Palmieri & Waxman 2005; Pienta et al. 1996; Cunliffe 2003; Ok et al. 2005) and the integration of curative and palliative treatment (Ok et al. 2005; Pienta et al. 1996) during the often long course of the disease (Green et al. 2002).

 

 

7.6.1.2 EvT 7.56

Quelle

Fazit/Empfehlung

Empfehlungsgrad

NICE 2008

 

Rectal Problems after Radiotherapy

Radiotherapy for prostate cancer may lead to a range of adverse effects on the bowel. Men receiving radiotherapy to pelvic lymph nodes may experience problems from irradiation of the small bowel. More commonly, radiotherapy is targeted at the prostate alone (and not the lymph nodes) and it is the rectum that is at risk of radiation effects.

Acute and late stage toxicity in the bowel is an important complication of radiotherapy for prostate cancer.

Radiation-induced injury to the bowel may be functional without underlying anatomical disturbance, and symptoms and signs may well be due to treatable causes or intercurrent pathology. There is an increased risk of rectal cancer after pelvic radiation but faecal occult blood testing is a poor discriminator due to telangiectasis and the emerging National Screening Programe for bowel cancer is inappropriate for these men.

There is a relative lack of research and specialisation by oncologists and gastroenterologists in radiation-induced gastrointestinal (GI) tract injury. In consequence, there is no structured way for patients with GI toxicity to be assessed and potential protective treatments have not been tested adequately in man.

Recommendations:

- Men presenting with symptoms consistent with radiation-induced enteropathy should be fully investigated (including using flexible sigmoidoscopy) to exclude inflammatory bowel disease or malignancy of the large bowel and to ascertain the nature of the radiation injury. Particular caution should be taken with anterior wall rectal biopsy following brachytherapy because of the risk of fistulation.

-Men treated with radical radiotherapy for prostate cancer should be offered flexible sigmoidoscopy every 5 years.

- Steroid enemas should not be used for treating men with radiation proctopathy.

- The nature and treatment of radiation-induced injury to the gastrointestinal (GI) tract should be included in the training programmes for oncologists and gastroenterologists. Qualifying statement: These recommendations are based on expert opinion and GDG consensus.

Clinical Evidence

Many of the trials were not restricted to prostate cancer but included any patients with any malignancy requiring pelvic EBRT. There was inconsistent evidence for the use of aminosalicylatessucralfate and misoprostol for the prevention of acute bowel toxicity during pelvic radiotherapy. Other trials reported effective interventions for treatment of acute bowel toxicity but each intervention was only tested in a single trial.

There was no evidence, from fifteen randomised trials in patients receiving pelvic radiotherapy, to support the use of radioprotective agents (see evidence review). Other randomised trials demonstrated clinical effectiveness of loperamide (Sherman et al. 1989), octreotide (Yavuz et al. 2002) and butyrate (Vernia et al. 2000) for acute radiation-induced diarrhoea.

A systematic review of non-surgical interventions for late radiation proctopathy (Denton et al. 2002) identified six randomised trials. Although some of studies reported positive results, the trials were small and each examined a different intervention. There was insufficient evidence,therefore, to recommend any specific intervention.

A systematic review (McGough et al. 2004) concluded there was little evidence to support the use of nutritional interventions for acute or chronic gastrointestinal symptoms. Due to the lack of good evidence for this question the GDG commissioned an expert position paper (see Appendix B of the evidence review).

Recommendation:

Men with metastatic prostate cancer should be offered tailored information and access to specialist urology and palliative care teams to address the specific needs of men with metastatic cancer. They should have the opportunity to discuss any significant changes in their disease status or symptoms as these occur.

The regular assessment of needs should be applied systematically to men with metastatic prostate cancer.

Palliative interventions at any stage should be integrated into coordinated care, and any transitions between care settings should be facilitated as smoothly as possible.

Healthcare professionals should discuss personal preferences for palliative care as early as possible with men with metastatic prostate cancer, their partners and carers. Treatment/care plans should be tailored accordingly and the preferred place of care should be identified.

Healthcare professionals should ensure that palliative care is available when needed and is not limited to the end of life. It should not be restricted to being associated with hospice care.

Qualifying statement: There is evidence from qualitative studies and GDG (group) consensus to support these recommendations

Evidence:

Literature searches did not find any studies that compared palliative care settings or models in prostate cancer. Several observational studies described experiences with palliative care in particular settings. Although this shows that care is possible in such settings, without comparative studies there was no evidence about which palliative care model or setting was best.Several themes emerged: the need for multidisciplinary delivery of palliative care (Palmieri & Waxman 2005; Pienta et al. 1996; Cunliffe 2003; Ok et al. 2005) and the integration of curative and palliative treatment (Ok et al. 2005; Pienta et al. 1996) during the often long course of the disease (Green et al. 2002).

 

 

7.6.2 EvT 7.60

Quelle

Fazit/Empfehlung

Empfehlungsgrad

ACP 2008

In patients with serious illness at the end of life, clinicians should regularly assess patients for pain, dyspnea, and depression. (GRADE: moderate quality of evidence)

Although each patient and family will require individualized assessment and care, the evidence shows that a set of general issues is shared widely among patients needing end-of-life care. These issues include pain and other symptom management, psychological well-being, care coordination and advance care planning, and caregiver burden. The evidence was classified as moderate quality because it was mostly derived from studies of patients with cancer or cancer-predominant populations in addition to being heterogeneous. (Patrick DL, et al. Ann Intern Med. 2003; Steinhauser KE, et al. JAMA. 2000; Hanson LC, et al. J Am Geriatr Soc. 1997; Singer PA, et al. JAMA. 1999; Wenrich MD, et al. J Pain Symptom Manage. 2003; Teno JM, et al. J Pain Symptom Manage. 2001; Teno JM, et al. JAMA. 2004; Field MJ, Cassel CK, eds. Institute of Medicine. Washington, DC: National Academies Pr; 1997)

In patients with serious illness at the end of life, clinicians should regularly assess patients for pain, dyspnea, and depression. (GRADE: moderate quality of evidence)

Although each patient and family will require individualized assessment and care, the evidence shows that a set of general issues is shared widely among patients needing end-of-life care. These issues include pain and other symptom management, psychological well-being, care coordination and advance care planning, and caregiver burden. The evidence was classified as moderate quality because it was mostly derived from studies of patients with cancer or cancer-predominant populations in addition to being heterogeneous. (Patrick DL, et al. Ann Intern Med. 2003; Steinhauser KE, et al. JAMA. 2000; Hanson LC, et al. J Am Geriatr Soc. 1997; Singer PA, et al. JAMA. 1999; Wenrich MD, et al. J Pain Symptom Manage. 2003; Teno JM, et al. J Pain Symptom Manage. 2001; Teno JM, et al. JAMA. 2004; Field MJ, Cassel CK, eds. Institute of Medicine. Washington, DC: National Academies Pr; 1997)

In patients with serious illness at the end of life, clinicians should use therapies of proven effectiveness to manage pain. For patients with cancer, this includes nonsteroidal anti-inflammatory drugs, opioids, and bisphosphonates. (GRADE: moderate quality of evidence)

Clinicians should use specific effective therapies for all patients with acute and chronic pain. Strong evidence supports using nonsteroidal anti-inflammatory drugs, opioids, and bisphosphonates for pain relief in patients with cancer. Bisphosphonates are effective for bone pain relief in patients with breast cancer and myeloma. (Carr D, et al. AHRQ publication no. 02-E032, 2002; Knols R, et al. J Clin Oncol. 2005; Lee H, et al. Eur J Pain. 2005; Higginson IJ, et al. University of Wales College of Medicine; 2000; Detmar SB, et al. JAMA. 2002; Jordhøy MS, et al. J Clin Oncol. 2001)

In patients with serious illness at the end of life, clinicians should use therapies of proven effectiveness to manage dyspnea, which include opioids in patients with unrelieved dyspnea and oxygen for short-term relief of hypoxemia. (Grade: moderate quality of evidence)

Opioids should be considered in patients with severe and unrelieved dyspnea, for example, in cancer and cardiopulmonary disease at the end of life. Clinicians should consider the use of oxygen for hypoxemia for advanced chronic obstructive pulmonary disease. Evidence also supports the use of beta-agonists for treating dyspnea in chronic obstructive pulmonary disease, but this use has not been studied for end-of-life care. (Detmar SB, et al. JAMA. 2002; Jordhøy MS, et al. J Clin Oncol. 2001; Jennings AL, et al. Thorax. 2002; Abernethy AP, et al. BMJ. 2003; Bruera E, et al. Palliat Med. 2003; Shukla V, et al. Technology report no 65. Ottawa: Canadian Coordinating Office for Health Technology Assessment; 2006; Grande GE, et al. Palliat Med. 2000; Grande GE, et al. Palliat Med. 1997)

strong recommendation

 

 

 

 

strong recommendation

 

 

 

 

strong recommendation

 

 

 

strong recommendation

LG Hessen 2009

Schwäche:

reversible Ursachen, die zum Schwächegefühl beitragen, identifizieren und beheben

nur bei entsprechender Prognose oder dem klinischen Verdacht auf eine potenziell reversible Ursache weitere Diagnostik betreiben

Intravenöse Hyperalimentation ohne nachgewiesenen Effekt

mit Patienten gemeinsam nach Lösungen suchen, z. B. Erklärung, dass das Gefühl der Schwäche fluktuiert. Vorschläge machen, die Kraft einzuteilen, Ruhepausen mit Aktivität abzuwechseln, den Tagesablauf anzupassen. Vor wichtigen Ereignissen (z. B. Geburtstage, Hochzeit, Hochzeitstag) kann eine Erhöhung der Steroide oder ggf. eine Bluttransfusion kurzfristig eine Besserung des Schwächegefühls bringen. Körperliche Aktivierung (z. B. Gymnastik im Bett, Stretchbänder).

Gabe von Steroiden unterschiedlich beurteilt, jedoch kann als allgemein roborierende Maßnahme ein Versuch mit 4 mg Dexamethason indiziert sein (Ream E, Stone P. Clinical interventions for fatigue. In: Fatigue in cancer. Armes J, et al. Oxford University Press, Oxford, 2004: 255-277)

Wirksamkeit einer hormonellen Therapie (z. B. Medroxyprogesteronacetat, Megestrolacetat, Testosteron, Erythropoetin oder Melatonin) nicht ausreichend belegt, kann nicht empfohlen werden (Ream E, Stone P. Clinical interventions for fatigue. In: Fatigue in cancer. Armes J, et al. Oxford University Press, Oxford, 2004: 255-277)

Therapie potenziell behandelbarer Ursachen bei Schwäche

Anämie: Transfusion bei Hb < 8 g/l, weitere Anämiezeichen wie Kurzatmigkeit, Müdigkeit

Hypokaliämie: ggf. Schleifendiuretikum durch ein kaliumsparendes Diuretikum ersetzen, Substitution über Nahrung (Bananen, Tomaten, Zitrusfrüchte)

Hyperkalzämie: Hydratation, Bisphosphonate

Hypotonie: Flüssigkeit, Medikamente überprüfen/absetzen

Schilddrüsenfunktionsstörung: Medikamente überprüfen, ggf. Therapie

Medikamente: Reduktion oder Wechsel (Steroide, Antidepressiva, Benzodiazepine, Opioide), sedierende Medikamente möglichst nur zur Nacht geben

Schlaflosigkeit: Ursachen besprechen (Sorgen, Angst etc.), für ruhige, entspannte Umgebung sorgen, ggf. Benzodiazepine

Anorexie, Kachexie: Steroide (z. B. 4 mg Dexamethason p.o.)

Depressives Syndrom: Antidepressiva

Inaktivität: Krankengymnastik

Gehirnmetastase: Dexamethason, ggf. Bestrahlung/systemische Chemotherapie

Rückenmarkskompression: Dexamethason (Bausewein C, et al. Arzneitherapie in der Palliativmedizin. 1. Aufl. Urban und Fischer, München, Jena, 2005), z. B. beginnend mit 10-20 mg i.v., dann p.o.16 mg/die, ggf. Bestrahlung, ggf. operative Entlastung

Fatigue:

vor Einleitung »therapeutischer« Maßnahmen immer unrealistische Erwartungen an den Therapieerfolg verhindern

Behandlungsversuche mit Amphetaminen (Methylphenidat) in randomisierten Studien nicht erfolgreicher als Placebogabe (Bruera E, et al. J Clin Oncol 2006)

Viele Betroffene profitieren von Informationsbroschüren (Deutsche Krebshilfe oder andere Patientenorganisationen). Besonders für Angehörige hilfreich, da sie sich weniger hilflos im Umgang mit dem erkrankten Familienmitglied

fühlen (Glaus A, et al. Pflege 2002)

klinische Unterscheidung von Depression und Fatigue im Einzelfall schwierig (Visser M, Smets E. Support Care Cancer 1998), jedoch Fatigue nicht erfolgreich mit Antidepressiva zu behandeln (Morrow G, et al. Proceedings American Society of Clinical Oncology 2001)

wesentliche Aufgabe der behandelnden Ärzte: Vermittlung der Tatsache, dass Fatigue Ausdruck der fortschreitenden Krankheit ist (Glaus A. Fatigue. In: Lehrbuch der Palliativmedizin. Aulert E, Nauck F, Radbruch L (Hrsg.). Schattauer Verlag, Stuttgart, 2007)

Unterstützende Maßnahmen

kein Verzicht auf gewohnte kleine »Alltagsfreuden« wie Alkohol oder andere Genussmittel

deutlich stärkerer Profit von gezielter Suche nach »Womit könnten Sie sich denn heute etwas Gutes tun?« als von medikamentösen Therapieversuchen

Armbewegungen im Sitzen oder Liegen, Hin- und Herwiegen zu Musik sowie passive Bewegungsübungen können selbst bei schwächeren Patienten das Wohlbefinden günstig beeinflussen (Mock V, et al. Oncology Nursing Forum 1997)

Unruhe:

möglichst Behandlung der Ursachen (Schmerzen, Atemnot, Augen- und Mundtrockenheit, Harnverhalt, Exsikkose, Hypoglykämie, Hyperkalzämie, Hirnmetastasen, Bewegungsunfähigkeit aufgrund von Schwäche, Medikamente (paradoxe Reaktion auf Benzodiazepine), Psychosoziale Ursachen (unerledigte Angelegenheiten, keine oder »falsche« Sitzwache))

Schaffen einer vertrauten Atmosphäre (bekannte Musik, Bettdecke, ruhige Anwesenheit von Angehörigen oder vertrauten Personen)

medikamentöse Therapie:

Promethazin: 15-75 mg p.o

Diazepam: 2-10 mg p.o., i.v. (nicht s.c.), 10-20 mg rektal 8-stdl.

Lorazepam: 0,5-2,5 mg p.o., i.m., i.v.

Midazolam: bei Bedarf: 2,5-5 mg s.c. (nicht für s.c. Gabe zugelassen), p.o. oder 1-2,5 mg i.v. oder kontinuierlich: 10-60 mg/24 h in subkutaner Spritzenpumpe. Kurze HWZ, stark sedierend

Flunitrazepam: 1-2 mg s.c. (nicht für s.c. Gabe zugelassen), p.o., 0,5-2 mg i.v., i.m., stark sedierend

Prothipendyl: (z. B. Dominal®) 20-80 mg p.o., i.v.

Pipamperon: 20-80 mg p.o.

Melperon: 25-50 mg 8-stdl. p.o.

Bei Halluzinationen, Alpträumen, paranoiden Symptomen (v.a. delirantes Syndrom)

Haloperidol (z. B. als Generikum verfügbar) 1,5-3 mg s.c. 8-stdl. bei älteren Patienten, 5 mg s.c. 8-stdl. bei jüngeren Patienten, 10-30 mg/24 h s.c., i.v.

Levomepromazin (z. B. als Generikum verfügbar) 10-50 mg 4-stdl. p.o., s.c., i.m., sedierend, als Tropfen gut dosierbar

parenterale Gabe von Medikamenten notwendig (Patienten meist nicht mehr in der Lage sind zu schlucken)

subkutane Gabe zu bevorzugen (einfachere Applikation (auch zuhause möglich))

bei überwiegender Unruhe vor allem z. B. bei Patienten mit Hirnmetastasen Benzodiazepine bevorzugen (erniedrigen im Vergleich zu Neuroleptika nicht die Krampfschwelle), bei diesen bietet sich auch Therapieversuch mit Kortison an

Midazolam (nicht für s.c. Gabe zugelassen) und Haloperidol können in der Spritzenpumpe mit Morphin gemischt werden

Falls Patient unter Midazolam (120 mg/24 h) und Haloperidol (30 mg/24 h) nicht deutlich ruhiger wird, kann Phenobarbital (200 mg s.c. oder i.m. bei Bedarf, oder 800-1600 mg/24 h) in Spritzenpumpe gegeben werden.

aktives Vorgehen notwendig, einerseits um des Patienten Willen, andererseits aber auch wegen der möglichen Beunruhigung der Angehörigen, da sie dieses Bild der letzten Stunden des Patienten oft im Gedächtnis behalten

-

DKG 2008

D 6 Palliativmedizin

Die therapeutische Strategie in der Palliativsitution sollte interdisziplinar festgelegt werden, z. B. im Rahmen einer Tumorkonferenz. Patientinnen sind ihren individuellen Bedurfnissen entsprechend ausfuhrlich uber die Therapieoptionen aufzuklaren und in die Entscheidung miteinzubeziehen. Tumorspezifische Therapieoptionen (siehe Abschnitt C dieser Leitlinie), supportive Masnahmen, eine bedarfsgerechte psychosoziale und psychoonkologische Versorgung (siehe Abschnitt D1 bis D3) sowie die Sicherstellung von adaquater Schmerztherapie und Ernahrung sind integrale Bestandteile des palliativen Therapiekonzepts.

Dem Wunsch der Patientin nach Informationen uber alle relevanten verfugbaren Masnahmen, Hilfs- und Betreuungsangebote (z. B. Krebsberatungsstellen, Selbsthilfegruppen, Hospize) ist nachzukommen. Auch komplementare/unkonventionelle Behandlungsmethoden sollten offen mit den Patientinnen besprochen werden, um auch ungunstige Wechselwirkungen mit anderen Therapeutika zu vermeiden.

Zu den Aspekten der palliativmedizinischen Versorgung wird auch auf themenspezifische Leitlinien verwiesen:

– S3-Leitlinien der Deutschen Gesellschaft fur Ernahrungsmedizin/ESPEN Guidelines on Enteral Nutrition unter www.awmf-leitlinien.de, Registernr. 073/006e und 073/005e;

– Leitlinie Medikamentose Schmerztherapie der DKG unter www.awmf-leitlinien.de, Registernr. 032/039, zur Zeit in Uberarbeitung mit der Deutschen Interdisziplinaren Vereinigung fur Schmerztherapie;

– Hausarztliche Leitlinie Palliativversorgung der Leitliniengruppe Hessen unter http://www.leitlinien.de/leitlinienanbieter/deutsch/pdf/hessenpalliativ.

 

 

7.6.2 EvT 7.61

Quelle

Fazit/Empfehlung

Empfehlungsgrad

ACP 2008

In patients with serious illness at the end of life, clinicians should regularly assess patients for pain, dyspnea, and depression. (GRADE: moderate quality of evidence)

Although each patient and family will require individualized assessment and care, the evidence shows that a set of general issues is shared widely among patients needing end-of-life care. These issues include pain and other symptom management, psychological well-being, care coordination and advance care planning, and caregiver burden. The evidence was classified as moderate quality because it was mostly derived from studies of patients with cancer or cancer-predominant populations in addition to being heterogeneous. (Patrick DL, et al. Ann Intern Med. 2003; Steinhauser KE, et al. JAMA. 2000; Hanson LC, et al. J Am Geriatr Soc. 1997; Singer PA, et al. JAMA. 1999; Wenrich MD, et al. J Pain Symptom Manage. 2003; Teno JM, et al. J Pain Symptom Manage. 2001; Teno JM, et al. JAMA. 2004; Field MJ, Cassel CK, eds. Institute of Medicine. Washington, DC: National Academies Pr; 1997)

In patients with serious illness at the end of life, clinicians should regularly assess patients for pain, dyspnea, and depression. (GRADE: moderate quality of evidence)

Although each patient and family will require individualized assessment and care, the evidence shows that a set of general issues is shared widely among patients needing end-of-life care. These issues include pain and other symptom management, psychological well-being, care coordination and advance care planning, and caregiver burden. The evidence was classified as moderate quality because it was mostly derived from studies of patients with cancer or cancer-predominant populations in addition to being heterogeneous. (Patrick DL, et al. Ann Intern Med. 2003; Steinhauser KE, et al. JAMA. 2000; Hanson LC, et al. J Am Geriatr Soc. 1997; Singer PA, et al. JAMA. 1999; Wenrich MD, et al. J Pain Symptom Manage. 2003; Teno JM, et al. J Pain Symptom Manage. 2001; Teno JM, et al. JAMA. 2004; Field MJ, Cassel CK, eds. Institute of Medicine. Washington, DC: National Academies Pr; 1997)

In patients with serious illness at the end of life, clinicians should use therapies of proven effectiveness to manage pain. For patients with cancer, this includes nonsteroidal anti-inflammatory drugs, opioids, and bisphosphonates. (GRADE: moderate quality of evidence)

Clinicians should use specific effective therapies for all patients with acute and chronic pain. Strong evidence supports using nonsteroidal anti-inflammatory drugs, opioids, and bisphosphonates for pain relief in patients with cancer. Bisphosphonates are effective for bone pain relief in patients with breast cancer and myeloma. (Carr D, et al. AHRQ publication no. 02-E032, 2002; Knols R, et al. J Clin Oncol. 2005; Lee H, et al. Eur J Pain. 2005; Higginson IJ, et al. University of Wales College of Medicine; 2000; Detmar SB, et al. JAMA. 2002; Jordhøy MS, et al. J Clin Oncol. 2001)

In patients with serious illness at the end of life, clinicians should use therapies of proven effectiveness to manage dyspnea, which include opioids in patients with unrelieved dyspnea and oxygen for short-term relief of hypoxemia. (Grade: moderate quality of evidence)

Opioids should be considered in patients with severe and unrelieved dyspnea, for example, in cancer and cardiopulmonary disease at the end of life. Clinicians should consider the use of oxygen for hypoxemia for advanced chronic obstructive pulmonary disease. Evidence also supports the use of beta-agonists for treating dyspnea in chronic obstructive pulmonary disease, but this use has not been studied for end-of-life care. (Detmar SB, et al. JAMA. 2002; Jordhøy MS, et al. J Clin Oncol. 2001; Jennings AL, et al. Thorax. 2002; Abernethy AP, et al. BMJ. 2003; Bruera E, et al. Palliat Med. 2003; Shukla V, et al. Technology report no 65. Ottawa: Canadian Coordinating Office for Health Technology Assessment; 2006; Grande GE, et al. Palliat Med. 2000; Grande GE, et al. Palliat Med. 1997)

strong recommendation

 

 

 

 

strong recommendation

 

 

 

 

strong recommendation

 

 

 

strong recommendation

NICE 2008

 

Rectal Problems after Radiotherapy

Radiotherapy for prostate cancer may lead to a range of adverse effects on the bowel. Men receiving radiotherapy to pelvic lymph nodes may experience problems from irradiation of the small bowel. More commonly, radiotherapy is targeted at the prostate alone (and not the lymph nodes) and it is the rectum that is at risk of radiation effects.

Acute and late stage toxicity in the bowel is an important complication of radiotherapy for prostate cancer.

Radiation-induced injury to the bowel may be functional without underlying anatomical disturbance, and symptoms and signs may well be due to treatable causes or intercurrent pathology. There is an increased risk of rectal cancer after pelvic radiation but faecal occult blood testing is a poor discriminator due to telangiectasis and the emerging National Screening Programe for bowel cancer is inappropriate for these men.

There is a relative lack of research and specialisation by oncologists and gastroenterologists in radiation-induced gastrointestinal (GI) tract injury. In consequence, there is no structured way for patients with GI toxicity to be assessed and potential protective treatments have not been tested adequately in man.

Recommendations:

- Men presenting with symptoms consistent with radiation-induced enteropathy should be fully investigated (including using flexible sigmoidoscopy) to exclude inflammatory bowel disease or malignancy of the large bowel and to ascertain the nature of the radiation injury. Particular caution should be taken with anterior wall rectal biopsy following brachytherapy because of the risk of fistulation.

-Men treated with radical radiotherapy for prostate cancer should be offered flexible sigmoidoscopy every 5 years.

- Steroid enemas should not be used for treating men with radiation proctopathy.

- The nature and treatment of radiation-induced injury to the gastrointestinal (GI) tract should be included in the training programmes for oncologists and gastroenterologists. Qualifying statement: These recommendations are based on expert opinion and GDG consensus.

Clinical Evidence

Many of the trials were not restricted to prostate cancer but included any patients with any malignancy requiring pelvic EBRT. There was inconsistent evidence for the use of aminosalicylates,  sucralfate and misoprostol for the prevention of acute bowel toxicity during pelvic radiotherapy. Other trials reported effective interventions for treatment of acute bowel toxicity but each intervention was only tested in a single trial.

There was no evidence, from fifteen randomised trials in patients receiving pelvic radiotherapy, to support the use of radioprotective agents (see evidence review). Other randomised trials demonstrated clinical effectiveness of loperamide (Sherman et al. 1989), octreotide (Yavuz et al. 2002) and butyrate (Vernia et al. 2000) for acute radiation-induced diarrhoea.

A systematic review of non-surgical interventions for late radiation proctopathy (Denton et al. 2002) identified six randomised trials. Although some of studies reported positive results, the trials were small and each examined a different intervention. There was insufficient evidence,therefore, to recommend any specific intervention.

A systematic review (McGough et al. 2004) concluded there was little evidence to support the use of nutritional interventions for acute or chronic gastrointestinal symptoms. Due to the lack of good evidence for this question the GDG commissioned an expert position paper (see Appendix B of the evidence review).

Recommendation:

Men with metastatic prostate cancer should be offered tailored information and access to specialist urology and palliative care teams to address the specific needs of men with metastatic cancer. They should have the opportunity to discuss any significant changes in their disease status or symptoms as these occur.

The regular assessment of needs should be applied systematically to men with metastatic prostate cancer.

Palliative interventions at any stage should be integrated into coordinated care, and any transitions between care settings should be facilitated as smoothly as possible.

Healthcare professionals should discuss personal preferences for palliative care as early as possible with men with metastatic prostate cancer, their partners and carers. Treatment/care plans should be tailored accordingly and the preferred place of care should be identified.

Healthcare professionals should ensure that palliative care is available when needed and is not limited to the end of life. It should not be restricted to being associated with hospice care.

Qualifying statement: There is evidence from qualitative studies and GDG (group) consensus to support these recommendations

Evidence:

Literature searches did not find any studies that compared palliative care settings or models in prostate cancer. Several observational studies described experiences with palliative care in particular settings. Although this shows that care is possible in such settings, without comparative studies there was no evidence about which palliative care model or setting was best.Several themes emerged: the need for multidisciplinary delivery of palliative care (Palmieri & Waxman 2005; Pienta et al. 1996; Cunliffe 2003; Ok et al. 2005) and the integration of curative and palliative treatment (Ok et al. 2005; Pienta et al. 1996) during the often long course of the disease (Green et al. 2002).

 

 

7.6.2 EvT 7.62

Quelle

Fazit/Empfehlung

Empfehlungsgrad

LG Hessen 2009

Schwäche:

reversible Ursachen, die zum Schwächegefühl beitragen, identifizieren und beheben

nur bei entsprechender Prognose oder dem klinischen Verdacht auf eine potenziell reversible Ursache weitere Diagnostik betreiben

Intravenöse Hyperalimentation ohne nachgewiesenen Effekt

mit Patienten gemeinsam nach Lösungen suchen, z. B. Erklärung, dass das Gefühl der Schwäche fluktuiert. Vorschläge machen, die Kraft einzuteilen, Ruhepausen mit Aktivität abzuwechseln, den Tagesablauf anzupassen. Vor wichtigen Ereignissen (z. B. Geburtstage, Hochzeit, Hochzeitstag) kann eine Erhöhung der Steroide oder ggf. eine Bluttransfusion kurzfristig eine Besserung des Schwächegefühls bringen. Körperliche Aktivierung (z. B. Gymnastik im Bett, Stretchbänder).

Gabe von Steroiden unterschiedlich beurteilt, jedoch kann als allgemein roborierende Maßnahme ein Versuch mit 4 mg Dexamethason indiziert sein (Ream E, Stone P. Clinical interventions for fatigue. In: Fatigue in cancer. Armes J, et al. Oxford University Press, Oxford, 2004: 255-277)

Wirksamkeit einer hormonellen Therapie (z. B. Medroxyprogesteronacetat, Megestrolacetat, Testosteron, Erythropoetin oder Melatonin) nicht ausreichend belegt, kann nicht empfohlen werden (Ream E, Stone P. Clinical interventions for fatigue. In: Fatigue in cancer. Armes J, et al. Oxford University Press, Oxford, 2004: 255-277)

Therapie potenziell behandelbarer Ursachen bei Schwäche

Anämie: Transfusion bei Hb < 8 g/l, weitere Anämiezeichen wie Kurzatmigkeit, Müdigkeit

Hypokaliämie: ggf. Schleifendiuretikum durch ein kaliumsparendes Diuretikum ersetzen, Substitution über Nahrung (Bananen, Tomaten, Zitrusfrüchte)

Hyperkalzämie: Hydratation, Bisphosphonate

Hypotonie: Flüssigkeit, Medikamente überprüfen/absetzen

Schilddrüsenfunktionsstörung: Medikamente überprüfen, ggf. Therapie

Medikamente: Reduktion oder Wechsel (Steroide, Antidepressiva, Benzodiazepine, Opioide), sedierende Medikamente möglichst nur zur Nacht geben

Schlaflosigkeit: Ursachen besprechen (Sorgen, Angst etc.), für ruhige, entspannte Umgebung sorgen, ggf. Benzodiazepine

Anorexie, Kachexie: Steroide (z. B. 4 mg Dexamethason p.o.)

Depressives Syndrom: Antidepressiva

Inaktivität: Krankengymnastik

Gehirnmetastase: Dexamethason, ggf. Bestrahlung/systemische Chemotherapie

Rückenmarkskompression: Dexamethason (Bausewein C, et al. Arzneitherapie in der Palliativmedizin. 1. Aufl. Urban und Fischer, München, Jena, 2005), z. B. beginnend mit 10-20 mg i.v., dann p.o.16 mg/die, ggf. Bestrahlung, ggf. operative Entlastung

Fatigue:

vor Einleitung »therapeutischer« Maßnahmen immer unrealistische Erwartungen an den Therapieerfolg verhindern

Behandlungsversuche mit Amphetaminen (Methylphenidat) in randomisierten Studien nicht erfolgreicher als Placebogabe (Bruera E, et al. J Clin Oncol 2006)

Viele Betroffene profitieren von Informationsbroschüren (Deutsche Krebshilfe oder andere Patientenorganisationen). Besonders für Angehörige hilfreich, da sie sich weniger hilflos im Umgang mit dem erkrankten Familienmitglied

fühlen (Glaus A, et al. Pflege 2002)

klinische Unterscheidung von Depression und Fatigue im Einzelfall schwierig (Visser M, Smets E. Support Care Cancer 1998), jedoch Fatigue nicht erfolgreich mit Antidepressiva zu behandeln (Morrow G, et al. Proceedings American Society of Clinical Oncology 2001)

wesentliche Aufgabe der behandelnden Ärzte: Vermittlung der Tatsache, dass Fatigue Ausdruck der fortschreitenden Krankheit ist (Glaus A. Fatigue. In: Lehrbuch der Palliativmedizin. Aulert E, Nauck F, Radbruch L (Hrsg.). Schattauer Verlag, Stuttgart, 2007)

Unterstützende Maßnahmen

kein Verzicht auf gewohnte kleine »Alltagsfreuden« wie Alkohol oder andere Genussmittel

deutlich stärkerer Profit von gezielter Suche nach »Womit könnten Sie sich denn heute etwas Gutes tun?« als von medikamentösen Therapieversuchen

Armbewegungen im Sitzen oder Liegen, Hin- und Herwiegen zu Musik sowie passive Bewegungsübungen können selbst bei schwächeren Patienten das Wohlbefinden günstig beeinflussen (Mock V, et al. Oncology Nursing Forum 1997)

Unruhe:

möglichst Behandlung der Ursachen (Schmerzen, Atemnot, Augen- und Mundtrockenheit, Harnverhalt, Exsikkose, Hypoglykämie, Hyperkalzämie, Hirnmetastasen, Bewegungsunfähigkeit aufgrund von Schwäche, Medikamente (paradoxe Reaktion auf Benzodiazepine), Psychosoziale Ursachen (unerledigte Angelegenheiten, keine oder »falsche« Sitzwache))

Schaffen einer vertrauten Atmosphäre (bekannte Musik, Bettdecke, ruhige Anwesenheit von Angehörigen oder vertrauten Personen)

medikamentöse Therapie:

Promethazin: 15-75 mg p.o

Diazepam: 2-10 mg p.o., i.v. (nicht s.c.), 10-20 mg rektal 8-stdl.

Lorazepam: 0,5-2,5 mg p.o., i.m., i.v.

Midazolam: bei Bedarf: 2,5-5 mg s.c. (nicht für s.c. Gabe zugelassen), p.o. oder 1-2,5 mg i.v. oder kontinuierlich: 10-60 mg/24 h in subkutaner Spritzenpumpe. Kurze HWZ, stark sedierend

Flunitrazepam: 1-2 mg s.c. (nicht für s.c. Gabe zugelassen), p.o., 0,5-2 mg i.v., i.m., stark sedierend

Prothipendyl: (z. B. Dominal®) 20-80 mg p.o., i.v.

Pipamperon: 20-80 mg p.o.

Melperon: 25-50 mg 8-stdl. p.o.

Bei Halluzinationen, Alpträumen, paranoiden Symptomen (v.a. delirantes Syndrom)

Haloperidol (z. B. als Generikum verfügbar) 1,5-3 mg s.c. 8-stdl. bei älteren Patienten, 5 mg s.c. 8-stdl. bei jüngeren Patienten, 10-30 mg/24 h s.c., i.v.

Levomepromazin (z. B. als Generikum verfügbar) 10-50 mg 4-stdl. p.o., s.c., i.m., sedierend, als Tropfen gut dosierbar

parenterale Gabe von Medikamenten notwendig (Patienten meist nicht mehr in der Lage sind zu schlucken)

subkutane Gabe zu bevorzugen (einfachere Applikation (auch zuhause möglich))

bei überwiegender Unruhe vor allem z. B. bei Patienten mit Hirnmetastasen Benzodiazepine bevorzugen (erniedrigen im Vergleich zu Neuroleptika nicht die Krampfschwelle), bei diesen bietet sich auch Therapieversuch mit Kortison an

Midazolam (nicht für s.c. Gabe zugelassen) und Haloperidol können in der Spritzenpumpe mit Morphin gemischt werden

Falls Patient unter Midazolam (120 mg/24 h) und Haloperidol (30 mg/24 h) nicht deutlich ruhiger wird, kann Phenobarbital (200 mg s.c. oder i.m. bei Bedarf, oder 800-1600 mg/24 h) in Spritzenpumpe gegeben werden.

aktives Vorgehen notwendig, einerseits um des Patienten Willen, andererseits aber auch wegen der möglichen Beunruhigung der Angehörigen, da sie dieses Bild der letzten Stunden des Patienten oft im Gedächtnis behalten

-

 

7.6.2 EvT 7.63

Quelle

Fazit/Empfehlung

Empfehlungsgrad

ACP 2008

In patients with serious illness at the end of life, clinicians should regularly assess patients for pain, dyspnea, and depression. (GRADE: moderate quality of evidence)

Although each patient and family will require individualized assessment and care, the evidence shows that a set of general issues is shared widely among patients needing end-of-life care. These issues include pain and other symptom management, psychological well-being, care coordination and advance care planning, and caregiver burden. The evidence was classified as moderate quality because it was mostly derived from studies of patients with cancer or cancer-predominant populations in addition to being heterogeneous. (Patrick DL, et al. Ann Intern Med. 2003; Steinhauser KE, et al. JAMA. 2000; Hanson LC, et al. J Am Geriatr Soc. 1997; Singer PA, et al. JAMA. 1999; Wenrich MD, et al. J Pain Symptom Manage. 2003; Teno JM, et al. J Pain Symptom Manage. 2001; Teno JM, et al. JAMA. 2004; Field MJ, Cassel CK, eds. Institute of Medicine. Washington, DC: National Academies Pr; 1997)

In patients with serious illness at the end of life, clinicians should regularly assess patients for pain, dyspnea, and depression. (GRADE: moderate quality of evidence)

Although each patient and family will require individualized assessment and care, the evidence shows that a set of general issues is shared widely among patients needing end-of-life care. These issues include pain and other symptom management, psychological well-being, care coordination and advance care planning, and caregiver burden. The evidence was classified as moderate quality because it was mostly derived from studies of patients with cancer or cancer-predominant populations in addition to being heterogeneous. (Patrick DL, et al. Ann Intern Med. 2003; Steinhauser KE, et al. JAMA. 2000; Hanson LC, et al. J Am Geriatr Soc. 1997; Singer PA, et al. JAMA. 1999; Wenrich MD, et al. J Pain Symptom Manage. 2003; Teno JM, et al. J Pain Symptom Manage. 2001; Teno JM, et al. JAMA. 2004; Field MJ, Cassel CK, eds. Institute of Medicine. Washington, DC: National Academies Pr; 1997)

In patients with serious illness at the end of life, clinicians should use therapies of proven effectiveness to manage pain. For patients with cancer, this includes nonsteroidal anti-inflammatory drugs, opioids, and bisphosphonates. (GRADE: moderate quality of evidence)

Clinicians should use specific effective therapies for all patients with acute and chronic pain. Strong evidence supports using nonsteroidal anti-inflammatory drugs, opioids, and bisphosphonates for pain relief in patients with cancer. Bisphosphonates are effective for bone pain relief in patients with breast cancer and myeloma. (Carr D, et al. AHRQ publication no. 02-E032, 2002; Knols R, et al. J Clin Oncol. 2005; Lee H, et al. Eur J Pain. 2005; Higginson IJ, et al. University of Wales College of Medicine; 2000; Detmar SB, et al. JAMA. 2002; Jordhøy MS, et al. J Clin Oncol. 2001)

In patients with serious illness at the end of life, clinicians should use therapies of proven effectiveness to manage dyspnea, which include opioids in patients with unrelieved dyspnea and oxygen for short-term relief of hypoxemia. (Grade: moderate quality of evidence)

Opioids should be considered in patients with severe and unrelieved dyspnea, for example, in cancer and cardiopulmonary disease at the end of life. Clinicians should consider the use of oxygen for hypoxemia for advanced chronic obstructive pulmonary disease. Evidence also supports the use of beta-agonists for treating dyspnea in chronic obstructive pulmonary disease, but this use has not been studied for end-of-life care. (Detmar SB, et al. JAMA. 2002; Jordhøy MS, et al. J Clin Oncol. 2001; Jennings AL, et al. Thorax. 2002; Abernethy AP, et al. BMJ. 2003; Bruera E, et al. Palliat Med. 2003; Shukla V, et al. Technology report no 65. Ottawa: Canadian Coordinating Office for Health Technology Assessment; 2006; Grande GE, et al. Palliat Med. 2000; Grande GE, et al. Palliat Med. 1997)

strong recommendation

 

 

 

 

strong recommendation

 

 

 

 

strong recommendation

 

 

 

strong recommendation

LG Hessen 2009

Schwäche:

reversible Ursachen, die zum Schwächegefühl beitragen, identifizieren und beheben

nur bei entsprechender Prognose oder dem klinischen Verdacht auf eine potenziell reversible Ursache weitere Diagnostik betreiben

Intravenöse Hyperalimentation ohne nachgewiesenen Effekt

mit Patienten gemeinsam nach Lösungen suchen, z. B. Erklärung, dass das Gefühl der Schwäche fluktuiert. Vorschläge machen, die Kraft einzuteilen, Ruhepausen mit Aktivität abzuwechseln, den Tagesablauf anzupassen. Vor wichtigen Ereignissen (z. B. Geburtstage, Hochzeit, Hochzeitstag) kann eine Erhöhung der Steroide oder ggf. eine Bluttransfusion kurzfristig eine Besserung des Schwächegefühls bringen. Körperliche Aktivierung (z. B. Gymnastik im Bett, Stretchbänder).

Gabe von Steroiden unterschiedlich beurteilt, jedoch kann als allgemein roborierende Maßnahme ein Versuch mit 4 mg Dexamethason indiziert sein (Ream E, Stone P. Clinical interventions for fatigue. In: Fatigue in cancer. Armes J, et al. Oxford University Press, Oxford, 2004: 255-277)

Wirksamkeit einer hormonellen Therapie (z. B. Medroxyprogesteronacetat, Megestrolacetat, Testosteron, Erythropoetin oder Melatonin) nicht ausreichend belegt, kann nicht empfohlen werden (Ream E, Stone P. Clinical interventions for fatigue. In: Fatigue in cancer. Armes J, et al. Oxford University Press, Oxford, 2004: 255-277)

Therapie potenziell behandelbarer Ursachen bei Schwäche

Anämie: Transfusion bei Hb < 8 g/l, weitere Anämiezeichen wie Kurzatmigkeit, Müdigkeit

Hypokaliämie: ggf. Schleifendiuretikum durch ein kaliumsparendes Diuretikum ersetzen, Substitution über Nahrung (Bananen, Tomaten, Zitrusfrüchte)

Hyperkalzämie: Hydratation, Bisphosphonate

Hypotonie: Flüssigkeit, Medikamente überprüfen/absetzen

Schilddrüsenfunktionsstörung: Medikamente überprüfen, ggf. Therapie

Medikamente: Reduktion oder Wechsel (Steroide, Antidepressiva, Benzodiazepine, Opioide), sedierende Medikamente möglichst nur zur Nacht geben

Schlaflosigkeit: Ursachen besprechen (Sorgen, Angst etc.), für ruhige, entspannte Umgebung sorgen, ggf. Benzodiazepine

Anorexie, Kachexie: Steroide (z. B. 4 mg Dexamethason p.o.)

Depressives Syndrom: Antidepressiva

Inaktivität: Krankengymnastik

Gehirnmetastase: Dexamethason, ggf. Bestrahlung/systemische Chemotherapie

Rückenmarkskompression: Dexamethason (Bausewein C, et al. Arzneitherapie in der Palliativmedizin. 1. Aufl. Urban und Fischer, München, Jena, 2005), z. B. beginnend mit 10-20 mg i.v., dann p.o.16 mg/die, ggf. Bestrahlung, ggf. operative Entlastung

Fatigue:

vor Einleitung »therapeutischer« Maßnahmen immer unrealistische Erwartungen an den Therapieerfolg verhindern

Behandlungsversuche mit Amphetaminen (Methylphenidat) in randomisierten Studien nicht erfolgreicher als Placebogabe (Bruera E, et al. J Clin Oncol 2006)

Viele Betroffene profitieren von Informationsbroschüren (Deutsche Krebshilfe oder andere Patientenorganisationen). Besonders für Angehörige hilfreich, da sie sich weniger hilflos im Umgang mit dem erkrankten Familienmitglied

fühlen (Glaus A, et al. Pflege 2002)

klinische Unterscheidung von Depression und Fatigue im Einzelfall schwierig (Visser M, Smets E. Support Care Cancer 1998), jedoch Fatigue nicht erfolgreich mit Antidepressiva zu behandeln (Morrow G, et al. Proceedings American Society of Clinical Oncology 2001)

wesentliche Aufgabe der behandelnden Ärzte: Vermittlung der Tatsache, dass Fatigue Ausdruck der fortschreitenden Krankheit ist (Glaus A. Fatigue. In: Lehrbuch der Palliativmedizin. Aulert E, Nauck F, Radbruch L (Hrsg.). Schattauer Verlag, Stuttgart, 2007)

Unterstützende Maßnahmen

kein Verzicht auf gewohnte kleine »Alltagsfreuden« wie Alkohol oder andere Genussmittel

deutlich stärkerer Profit von gezielter Suche nach »Womit könnten Sie sich denn heute etwas Gutes tun?« als von medikamentösen Therapieversuchen

Armbewegungen im Sitzen oder Liegen, Hin- und Herwiegen zu Musik sowie passive Bewegungsübungen können selbst bei schwächeren Patienten das Wohlbefinden günstig beeinflussen (Mock V, et al. Oncology Nursing Forum 1997)

Unruhe:

möglichst Behandlung der Ursachen (Schmerzen, Atemnot, Augen- und Mundtrockenheit, Harnverhalt, Exsikkose, Hypoglykämie, Hyperkalzämie, Hirnmetastasen, Bewegungsunfähigkeit aufgrund von Schwäche, Medikamente (paradoxe Reaktion auf Benzodiazepine), Psychosoziale Ursachen (unerledigte Angelegenheiten, keine oder »falsche« Sitzwache))

Schaffen einer vertrauten Atmosphäre (bekannte Musik, Bettdecke, ruhige Anwesenheit von Angehörigen oder vertrauten Personen)

medikamentöse Therapie:

Promethazin: 15-75 mg p.o

Diazepam: 2-10 mg p.o., i.v. (nicht s.c.), 10-20 mg rektal 8-stdl.

Lorazepam: 0,5-2,5 mg p.o., i.m., i.v.

Midazolam: bei Bedarf: 2,5-5 mg s.c. (nicht für s.c. Gabe zugelassen), p.o. oder 1-2,5 mg i.v. oder kontinuierlich: 10-60 mg/24 h in subkutaner Spritzenpumpe. Kurze HWZ, stark sedierend

Flunitrazepam: 1-2 mg s.c. (nicht für s.c. Gabe zugelassen), p.o., 0,5-2 mg i.v., i.m., stark sedierend

Prothipendyl: (z. B. Dominal®) 20-80 mg p.o., i.v.

Pipamperon: 20-80 mg p.o.

Melperon: 25-50 mg 8-stdl. p.o.

Bei Halluzinationen, Alpträumen, paranoiden Symptomen (v.a. delirantes Syndrom)

Haloperidol (z. B. als Generikum verfügbar) 1,5-3 mg s.c. 8-stdl. bei älteren Patienten, 5 mg s.c. 8-stdl. bei jüngeren Patienten, 10-30 mg/24 h s.c., i.v.

Levomepromazin (z. B. als Generikum verfügbar) 10-50 mg 4-stdl. p.o., s.c., i.m., sedierend, als Tropfen gut dosierbar

parenterale Gabe von Medikamenten notwendig (Patienten meist nicht mehr in der Lage sind zu schlucken)

subkutane Gabe zu bevorzugen (einfachere Applikation (auch zuhause möglich))

bei überwiegender Unruhe vor allem z. B. bei Patienten mit Hirnmetastasen Benzodiazepine bevorzugen (erniedrigen im Vergleich zu Neuroleptika nicht die Krampfschwelle), bei diesen bietet sich auch Therapieversuch mit Kortison an

Midazolam (nicht für s.c. Gabe zugelassen) und Haloperidol können in der Spritzenpumpe mit Morphin gemischt werden

Falls Patient unter Midazolam (120 mg/24 h) und Haloperidol (30 mg/24 h) nicht deutlich ruhiger wird, kann Phenobarbital (200 mg s.c. oder i.m. bei Bedarf, oder 800-1600 mg/24 h) in Spritzenpumpe gegeben werden.

aktives Vorgehen notwendig, einerseits um des Patienten Willen, andererseits aber auch wegen der möglichen Beunruhigung der Angehörigen, da sie dieses Bild der letzten Stunden des Patienten oft im Gedächtnis behalten

-

 

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